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1 maging reproducibility and interobserver and intraobserver variability.
2 t set conditions to establish a baseline for intraobserver variability.
3 stry suffer from a high degree of inter- and intraobserver variability.
4 omly selected patients was used to determine intraobserver variability.
5 months after the first session to determine intraobserver variability.
6 analysis of cellular morphology with inter-/intraobserver variability.
8 e ascending aorta (AA) with COVs of 3.6% for intraobserver variability, 10.7% for interobserver varia
10 ing aorta (DDA) with median COVs of 1.6% for intraobserver variability, 4.0% for interobserver variab
12 tablish interobserver variability, to assess intraobserver variability, and to evaluate means of impr
13 ion methods were tested in 100 patients, and intraobserver variabilities as well as comparison with c
14 assessment, the 6-SD threshold yielded less intraobserver variability (difference, 0.6 g +/- 8, kapp
17 al of this study was to determine inter- and intraobserver variability in measurement of pulmonary ar
21 ver variability of -0.1 g +/- 2.3 and a mean intraobserver variability of 0.2 g +/- 1.6 at every-sect
27 h recent carotid events, assessed inter- and intraobserver variability of such measurements, and expl
31 For the determination of HF-US volume, the intraobserver variability was 9.7% +/- 5.1% (n = 8), and
38 is affected by substantial interobserver and intraobserver variability, which often leads to inapprop