ライフサイエンスコーパス: intraluminal pressure

1 s-sectional area was increased by increasing intraluminal pressure.

2 region and muscles activated but also on the intraluminal pressure.

3 ve lymph pump under conditions of controlled intraluminal pressure.

4 trolled in lung organ cultures by the airway intraluminal pressure.

5 or pattern was not accompanied by changes in intraluminal pressure.

6 rat carotid arteries exposed to 1 h of high intraluminal pressure.

7 1 h exposure to high intraluminal pressure (120 mmHg) resulted in increased l

8 e, and in veins through NE across a range of intraluminal pressures (2-12 cmH(2)O).

9 receptor (alpha1AR) stimulation and elevated intraluminal pressure: 2 main physiologic stimuli that c

10 High intraluminal pressure also resulted in a downstream sign

11 sors, and a balloon, simultaneously measures intraluminal pressure and cross-sectional areas.

12 es to constrict in response to elevations in intraluminal pressure and is a fundamental determinant o

13 The increases in intraluminal pressure and muscle cross-sectional area (C

14 total tension within the esophageal wall to intraluminal pressure and wall geometry.

15 dherin AJs in the vessel wall depends on the intraluminal pressure and was responsive to treatment wi

16 Interestingly, temperature and intraluminal pressure are sensed largely independently a

17 Decreasing intraluminal pressure below 50 mmHg increased the freque

18 l model incorporating both cell geometry and intraluminal pressure can account for our observations a

19 inal cord were investigated by measuring the intraluminal pressure change via saline filled balloons

20 eveal that tissue temperature, combined with intraluminal pressure, critically determines myogenic to

21 cle cells from cerebral arteries, increasing intraluminal pressure engages a signalling cascade that

22 ical stimulation of the artery, by increased intraluminal pressure, flattened the endothelial cells a

23 At intraluminal pressures from 6 to -6 cm H2O, maximum ante

24 Intraluminal pressures greater than 40 mmHg generated st

25 etch of arteriolar myocytes and increases in intraluminal pressure in arteries triggered rapid Ca(2+)

26 r type 1b (AT1 Rb ) is the primary sensor of intraluminal pressure in cerebral arteries.

27 of TRPM4 channels, is the primary sensor of intraluminal pressure in cerebral artery smooth muscle c

28 for either isoform as the primary sensor of intraluminal pressure in peripheral arteries.

29 tina preparation, we found that increases in intraluminal pressure in the physiological range induce

30 Myogenic tone was assessed over a range of intraluminal pressures in the presence and absence of hi

31 pressure-sensitive valves, opening whenever intraluminal pressure increases above a specific value.

32 may contribute to detection of distention or intraluminal pressure increases and initiation of reflex

33 These findings demonstrate an increase in intraluminal pressure induced increase in ROS production

34 An increase in intraluminal pressure induced oxidation of PTEN and acti

35 control animals, and this is independent of intraluminal pressure-induced depolarization.

36 Intraluminal pressure-induced reflex contractions (peris

37 Intraluminal pressure-induced TRPV4(SMC) channel activit

38 These data show that intraluminal pressure influences an endothelial microdom

39 onstrate a crucial role of blood flow-driven intraluminal pressure (IP) in regulating wound angiogene

40 her, these results suggest that elevation of intraluminal pressure is associated with generation of 2

41 ells to contract in response to increases in intraluminal pressure, is critically important for the a

42 he global equations with space-time-resolved intraluminal pressure measured manometrically and videof

43 stimated using geometric center analysis and intraluminal pressure monitoring.

44 Myogenic tone developed at an intraluminal pressure of 30 to 40 mm Hg, continued to in

45 At an intraluminal pressure of 50 mmHg and flow rate of 10 ul/

46 tor and dilator agonists were obtained at an intraluminal pressure of 70 mm Hg.

47 Step increases in intraluminal pressure of cannulated cerebral arteries in

48 The effect of intraluminal pressure on signaling mechanisms including

49 The effect of intraluminal pressure on the diameter was assessed and c

50 Here we report on elucidation of intraluminal pressure patterns using High Resolution Col

51 Intraluminal pressure, pH, and ultrasound images of the

52 Thus, we tested the hypothesis that high intraluminal pressure (Pi) itself, by activating vascula

53 the pressure-diameter relationship over the intraluminal pressure range, 30-110 mmHg.

54 At given intraluminal pressures ranging from +6 to -6 cm H2O, cro

55 Intraluminal pressure recording systems have not demonst

56 On the basis of the ultrasound images and intraluminal pressure recordings, it seems that there is

57 This study sought to determine whether high intraluminal pressure results in vascular inflammation.

58 n's activation elicited increases in colonic intraluminal pressure that were eliminated by scopolamin

59 spatially separated alpha1AR-TRPV4(SMC) and intraluminal pressure-TRPV4(SMC)-BK channel signaling ha

60 cular smooth muscle contractile responses to intraluminal pressure was examined in resistance vessels

61 Intraluminal pressure was measured using a Millar Mikro-

62 easurable CSA within the distal esophagus vs intraluminal pressure was significantly reduced in EoE p

63 which will increase wall stress for a given intraluminal pressure, which may be a stimulus for vascu

64 ed, cannulated, and pressurized to 60 cm H2O intraluminal pressure without flow for in vitro study.