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1 s, was increased three-fold by high doses of iron dextran.
2 one to 15 weekly subcutaneous injections of iron dextran.
3 ability shift were revealed below 500 mug/mL iron dextran.
4 t mice that included daily administration of iron dextran.
5 iron carbonyl in the diet, or injected with iron dextran.
6 ongolian gerbil given repeated injections of iron dextran.
7 phrine administrations, both associated with iron dextran.
9 ly subcutaneous injections of either 1 mL of iron dextran (5 mg/mL) or 0.9% NaCl for a total of five
10 red within a 12-week period was highest with iron dextran (82 per 100,000 persons, 95% CI, 70.5- 93.1
11 irst exposure was 68 per 100,000 persons for iron dextran (95% CI, 57.8-78.7 per 100,000) and 24 per
14 omozygosity for an Hfe-null mutation but not iron-dextran administration was porphyrinogenic in anima
18 /-) zebrafish were rescued by injection with iron dextran and studied in comparison with injected and
23 erated an iron-overloaded mouse by injecting iron dextran at 2 doses into C57/BL6 mice for 2 months.
25 r complexes were introduced, e.g., InFed(R) (iron-dextran complex injection) in 1974 and Abraxane(R)
27 against KIM5 infections in mice administered iron dextran doses leading to light or severe pneumonia,
28 MDA was considerably increased even at low iron dextran doses, but without any increase in URO accu
31 f HIF-2alpha or parenteral administration of iron-dextran in HIF-2alpha knockout mothers ameliorated
32 ethanol for 6 to 7 months, administration of iron dextran increased hepatic URO accumulation and decr
33 ) deformability in vitro was assessed during iron dextran (INFeD) loading and/or ethanol co-administr
34 ls < 600 ng/mL were treated with intravenous iron dextran (INFeD; Schein Pharmaceutical Inc., Florham
39 nes the effects of a maintenance intravenous iron dextran (ivID) protocol that increased TSAT in ESRD
41 ed significant cell stiffening at 500 mug/mL iron dextran loading concentration (p < 0.05, Tukey test
42 dequacy of dialysis and received intravenous iron dextran more often than those in the low-hematocrit
43 increased systemic iron availability (e.g., iron dextran or phenylhydrazine) on the induction, kinet
44 erloaded mice, including models of acquired (iron-dextran or stored red blood cells) and primary (Hfe
47 iron; (2). oral iron 325 mg twice daily; (3) iron dextran repeated 100mg IV bolus; or (4) iron dextra
48 high-iron diet or by parenteral injection of iron dextran; rescue can also be achieved by providing a
50 n sucrose, 3.8% (n = 345, 95% CI: 3.4%-4.2%) iron dextran (test and full doses or test dose alone), 1
53 , comprising 22 309 iron sucrose doses, 9067 iron dextran total doses (1771 preceded by test dose, 56
55 lesions in subcutaneously (s.c.) inoculated iron dextran-treated mice in terms of numbers of recover
56 results demonstrate that s.c. inoculation of iron dextran-treated mice is a useful model for studying
63 n period were not significantly different in iron dextran-treated mice; however, the rate of death of
65 ly attenuated in a subcutaneously inoculated iron dextran-treated mouse model of V. vulnificus diseas
68 erapeutic agents against V. vulnificus in an iron-dextran-treated mouse model of V. vulnificus infect
70 ins demonstrated that the greatest effect of iron dextran-treatment was increased growth rate, while
71 sucrose, the adjusted OR of anaphylaxis for iron dextran was 3.6 (95% CI, 2.4-5.4); for iron glucona