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1 strema of rats fed a high-fat diet increased intravenous glucose tolerance and insulin sensitivity to
2 had normal levels of fasting blood glucose, intravenous glucose tolerance, and HbA1c, and 15 of 16 s
3 asurements of fasting plasma glucose, HbA1c, intravenous glucose tolerance, and insulin secretory res
5 minimal model analyses of frequently sampled intravenous glucose tolerance (FSIGT) from the Insulin R
6 ous adipose biopsies, and frequently sampled intravenous glucose tolerance (FSIGT) testing were perfo
7 eloping T2D with annual measures of oral and intravenous glucose tolerance (IVGTT), body composition,
8 vity index (SI), disposition index (DI), and intravenous glucose tolerance (kg) were compared for eac
9 tion was assessed using a frequently sampled intravenous glucose tolerance test (first-phase insulin
10 aits were measured by the frequently sampled intravenous glucose tolerance test (four cohorts) or eug
11 ients were submitted to a frequently sampled intravenous glucose tolerance test (FSIGT) with the stim
12 rom the insulin-modified, frequently sampled intravenous glucose tolerance test (FSIGT), we estimated
15 ex (SI) calculated from a frequently sampled intravenous glucose tolerance test (FSIVGTT) after the m
16 lowing tests: 1) frequently sampled 0.3-g/kg intravenous glucose tolerance test (FSIVGTT) with MinMod
19 euglycemic hyperinsulinemic clamp (EHC), by intravenous glucose tolerance test (IVGTT) and by oral g
20 condition, we assessed glucose metabolism by intravenous glucose tolerance test (IVGTT) and euglycemi
22 exes with analogous indexes obtained from an intravenous glucose tolerance test (IVGTT) and hyperglyc
23 mpensation (AIR(G)) were assessed through an intravenous glucose tolerance test (IVGTT) and serum bio
26 sing clamp and minimal model analysis of the intravenous glucose tolerance test (IVGTT) to document p
27 e 2 diabetes and physiologic responses to an intravenous glucose tolerance test (IVGTT) to identify n
28 an oral glucose tolerance test (OGTT) and an intravenous glucose tolerance test (IVGTT) were performe
31 dministration of exogenous insulin during an intravenous glucose tolerance test allows the use of the
32 ivity index (S(i)) from a frequently sampled intravenous glucose tolerance test among African-America
33 nsitivity (S(i)) from the frequently sampled intravenous glucose tolerance test among African-America
34 easured directly from the frequently sampled intravenous glucose tolerance test among black, Hispanic
35 s-sectional study in which we carried out an intravenous glucose tolerance test and an oral glucose t
36 an oral glucose tolerance test (OGTT) and an intravenous glucose tolerance test and by a dual-energy
37 sulin secretion using the frequently sampled intravenous glucose tolerance test and insulin sensitivi
38 ects were examined with a frequently sampled intravenous glucose tolerance test and meal tolerance te
39 ulin sensitivity (determined by the modified intravenous glucose tolerance test and minimal model ana
40 were determined by the tolbutamide-modified intravenous glucose tolerance test and minimal modeling,
41 veness (S(G)), which were determined from an intravenous glucose tolerance test and minimal modeling.
42 ody insulin sensitivity index (S(I)) with an intravenous glucose tolerance test and minimal modeling.
44 second phase insulin release in response to intravenous glucose tolerance test and suppressed postpr
45 x ml(-1)), estimated by a frequently sampled intravenous glucose tolerance test and the minimal model
46 vity (SI), estimated by a frequently sampled intravenous glucose tolerance test and the minimal model
48 ixed meal and underwent a frequently sampled intravenous glucose tolerance test before and after 2 ye
49 s undergoing SG or RYGB were studied with an intravenous glucose tolerance test before surgery and at
53 point was change in SI by frequently sampled intravenous glucose tolerance test from entry to week 12
54 mal model analysis of the frequently sampled intravenous glucose tolerance test in 1,625 men and wome
55 ulin sensitivity (S(I)) was measured with an intravenous glucose tolerance test in obese HIV+ women r
56 tance was measured with a frequently sampled intravenous glucose tolerance test in the Insulin Resist
59 ulin action (Si), measured with the meal and intravenous glucose tolerance test models, was highly co
60 y and an insulin-modified frequently sampled intravenous glucose tolerance test on the second day.
63 l model analysis with the frequently sampled intravenous glucose tolerance test provides an effective
64 of EXN during mixed meal tolerance test and intravenous glucose tolerance test results in improved f
67 haracteristic loss of first-phase GSIS in an intravenous glucose tolerance test that is diagnostic of
68 = 389) had first-phase insulin release on an intravenous glucose tolerance test that was higher than
73 lucose levels, urine glucose levels, and the intravenous glucose tolerance test were used to monitor
74 n sensitivity (SI) by the frequently sampled intravenous glucose tolerance test with analysis by the
75 th a validated, 12-sample, insulin-enhanced, intravenous glucose tolerance test with minimal model an
76 the tolbutamide-modified, frequently sampled intravenous glucose tolerance test with minimal modeling
77 mes were changes in Si (measured by using an intravenous glucose tolerance test) and cardiovascular r
78 changes of portal insulin (as measured by an intravenous glucose tolerance test) versus slow changes
79 rinsulinemic clamp), insulin secretion (25-g intravenous glucose tolerance test), and endogenous gluc
80 ) in gSAT and aSAT, S(I) (frequently sampled intravenous glucose tolerance test), body composition (d
83 the curve and glucose disappearance rate on intravenous glucose tolerance test, all of which worsene
84 abolic testing by mixed meal tolerance test, intravenous glucose tolerance test, and arginine stimula
85 (S(I)) was measured by a frequently sampled intravenous glucose tolerance test, and CRP was measured
86 [AIR]), as derived from a frequently sampled intravenous glucose tolerance test, as well as common ca
89 index (SI) assessed by a frequently sampled intravenous glucose tolerance test, insulin secretion ra
90 ic clamp), acute insulin response (AIR, 25-g intravenous glucose tolerance test, n = 118 normal gluco
91 ive insulin) had higher DRs than first-phase intravenous glucose tolerance test-derived incremental i
119 tabolism as assessed by a frequently sampled intravenous glucose tolerance test.RESULTSChronic mirabe
120 phy, respectively; S(i) was assessed with an intravenous-glucose-tolerance test and minimal modeling.
121 were measured by using a frequently sampled intravenous-glucose-tolerance test and minimal modeling.
122 nemic-euglycemic glucose clamp technique and intravenous-glucose-tolerance test have indicated that i
123 tion were assessed with a frequently sampled intravenous-glucose-tolerance test, dual-energy X-ray ab
128 e examined, and at 5-8 d postpartum (PP), an intravenous glucose-tolerance-test (GTT) and AT biopsies
129 asured insulin sensitivity index (S(I)) from intravenous glucose tolerance testing among African-Amer
131 d first-phase insulin release in response to intravenous glucose tolerance testing, was observed afte
133 virtually identical to that obtained during intravenous glucose tolerance tests (71.6+/-6.1% of tota
135 lerance tests (OGTTs) and frequently sampled intravenous glucose tolerance tests (FSIGTs) were conduc
136 ch subject underwent four frequently sampled intravenous glucose tolerance tests (FSIGTT), one with t
138 and normalization of glucose disposal during intravenous glucose tolerance tests (IVGTT) remains crit
140 dlimb lymph insulin profile during simulated intravenous glucose tolerance tests (IVGTTs) in anesthet
143 Oral glucose tolerance tests (OGTTs) and intravenous glucose tolerance tests (IVGTTs) were perfor
145 l antibody-negative women underwent oral and intravenous glucose tolerance tests (OGTT; IVGTT), hyper
147 m a large sample of individuals studied with intravenous glucose tolerance tests demonstrated that in
148 administration protocols, we performed three intravenous glucose tolerance tests in each of seven obe
150 cutaneous and intraperitoneal sensors during intravenous glucose tolerance tests in eight swine.
152 ed beta-cell function, we performed oral and intravenous glucose tolerance tests on mutation carriers
156 emic clamps in adults and frequently sampled intravenous glucose tolerance tests using Bergman minima
157 r sensor lag times (<4.2 min) in response to intravenous glucose tolerance tests versus burst NO-rele
158 ity (SI) as determined by frequently sampled intravenous glucose tolerance tests was measured over a
164 abetic, non-Amish subjects (n = 48), in whom intravenous glucose tolerance tests were performed, and
165 n levels returned to normal, and K values of intravenous glucose tolerance tests were significantly h
166 insulin secretion rates during both oral and intravenous glucose tolerance tests were used to generat
167 BCF) were determined from frequently sampled intravenous glucose tolerance tests, and total body fat
170 irst-phase insulin secretion, as measured by intravenous glucose tolerance tests, using up to 5,567 i
175 glycemic clamps), and insulin secretion [via intravenous-glucose-tolerance tests (IVGTTs)].Fifty-four