ライフサイエンスコーパス: intraretinal

1 cal coherence tomography, 83% had fluid (61% intraretinal, 38% subretinal, and 36% sub-retinal pigmen

2 Macular hemorrhages most often were intraretinal (40%).

3 showed a demonstrable communication with the intraretinal abnormal vascular plexus in 6 of 7 eyes.

4 Intraretinal accumulations of fluid with increased OCT s

5 Intraretinal activation of immune modulators was assesse

6 monstrated minimal choroidal contribution to intraretinal analysis.

7 The distinctive intraretinal anatomy suggests that MME is caused by retr

8 segmentation we quantitatively measured the intraretinal anatomy.

9 curately detect, differentiate, and quantify intraretinal and SRF using area under the receiver opera

10 e examined immunohistochemically to identify intraretinal and subretinal exudative cells.

11 th gas tamponade for creation of a permanent intraretinal and subretinal fluid barrier.

12 on of brolucizumab-treated eyes had resolved intraretinal and subretinal fluid compared with afliberc

13 in eyes with persistent fluid by fluid type (intraretinal and subretinal fluid).

14 retinal thickness (CRT) and the presence of intraretinal and subretinal fluid.

15 each SD-OCT scan for the presence/absence of intraretinal and subretinal fluid.

16 emorrhages involving the optic nerve sheath, intraretinal and subretinal hemorrhages, and macular fol

17 Intraretinal and subretinal hyperreflective foci as seen

18 Intraretinal and subretinal temperature increases were m

19 nt epithelium), type 2 (subretinal), type 3 (intraretinal), and mixed neovascularization (NV), respec

20 hout all 3 layers of the retina (preretinal, intraretinal, and subretinal layers).

21 evaluated while performing retinal surface, intraretinal, and subretinal maneuvers in cadaveric porc

22 ic eye was developed to evaluate preretinal, intraretinal, and subretinal stresses during repetitive

23 eeks of follow-up with gradual resolution of intraretinal- and subretinal fluid, and remained stable

24 ies featuring an isolated, perifoveal, large intraretinal aneurysm surrounded by capillary rarefactio

25 thologic angiogenesis but also extend normal intraretinal angiogenesis by ordering the development of

26 uingly, a small population of M1 ipRGCs have intraretinal axon collaterals that project toward the ou

27 and Slit2, regulate two distinct aspects of intraretinal axon guidance in a region-specific manner.

28 In mice lacking robo1 intraretinal axon guidance occurs normally.

29 The intraretinal axon guidance thus serves as an excellent m

30 ay, Robo2 is the major receptor required for intraretinal axon guidance.

31 requirement for Hedgehog (Hh) signaling for intraretinal axon pathfinding and show that Shh acts to

32 regional specificity of Slit function during intraretinal axon pathfinding.

33 idance molecule ephrinB2, was increased, and intraretinal axons were disorganised resulting in a fail

34 cell axons in living rats for 4 weeks after intraretinal axotomy.

35 The intraretinal b- and c-wave amplitudes decreased most dra

36 Longer, weaker flashes were used to elicit intraretinal b- and c-waves.

37 gas tamponade can safely create an effective intraretinal barrier to fluid egress from the optic disc

38 An effective intraretinal barrier to fluid migration from cavitary op

39 tantial and boosts the membrane potential of intraretinal blood vessels to a suprahyperpolarized leve

40 ied quantum dots in the choriocapillaris and intraretinal capillaries upon i.v. injection and 1-h cir

41 distinctive traits included the presence of intraretinal cavitation that could affect all retinal la

42 Multiple RPE fates in AMD, including intraretinal cells that are highly prognostic for progre

43 developed into preretinal NV with contiguous intraretinal components.

44 Intraretinal concentrations of glutamate and its main tr

45 Intraretinal crystals, visible as hyperreflective dots,

46 foci numbers (P = 0.038) and mean ODR of the intraretinal cyst (P = 0.006).

47 The optical density ratio (ODR) of the intraretinal cyst and the numbers of hyperreflective foc

48 ased macular thickness and the absence of an intraretinal cyst.

49 plete closure of the FTMH with resolution of intraretinal cystic changes was confirmed on OCT at 16 m

50 cribed features such as fluorescein-negative intraretinal cystic changes, choroidal neovascularizatio

51 ic patterns of fluid presentation, including intraretinal cystic spaces (ICS), retinal pigment epithe

52 tical coherence tomography revealed multiple intraretinal cystic spaces and hyperreflective deposit i

53 yporeflective lumen, typically surrounded by intraretinal cystic spaces.

54 earning to automatically detect and quantify intraretinal cystoid fluid (IRC) and subretinal fluid (S

55 Assessed morphologic parameters included intraretinal cystoid fluid (IRC), subretinal fluid (SRF)

56 y masked reading centers for the presence of intraretinal cystoid fluid (IRC), subretinal fluid (SRF)

57 Tractional intraretinal cystoid spaces (24/72 eyes, 33.3%), display

58 Exudative intraretinal cystoid spaces (36/72 eyes, 50%) displayed

59 P < .001), maxi peaks (5% vs 88%, P < .001), intraretinal cystoid spaces (72% vs 40%, P < .038), oute

60 A cohort of patients diagnosed with intraretinal cystoid spaces and imaged with optical cohe

61 Exudative and tractional intraretinal cystoid spaces displayed characteristic mul

62 Intraretinal cystoid spaces were observed in 34 eyes (68

63 n sham-treated patients by submacular fluid, intraretinal cystoid spaces, and renal disease.

64 escence, full-thickness retinal involvement, intraretinal cystoid spaces, ellipsoid zone disruption,

65 d patients, and by poor baseline BCVA, large intraretinal cystoid spaces, renal disease, and absence

66 was assessed by standardized OCT, including intraretinal cysts (IRCs), subretinal fluid (SRF), and p

67 showing retinal morphologic changes, such as intraretinal cysts (IRCs), subretinal fluid (SRF), and p

68 = 0.0045), thicker SRF (P = 0.0006), larger intraretinal cysts (P = 0.0015), and higher percentage o

69 esence of features of nAMD disease activity (intraretinal cysts [IRC], subretinal fluid [SRF], diffus

70 Patients with intraretinal cysts and intraretinal fluid at baseline ha

71 phy (OCT) features such as subretinal fluid, intraretinal cysts and intraretinal fluid were assessed

72 Baseline OCT features (intraretinal cysts and subretinal fluid) are useful pred

73 fidence interval [CI] 0.32-0.93, p = 0.025), intraretinal cysts at baseline (OR 2.95, 95% CI 1.67-5.2

74 Intraretinal cysts consistently showed the lowest BCVA g

75 f active myopic CNV (either subretinal fluid/intraretinal cysts on SD OCT or dye leakage on fluoresce

76 Intraretinal cysts resolved most rapidly followed by SRF

77 coherence tomography can clearly demonstrate intraretinal cysts which may not be clinically detectabl

78 Elimination of submacular fluid, intraretinal cysts, and severe thickening are important

79 Occurrence of intraretinal cysts, DRIL length, and lens status were si

80 l 1-mm subfield thickness, the occurrence of intraretinal cysts, ellipsoid zone disruption, and disor

81 Patients with DME and submacular fluid, intraretinal cysts, severe thickening, or renal disease

82 The exact intraretinal depth of microaneurysms on OCTA was localiz

83 roaneurysms than FA, but located their exact intraretinal depth.

84 ing degrees of venous stasis retinopathy and intraretinal edema overlying the macular detachment.

85 f these features include photoreceptor loss, intraretinal edema, and retinal thinning overlying choro

86 oreceptors overlying choroidal melanoma; and intraretinal edema, retinoschisis, and retinal thinning

87 increased retinal vessel leakage and caused intraretinal edema.

88 Vitreal and intraretinal ERGs were recorded from eight dark-adapted,

89 l architecture, cystoid macular edema (CME), intraretinal exudates and subretinal lipid aggregation,

90 ge (n = 4), subretinal fluid (n = 4), and/or intraretinal exudation (n = 1).

91 ssociated with small retinal hemorrhages and intraretinal exudation.

92 as found for eyes that displayed fluid, NSD, intraretinal flecks, and low reflectivity or undefined b

93 fluid; 68% and 88% for NSD; 81% and 83% for intraretinal flecks; 63% and 92% for undefined boundarie

94 8 mm2, P < .001), and higher proportions of intraretinal fluid (82.5% vs 51.0%, P < .001), subretina

95 Our model can also detect the presence of intraretinal fluid (AUC: 0.81; 95% CI: 0.81-0.86) and su

96 ere created on the basis of baseline CME and intraretinal fluid (IRF) status: (1) CME, (2) IRF withou

97 At 5 years, 60% of eyes had intraretinal fluid (IRF), 38% had subretinal fluid (SRF)

98 ence of persistent subretinal fluid (SRF) or intraretinal fluid (IRF), and on-study events (atrophy s

99 Three-dimensional volumes (nanoliters) for intraretinal fluid (IRF), subretinal fluid (SRF), and pi

100 dependently graded OCT scans for presence of intraretinal fluid (IRF), subretinal fluid (SRF), and su

101 tal thickness at the foveal center point and intraretinal fluid (IRF), subretinal fluid (SRF), and su

102 At 2 years, intraretinal fluid (IRF), subretinal fluid (SRF), sub-re

103 s such as neurosensory retina (NSR), drusen, intraretinal fluid (IRF), subretinal fluid (SRF), subret

104 Intraretinal fluid (IRF), subretinal fluid (SRF), subret

105 until either complete resolution of SRF and intraretinal fluid (IRF; intensive arm: SRF intolerant)

106 arameters (central subfield thickness [CST], intraretinal fluid [IRF], or subretinal fluid [SRF]) ver

107 TA) signals corresponding to hyperreflective intraretinal fluid across various exudative maculopathie

108 teristics included subretinal fluid (n = 5), intraretinal fluid and cysts (n = 1), and subretinal hyp

109 This was particularly true for intraretinal fluid and difficult cases (with lower fluid

110 reported to be "inactive" (i.e., absence of intraretinal fluid and hemorrhages).

111 first sustained absence of retinal fluid and intraretinal fluid as evaluated by OCT with respect to C

112 Patients with intraretinal cysts and intraretinal fluid at baseline had worse BCVA at month 3

113 differences in the presence of subretinal or intraretinal fluid at final evaluation, dye leakage on a

114 llow eye (aHR, 2.07; 95% CI, 1.40-3.08), and intraretinal fluid at the foveal center (aHR, 2.10; 95%

115 osits, subretinal fibrous scars, and cystoid intraretinal fluid collections in the macula.

116 For C3, the proportion with intraretinal fluid decreased from 78% to 69% to 64% (P =

117 circumscribed vessels, subretinal fluid, and intraretinal fluid each were seen in 71% (22/31).

118 nts (50%) had edema resolution defined as no intraretinal fluid for 6 months or more after the last i

119 here was a reduction of either subretinal or intraretinal fluid in 18 of 36 (50.0%) of the treated ey

120 At M01, subretinal fluid was seen in 28.5% intraretinal fluid in 67.2%, DRIL was seen in 73.8%, mos

121 0% to 12% (P = .05), and the proportion with intraretinal fluid increased from 72% to 71% to 82% (P =

122 7 25-line raster scans confirmed subretinal/intraretinal fluid not identified by the 6-line radial (

123 oth eyes showed improvement in subretinal or intraretinal fluid on OCT.

124 RPE drusen complex abnormal thinning volume, intraretinal fluid or cystoid spaces, hyperreflective fo

125 brane; (3) presence, location, and amount of intraretinal fluid or subretinal fluid (SRF); (4) presen

126 rns for each fluid compartment individually: Intraretinal fluid showed the greatest and most rapid re

127 st sustained absence of retinal fluid and of intraretinal fluid than eyes with predominantly classic

128 nable immunoglobulins along with accumulated intraretinal fluid to flow into the subretinal space, cr

129 At week 100, central macular intraretinal fluid volume was reduced by >65% (P < 0.001

130 as subretinal fluid, intraretinal cysts and intraretinal fluid were assessed by reading-center certi

131 tion (RAP) lesion, GA in the fellow eye, and intraretinal fluid were associated with a higher risk of

132 s underwent OCT imaging; 7 eyes demonstrated intraretinal fluid, 4 eyes were found to have an epireti

133 tip (COST) visibility, cysts, subretinal and intraretinal fluid, and epiretinal membranes.

134 ive material, pigment epithelial detachment, intraretinal fluid, and sub-retinal pigment epithelium f

135 ity on SD OCT (presence of subretinal fluid, intraretinal fluid, and/or cystoid spaces); (2) evidence

136 indings, patients were categorized as having intraretinal fluid, epiretinal membrane, or optic nerve

137 sed macular thickness and an accumulation of intraretinal fluid, indicating macular oedema.

138 e risk factors included poor VA, RAP, foveal intraretinal fluid, monthly dosing, and treatment with r

139 5 years, NFS eyes demonstrated less GA, less intraretinal fluid, more subretinal fluid, and less subr

140 reasing age, increasing CST, the presence of intraretinal fluid, pigment epithelial detachment, and s

141 New foveal scar, CNV, intraretinal fluid, SHRM and retinal thinning, developme

142 -scans of each cube scan for the presence of intraretinal fluid, subretinal fluid, and sub-retinal pi

143 For detecting intraretinal fluid, the investigator metrics were 0.815

144 subfield thickness (CST), subretinal fluid, intraretinal fluid, vitreoretinal interface abnormalitie

145 ubretinal fluid when compared with eyes with intraretinal fluid.

146 lectivity and the presence of subretinal and intraretinal fluid.

147 6/7) had subretinal fluid, and 14% (1/7) had intraretinal fluid.

148 ed 7 eyes without subretinal fluid, but with intraretinal fluid.

149 rcumscribed), and presence of subretinal and intraretinal fluid.

150 in year 2 were treated with ranibizumab for intraretinal fluid.

151 ntage of retinal volume that was occupied by intraretinal fluid.

152 (ONL) thinning; presence of hyper-reflective intraretinal foci; dome-shaped pigment epithelium detach

153 >/=20/40), scar (OR 2.21, 95% CI:1.22-4.01), intraretinal foveal fluid on optical coherence tomograph

154 subtle but significant mistakes during their intraretinal growth and inappropriately defasciculate al

155 he retina, the optic disc, a process termed "intraretinal guidance".

156 bullous retinal schisis with pre-retinal and intraretinal haemorrhages.

157 hments were identified, with the presence of intraretinal hemmorhage predicting a false-positive exam

158 t most features; however, with limitation to intraretinal hemorrhage and pigment migration.

159 namic therapy-related complications included intraretinal hemorrhage in 1 eye.

160 hy; however, lower for pigment migration (or intraretinal hemorrhage).

161 le a vaso-occlusive event and include edema, intraretinal hemorrhage, and nonperfusion detected by fl

162 leads to impaired blood vessel sprouting and intraretinal hemorrhage, particularly during formation o

163 g of the optic nerve, macular edema, diffuse intraretinal hemorrhages, and dilated and tortuous retin

164 Cotton-wool spots, intraretinal hemorrhages, and hard exudates in the macul

165 ections resulted in significant reduction in intraretinal HEs that paralleled reductions in macular t

166 This study sought to quantify the change in intraretinal HF distribution and its correlation with ag

167 ith an increased risk of development of CNV: intraretinal hyper-reflective foci had an HR of 11.58 (9

168 The volume of intraretinal hyper-reflective foci was significantly lar

169 presence of ultrastructural features such as intraretinal hyperreflective flecks and the inherent ref

170 Intraretinal hyperreflective foci associated with acquir

171 e use of cholesterol-lowering medication and intraretinal hyperreflective foci attributable to RPE ce

172 Intraretinal hyperreflective foci correlated with intrar

173 ons were observed prior to the occurrence of intraretinal hyperreflective foci in 75% of cases.

174 tion was significantly associated with SDOCT intraretinal hyperreflective foci in the 314 study eyes

175 retinal anatomic changes and the pattern of intraretinal hyperreflective foci migration were documen

176 defined by the presence of depolarization at intraretinal hyperreflective foci on PS-SLO and PS-OCT i

177 Histologic evaluation showed that intraretinal hyperreflective foci represent cells of ret

178 One donor eye with intraretinal hyperreflective foci was identified in a pa

179 The occurrence of intraretinal hyperreflective foci was not a significant

180 The appearance of intraretinal hyperreflective foci was preceded by thicke

181 Intraretinal hyperreflective foci were associated with a

182 the abundance of lines and association with intraretinal hyperreflective foci.

183 surrounded by thick delaminated retina with intraretinal hyperreflective lesions.

184 irregularities, abnormal retinal thickness, intraretinal hyperreflective/hyporeflective features, an

185 r than FP for abnormal retinal thickness (or intraretinal hyporeflective features); similar as FP for

186 Intraretinal immunofluorescence of ApoB100 increased wit

187 ation within the retina and exert widespread intraretinal influence.

188 Central intraretinal ion activity and retinal thickness were mea

189 Central retinal thickness and intraretinal ion activity were measured from the MEMRI d

190 Central retinal thickness and intraretinal ion channel regulation were measured from t

191 Intraretinal ion demand and retinal thickness were measu

192 Panretinal TI(v) was not correlated with intraretinal ion demand in any case.

193 owerful approach for measuring alteration in intraretinal ion demand in models of ocular injury.

194 nhanced MRI (MEMRI), assesses alterations in intraretinal ion demand in models of ocular insult.

195 irst-time evidence for changes (P < 0.05) in intraretinal ion regulation before and during pathologic

196 tive foci (HRF), average and largest area of intraretinal (IR) cysts, and extent of disruption of ext

197 Enzymatic cleavage of intraretinal laminin is a biologically plausible mechani

198 nce Algorithms (version 3.8.0) were used for intraretinal layer segmentation, and mean thickness of i

199 OCT scanning, including automated intraretinal layer segmentation, yielded thicknesses of

200 t algorithm (OCTRIMA) to measure locally the intraretinal layer thickness.

201 eflectance and fractal dimension) of various intraretinal layers extracted from optical coherence tom

202 al layer segmentation, and mean thickness of intraretinal layers was rescaled with magnification corr

203 ne photoreceptor length and the thickness of intraretinal layers were measured and compared to previo

204 Among the intraretinal layers, the inner nuclear layer was identif

205 murine retina and visualization of all major intraretinal layers.

206 In Akita mouse retinas, diabetes increased intraretinal levels of oxidized LDL and glycated LDL, in

207 inal injection of 1% hyaluronic acid and the intraretinal levels of the autophagy proteins LC3 and At

208 acute retinal injury (consisting of abnormal intraretinal light scattering) were visualized in vivo i

209 These findings suggest that intraretinal macular hemorrhage is an important indicato

210 Intraretinal manganese ion uptake and retinal thickness

211 of young and aged mice revealed a subnormal intraretinal manganese uptake (P < 0.05) in aged DBA/2J

212 provide proof-of-concept that the extent of intraretinal manganese uptake after systemic MnCl(2) inj

213 In diabetic WT mice, intraretinal manganese uptake became subnormal between 1

214 In separate experiments, intraretinal manganese uptake was also measured in adult

215 After sodium iodate exposure, intraretinal manganese uptake was supernormal (P < 0.05)

216 adult rats, diltiazem suppressed (P < 0.05) intraretinal manganese uptake.

217 These intraretinal measurements in cats provide further eviden

218 However, macular intraretinal measurements still have not overcome standa

219 raphy showed a normal foveal contour without intraretinal microcystic spaces and a resolution of the

220 In severe NPDR, the eyes with intraretinal microvascular abnormalities (IRMA) had a si

221 n area, vessel density (VD), and presence of intraretinal microvascular abnormalities (IRMA).

222 uous intraretinal vascular segments known as intraretinal microvascular abnormalities (IRMAs) are a k

223 Microaneurysms, intraretinal microvascular abnormalities (IRMAs), and ne

224 p hemorrhages (DH), venous beading (VB), and intraretinal microvascular abnormalities (IRMAs).

225 Moderate agreement of intraretinal microvascular abnormalities and venous bead

226 For soft exudates, intraretinal microvascular abnormalities, and venous bea

227 microaneurysm [ma], cotton wool spot [CWS], intraretinal microvascular abnormality [IRMA]) were manu

228 f the disc, neovascularization elsewhere, or intraretinal microvascular abnormality was associated wi

229 ose of the present study was to evaluate the intraretinal migration of the retinal pigment epithelium

230 dal anastomosis was found, 3 patients showed intraretinal neovascularization connected with a pigment

231 Abnormal flow was defined as intraretinal neovascularization or retinal choroidal ana

232 tion of Srf in adult murine vessels elicited intraretinal neovascularization that was reminiscent of

233 pithelial detachment, 2 patients showed only intraretinal neovascularization, and in 2 patients flow

234 blood-retinal barrier breakdown and, later, intraretinal neovascularization.

235 nting extrinsic macrophages, were present in intraretinal ON region, but not in the retroscleral (iso

236 t was worst for subretinal fluid compared to intraretinal or sub-retinal pigment epithelial fluid.

237 within 6 months with classic features of new intraretinal or sub-retinal pigment epithelium infiltrat

238 es with and without persistent fluid (cystic intraretinal or subretinal fluid at all 4 initial visits

239 In 11 of 19 patients with intraretinal or subretinal fluid at baseline judged to b

240 eneration who exhibit recurrent or resistant intraretinal or subretinal fluid following multiple inje

241 line at the last follow-up and/or persistent intraretinal or subretinal fluid or detectable choroidal

242 Retreatment indication was recurrence of intraretinal or subretinal fluid or new hemorrhage.

243 Retreatment criteria relying on intraretinal or subretinal fluid or new hemorrhages may

244 nthly with intravitreal bevacizumab until no intraretinal or subretinal fluid was observed on optical

245 ts with resistant or multiple recurrences of intraretinal or subretinal fluid while receiving monthly

246 ted visual acuity (BCVA) 20/40 or worse, and intraretinal or subretinal fluid with central foveal thi

247 including a thinned outer nuclear layer and intraretinal or subretinal fluid.

248 r irregularity of each category (epiretinal, intraretinal, or RPE/choroidal irregularity), 3D-OCT was

249 n of tempol, a superoxide scavenger, reduced intraretinal oxidized LDL and glycated LDL levels, PGIS

250 Intraretinal oxidized LDL was absent in nondiabetic subj

251 Intraretinal oxygen (PO2) profiles were recorded with ox

252 Intraretinal (P < .001) and subretinal (P < .001) fluid

253 Intraretinal (P = .003) and subretinal (P = .046) fluid

254 e qualitatively and quantitatively identical intraretinal pathfinding errors to those reported previo

255 PL appeared to have a saw-tooth appearance ("intraretinal peaks") in 12 eyes (75%).

256 After 1 day, the intraretinal photocoagulation lesions were sharply demar

257 zed retinal dysfunction, peripheral lacunae, intraretinal pigment migration, and hyperautofluorescent

258 nation features included macular edema, mild intraretinal pigment migration, and widespread atrophy i

259 heral retinal pigment epithelial atrophy and intraretinal pigment migration.

260 eposits, differing from the classic spicular intraretinal pigmentation observed in other individuals

261 ensitive microelectrodes were used to record intraretinal Po(2) profiles from healed photocoagulation

262 does not cause the immediate degeneration of intraretinal portions of axons or the immediate death of

263 s used to calculate the fractal dimension in intraretinal regions of interest identified in the image

264 Increased peripheral intraretinal retinal manganese uptake was associated wit

265 The solid intraretinal retinoblastoma and subretinal seeds showed

266 extent of preretinal neovascularization and intraretinal revascularization was quantified by image a

267 rovascular networks were analyzed to examine intraretinal revascularization, capillary sprouting, and

268 retinal hyperreflective foci correlated with intraretinal RPE and lipid-filled cells of probable mono

269 Intraretinal RPE migration was defined by the presence o

270 Overall, our results showed that intraretinal RPE migrations occurred in various AMD stag

271 Each model developed intraretinal schisis and reductions in the ERG that were

272 The OCT showed intraretinal schitic cavities in the majority of eyes.

273 Intraretinal signal intensity returned to baseline by 7

274 Hyporeflective intraretinal spaces, indicating cystoid or schitic fluid

275 Here, we test the hypothesis that intraretinal spin-lattice relaxation rate in the rotatin

276 Intraretinal steric interactions as well as electronic e

277 Blood flow was mostly confined to the intraretinal structures with or without a connecting pig

278 and progressing to the subretinal space with intraretinal, subretinal, and choroidal angiogenic stage

279 in optical coherence tomography (presence of intraretinal, subretinal, and subretinal pigment epithel

280 omic improvement in patients with persistent intraretinal, subretinal, or subretinal pigment epitheli

281 aflibercept injection due to the presence of intraretinal/subretinal fluid and pigment epithelial det

282 macular thickness (CMT), and the presence of intraretinal/subretinal fluid and the height and presenc

283 and 25-line raster scans were evaluated for intraretinal/subretinal fluid and, when applicable, vitr

284 dth, as well as of peripapillary and macular intraretinal thickness measurements.

285 nfirmed that the precursor IRMA lesions were intraretinal tortuous vascular lesions at baseline and t

286 ver a mean of 13 months follow-up, there was intraretinal tumor recurrence (n = 1), subretinal seed r

287 Peripheral intraretinal uptake of manganese was significantly super

288 , and a surrogate of retinal ion regulation (intraretinal uptake of manganese) were assessed from MEM

289 Intraretinal variation in opsin abundance consisted of g

290 roduction leads to characteristic defects in intraretinal vascular architecture.

291 eovascular complexes is transmitted into the intraretinal vascular network.

292 Tortuous intraretinal vascular segments known as intraretinal mic

293 cell dysfunction induces alterations to the intraretinal vasculature and substantial visual deficits

294 e that specific retinal interneurons and the intraretinal vasculature are highly interdependent, and

295 required for generating and maintaining the intraretinal vasculature through precise regulation of h

296 Intraretinal vessel development was not altered by the i

297 cytes, and staining was increased around new intraretinal vessels in mouse OIR and rat retinopathy of

298 angiogenesis by ordering the development of intraretinal vessels.

299 ark adaptation of M-cones driven by both the intraretinal visual cycle and the retinal pigmented epit

300 uration are believed to depend in part on an intraretinal visual cycle that supplies 11- cis-retinald