1 The drFSRS data from the ethanol in
1%
intralipid solution demonstrate less signal strength but
2 fluoroethylene) microbeads and ethanol in
1%
intralipid solutions.
3 ure to isoflurane, control, propofol, or
10%
intralipid.
4 ng single exponential decay kinetics in a
2%
intralipid solution.
5 while imaging the CAs before and after a
20%
intralipid infusion to induce hypertriglyceridemia.
6 en 0 and 120 minutes, high-fat nutrient (
20%
intralipid) was infused via a postpyloric catheter into
7 sion of normal saline and 2) infusion of
20%
intralipid (IL).
8 NS + FAT; n = 6) received an infusion of
20%
intralipid + heparin (0.5 U x kg(-1) x min(-1)) to clamp
9 Two-hour intakes of
5%
intralipid were measured (5-120 min) in seven inbred (BA
10 0) of the doses tested, but failed to
affect intralipid intake in BALB/c mice.
11 the doses tested, and also failed to
affect intralipid intake in BALB/cJ mice.
12 ation of the fluorophore rhodamine 800 in
an intralipid suspension and in chicken tissue.
13 al anesthetic systemic toxicity requiring
an intralipid was reported.
14 els were raised approximately twofold via
an intralipid/heparin infusion in eight lean women.
15 n both insulin/glucose (by 34%, P = .05)
and intralipid (by 36%, P = .03).
16 analysis on infusions of insulin/glucose
and intralipid.
17 Fat-free milk
and intralipid, a source of vegetable triglycerides, were wi
18 ptical properties of water, mineral oil,
and intralipid droplets and to investigate the optical mecha
19 Both intralipid infusion and a low-carbohydrate diet resulted
20 rate and leg glucose uptake was decreased
by intralipid.
21 Plasma FFAs were increased
by intralipid/heparin infusion (3 h), insulin was increased
22 free fatty acid (FFA) levels by twofold (
by intralipid infusion during 11 mmol/l glucose clamp) resu
23 signaling mechanisms differentially
control intralipid intake across different mouse strains, sugges
24 or combined with phenylephrine,
dobutamine,
intralipid infusion, or intralipid infusion and dobutami
25 In vivo in
dogs,
intralipid administration significantly increased the in
26 xybutyrate uptake (R = 0.81, P = .05)
during intralipid infusion.
27 Three dogs were studied
during intralipid infusion alone under resting conditions.
28 antagonism to alter intake of fat
emulsions (
intralipid) in mice.
29 for and intake of solid and emulsified
fat (
intralipid) solutions vary across different mouse strain
30 ile CPT was undetectable in NNP-CPT or
free (
intralipid) CPT-injected tumors at that time.
31 Eighteen patients underwent 8-
hour intralipid/heparin-infusion (high FFA) and hyperinsuline
32 used to measure the concentration of RhB
in intralipid and to measure pH using 6-carboxyfluorescein.
33 lities of SCH23390 and naltrexone to
inhibit intralipid intake across strains.
34 -fold and 9.3-fold more potent in
inhibiting intralipid intake in the most sensitive (DBA/2) relative
35 stigated sites compared with the rested
leg,
intralipid impaired TBC1D4 S341 phosphorylation compared
36 +/- 0.25 mmol/l; P = 0.009), despite
matched intralipid infusion rates.
37 Using a combination
of intralipid and/or nicotinic acid infusions and a pancrea
38 Interestingly, this effect
of intralipid in the exercised leg was abolished by DCA, wh
39 from whole-body exercise, and 3) infusion
of intralipid + oral PDC activator, dichloroacetate (DCA).
40 e with 1) infusion of saline, 2) infusion
of intralipid imitating circulating FA levels during recove
41 ake, and increased ketone uptake with CRT
on intralipid (R = 0.79, P = .05).
42 a clamp with either infusion of glycerol
or intralipid.
43 ephrine, dobutamine, intralipid infusion,
or intralipid infusion and dobutamine.
44 or exercise was followed by 7 h of saline
or intralipid infusion.
45 rimental trials, differing only by saline
or intralipid infusion.
46 The present simulations reveal O-H:::O-
P intralipid hydrogen bonding occurs 99% of the time, and
47 dose-dependently and differentially
reduced intralipid intake at all five (DBA/2, SWR, CD-1), four (
48 dose-dependently and differentially
reduced intralipid intake at all four (DBA/2), three (SWR, SJL),
49 s decreased in both acute insulin-
resistant (
intralipid infusion) and chronic insulin-resistant state
50 arized emission of Rh800 from the
scattering intralipid or tissue.
51 , SGU was significantly decreased during
the intralipid/heparin versus saline infusion (30 +/- 2 vs.
52 is accompanied by significant changes in
the intralipid interactions within the bilayer.
53 icantly higher during the OGL clamp with
the intralipid/heparin infusion than with the saline infusio
54 eport that kidney GDF15 is necessary for
USI intralipid sensing to trigger an area postrema axis to i
55 eated animals (TZD + FFA2) were infused
with intralipid at a higher infusion rate (44%) to match the
56 efore and after elevation of FFA levels
with intralipid infusion (by +535%, n=9) and a 5-day high-fat
57 159 and 787 +/- 187 ml/min with and
without intralipid infusion, respectively).
58 A levels decreased during the clamps
without intralipid but increased above basal during the clamps w