ライフサイエンスコーパス: ionotropic glutamate receptor

1 e the N-methyl-D-aspartate (NMDA) subtype of ionotropic glutamate receptor.

2 face maintenance of the N-methyl-D-aspartate ionotropic glutamate receptor.

3 ic transmission, modulating the postsynaptic ionotropic glutamate receptors.

4 ystemic elimination and the kidney expresses ionotropic glutamate receptors.

5 key neuronal signaling molecules, including ionotropic glutamate receptors.

6 provide target validation for this family of ionotropic glutamate receptors.

7 trafficking, or inactivation of postsynaptic ionotropic glutamate receptors.

8 is resistant to blockade of metabotropic and ionotropic glutamate receptors.

9 ease of ascorbate is delicately regulated by ionotropic glutamate receptors.

10 is mediated by glutamate acting on AMPA-type ionotropic glutamate receptors.

11 nnels, suggesting a link in the evolution of ionotropic glutamate receptors.

12 azole propionate and kainate subtypes of the ionotropic glutamate receptors.

13 occurs primarily through AMPA- and NMDA-type ionotropic glutamate receptors.

14 es were largely insensitive to block of fast ionotropic glutamate receptors.

15 tion by signaling through platelet-expressed ionotropic glutamate receptors.

16 c processes are essential to the function of ionotropic glutamate receptors.

17 coupled to an altered expression profile of ionotropic glutamate receptors.

18 ve deficits by increasing internalization of ionotropic glutamate receptors.

19 necessary for plasma membrane expression of ionotropic glutamate receptors.

20 mmalian central nervous system by activating ionotropic glutamate receptors.

21 onstrating functional activation of specific ionotropic glutamate receptors.

22 ned potential interactions between EAAC1 and ionotropic glutamate receptors.

23 nd GRIK2, respectively, both of which encode ionotropic glutamate receptors.

24 ast synaptic potentials mediated by GABAA or ionotropic glutamate receptors.

25 which has not previously been described for ionotropic glutamate receptors.

26 Blockers of ionotropic glutamate receptors, 2,3-dihydroxy-6-nitro-7-

27 ype of positive allosteric modulators of the ionotropic glutamate receptor A2 (GluA2) are promising l

28 Ionotropic glutamate receptors activate cell signaling i

29 Subsequent activation of ionotropic glutamate receptors activates MAP kinases, th

30 Here, we show that treatment of neurons with ionotropic glutamate receptor agonists causes CPEB4 to a

31 sion, but it remains uncertain whether these ionotropic glutamate receptors also have essential subun

32 studied whether a prolonged inactivation of ionotropic glutamate receptors also induces cholinergic

33 Using heterologous expression of excitatory ionotropic glutamate receptor AMPA subunits in Xenopus o

34 methyl-4-isoxazolepropionic acid (AMPA)-type ionotropic glutamate receptors (AMPA receptors) predeter

35 Among ionotropic glutamate receptors (AMPA, kainate, NMDA), AM

36 Among the three classes of ionotropic glutamate receptors (AMPA, NMDA, and kainate

37 Numerous in vitro findings indicate that the ionotropic glutamate receptor, AMPAR, can rapidly traffi

38 rain is carried by AMPA and NMDA subtypes of ionotropic glutamate receptors (AMPARs and NMDARs).

39 orylation and dephosphorylation of AMPA-type ionotropic glutamate receptors (AMPARs) by kinases and p

40 AMPA-subtype ionotropic glutamate receptors (AMPARs) mediate fast exc

41 KEY POINTS: The AMPA-type ionotropic glutamate receptors (AMPARs) mediate the majo

42 alian brain is largely mediated by AMPA-type ionotropic glutamate receptors (AMPARs), which are activ

43 ry synaptic signaling mediated by AMPA-class ionotropic glutamate receptors (AMPARs).

44 eurotransmission is mediated by AMPA-subtype ionotropic glutamate receptors (AMPARs).

45 GRIA3 encodes GluA3, a subunit of AMPA-type ionotropic glutamate receptors (AMPARs).

46 onents of neural signalling (specifically an ionotropic glutamate receptor and two regucalcins), and

47 at may permit calcium influx such as certain ionotropic glutamate receptors and canonical transient r

48 Adenosine release required the activation of ionotropic glutamate receptors and could be evoked by lo

49 populated by glial progenitors that express ionotropic glutamate receptors and extend numerous proce

50 ynaptic neurexins and postsynaptic AMPA-type ionotropic glutamate receptors and induced the formation

51 lts extend the influence of PIP2 to the NMDA ionotropic glutamate receptors and introduce a novel mec

52 nomycin D and tetrodotoxin, by inhibitors of ionotropic glutamate receptors and L-type voltage-gated

53 l function and pharmacological properties of ionotropic glutamate receptors and likely are important

54 These results identified ionotropic glutamate receptors and NMDA-Rs, specifically

55 in the PSD fraction, whereas the amounts of ionotropic glutamate receptors and other prominent PSD p

56 the dimeric NTD of GluN1 is unique among the ionotropic glutamate receptors and predicts that the str

57 elevated glutamate may subsequently activate ionotropic glutamate receptors and result in the dephosp

58 ltage-gated Ca2+ channels but persisted when ionotropic glutamate receptors and sodium spikes were bl

59 or understanding gating across the family of ionotropic glutamate receptors and the role of AMPA rece

60 We focus particularly on three classes of ionotropic glutamate receptors and their transmembrane i

61 on of voltage-gated Na(+) (NaV) channels and ionotropic glutamate receptors, and formation of synapse

62 Theories attribute an important role to ionotropic glutamate receptors, and it has been suggeste

63 contain beta-adrenergic receptors as well as ionotropic glutamate receptors, and retromer knockdown r

64 s ([glutamate]e) and was prevented by either ionotropic glutamate receptor antagonism or removal of L

65 structure-activity study of the broad-acting ionotropic glutamate receptor antagonist 1a.

66 cell degeneration, or in combination with an ionotropic glutamate receptor antagonist 6-cyano-7-nitro

67 no-4-phosphonobutyric acid (APB), and/or the ionotropic glutamate receptor antagonist cis-2,3 piperid

68 y-NH2 , d(CH2 )5 [D-Tyr(2) ,Thr(4) ]OVT, the ionotropic glutamate receptor antagonist kynurenate or t

69 Injection of kynurenic acid (Kyn, ionotropic glutamate receptor antagonist) into RVLM or t

70 agonist) or kynurenic acid (a non-selective ionotropic glutamate receptor antagonist) microinjected

71 ficantly inhibited by bath application of an ionotropic glutamate receptor antagonist, indicating tha

72 ent of complex EPSCs was markedly reduced by ionotropic glutamate receptor antagonists (2-amino-5-pho

73 rolactin were abolished by coinfusion of the ionotropic glutamate receptor antagonists 6-cyano-7-nitr

74 Ionotropic glutamate receptor antagonists are valuable t

75 Since ionotropic glutamate receptor antagonists can partially

76 increased proMMP-2 release, whereas non-NMDA ionotropic glutamate receptor antagonists reduced IL-6 p

77 rficial layers of the SC, in the presence of ionotropic glutamate receptor antagonists, evoked IPSCs

78 ves, MC calcium transients were inhibited by ionotropic glutamate receptor antagonists, indicating th

79 ions on hypocretin neurons were abolished by ionotropic glutamate receptor antagonists.

80 ate and not 5-HT because it was abolished by ionotropic glutamate receptor antagonists.

81 Importantly, synchrony was resistant to ionotropic glutamate receptors antagonists but was stron

82 We conclude that the two types of ionotropic glutamate receptor are built in different way

83 chitecture and atomic structure of an intact ionotropic glutamate receptor are unknown.

84 Ionotropic glutamate receptors are a family of tetrameri

85 Thus, VP ionotropic glutamate receptors are critical mediators of

86 Ionotropic glutamate receptors are functionally diverse

87 Ionotropic glutamate receptors are important for estradi

88 Ionotropic glutamate receptors are ligand-gated ion chan

89 Ionotropic glutamate receptors are ligand-gated ion chan

90 Ionotropic glutamate receptors are ligand-gated ion chan

91 The ionotropic glutamate receptors are localized in the pre-

92 Ionotropic glutamate receptors are postsynaptic tetramer

93 Positive allosteric modulators of ionotropic glutamate receptors are potential compounds f

94 The ionotropic glutamate receptors are primary mediators of

95 ng, and synaptic targeting of KARs and other ionotropic glutamate receptors are processes controlled,

96 Ionotropic glutamate receptors are thought to play an es

97 Ionotropic glutamate receptors are widely distributed in

98 MPA) receptors, one subtype in the family of ionotropic glutamate receptors, are the main receptors r

99 Kainate receptors (KARs), a family of ionotropic glutamate receptors, are widely expressed in

100 s NL1 isoform-specific cis-interactions with ionotropic glutamate receptors as a key mechanism for co

101 was not due to a differential expression of ionotropic glutamate receptors, as evidenced by similar

102 eceptors (KARs), one of three subfamilies of ionotropic glutamate receptors, as well as the putative

103 -93, and SAP97, are scaffolding proteins for ionotropic glutamate receptors at excitatory synapses.

104 generation was blocked by OT antagonist and ionotropic glutamate receptor blockers or tetanus toxin.

105 einstated by 4-aminopyridine, and blocked by ionotropic glutamate receptor blockers.

106 ological diseases, is not mediated merely by ionotropic glutamate receptors but also by heteromeric T

107 al D1R/PKA/MEK1/2 pathway and independent of ionotropic glutamate receptors but blocked by antagonist

108 afficking is regulated by Ca2+ entry through ionotropic glutamate receptors, but the underlying mecha

109 Here, we design a light-controlled ionotropic glutamate receptor by genetically encoding a

110 We conclude that activation of ionotropic glutamate receptors by excitatory temporoammo

111 binding core (S1S2) of the GluR2 subtype of ionotropic glutamate receptors can be produced as a solu

112 The many divergent features of the different ionotropic glutamate receptor classes and different subu

113 Ionotropic glutamate receptors comprise two conformation

114 The N-methyl-d-aspartate (NMDA) subtype of ionotropic glutamate receptors comprises both NR1 and NR

115 rons after I(h) block required activation of ionotropic glutamate receptors; consistent with this was

116 The delta family of ionotropic glutamate receptors consists of glutamate del

117 The overstimulated ionotropic glutamate receptors exert their neurotoxic ef

118 We used RT-PCR to profile ionotropic glutamate receptor expression in cultured SCs

119 calcium elevations and Western blots reveal ionotropic glutamate receptor expression prior to immuno

120 s and MEF2C in the signal cascade leading to ionotropic glutamate receptor expression.

121 The members of the ionotropic glutamate receptor family, namely, a-amino-3-

122 utamate delta (GluD) receptors belong to the ionotropic glutamate receptor family, yet they don't bin

123 n is necessary and sufficient to up-regulate ionotropic glutamate receptors from a pool of different

124 We focus here on the control of ionotropic glutamate receptor function by GPCR signaling

125 NMDA ionotropic glutamate receptors gate the cytoplasmic infl

126 ome abnormality disrupting the kainate class ionotropic glutamate receptor gene, GRIK4/KA1, in an ind

127 NMDA receptor subunit NR2B/Grin2B and other ionotropic glutamate receptor genes.

128 Ionotropic glutamate receptors (GluRs) are ligand-gated

129 Ionotropic glutamate receptors (GluRs) are ligand-gated

130 We investigated whether directly stimulating ionotropic glutamate receptors (GluRs) within the nucleu

131 lence of somatic mutations within one of the ionotropic glutamate receptors, GRIN2A, in malignant mel

132 The kainate (KA) subtype of ionotropic glutamate receptor has been shown to potently

133 e structure of the agonist binding domain of ionotropic glutamate receptors has led to an improved un

134 hypothesis of addiction and the role of the ionotropic glutamate receptors have been emphasized.

135 Structural and functional studies of ionotropic glutamate receptors have offered detailed ins

136 ory synaptic transmission is mediated by the ionotropic glutamate receptor homolog cation channel, de

137 nnels, cyclic nucleotide-gated channels, and ionotropic glutamate receptor homologs.

138 ptic depression is modulated by postsynaptic ionotropic glutamate receptor (iGluR) activity.

139 Exposure to ionotropic glutamate receptor (iGluR) agonists, kainic a

140 ficial cerebrospinal fluid, or a cocktail of ionotropic glutamate receptor (iGluR) antagonists each b

141 near 0 mV or by recording in the presence of ionotropic glutamate receptor (iGluR) antagonists to iso

142 dria in astrocytic processes were blocked by ionotropic glutamate receptor (iGluR) antagonists, tetro

143 bitory currents from RGCs in the presence of ionotropic glutamate receptor (iGluR) antagonists.

144 bitory currents from RGCs in the presence of ionotropic glutamate receptor (iGluR) antagonists.

145 Ionotropic glutamate receptor (iGluR) channels control s

146 anisms regulating the extent of postsynaptic ionotropic glutamate receptor (iGluR) clustering have be

147 reconfiguration of the subunits constituting ionotropic glutamate receptor (iGluR) complexes.

148 Ionotropic glutamate receptor (iGluR) desensitization ca

149 Ionotropic glutamate receptor (iGluR) family members are

150 as seen a revolution in our understanding of ionotropic glutamate receptor (iGluR) structure, startin

151 The GluD1 and GluD2 receptors form the GluD ionotropic glutamate receptor (iGluR) subfamily.

152 postmortem study examined mRNA expression of ionotropic glutamate receptor (iGluR) subunits and PSD95

153 Loss of specific ionotropic glutamate receptor (iGluR) subunits during in

154 The amino terminal domain (ATD) of ionotropic glutamate receptor (iGluR) subunits resides a

155 Within ionotropic glutamate receptor (iGluR) subunits, this pro

156 lated GluD1 are classified as members of the ionotropic glutamate receptor (iGluR) superfamily on the

157 determine the efficacy of PTL gating in the ionotropic glutamate receptor iGluR6 using a family of p

158 Ionotropic glutamate receptors (iGluRs) are glutamate-ga

159 Ionotropic glutamate receptors (iGluRs) are key molecule

160 Ionotropic glutamate receptors (iGluRs) are ligand-gated

161 Ionotropic glutamate receptors (iGluRs) are ligand-gated

162 Ionotropic glutamate receptors (iGluRs) are responsible

163 Ionotropic glutamate receptors (iGluRs) are tetrameric c

164 Ionotropic glutamate receptors (iGluRs) are ubiquitous i

165 ation followed the arrival and clustering of ionotropic glutamate receptors (iGluRs) at NMJ synapses.

166 e protein synthesis critical for trafficking ionotropic glutamate receptors (iGluRs) at synapses.

167 generated an extensive sequence alignment of ionotropic glutamate receptors (iGluRs) from diverse ani

168 hores have revealed the presence of numerous ionotropic glutamate receptors (iGluRs) in Mnemiopsis le

169 Ionotropic glutamate receptors (iGluRs) mediate excitato

170 Ionotropic glutamate receptors (iGluRs) mediate fast exc

171 Ionotropic glutamate receptors (iGluRs) mediate most exc

172 Ionotropic glutamate receptors (iGluRs) mediate neuronal

173 Ionotropic glutamate receptors (iGluRs) mediate neurotra

174 Ionotropic glutamate receptors (iGluRs) mediate the majo

175 Ionotropic glutamate receptors (iGluRs) mediate the majo

176 Ionotropic glutamate receptors (iGluRs) play important r

177 Presynaptic ionotropic glutamate receptors (iGluRs) play important r

178 is comb jelly encodes homologs of vertebrate ionotropic glutamate receptors (iGluRs) that are distant

179 NMDA receptors (NMDARs) are a class of ionotropic glutamate receptors (iGluRs) that are essenti

180 ing in the brain is mediated by postsynaptic ionotropic glutamate receptors (iGluRs) that are gated o

181 Kainate receptors (KARs) are a subfamily of ionotropic glutamate receptors (iGluRs) that mediate exc

182 A) receptors (AMPARs) are a major subtype of ionotropic glutamate receptors (iGluRs) that mediate rap

183 ate (NMDA) receptors belong to the family of ionotropic glutamate receptors (iGluRs) that mediate the

184 Kainate receptors (KARs) are ionotropic glutamate receptors (iGluRs) that modulate sy

185 Ionotropic glutamate receptors (iGluRs) transduce the ch

186 Ionotropic glutamate receptors (iGluRs) underlie rapid,

187 lso attenuated by the blockade of the spinal ionotropic glutamate receptors (iGLURs) which was accomp

188 Ionotropic glutamate receptors (iGluRs), a family of lig

189 osstalk between Wnt receptors, activation of ionotropic glutamate receptors (iGluRs), and localized c

190 Ionotropic glutamate receptors (iGluRs), including the N

191 ory neurotransmission is mediated largely by ionotropic glutamate receptors (iGluRs), tetrameric, lig

192 americ ion channels that together with other ionotropic glutamate receptors (iGluRs), the NMDA and ka

193 Here, we studied ionotropic glutamate receptors (iGluRs), which are ion c

194 stem is mediated by glutamate acting through ionotropic glutamate receptors (iGluRs).

195 s excitatory synaptic transmission by gating ionotropic glutamate receptors (iGluRs).

196 oduces a robust learning that is mediated by ionotropic glutamate receptors (iGluRs).

197 citatory synaptic transmission by activating ionotropic glutamate receptors (iGluRs).

198 Glutamate-gated ion channels (ionotropic glutamate receptors, iGluRs) sense the extrac

199 ed by activation of either NMDA or AMPA-type ionotropic glutamate receptors in a calcium-dependent ma

200 selective genetic inactivation of NMDA-type, ionotropic glutamate receptors in DA neurons.

201 s been designed to enable optical control of ionotropic glutamate receptors in neurons via sensitized

202 Recently, several full-length structures of ionotropic glutamate receptors in putative desensitized

203 discovery of 20 genes encoding for putative ionotropic glutamate receptors in the Arabidopsis (Arabi

204 Blockade of ionotropic glutamate receptors in the DMH, or brain tran

205 s strongly correlated with calcium-permeable ionotropic glutamate receptors in the neurons of the sen

206 al agent that helped unravel the key role of ionotropic glutamate receptors, including the kainate re

207 s of anti-BDNF were abolished by blockade of ionotropic glutamate receptors, indicating a role for gl

208 e effects were blocked by the antagonists of ionotropic glutamate receptors, indicating the involveme

209 d Ca(2+) imaging, we show that activation of ionotropic glutamate receptors induces a selective inter

210 that a long-term decrease in the activity of ionotropic glutamate receptors induces cholinergic activ

211 nfusion of kynurenic acid (an antagonist for ionotropic glutamate receptors) into core but not shell

212 trated that a member of the newly discovered ionotropic glutamate receptor (IR) family, IR76b, functi

213 he responsible sensory receptor (the variant ionotropic glutamate receptor IR75b) and attraction-medi

214 neurons express either odorant receptors or ionotropic glutamate receptors (IRs).

215 The NMDA ionotropic glutamate receptor is ubiquitous in mammalian

216 vidence that a family of proteins related to ionotropic glutamate receptors is a previously unrecogni

217 Whereas overactivation of ionotropic glutamate receptors is neurotoxic, the role o

218 e N-methyl-d-aspartate (NMDA) subtype of the ionotropic glutamate receptors is the primary mediator o

219 Zn(2+) inhibition of the NMDA subtype of the ionotropic glutamate receptors is well characterized, th

220 Among the three classes of ionotropic glutamate receptors, kainate receptors (KARs)

221 iod of synaptic development, kainate-type of ionotropic glutamate receptors (KARs) are highly express

222 The mechanism by which agonist binding to an ionotropic glutamate receptor leads to channel opening i

223 y SNAG-mGluR2 and excitatory light-activated ionotropic glutamate receptor LiGluR yielded a distribut

224 -shifted PTL, L-MAG0460, for the light-gated ionotropic glutamate receptor LiGluR.

225 excitatory mammalian ion channel light-gated ionotropic glutamate receptor (LiGluR) in retinal gangli

226 We show that the engineered light-gated ionotropic glutamate receptor (LiGluR), when introduced

227 and bradycardic responses via activation of ionotropic glutamate receptors located on secondary mNTS

228 Therefore, our results suggest that ionotropic glutamate receptors may have been conserved t

229 The ionotropic glutamate receptors mediate excitatory neurot

230 N-methyl-d-aspartate (NMDA)-type ionotropic glutamate receptors mediate excitatory neurot

231 5-methyl-4-isoxazole propionic acid)-subtype ionotropic glutamate receptors mediate fast excitatory n

232 AMPA subtype ionotropic glutamate receptors mediate fast excitatory n

233 Ionotropic glutamate receptors mediate fast excitatory s

234 Ionotropic glutamate receptors mediate fast synaptic tra

235 Ionotropic glutamate receptors mediate most excitatory n

236 AMPA-type ionotropic glutamate receptors mediate the majority of f

237 Ionotropic glutamate receptors mediate the majority of v

238 Ionotropic glutamate receptors mediate the majority of v

239 possibility that plant homologs of neuronal ionotropic glutamate receptors mediate these neuron-like

240 NMDA receptors (NMDAR), a type of ionotropic glutamate receptor, mediate synaptic plastici

241 isoxazole-4-propionic acid (AMPA) subtype of ionotropic glutamate receptors mediates much of the fast

242 cumulation leading to overstimulation of the ionotropic glutamate receptors mediates neuronal injury

243 Presynaptic ionotropic glutamate receptors modulate transmission at

244 The ionotropic glutamate receptors (N-methyl-D-aspartate rec

245 and memory and a reduction in the amounts of ionotropic glutamate receptors (NMDA and AMPA receptors)

246 Ca(2+) entry through extrasynaptic NMDA-type ionotropic glutamate receptors (NMDARs).

247 the central network by glutamate binding to ionotropic glutamate receptors on second-order barorecep

248 n addition, the effect of antagonists to the ionotropic glutamate receptors on TAI was evaluated.

249 the activation of N-methyl-d-aspartate-type ionotropic glutamate receptors opposed increases in stri

250 ced by systemic injections of antagonists of ionotropic glutamate receptors or metabotropic glutamate

251 at central synapses depends on the number of ionotropic glutamate receptors, particularly the class g

252 Ionotropic glutamate receptors perform diverse functions

253 Some forms of ionotropic glutamate receptors permit passage of Ca++ io

254 ptors (IRs) are a large subfamily of variant ionotropic glutamate receptors present across Protostomi

255 Ionotropic glutamate receptors principally mediate fast

256 structural basis of allosteric inhibition in ionotropic glutamate receptors, providing key insights i

257 enhanced by DEX (10 microM), and blockade of ionotropic glutamate receptors reduced the DEX effect on

258 e suggests that surface trafficking of other ionotropic glutamate receptors requires ligand binding f

259 -4-isoxazole-propionate receptor (AMPAR) are ionotropic glutamate receptors responsible for excitator

260 ations in melanoma and the significance that ionotropic glutamate receptor signaling has in malignant

261 Electrophysiological testing at three ionotropic glutamate receptor subtypes reveals that two

262 onses, short-term dynamics and expression of ionotropic glutamate receptor subtypes.

263 The distribution of ionotropic glutamate receptor subunit 4 (iGluR4) was exa

264 tations disrupt a previously uncharacterized ionotropic glutamate receptor subunit, named here "GluRI

265 Other ionotropic glutamate receptor subunits are expressed nor

266 ellular amino-terminal domains (ATDs) of the ionotropic glutamate receptor subunits form a semiautono

267 dings and showed expression of mRNA encoding ionotropic glutamate receptor subunits NR2A, NR2D, GluR4

268 y advance our physiological understanding of ionotropic glutamate receptor subunits, but is generally

269 a(v)beta(3) integrin-Lyn kinase complex with ionotropic glutamate receptor subunits, GluR2 and GluR4,

270 he expression of non-NMDA (AMPA and kainate) ionotropic glutamate receptor subunits.

271 viously observed for the AMPA subtype of the ionotropic glutamate receptors, suggesting a similar mec

272 nit of N-methyl-d-aspartate receptors in the ionotropic glutamate receptor superfamily have been targ

273 eptors (AMPARs) constitute a subclass of the ionotropic glutamate receptor superfamily, which functio

274 ivo; and (3) constitutive desensitization of ionotropic glutamate receptors suppresses their ability

275 Here we report the discovery of an ionotropic glutamate receptor that combines the typical

276 xazoleproprionic acid receptor (AMPAR) is an ionotropic glutamate receptor that governs most of excit

277 N2A subunit of the NMDA receptor (NMDAR), an ionotropic glutamate receptor that has important roles i

278 We developed a K+-selective ionotropic glutamate receptor that reversibly inhibits n

279 Kainate receptors (KARs) are ionotropic glutamate receptors that also activate noncan

280 NMDA receptors (NMDARs) are ionotropic glutamate receptors that are crucial for neur

281 NMDA receptors (NMDARs) are a major class of ionotropic glutamate receptors that can undergo activity

282 sts of N-methyl D-aspartate (NMDA)-selective ionotropic glutamate receptors that contain the (E)-3-ph

283 NMDA receptors are ionotropic glutamate receptors that function as heterote

284 ptors (NMDARs) are a subtype of postsynaptic ionotropic glutamate receptors that function as molecula

285 N-methyl-D-aspartate-receptors (NMDARs) are ionotropic glutamate receptors that function in synaptic

286 ate (NMDA) receptors belong to the family of ionotropic glutamate receptors that mediate a majority o

287 NMDA receptors are ionotropic glutamate receptors that mediate a slow, Ca2+

288 NMDA receptors are ionotropic glutamate receptors that mediate excitatory s

289 These results highlight the diversity of ionotropic glutamate receptor trafficking rules at a sin

290 ction and its proposed role in AMPA and NMDA ionotropic glutamate receptor trafficking, we believe th

291 biophysical characteristics of postsynaptic ionotropic glutamate receptors via changes in subunit co

292 inate receptors (KARs) consist of a class of ionotropic glutamate receptors, which exert diverse pre-

293 ate (NMDA) receptors belong to the family of ionotropic glutamate receptors, which mediate most excit

294 Correspondingly, AMPA-subtype ionotropic glutamate receptors, which mediate the majori

295 osed to the hippocampus proper, also express ionotropic glutamate receptors, which might provide addi

296 Ionotropic glutamate receptors, which underlie a majorit

297 king the N-methyl-d-aspartate (NMDA) type of ionotropic glutamate receptor with D-AP5 attenuated this

298 Kainate receptor subunits, which form ionotropic glutamate receptors with diverse roles in the

299 During apnea, induced by blockade of ionotropic glutamate receptors within the same region, m

300 The role of ionotropic glutamate receptors within the ventral pallid