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1 died during the first nymphal stages (hybrid inviability).
2 re of hybrid inferiority (both sterility and inviability).
3 ecessary nor sufficient for hybrid embryonic inviability.
4 one of the proximate causes of hybrid larval inviability.
5 xpression levels but relieved mec1 and rad53 inviability.
6 tem for investigating the genetics of hybrid inviability.
7 sult in reduced male fertility, sterility or inviability.
8 ation of glycoproteins in the Golgi leads to inviability.
9 es show Haldane's rule for sterility but not inviability.
10  obeys Haldane's rule for both sterility and inviability.
11 igh-copy-number suppressors of the mot1 spt3 inviability.
12 severe delay of anther dehiscence and pollen inviability.
13 es whose overexpression also suppresses mec1 inviability.
14 bination with an spt3 null mutation, lead to inviability.
15 ssion of RED1 exacerbates the mek1-974 spore inviability.
16  hybrid male and hybrid female sterility and inviability.
17 s that are likely responsible for their high inviability.
18 viving to the adult stage, confirming hybrid inviability.
19 ER-ER fusion pathways caused almost complete inviability.
20 tive stress, floral bud abortion, and pollen inviability.
21 ely to morphological abnormalities and adult inviability.
22  thus overestimating the frequency of hybrid inviability.
23 y(A) polymerase I (PAP I), which exacerbates inviability.
24 n in the absence of histone synthesis causes inviability.
25 ially faster than female sterility or hybrid inviability.
26 tiations via elevated dnaA expression causes inviability.
27 sking two questions about its role in hybrid inviability.
28 with genetic studies of hybrid sterility and inviability.
29        (2) Does Hmr(mel) cause hybrid larval inviability?
30 lanogaster (Hmr(mel)) cause hybrid embryonic inviability?
31 al races, predominantly in the form of spore inviability (23.7% F(1) spore viability).
32 rotection, chromosome end-to-end fusions and inviability(3-9).
33  initial pds1-1 allele was identified by its inviability after transient exposure to microtubule inhi
34  alternative screen for mutants that exhibit inviability after transient exposure to nocodazole and p
35 lex in ncr1Delta strains conferred anaerobic inviability and accumulation of multiple sterol intermed
36 results add to previous studies of F2 hybrid inviability and behavioral sterility, and indicate that
37 specially in contrast with the low degree of inviability and female sterility, is far greater than ex
38 ng isolating mechanisms (for example, hybrid inviability and hybrid sterility).
39 ssion of HOP1 partially suppresses the spore inviability and recombination defects of mek1-974; in co
40  premating behavioral isolation, postzygotic inviability and Wolbachia-induced cytoplasmic incompatib
41 ng both 3' to 5' and 5' to 3' decay leads to inviability, and conditional double mutants show extreme
42  of induced dnaC expression and a model that inviability arises by the binding of DnaC to DnaB to inh
43                                          The inviability associated with deletion of the yeast genes
44  Taken together, these data suggest that the inviability associated with inactivation of RNase E is n
45 oth 30 and 44 degrees C, suggesting that the inviability associated with the mrsC505 allele is not re
46 e biology, viable null mutations that confer inviability at 36 degrees have been identified for multi
47 tations affecting microtubules in yeast, and inviability at extreme temperatures (i.e., >/=37 degrees
48 re-accumulation of 4-carboxysterols and cell inviability at high temperature.
49 npA49) and or RNA (rnpB709) subunits lead to inviability at nonpermissive temperatures.
50 se IV mutants in Escherichia coli results in inviability at the permissive temperature.
51 gested by work on other species, that hybrid inviability between closely related species might be cau
52 uctive barriers, such as hybrid sterility or inviability between populations.
53  mitochondrial genome in evolution of hybrid inviability between S. nutans lineages.
54 bias in the evolution of hybrid sterility or inviability but do find a very strong sex bias in the ev
55                                         This inviability can be suppressed in fission yeast by overex
56                            The rapid loss of inviability displayed by a yku80delta est2delta strain w
57 arabiensis suffer from hybrid sterility, and inviability effects are sometimes present as well.
58                         Generally, recessive inviability effects were found on the X chromosome of ga
59 ithin populations, may be aided by immigrant inviability, especially when a long period separates lar
60       dnaC suppressors of priA overcome this inviability, especially when RecF, RecO or RecR is inact
61  is complete when the gambiae and arabiensis inviability factors are hemi- or homozygous.
62 more rapidly than either female sterility or inviability factors.
63  or the histone deacetylase RPD3 gene, shows inviability following induction of an HO lesion that is
64                                  Hybrid seed inviability forms a potent reproductive barrier between
65                     The relative paucity of "inviability genes" supports the idea, suggested by work
66                      A single case of hybrid inviability has been tracked down to a 3-Kb element that
67 ividual genes that cause hybrid sterility or inviability have been identified in a few cases, my anal
68                         Hybrid sterility and inviability have been unimportant in the early stages of
69 rgely reproductively isolated by hybrid seed inviability (HSI).
70 ed that different events are responsible for inviability in checkpoint-deficient cells harboring muta
71  one of the underlying causes of male hybrid inviability in Drosophila, wherein the fate of hybrid ma
72 compensation could contribute to male hybrid inviability in Drosophila.
73 ere we identify a gene that causes epistatic inviability in hybrids between two fruitfly species, Dro
74 l cycle-regulation gene as the cause of male inviability in hybrids resulting from a cross between Dr
75 netic incompatibilities causing sterility or inviability in interspecies hybrids grows faster than li
76 es to study the genetics of intrinsic hybrid inviability in male F2 hybrids of Nasonia giraulti and N
77 7, which encodes a flap endonuclease, causes inviability in mre11 strains.
78 ads to loss of sister chromatid cohesion and inviability in nocodazole.
79 ic and epigenetic mechanisms underlie hybrid inviability in Peromyscus and hence have a role in the e
80                            Much of the spore inviability in sgs1 results from PSSC, and these events
81  loss of anionic phospholipids leads to cell inviability in the absence of mitochondrial DNA.
82 studies of the developmental basis of hybrid inviability in the Drosophila melanogaster complex.
83 asm account for most of the intrinsic hybrid inviability in this cross.
84 a high-expression suppressor of Sir2-induced inviability in yeast cells.
85 ytological analyses to show that hybrid male inviability is associated with, and probably caused by,
86                                              Inviability is complete when the gambiae and arabiensis
87 ains of Saccharomyces cerevisiae, most spore inviability is due to precocious separation of sister ch
88 double-mutant phenotype, suggesting that the inviability is not solely the result of precocious expre
89               Our studies reveal that hybrid inviability is partially due to a maternally expressed X
90    We present a simple model in which hybrid inviability is partly or entirely caused by a mitotic de
91       In Saccharomyces cerevisiae, dna2Delta inviability is reversed by deletion of the conserved hel
92                     One explanation for this inviability is that one or more sumoylated proteins accu
93 of fertilization success or hybrid sterility/inviability, is very common.
94 isolating mechanisms of hybrid sterility and inviability, little is known about the genetic basis of
95 isolating mechanisms of hybrid sterility and inviability, little is known about the genetic basis of
96 several genes that cause hybrid sterility or inviability-many of which have evolved rapidly under pos
97      Theory predicts that early-onset hybrid inviability may stem from conflict between parents for r
98                                          The inviability of a dam lexA (Ind(-)) host was abrogated by
99 ne-rich (G-F) region, was able to rescue the inviability of a Deltasis1 strain.
100                       Synthetic lethality is inviability of a double-mutant combination of two fully
101 ther cdc33 or ceg1 and no suppression of the inviability of a pab1 null mutation by xrn1Delta.
102 selection for mutations overcoming the spore inviability of a rad52 spo13 haploid strain.
103 tion, was found to specifically suppress the inviability of a ydr1 deletion, demonstrating that the e
104           Overexpression of New1 rescues the inviability of a yeast strain lacking the otherwise stri
105 olism is the primary underlying cause of the inviability of apn1Delta apn2Delta rad1Delta and apn1Del
106 tion but instead are capable of rescuing the inviability of bem2 mutants at 35 degrees C.
107 s Cdc28) phosphorylation also suppresses the inviability of Bir1 deletion.
108                            Here we show that inviability of double-mutant offspring biased inferences
109 hat deletion of KIP2 could also suppress the inviability of dyn1Delta kar3Delta cells suggests that k
110 2-1 (D530N, V615E) alleles that suppress the inviability of erg26-1ts at high temperature, and cause
111 have established a requirement for Xist with inviability of female embryos that inherit an Xist delet
112 a close relative (human KChIP2), rescues the inviability of frq1 cells.
113 ted Frq1(G2A,C15A) double mutant rescued the inviability of frq1Delta cells.
114                                          The inviability of gcn5 yng2 double mutants suggests overlap
115  taxa, including the intrinsic sterility and inviability of hybrids.
116                                          The inviability of KLP64D mutations can be rescued by expres
117 egulator causes mitotic arrest and, thereby, inviability of male hybrid larvae.
118 omes, aneuploidy of some daughter cells, and inviability of most other daughter cells.
119 nent nuclear accumulation and did not rescue inviability of pik1Delta cells.
120 ded from the nucleus and also did not rescue inviability of pik1Delta cells.
121 ts are viable, suggesting, together with the inviability of pta/ackA recBC mutants, that chromosomal
122              To determine the reason for the inviability of Saccharomyces cerevisiae with skeletal mu
123                            The sterility and inviability of species hybrids can be explained by betwe
124                            The sterility and inviability of species hybrids is thought to evolve by t
125  have long recognized that the sterility and inviability of species hybrids must involve incompatible
126 dane's rule - the preferential sterility and inviability of species hybrids of the heterogametic (XY)
127 2Delta and rqh1Delta mutants and rescues the inviability of srs2Delta rqh1Delta cells.
128                                 The supposed inviability of such intermediates gave pleuronectiforms
129 of Clns, because a sic1 deletion rescued the inviability of the cln1 cln2 cln3 triple mutant.
130                       In contrast, the spore inviability of the mum4-1 mutant is rescued by the spo13
131       Overexpression of TRF5 complements the inviability of top1 trf4 double mutants.
132                              The conditional inviability of two mutants, msh2-L560S pol3-01 and msh2-
133  stability through several diverse pathways, inviability of vertebrate cells that lack Mre11 has hind
134 tic stages of pollen development, leading to inviability of xri(-) pollen and abnormal segregation of
135       A screen for bypass suppressors of the inviability of xrn1 ski2 double mutants identified domin
136 progressive, with floral abscission and seed inviability ongoing during backcrossing cycles.
137 iability suggest that bacteria contribute to inviability only because intrinsic hybrid dysfunction in
138 gent cid alleles are not sufficient to cause inviability or female sterility in hybrid crosses.
139 t postzygotic reproductive isolation (hybrid inviability or sterility) evolves by the accumulation of
140 cdc13-ts alleles argues that the accelerated inviability previously observed at 36 degrees in cdc13-1
141 ies or subspecies and on hybrid sterility or inviability rather than on ecologically based barriers t
142 c distances between taxa that exhibit female inviability/sterility show no differences between "large
143 gence leading to gametic isolation or hybrid inviability/sterility, and/or ecological divergence over
144                   Antioxidants suppress this inviability, suggesting that reactive oxygen species (RO
145 h central to evolution, the causes of hybrid inviability that drive reproductive isolation are poorly
146 tes that in instances of hybrid sterility or inviability, the heterogametic sex tends to be more seve
147 t leads hybrid individuals to infertility or inviability, the world awoken to the dawn of new species
148  condition that can lead to sterility and/or inviability through improper gene regulation in Drosophi
149                  The severity of hybrid seed inviability varies among these crosses, which we inferre
150                              Complete hybrid inviability was also associated with some regions of the
151  otherwise nonessential genes cause cellular inviability when knocked out simultaneously.
152 bstantially suppresses the sid2-1 sic1 Delta inviability, while stabilizing Clb5 protein exacerbates
153  in which presumed nicks in DNA do not cause inviability with recA, suggesting that nicks stimulate h

 
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