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1 almost 50 years after it was approved as an intravenous anesthetic.
2 rgets for the sedative action of ethanol and intravenous anesthetics.
3 little or no effect on the actions of these intravenous anesthetics.
4 ative, immobilizing, and hypnotic effects of intravenous anesthetics, a role for those receptors is l
7 re intravenous antiseizure drug, more than 1 intravenous anesthetic, and intensive care unit admissio
9 uced the patient's anxiety to the point that intravenous anesthetic/anxiolytic medications were disco
11 ed arousal states induced by five classes of intravenous anesthetics by relating their behavioral and
16 nally, TLR4 activation was not attenuated by intravenous anesthetics, except for a high concentration
19 addition, hypnotic sensitivity to two other intravenous anesthetics in HCN1 knock-out mice matched e
24 nd assayed for their activation by GABA, the intravenous anesthetic propofol and the endogenous neuro
27 , the volatile anesthetic isoflurane and the intravenous anesthetic propofol inhibit voltage-gated Na
31 voflurane), a haloalkane (halothane), and an intravenous anesthetic (propofol) to bovine and human se
33 annels, a diazirine derivative of the potent intravenous anesthetic, R-(+)-etomidate (2-ethyl 1-(1-ph
35 are part of a binding site for propofol, an intravenous anesthetic, we probed propofol's ability to