ライフサイエンスコーパス: intracellular calcium

1 ade is one of the major pathways governed by intracellular calcium.

2 e further membrane depolarisation and rising intracellular calcium.

3 ) mice showed no hypoxia-induced increase of intracellular calcium.

4 Application of Ang(1-7) had no effect on intracellular calcium.

5 ly inhibiting glutamate-induced increases in intracellular calcium.

6 events are similarly tightened by buffering intracellular calcium.

7 n, apoptosis, proliferation and increases in intracellular calcium.

8 sine kinases, p38 MAPK, phospholipase C, and intracellular calcium.

9 tion caused by frequency-induced increase in intracellular calcium.

10 ges in membrane potential and an increase in intracellular calcium.

11 caused a rapid and synergistic elevation of intracellular calcium.

12 esponse to environmental stimuli to increase intracellular calcium.

13 ence of the betaAR agonist isoproterenol and intracellular calcium.

14 ivation in murine macrophages via changes in intracellular calcium.

15 regulators of NRG3 signaling: (1) release of intracellular calcium, (2) activation of the BACE1 beta-

16 Most importantly, experimental increase in intracellular calcium abolished Flunarizine's effect.

17 ic acid acetoxymethyl ester, an inhibitor of intracellular calcium abundance, blocked BMP-2-induced t

18 nism involving plasma membrane targeting and intracellular calcium accumulation.

19 In Jurkat T cells, elevation of intracellular calcium activates CRACR2a-mediated dynein

20 of action potentials and consequent rises of intracellular calcium activity ([Ca(2+)]i).

21 neously track the position, deformation, and intracellular calcium activity of their multidendritic p

22 Intracellular calcium acts as a secondary messenger in a

23 ucible and reversible transient increases in intracellular calcium, allowing the generation of a conc

24 RG neurons, tacrolimus evokes an increase in intracellular calcium almost exclusively in cold-sensiti

25 and IL-33 stimulation led to an increase in intracellular calcium, altered gene expression, but had

26 However, the automatization of global intracellular calcium analysis at the single-cell level

27 ion potential stability and time to failure, intracellular calcium and ATP, mitochondrial depolarizat

28 nt lymphatic muscle cells exhibited elevated intracellular calcium and decreased SERCA2a expression a

29 us positive feedback loop involving elevated intracellular calcium and enhanced mGluR1 function, a me

30 lation, Rab46 senses localized elevations of intracellular calcium and evokes dispersal of microtubul

31 otides and their effectors in the control of intracellular calcium and exocytosis.

32 ated with a period of sustained elevation of intracellular calcium and formation of larger and more h

33 Intracellular calcium and membrane potential were evalua

34 interfered with serotonin-evoked increase in intracellular calcium and membrane potential, and blunte

35 protein-alpha-mediated signaling, mobilizing intracellular calcium and Nf-kappaB signaling, leading t

36 Our analysis indicates that in both intracellular calcium and NFkappaB signaling, response v

37 ate heterotrimeric G-proteins that stimulate intracellular calcium and oncogenic Kras signaling, ther

38 lement attack limits sustained elevations in intracellular calcium and prevents mitochondrial injury.

39 ssion of genes involved in the regulation of intracellular calcium and proliferation, and preventing

40 Blue/green produced a bigger increase in intracellular calcium and reactive oxygen species (ROS).

41 clinical significance since dysregulation of intracellular calcium and ROS signaling is implicated in

42 ow or absent because of a steady increase in intracellular calcium and sodium concentrations.

43 ceptor modulators (SERMs) potently stabilize intracellular calcium and thereby counteract atRAL-induc

44 increased phosphatidylserine exposure, high intracellular calcium, and elevated osmotic fragility.

45 lls, we imaged ciliary beat frequency (CBF), intracellular calcium, and nitric oxide (NO).

46 ROS production, a ROS-dependent increase of intracellular calcium, and the production of MCP 1 (CCL-

47 that matrix deprivation leads to a spike in intracellular calcium as well as oxidant signaling, and

48 cell imaging coupled with temporally precise intracellular calcium buffering.

49 mediated ATP release occurs independently of intracellular calcium but is sensitive to SRC family kin

50 While stretch-elicited X-ROS primes intracellular calcium (Ca(2+) ) channels for synchronize

51 termined by the frequency of oscillations of intracellular calcium (Ca(2+) ) concentration.

52 termined by the frequency of oscillations of intracellular calcium (Ca(2+) ) concentration.

53 egulate many biological processes, including intracellular calcium (Ca(2+) ) handling.

54 tion rate, and diminished the sensitivity to intracellular calcium (Ca(2+) ) in G protein-induced exo

55 mine the role of trefoil factor 2 (TFF2) and intracellular calcium (Ca(2+) ) mobilization in gastric

56 nel is remarkably sensitive to inhibition by intracellular calcium (Ca(2+) i) through binding of Ca(2

57 Intracellular calcium (Ca(2+)) alternans is a dynamical

58 The high-conductance intracellular calcium (Ca(2+)) channel RyR2 is essential

59 Intracellular calcium (Ca(2+)) cycling dynamics in cardi

60 Ca(2+) entry (SOCE) mediates the increase in intracellular calcium (Ca(2+)) in endothelial cells (ECs

61 -R1 with CP-154526 caused an accumulation of intracellular calcium (Ca(2+)) over time and cell death.

62 The type-1 ryanodine receptor (RyR1) is an intracellular calcium (Ca(2+)) release channel required

63 ositol 1,4,5-triphosphate receptor-dependent intracellular calcium (Ca(2+)) release.

64 es-all of which are consistent with aberrant intracellular calcium (Ca(2+)) response.

65 Intracellular calcium (Ca(2+)) signaling resulting from

66 he release of these granules is dependent on intracellular calcium (Ca(2+)) signals.

67 activated receptors (PAR) leads to increased intracellular calcium (Ca(2+)).

68 tors for neurotransmitters that can increase intracellular calcium (Ca(2+)).

69 tatic laser microscopy, and determination of intracellular calcium ([Ca(2+)] (i) ), we show that vari

70 iezo1 agonist, Yoda1, led to an elevation in intracellular calcium ([Ca(2+)](i)) and that application

71 iple odorous molecules capable of increasing intracellular calcium ([Ca(2+)](i)) in ASM cells, some o

72 Intracellular calcium ([Ca(2+)](i)) oscillation is a fun

73 levate pulmonary vascular tone by increasing intracellular calcium ([Ca(2+)](i)) through reduction-ox

74 (RyR), plays a major role in agonist-induced intracellular calcium ([Ca(2+)]cyt) dynamics in vascular

75 arized membrane potentials or with decreased intracellular calcium ([Ca(2+)]i) and recovered with dep

76 y [ via increased airway smooth muscle (ASM) intracellular calcium [Ca(2+)](i)] and remodeling (ASM p

77 iple cellular organelles tightly orchestrate intracellular calcium (Ca2+) dynamics to regulate cellul

78 Intracellular calcium ([Ca2+]i) is a basic and ubiquitou

79 In the pancreas, excessive intracellular calcium causes mitochondrial dysfunction,

80 2 (RyR2) macromolecular complex, which is an intracellular calcium channel and abundant in the brain.

81 slocate into cells and potently activate the intracellular calcium channel type 1 ryanodine receptor

82 Addition of an intracellular calcium chelator or an AMPK inhibitor to e

83 POINTS: For the heart to function as a pump, intracellular calcium concentration ([Ca(2+) ]i ) must i

84 neuron axon we found that activity-dependent intracellular calcium concentration ([Ca(2+)](i)) in the

85 , a specific alpha7 nAChR agonist, increases intracellular calcium concentration ([Ca(2+)]i) mainly r

86 s that exhibit activity-related increases in intracellular calcium concentration (FLiCRE).

87 ted that hemichannel activity depends on the intracellular calcium concentration and is associated wi

88 mechanotransduction is often an increase in intracellular calcium concentration associated with intr

89 filament, but inhibits contractility at high intracellular calcium concentration by disrupting the ac

90 shing the SR calcium store, the evolution of intracellular calcium concentration during a train of lo

91 the hyperpolarization-activated current and intracellular calcium concentration in both normal contr

92 thick filament stress but are independent of intracellular calcium concentration in the physiological

93 The subsequent increase in intracellular calcium concentration induces proteolytic

94 lex is a molecular switch that ties shifting intracellular calcium concentration to association and d

95 Notably, the rise of intracellular calcium concentration upon immunoglobulin

96 e to 4-CMC or caffeine, similar increases in intracellular calcium concentration were observed in Sta

97 lowed, within 205+/-34 seconds, by increased intracellular calcium concentration, [Ca(2+)](i).

98 osphorylation of TCRzeta, ZAP70, and LAT and intracellular calcium concentration, as well as IL-2 gen

99 uscle cell membrane, a transient increase of intracellular calcium concentration, binding of calcium

100 n of mitochondrial complex I, an increase in intracellular calcium concentration, or formation of rea

101 sed F-actin content, and increased the basal intracellular calcium concentration.

102 he transformation of membrane potential into intracellular calcium concentration.

103 analyze spontaneous dynamic fluctuations in intracellular calcium concentrations ([Ca(2+)](i)) in sm

104 ond to mechanical stimulation with increased intracellular calcium concentrations and increased inwar

105 arginine deiminase activity depends on high intracellular calcium concentrations occurring in dying

106 cardial mechanisms: calcitropes, which alter intracellular calcium concentrations; myotropes, which a

107 Using cryo-soft X-ray microscopy we imaged intracellular calcium-containing particles in the PMCs a

108 Consequently, TMEM33 reduces intracellular calcium content in a PC2-dependent manner,

109 HSCs are endowed with low intracellular calcium conveyed by elevated activity of g

110 Our findings suggest a key role for intracellular calcium cycling and excitation-transcripti

111 s study, we present a computational model of intracellular calcium cycling in three-dimensions (3-D),

112 Intracellular calcium cycling is a vital component of ca

113 maintain transcription of genes that control intracellular calcium cycling.

114 ubsequent P2Y(2)R activation, and downstream intracellular calcium-dependent signal transduction.

115 Calpains are a family of intracellular, calcium-dependent cysteine proteases invo

116 al of extracellular calcium, or chelation of intracellular calcium did not normalize the differences

117 Knocking down PMCA2 increases intracellular calcium, disrupts interactions between HER

118 rametric mapping of transmembrane potential, intracellular calcium dynamics and other parameters in i

119 Comparison of intracellular calcium dynamics between genotypes was ass

120 Model results show that agonist-induced intracellular calcium dynamics can be modified by changi

121 The model reproduces intracellular calcium dynamics during control pacing and

122 Abnormal intracellular calcium dynamics in cardiomyocytes have ma

123 Intracellular calcium dynamics of ventricular myocytes i

124 ltered levels of SERCA, IP3R, and RyR on the intracellular calcium dynamics of VSMC and to understand

125 Functional imaging, for example, of intracellular calcium dynamics, has led to the demonstra

126 onses with respect to their contributions to intracellular calcium dynamics, testing the 'unifying hy

127 isation of these structures tightly controls intracellular calcium dynamics.

128 n, resulting in decreased membrane fluidity, intracellular calcium dysregulation, depolarization, and

129 lipid scramblase 1 (PLSCR1) activity reduces intracellular calcium dysregulation, prevents PtdSer ext

130 which extracellular signals elicit prolonged intracellular calcium elevation to drive changes in fund

131 d used to perform transcriptome, metabolome, intracellular calcium, extracellular cathepsin activity,

132 unds significantly antagonized DAMGO-induced intracellular calcium flux and displayed varying degrees

133 n gene in HIV; TAT-4BB) affected LPS-induced intracellular calcium flux and excitation in sensory neu

134 These delays are regulated by intracellular calcium flux downstream of T cell activati

135 r of NF-kappa-B ligand-induced activation of intracellular calcium flux in vitro.

136 by altering IP3 receptor phosphorylation and intracellular calcium flux, and activating calcium-depen

137 trans retinoic acid, we measured chemotaxis, intracellular calcium flux, and alpha4beta7-mediated cel

138 that GCs suppress CCR9-mediated chemotaxis, intracellular calcium flux, and alpha4beta7-mediated cel

139 ted membrane currents or by instabilities in intracellular calcium fluxes.

140 O treatment and included a rapid increase of intracellular calcium ([Formula: see text]) and higher l

141 culum calcium ATPase (SERCA) establishes the intracellular calcium gradient across the sarcoplasmic r

142 her, we postulate that the maintenance of an intracellular calcium gradient by the calcium ATPase and

143 leotide targeted pathways linked to abnormal intracellular calcium handling and cardiac neurotransmis

144 erlying physiological and pathophysiological intracellular calcium handling phenomena at the whole-ce

145 bited distinct cardiac dysfunction, dampened intracellular calcium handling, alterations in cardiac m

146 pathway with no need to postulate defects in intracellular calcium handling.

147 These factors combined lead to disruption of intracellular calcium homeostasis and isoproterenol-indu

148 ace these new developments in the context of intracellular calcium homeostasis and signaling.

149 e effects: a direct damage linked to altered intracellular calcium homeostasis in muscle cells and an

150 n central nervous system, where it regulates intracellular calcium homeostasis in response to excitat

151 urprisingly high, and sufficient to maintain intracellular calcium homeostasis in the presence of com

152 The altered intracellular calcium homeostasis led to activation of c

153 y a key role for TMEM33 in the regulation of intracellular calcium homeostasis of renal proximal conv

154 These data link defects in neuronal intracellular calcium homeostasis to the vulnerability o

155 d to safeguard ER-targeted mRNA translation, intracellular calcium homeostasis, and stem cell differe

156 Because Tat disrupts intracellular calcium homeostasis, we investigated the i

157 through their roles in energy production and intracellular calcium homeostasis.

158 Impairments in neuronal intracellular calcium ((i)Ca(2+)) handling may contribut

159 (1 muM-1 mM), as indicted by an increase in intracellular calcium (iCa(2+)).

160 Although optical probes for intracellular calcium imaging have been available for de

161 hat, "Harnessing Hematopoietic Stem Cell Low Intracellular Calcium Improves Their Maintenance In Vitr

162 TRPM4 is activated by increased intracellular calcium in a voltage-dependent manner but,

163 Transient elevations of intracellular calcium in ASP cytonemes correlate with si

164 IDs are associated with a strong increase of intracellular calcium in astrocytes and neurons.

165 Finally, we show that conditionally reducing intracellular calcium in astrocytes impairs the homeosta

166 Consequently, elevated intracellular calcium in DIS3L2-deficient cells activate

167 Measurement of intracellular calcium in live cells is a key component o

168 hymal arteriole tone significantly increased intracellular calcium in perivascular astrocyte processe

169 rates in egg activation, including a rise of intracellular calcium in response to the trigger.

170 dynamics, highlighting the critical role of intracellular calcium in shaping the pERK1/2 signal.

171 s GAT-107 and B-973B stimulated increases in intracellular calcium in stably transfected HEK293 cells

172 e relationship between neuronal activity and intracellular calcium in these neurons is unknown.

173 store-operated calcium entry contributes to intracellular calcium increase, leading to reactive oxyg

174 ies were able to antagonize chemerin-induced intracellular calcium increase.

175 Intracellular calcium increases induced by TRPV4 agonist

176 un-protonated and TMEM16A is activated when intracellular calcium increases; however, under acidic c

177 so report that SMF stimulation increases the intracellular calcium influx in OPCs as well as the gene

178 podocytes can occur as a result of excessive intracellular calcium influx, and we have previously sho

179 Regulation of intracellular calcium involves purinergic receptors and

180 sphosphate receptor (ITPR3) is the principal intracellular calcium ion (Ca(2+) ) release channel in c

181 Recently, we showed that an increase of the intracellular calcium ion concentration [Ca(2+) ] causes

182 r disorder, especially by reports of altered intracellular calcium ion concentrations ([Ca(2+)]).

183 of articular cartilage via the generation of intracellular calcium ion transients.

184 nodol are complex diterpenoids that modulate intracellular calcium-ion release at ryanodine receptors

185 on of force triggered sustained elevation in intracellular calcium, leading to enzyme activation and

186 We identified a correlation between the intracellular calcium level and cell division, consisten

187 encoded calcium indicator, to determine the intracellular calcium level of this model organism.

188 MI 2650 or NCI-H520 squamous cells increased intracellular calcium levels and granulocyte macrophage-

189 Aberrant intracellular calcium levels and increased cAMP signalin

190 ciated with the highest basal and stimulated intracellular calcium levels and with increased cellular

191 Using a DNA polymerase to record intracellular calcium levels has been proposed as a nove

192 r cheek skin, (ii) acidified buffer elevated intracellular calcium levels in dorsal root ganglion pru

193 image-based assay that can detect changes in intracellular calcium levels in U2OS cells.

194 ing oocyte maturation, and yet, manipulating intracellular calcium levels interferes with first-polar

195 aling cascade and demonstrate that a rise in intracellular calcium levels is sufficient to modulate t

196 nally show that the LKB1/CaMKK-AMPK axis and intracellular calcium levels play a critical role in anc

197 ith AMPK and CaMKK2 and that the increase in intracellular calcium levels promotes AMPK colocalizatio

198 cells, undergo esterase cleavage, and allow intracellular calcium levels to be monitored by magnetic

199 As adenosine is known to cause changes in intracellular calcium levels upon addition to cell cultu

200 (AMPK)-dependent manner, and with increased intracellular calcium levels via PAR2.

201 ched in the brain and neurons that regulates intracellular calcium levels via signaling through the i

202 Acute nicotine exposure increased intracellular calcium levels, an effect primarily depend

203 acinar cells caused a prolonged elevation in intracellular calcium levels, mitochondrial depolarizati

204 logical processes that include disruption in intracellular calcium levels, so amelioration of the cal

205 ted alpha7 nicotinic ACh receptor may affect intracellular calcium levels, such effects are not likel

206 al growth factor receptor and an increase in intracellular calcium levels, under the permissive contr

207 pression of PGF(2alpha)-mediated increase in intracellular calcium levels.

208 ansient actin reset in response to increased intracellular calcium levels.

209 rs, such as thrombin and histamine, increase intracellular calcium levels.

210 By remodeling the intracellular calcium machinery and impairing signaling

211 pports the hypothesis that alpha7 effects on intracellular calcium may be independent of channel-medi

212 nction was determined with tissue myography, intracellular calcium measurements, and regulatory myosi

213 human P2X4 using a YOPRO-1 dye uptake assay, intracellular calcium measurements, and whole-cell patch

214 function in epithelial cells was examined by intracellular calcium measurements, wound healing assays

215 ion of Galphaq-coupled CysLT1, and sustained intracellular calcium mobilisation and extracellular sig

216 3 and 10 +/- 0.18 mug/ml, respectively) with intracellular calcium mobility similar to amlodipine.

217 dose-dependently inhibited CP55,940-induced intracellular calcium mobilization and [(35)S]GTP-gamma-

218 fficacy as an antagonist of chemerin induced intracellular calcium mobilization and a much higher pot

219 ligation of FcepsilonRI and CD300c increased intracellular calcium mobilization and phosphorylation o

220 (CMKLR1)-mediated ERK signaling and altered intracellular calcium mobilization mediated by these rec

221 IL-1beta had no detectable effect on intracellular calcium mobilization or endothelial cell v

222 harmacologically assessed mAChR-A to monitor intracellular calcium mobilization upon receptor activat

223 luding Fc receptor gamma-chain signaling and intracellular calcium mobilization.

224 ontaneously release exosomes, alterations in intracellular calcium or extracellular pH can release ad

225 ignalling, which is linked to an increase in intracellular calcium oscillation mediated by ryanodine

226 Lowering SERCA level will enable intracellular calcium oscillations at low agonist concen

227 Intracellular calcium oscillations tracked via real-time

228 ate and concentration-dependent reduction of intracellular calcium oscillations, while other caged in

229 nd RyR need higher agonist concentration for intracellular calcium oscillations.

230 We also find that a ring of elevated intracellular calcium overlaps the region where membrane

231 t a unifying hypothesis whereby depletion of intracellular calcium pools by crude oil-derived PAHs di

232 lular alkalinization was dependent on C5aR1, intracellular calcium, protein kinase C, and calmodulin,

233 serum treatment via a mechanism dependent on intracellular calcium, protein kinase C, and phosphatidy

234 gy transfer (FRET), confocal microscopy, and intracellular calcium quantitation.

235 and HFrEF to reveal distinct differences in intracellular calcium regulation and excitation-contract

236 7 in the context of moderate hyperoxia, with intracellular calcium regulation as a readout of contrac

237 brane lipid peroxidation, membrane fluidity, intracellular calcium regulation, passive membrane elect

238 ists in vitro, assessed by (a) inhibition of intracellular calcium release (IC(50) 10 nM) induced in

239 gets ryanodine receptors (RyRs), a family of intracellular calcium release channels essential for man

240 ed assay measuring inhibition of UTP-induced intracellular calcium release in 1321N1 astrocytoma cell

241 mpound 16 (CYD-1-79) potentiated 5-HT-evoked intracellular calcium release in cells stably expressing

242 logical activity of SDF1-ELP, as measured by intracellular calcium release in HL60 cells was dose dep

243 lu5 heteromers exacerbated PLC signaling and intracellular calcium release in response to either glut

244 f calcium-activated chloride conductances by intracellular calcium release is the key factor underlyi

245 jects and evaluated their ability to inhibit intracellular calcium release mediated by angiotensin II

246 is blocked by BAPTA chelation, and recruits intracellular calcium release on its way to activation o

247 e such innate behaviour that is modulated by intracellular calcium release through inositol 1,4,5-tri

248 the Ser23 variant, we show that 5-HT evokes intracellular calcium release with decreased potency and

249 een CMs, triggering membrane depolarization, intracellular calcium release, and actomyosin contractio

250 ssessing inositol triphosphate accumulation, intracellular calcium release, and directed cell migrati

251 de-out signaling events (eg, Akt activation, intracellular calcium release, and Ras-associated protei

252 membrane voltage changes and then triggering intracellular calcium release.

253 e cavitation-induced injury while evoking an intracellular calcium response, may be particularly usef

254 quantitation of cholesterol composition and intracellular calcium responses to CCK.

255 y in vitro, without influencing NMDA-induced intracellular calcium responses.

256 When the dosing of oligomers was stopped the intracellular calcium returned to basal levels or below.

257 ssible in vivo Following single cell damage, intracellular calcium selectively increases within cells

258 Together, these findings indicate that intracellular calcium serves as a readout of neuronal ac

259 haping the dynamics of infrared pulse-evoked intracellular calcium signal.

260 results showed that neuronal CALHM1 controls intracellular calcium signaling and cell excitability, t

261 pment of probes for noninvasive detection of intracellular calcium signaling in deep tissue and intac

262 localization of NFAT, a downstream target of intracellular calcium signaling using a reporter in live

263 teins, up-regulation of proteins involved in intracellular calcium signaling, and down-regulation of

264 P requires muscarinic 1 receptor activation, intracellular calcium signaling, and GluR2-lacking AMPAR

265 Systems as varied as blood clotting, intracellular calcium signaling, and tissue inflammation

266 ated calcium entry is a central regulator of intracellular calcium signaling.

267 chronic pain, and is critically dependent on intracellular calcium signaling.

268 expression of CysLT1 in LUVA cells augmented intracellular calcium signalling induced by LTE4 but did

269 Intracellular calcium signalling was recorded to compare

270 We show that intracellular calcium signals are critical for the regul

271 in regulating the spatiotemporal patterns of intracellular calcium signals.

272 ias, repolarization, calcium transients, and intracellular calcium sparks.

273 or maturation of secreted proteins and as an intracellular calcium storage compartment, facilitating

274 face sarcolemma and transverse-tubules), the intracellular calcium store (the sarcoplasmic reticulum)

275 ormation and length are controlled by actin, intracellular calcium stores, and components of the Wnt

276 ory responses depend on calcium release from intracellular calcium stores, and run down rapidly at re

277 rane potentials (RMPs) reflects depletion of intracellular calcium stores, while mAChR-driven excitat

278 d reinforcing signals mediated by mGluRs and intracellular calcium stores.

279 action molecule 1 (STIM1) after depletion of intracellular calcium stores.

280 s responsible for the sustained elevation in intracellular calcium that caused intracellular organell

281 Piezo1 produces only transient elevation in intracellular calcium that is insufficient to cause panc

282 ammalian species and requires the release of intracellular calcium through one or more unknown cold-s

283 ly protonated and works synergistically with intracellular calcium to activate the channel.

284 s with dose-dependent detrimental effects on intracellular calcium transient amplitude, contractility

285 llected to assess sympathetic postganglionic intracellular calcium transients ([Ca(2+) ]i ) and immun

286 the main factor responsible for the reduced intracellular calcium transients and contractility in VS

287 Intracellular calcium transients are a universal phenome

288 e present an instrument capable of recording intracellular calcium transients from the majority of ne

289 revealed a 4-fold faster decay of ATP-evoked intracellular calcium transients than GCaMP6f.

290 is dependent on and occurs coordinately with intracellular calcium transients, which are tightly asso

291 We modulated levels of katanin and intracellular calcium, two putative regulators of decili

292 Intracellular calcium was higher after 420 nm and 540 nm

293 Intracellular calcium was higher after blue/green, and c

294 llular calcium concentration associated with intracellular calcium waves (ICWs) in various physiologi

295 o the myofilament may promote arrhythmogenic intracellular calcium waves, we modified a mathematical

296 Since estrogen regulates intracellular calcium, we investigated the interaction b

297 tein kinase (AMPK) is regulated, in part, by intracellular calcium, we postulated that AMPK participa

298 , a modulator of Nrf2 stability regulated by intracellular calcium, were decreased.

299 Buffering intracellular calcium with EGTA-AM or BAPTA-AM reduced a

300 We observed an immediate, robust rise in intracellular calcium (within seconds) followed by a per