ライフサイエンスコーパス: intrathoracic

1 patients had positive (18)F-FDG PET results (intrathoracic (18)F-FDG uptake), including 4 patients wi

2 he time of transplantation (peripheral, 94%; intrathoracic, 6%).

3 ormance, cardiac pathology, and extracardiac intrathoracic abnormalities.

4 pe is safe and effective in the diagnosis of intrathoracic adenopathy in HIV-infected patients, and i

5 This was mediated by intrathoracic air redistribution to the left and non-gra

6 suscitation (CPR), but it can be affected by intrathoracic airway closure.

7 onship between gene expression in extra- and intrathoracic airway epithelial cells and extend the con

8 During chest compressions, intrathoracic airway patency greatly affects the deliver

9 were continuously monitored, and WL from the intrathoracic airways was calculated from published rela

10 Outcomes following intrathoracic anastomoses (n = 621) were analyzed by era

11 Clinical decisions regarding the use of intrathoracic anastomoses should not be affected by conc

12 rvention occurred in 15 of 122 patients with intrathoracic anastomosis (12.3%) and in 39 of 123 patie

13 udy was to evaluate the impact of performing intrathoracic anastomosis and/or thoracotomy on POM afte

14 erall anastomotic leak rate was 12.3% in the intrathoracic anastomosis group and 34.1% in the cervica

15 oral feeding (intervention) after a MIE with intrathoracic anastomosis or to receive nil-by-mouth and

16 Nowadays, intrathoracic anastomosis provides a lower 30-day POM ra

17 In this randomized clinical trial, intrathoracic anastomosis resulted in better outcome for

18 of life were comparable between groups, but intrathoracic anastomosis was associated with fewer seve

19 ontinues to be partial esophagectomy with an intrathoracic anastomosis, which was associated with low

20 wis resection, which provides a tension-free intrathoracic anastomosis.

21 of care, control group) following a MIE with intrathoracic anastomosis.

22 bility, effectiveness, and safety of EVT for intrathoracic anastomotic leakage following abdomino-tho

23 Especially intrathoracic anastomotic leakage leads to life-threaten

24 EVT is a safe and effective approach for intrathoracic anastomotic leakages following abdomino-th

25 Complication rates for intrathoracic and abdominal/pelvic solid organ biopsies

26 Leakage rates were similar in intrathoracic and cervical anastomoses (15.9% vs 17.2%,

27 Radiotherapy of intrathoracic and chest wall tumors may lead to exposure

28 ry artery calcification, cardiac morphology, intrathoracic and extrathoracic fat, and osteoporosis.

29 ghly aggressive and metastasized to multiple intrathoracic and extrathoracic sites in a pattern simil

30 In MCS, intracranial, intrathoracic and intra-abdominal compartment pressures

31 clude ICP control as well as minimization of intrathoracic and intra-abdominal pressure as clinically

32 Because intrathoracic and intraabdominal organs are usually reco

33 the kidneys are usually recovered after the intrathoracic and intraabdominal organs, careful palpati

34 Intrathoracic and intraabdominal pressures were measured

35 tricular interdependence and dissociation of intrathoracic and intracardiac pressures for the diagnos

36 ix steady-state impedance signals, utilizing intrathoracic and intracardiac vectors, were measured th

37 However, intrathoracic and pericardial fat are associated with va

38 Intrathoracic and pericardial fat volumes were examined

39 Both intrathoracic and pericardial fat were associated with h

40 Intrathoracic and pericardial fat were directly correlat

41 With DIAm contraction, a negative intrathoracic and positive abdominal pressure are genera

42 The MRI scans revealed the intrathoracic and subcutan masses as mainly hypointense

43 y was to present a case report of a combined intrathoracic and subcutaneous splenosis in a patient 19

44 Together they determine the intraabdominal, intrathoracic, and subglottic pressure, control of which

45 en-chest dogs underwent surgery in which the intrathoracic aorta was bypassed with a stiff plastic tu

46 The impact of manual segmentation of TR's intrathoracic area and enhancing contrast method on the

47 Results A total of 110 intrathoracic biopsies were performed, and 101 (91.8%) w

48 t, body surface area, body mass index, Pao2, intrathoracic blood volume, cardiac output, or dosage of

49 between extrathorcic (buccal and nasal) and intrathoracic (bronchial) epithelium in healthy current

50 Stellate ganglia within the intrathoracic cardiac control system receive and integra

51 Location of intrathoracic catheters was obscured on 44 radiographs.

52 s a long-term side effect of radiotherapy of intrathoracic, chest wall and breast tumors when radiati

53 lky disease (mass > 10 cm or 1/3 the maximum intrathoracic diameter on chest x-ray) received two cycl

54 al bulky mediastinal mass 33% or more of the intrathoracic diameter, and/or "B" symptoms and all stag

55 acoscopic approach in patients with possible intrathoracic difficulties.

56 hildren, particularly in young children with intrathoracic disease as this represents the most common

57 sites of disease and 49 with more extensive intrathoracic disease at CT.

58 Intrathoracic disease recurred within the radiation fiel

59 y invasive surgical techniques primarily for intrathoracic disease.

60 ylactic cranial irradiation, have persistent intrathoracic disease.

61 were identified in 966 patients after ttMIE: intrathoracic end-to-side circular-stapled technique in

62 Leakage rates after intrathoracic end-to-side double-stapling (23.3%) and ce

63 cted, assigning 112 patients with resectable intrathoracic esophageal cancer to either RAMIE or OTE.

64 9 and March 2011, patients with a resectable intrathoracic esophageal carcinoma, including the gastro

65 After intrathoracic esophageal mobilization (median 9 cm), TSE

66 ruction was done by gastric pull-up and high intrathoracic esophagogastrostomy.

67 ment of epicardial adipose tissue (EATv) and intrathoracic fat (ITFv) volumes.

68 , pericardial fat (r=0.19 to 0.37, P<0.001), intrathoracic fat (r=0.17 to 0.31, P<0.001), visceral ad

69 , pericardial fat (r=0.20 to 0.35, P<0.001), intrathoracic fat (r=0.25 to 0.37, P<0.001), visceral ad

70 fined by pericardial, visceral, hepatic, and intrathoracic fat); and (3) muscle attenuation that expl

71 Pericardial fat, intrathoracic fat, and visceral adipose tissue quantifie

72 nce interval 1.005 to 1.46, P=0.04), whereas intrathoracic fat, but not pericardial fat, was associat

73 tomography study underwent quantification of intrathoracic fat, pericardial fat, visceral abdominal f

74 Pericardial fat, but not intrathoracic fat, was associated with coronary artery c

75 an-based depiction of common and less common intrathoracic findings directly caused by leukemic invol

76 l/familial disease, pre-existing lung cysts, intrathoracic findings, and treatments (surgery or surge

77 s of alert-driven interventions triggered by intrathoracic fluid index threshold crossings (FTC) were

78 ormed for a malignant tumor and multinodular intrathoracic goiters.

79 hage (16%), delayed intervention for ongoing intrathoracic hemorrhage (9%), inadequate DVT or gastroi

80 n/dehiscence, 3%, atelectasis/pneumonia, 2%, intrathoracic hemorrhage, recurrent laryngeal nerve para

81 otic leak (13%), atelectasis/pneumonia (2%), intrathoracic hemorrhage, recurrent laryngeal nerve para

82 obtained during a follow-up of 10 h using an intrathoracic high-resolution coil.

83 tability of pulmonary C-fibres is induced by intrathoracic hyperthermia, and this enhanced sensitivit

84 breast, 1 cervical, 1 bladder), and 3 had an intrathoracic imaging abnormality.

85 Direct measurements of intrathoracic impedance using an implanted device can be

86 ding pulmonary and left atrial pressures, or intrathoracic impedance, which is related to pulmonary c

87 Intrathoracic impedance-derived OptiVol fluid index calc

88 dgut; bypassing this barrier by experimental intrathoracic infection of the mosquito eliminates the n

89 Intrathoracic injection of function-blocking antibodies

90 After intrathoracic injection, ONNV-eGFP slowly spread to othe

91 ection rates are not affected by sfRNA after intrathoracic injection, thereby identifying sfRNA as a

92 rison of infection via the blood meal versus intrathoracic injection, which bypasses the midgut, reve

93 ito infection via infectious blood meals and intrathoracic injections showed that sfRNA is important

94 ation was pneumothorax (at 32 [28.6%] of 112 intrathoracic injections), for which only one patient re

95 mutF showed restricted replication following intrathoracic inoculation in the mosquito Toxorhynchites

96 After intrathoracic inoculation into mosquitoes, both viruses

97 Intrathoracic inoculation of VLPs into mosquitoes demons

98 to cells and in Culex quinquefasciatus after intrathoracic inoculation.

99 /ml) in mosquitoes 4 days after infection by intrathoracic inoculation.

100 ve pulmonary disease, and to dissociation of intrathoracic-intracardiac pressure changes in constrict

101 High intrathoracic (Ivor Lewis) and cervical esophagogastrost

102 m scans in AIDS patients for differentiating intrathoracic kaposi sarcoma from malignant lymphoma and

103 An intrathoracic leak following esophagectomy has historica

104 antly different for patients with or without intrathoracic leaks (3.3% versus 2.5%, P = 0.55), nor is

105 Assess outcomes following intrathoracic leaks after esophagectomy from 1970 to 200

106 Modern surgical management of intrathoracic leaks results in no increased mortality an

107 Eleven NSCLC patients, with at least 1 intrathoracic lesion 3 cm or greater, underwent double b

108 Location of an intrathoracic lesion on chest radiograph is facilitated

109 es suggests that even simple-appearing fatty intrathoracic lesions may lead to the development of mal

110 o underwent CT-guided transsternal biopsy of intrathoracic lesions were evaluated retrospectively.

111 y is the most common direct manifestation of intrathoracic leukemia.

112 ciated with prenatal diagnosis, CDHSG stage, intrathoracic liver, and patch repair (all P < 0.001).

113 e FEV1 decreased as WL rose, but the largest intrathoracic losses were associated with the smallest o

114 ed in 24 patients with histologically proved intrathoracic LPD and with positive serologic findings o

115 th size of the primary tumor, cell type, and intrathoracic lymph node stage.

116 Extraparenchymal perflubron was seen in intrathoracic lymph nodes (n = 4), supraclavicular nodes

117 Transbronchial needle aspiration (TBNA) of intrathoracic lymph nodes has been shown to be useful in

118 , one through Xpert MTB/RIF Ultra test) from intrathoracic lymph nodes or bronchial wash and received

119 e-positive with no metastases found in other intrathoracic lymph nodes without concurrent SN involvem

120 ronchial ultrasonography) with aspiration of intrathoracic lymph nodes.

121 CD11c(+hi)/MHC class II(+hi) cell numbers in intrathoracic lymph nodes.

122 tiology that predominantly affects lungs and intrathoracic lymph nodes; in rare cases (approx. 10%),

123 om cutaneous infections, such as cervical or intrathoracic lymphadenitis in children, to disseminated

124 pacity (32%), mass-like consolidation (20%), intrathoracic lymphadenopathy (16%), pleural effusion (1

125 (2.2%) participants were diagnosed with any intrathoracic malignancy after a positive baseline scree

126 f myelomeningocele (MMC, n=51), resection of intrathoracic masses (ITM, n=15), tracheal occlusion for

127 s demonstrated, with the majority exhibiting intrathoracic migration of the wrap with or without disr

128 t masses were infradiaphragmatic (n = 11) or intrathoracic (n = 1).

129 uble-stapling n = 90, purse-string n = 337), intrathoracic (n = 109) or cervical (n = 255) side-to-si

130 Patients had either cervical (n = 548) or intrathoracic (n = 2738) anastomosis.

131 extremity (n = 7), lower extremity (n = 4), intrathoracic (n = 3), sternal (n = 34), breast (n = 3),

132 All of 9 adults followed had an intrathoracic neoplasm, seven biopsied within 7 months (

133 ary to various acquired diseases, especially intrathoracic neoplasm.

134 rine tumors of the pancreas/duodenum and the intrathoracic neuroendocrine tumors that occur in MEN 1

135 .5% for recurrence, 5.6% for NLSC, 16.8% for intrathoracic new cancer, and 10.4% for extrathoracic ca

136 re incidence and timing of recurrence, NLSC, intrathoracic new cancer, extrathoracic cancer, or death

137 was clinically meaningful and distinct from intrathoracic new cancers.

138 Endosonography with intrathoracic nodal aspiration appears to be a promising

139 response to initial therapy, CNS metastases, intrathoracic nodal status, and EGFR and ALK status.

140 the Mlh1-/-;Nf1+/- mice were found to harbor intrathoracic NOS2-immunoreactive myeloid leukemias simi

141 mly assigned (1:1) to transthoracic MIE with intrathoracic or cervical anastomosis.

142 portant additional findings (n=35) about the intrathoracic or intraabdominal organs.

143 ]), vascular (OR, 1.6 [CI, 1.1 to 2.4]), and intrathoracic (OR, 9.2 [CI, 6.7 to 13]) procedures.

144 t diagnosis, the tumor is usually limited to intrathoracic organs.

145 ast cancer; cancers of the lung, pharynx, or intrathoracic organs; other cancer; respiratory disease;

146 d in cats with electrical stimulation of the intrathoracic phrenic nerve and C(5) root of the phrenic

147 h stimulation of myelinated afferents of the intrathoracic phrenic nerve in the contralateral post-cr

148 ied pneumothorax (86% correct) and increased intrathoracic positive end-expiratory pressure (93% corr

149 Intrathoracic pressure (ITP) swings that permit spontane

150 entilation is accomplished by alterations in intrathoracic pressure (ITP), which have physiological i

151 12, 20, and 30 breaths per minute, the mean intrathoracic pressure (mm Hg/min) and coronary perfusio

152 on-decompression CPR with augmented negative intrathoracic pressure (via an impedance-threshold devic

153 to exercise requires substantial changes in intrathoracic pressure and in the work output and metabo

154 al of patients of cardiac arrest by lowering intrathoracic pressure and increasing cardiac output.

155 on rates resulted in significantly increased intrathoracic pressure and markedly decreased coronary p

156 a-induced increase in CFV; however, negative intrathoracic pressure and the small amount of oxyhaemog

157 s an inspiratory pump to generate a negative intrathoracic pressure and thus pull air into the lungs

158 ely, by mechanical effects of respiration on intrathoracic pressure and/or cardiac filling; (3) BP va

159 onomic tone, lung volume, heart location and intrathoracic pressure are all varying during the respir

160 It may relate to increased intrathoracic pressure associated with retching and vomi

161 ves were compared between data obtained with intrathoracic pressure at atmospheric and with a phasic

162 on, hypoxia, hypoventilation, and changes in intrathoracic pressure can lead to severe hemodynamic in

163 Intrathoracic pressure changes are of particular importa

164 tions, with closed-chest and phasic negative intrathoracic pressure changes similar to those associat

165 onary arterial baroreceptors were altered by intrathoracic pressure changes similar to those encounte

166 acic pressure was at atmospheric, the phasic intrathoracic pressure decreased the pulmonary arterial

167 This new device enhances negative intrathoracic pressure during chest wall recoil or the d

168 d neck tissues as the generation of negative intrathoracic pressure during inspiration increases veno

169 Increased negative intrathoracic pressure during spontaneous inspiration th

170 Generation of negative intrathoracic pressure during the decompression phase of

171 pulmonary resuscitation (CPR) with decreased intrathoracic pressure in the decompression phase can le

172 downward flow of venous blood due to reduced intrathoracic pressure is counterbalanced by an upward m

173 Elevated intrathoracic pressure may be similarly associated with

174 ro which may be related to the effect of the intrathoracic pressure on cardiac afterload and blood ej

175 here is a negative impact of a high level of intrathoracic pressure on hemodynamic and cardiac tolera

176 An increase in intrathoracic pressure played a deleterious role in Font

177 ic pressure at atmospheric and with a phasic intrathoracic pressure ranging from atmospheric to aroun

178 A novel device, the intrathoracic pressure regulator (ITPR), combines an ins

179 r with active compression-decompression plus intrathoracic pressure regulator compared with active co

180 w with active compression-decompression plus intrathoracic pressure regulator plus epinephrine were s

181 r, and active compression-decompression plus intrathoracic pressure regulator plus epinephrine.

182 t with active compression-decompression plus intrathoracic pressure regulator significantly improved

183 evice, active compression-decompression plus intrathoracic pressure regulator, and active compression

184 t with active compression-decompression plus intrathoracic pressure regulator.

185 t with active compression-decompression plus intrathoracic pressure regulator; and group C-3 minutes

186 n very severe COPD, the impressive swings in intrathoracic pressure resulting from deranged ventilato

187 ompression CPR with augmentation of negative intrathoracic pressure should be considered as an altern

188 g expiration to take advantage of changes in intrathoracic pressure that assist in postural maintenan

189 disease (COPD) may contribute to changes in intrathoracic pressure that increase LV wall stress.

190 ic vascular resistance and abrupt changes in intrathoracic pressure that occur with resistive exercis

191 Because obstructive events generate negative intrathoracic pressure that reduces left ventricular (LV

192 t the ITD would result in a greater negative intrathoracic pressure to enhance cardiac venous return,

193 Compared to the values obtained when intrathoracic pressure was at atmospheric, the phasic in

194 Furthermore, qualitative changes in intrathoracic pressure were without influence on the res

195 se findings suggest that increasing negative intrathoracic pressure with ITD breathing improves heart

196 tory variation is due to increased change in intrathoracic pressure with respiration in chronic obstr

197 s/min combined with augmentation of negative intrathoracic pressure would lower intracranial pressure

198 ttern in the superior vena cava (affected by intrathoracic pressure) would be different in these two

199 is, hypothermia, hypervolemia, and increased intrathoracic pressure).

200 ransfusion, mechanical ventilation with high intrathoracic pressure, and acidosis, among others.

201 in end-expiratory lung volume and increased intrathoracic pressure, eventually exacerbated by expira

202 These results have shown that a phasic intrathoracic pressure, which simulates respiratory osci

203 e end-expiratory pressure, without affecting intrathoracic pressure.

204 airflow and helps maintain higher levels of intrathoracic pressure.

205 y are influenced by physiological changes in intrathoracic pressure.

206 intraabdominal pressure leading to increased intrathoracic pressure.

207 usion also occurs in the absence of negative intrathoracic pressure.

208 n (CPR) by increasing the degree of negative intrathoracic pressure.

209 this effect by augmenting pleural and other intrathoracic pressures and causing a functional obstruc

210 wall compliance both increase the change in intrathoracic pressures and the value of the dynamic ind

211 negative inspiratory and positive expiratory intrathoracic pressures cancel each other out, so averag

212 ture, and accompanying increases in negative intrathoracic pressures directly affecting cardiac funct

213 Application of phasic negative intrathoracic pressures further reduced the threshold an

214 d and resealed, and (c) with phasic negative intrathoracic pressures in the resealed chest.

215 olume (4, 6, 8, and 10 mL/kg), the change in intrathoracic pressures increased linearly with 0.9 +/-

216 as exchange was achieved at lower airway and intrathoracic pressures than those that developed during

217 Piglets' hemodynamic and intrathoracic pressures were continuously monitored duri

218 od gases, arousals, large negative swings in intrathoracic pressures, and increased sympathetic activ

219 iratory muscle function to generate elevated intrathoracic pressures.

220 magnitude of respiratory phasic variation of intrathoracic pressures.

221 piratory event common to all humans, but the intrathoracic processes remain poorly understood.

222 Intrathoracic progression remains the predominant patter

223 Seventeen (55%) of 31 episodes involved intrathoracic PTLD manifesting as multiple pulmonary nod

224 Intrathoracic PTLD occurred more commonly in lung transp

225 Intrathoracic PTLD tended to manifest early.

226 ) 0-36] vs 29 [IQR 0-40], P = 0.019} favored intrathoracic reconstructions.

227 ents were excluded if it was determined that intrathoracic recurrence had an impact on lung function.

228 ocal recurrence occurred in 7% (five of 72), intrathoracic recurrence in 22% (16 of 72), and extratho

229 and etoposide does not decrease the risk of intrathoracic recurrence or prolong survival in patients

230 ead through air spaces' were associated with intrathoracic recurrence, in contrast to the presence of

231 ques were significantly higher compared with intrathoracic side-to-side linear (15.6%), end-to-side p

232 Intrathoracic splenosis is a rare condition resulting fr

233 e in 93% of patients and involved contiguous intrathoracic structures and/or distant sites, including

234 ies, aorta, pulmonary arteries, and adjacent intrathoracic structures for the patient with acute ches

235 non-treatment-related effects of leukemia on intrathoracic structures will be the focus of this imagi

236 Among patients who had intrathoracic surgery, those receiving digoxin were at l

237 In children investigated for intrathoracic TB enlargement of perihilar or paratrachea

238 lyzed CR data from children with presumptive intrathoracic TB prospectively enrolled in a cohort stud

239 In children with presumptive intrathoracic TB, we aimed to identify CR features that

240 ed and artificially ventilated rats when the intrathoracic temperature (T(it)) was maintained at thre

241 re elicited by mechanical stimulation of the intrathoracic trachea.

242 re elicited by mechanical stimulation of the intrathoracic trachea.

243 infant trials; (2) symptomatic, complicated intrathoracic tuberculosis as an uncommon but clinically

244 should enhance harmonized classification for intrathoracic tuberculosis disease in children across st

245 dized clinical research case definitions for intrathoracic tuberculosis in children to enable harmoni

246 ses in research focusing on the diagnosis of intrathoracic tuberculosis in children.

247 ted children aged </=13 years with suspected intrathoracic tuberculosis were enrolled in 8 hospitals

248 Children with newly diagnosed intrathoracic tuberculosis were enrolled, and they recei

249 icipants agreed that radiologic diagnosis of intrathoracic tuberculosis would be based primarily on h

250 Among patients with pulmonary or intrathoracic tuberculosis, 9% of HIV-seropositive and 1

251 ptomatic children with clinical suspicion of intrathoracic tuberculosis, and were not intended to pre

252 on symptomatic children suspected of having intrathoracic tuberculosis.

253 clearance of lesions on CXR in children with intrathoracic tuberculosis.

254 ent may improve height gain in children with intrathoracic tuberculosis.

255 diagnostics among children, with a focus on intrathoracic tuberculosis.

256 lude mechanisms of injury, potentially fatal intrathoracic vascular injuries, anesthetic management,

257 ers for recording arterial pressure (AP) and intrathoracic vena caval pressure (VP).

258 of a systemic hypercoagulable state over an intrathoracic venous compression mechanism.

259 of lung aeration, tidal flow conditions, and intrathoracic volume distribution calculated for each in

260 rical impedance tomography was used to image intrathoracic volume patterns for every breath until 6 m

261 layed midexpiratory gas flow associated with intrathoracic volume redistribution (pendelluft flow) wi

262 Intrathoracic (vs cervical) anastomosis and a thoracotom

263 30-day POM between patients having received intrathoracic (vs cervical) anastomosis and between thos