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1 ar to represent a unique form of prokaryotic intracytoplasmic inclusions.
2 e first step in the formation of fibrils and intracytoplasmic inclusions.
3 ons diminish microtubule binding and lead to intracytoplasmic inclusions.
5 f the protein, leading to the development of intracytoplasmic inclusion bodies and eventually to neur
7 om 9 of 11 patients recognize antigen within intracytoplasmic inclusion bodies in ciliated bronchial
8 disorder characterized by the appearance of intracytoplasmic inclusions called Lewy bodies (LB) in d
10 sease (PD) patients progressively accumulate intracytoplasmic inclusions formed by misfolded alpha-sy
11 tification, we used laser microdissection of intracytoplasmic inclusions identified in patient muscle
12 g beta-sheet-rich fibrils that accumulate as intracytoplasmic inclusions in Parkinson disease and cer
13 ntotemporal cortices and ubiquitin-positive, intracytoplasmic inclusions in the granule cells of the
14 llmark of the disease is the accumulation of intracytoplasmic inclusions known as Lewy bodies that co
18 , and endomysial fibrosis, in the absence of intracytoplasmic inclusions, rimmed vacuoles, or any evi
19 yn by nitration can promote the formation of intracytoplasmic inclusions that constitute the hallmark
20 On routine staining, there were numerous intracytoplasmic inclusions, which ranged from eosinophi
21 y demonstrated pale pink, oval to crescentic intracytoplasmic inclusions with a predilection for peri