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6 ructive pathologies of the two main forms of irritable bowel disease (IBD), ulcerative colitis (UC),
7 individuals, and those with colon cancer and irritable bowel disease (IBD), we demonstrated that CD4C
12 evels of comorbidity included migraine (33), irritable bowel syndrome (17), functional neurological d
14 ), 6.78% in Crohn's disease (4/59), 5.82% in irritable bowel syndrome (51/877), and 4.9% in the remai
15 subjects (aged 24-61 y, 6 men) with NCGS and irritable bowel syndrome (based on Rome III criteria), b
16 ota of patients with constipated-predominant irritable bowel syndrome (C-IBS) displays chronic dysbio
17 (FC), Gastrointestinal Symptoms Rating Scale-Irritable Bowel Syndrome (GSRS-IBS) and Hospital Anxiety
18 he Gastrointestinal Symptom Rating Scale for Irritable Bowel Syndrome (GSRS-IBS) into German and to e
21 ed by the intestinal mucosa of patients with irritable bowel syndrome (IBS) affect the function of en
23 tudies have shown an increased prevalence of irritable bowel syndrome (IBS) after acute gastroenterit
24 patients (63.8%) had a previous diagnosis of irritable bowel syndrome (IBS) and 23 (28.8%) had one of
25 e Rome IV Diagnostic Questionnaire, Rome III irritable bowel syndrome (IBS) and constipation question
26 es from patients of colorectal cancer (CRC), irritable bowel syndrome (IBS) and controls to be run th
28 symptoms, which occasionally mimic those of Irritable Bowel Syndrome (IBS) and Fibromyalgia Syndrome
29 hypnotherapy (HT) is effective in pediatric irritable bowel syndrome (IBS) and functional abdominal
30 tious gastroenteritis increases the risk for irritable bowel syndrome (IBS) and functional dyspepsia
31 ists are effective in treating patients with irritable bowel syndrome (IBS) and have anxiolytic effec
34 Very few studies report on the prevalence of irritable bowel syndrome (IBS) and its correlates in the
37 isease (GERD), functional dyspepsia (FD) and irritable bowel syndrome (IBS) are common functional gas
38 healthy children and pediatric patients with irritable bowel syndrome (IBS) are not well defined.
39 cture is used by patients as a treatment for irritable bowel syndrome (IBS) but the evidence on effec
42 pathophysiology, diagnosis, and treatment of irritable bowel syndrome (IBS) convened to audit the cur
44 suggestive of functional dyspepsia (FD) and irritable bowel syndrome (IBS) frequently overlap with t
48 odify pain end points in clinical trials for irritable bowel syndrome (IBS) highlights the knowledge
50 and its degree of overlap with dyspepsia and irritable bowel syndrome (IBS) in Nigeria, a typical Afr
51 care consumption for patients diagnosed with Irritable Bowel Syndrome (IBS) in primary and secondary
74 role of the microbiota in the development of irritable bowel syndrome (IBS) only recently has been co
76 HPA axis response to a visceral stressor in irritable bowel syndrome (IBS) patients and healthy cont
77 Syndrome (FMS) is a frequent comorbidity in Irritable Bowel Syndrome (IBS) patients with a higher fu
81 sorbed fermentable carbohydrates can provoke irritable bowel syndrome (IBS) symptoms by escaping abso
82 suspected food intolerances in patients with irritable bowel syndrome (IBS) using confocal laser endo
83 entable carbohydrates may induce symptoms of irritable bowel syndrome (IBS) via unclear mechanisms.
84 entable carbohydrates may induce symptoms of irritable bowel syndrome (IBS) via unclear mechanisms.
85 ders state that children suspected of having Irritable Bowel Syndrome (IBS) with Constipation (IBS-C)
86 ept study for the treatment of patients with irritable bowel syndrome (IBS) with constipation (IBS-C)
89 of PI sequelae among exposed was as follows: irritable bowel syndrome (IBS), 3.0; dyspepsia, 1.8; con
90 and male healthy subjects and patients with irritable bowel syndrome (IBS), a common chronic abdomin
91 d neural mechanisms are well-acknowledged in irritable bowel syndrome (IBS), a disorder of brain-gut-
92 ve been implicated in the pathophysiology of irritable bowel syndrome (IBS), a visceral pain syndrome
93 including inflammatory bowel disease (IBD), irritable bowel syndrome (IBS), and chronic constipation
95 s are abnormal in the ileum of patients with irritable bowel syndrome (IBS), and whether any abnormal
96 d genetic factors contribute to variation in irritable bowel syndrome (IBS), anxiety and depression.
98 ND & AIMS: Probiotics can reduce symptoms of irritable bowel syndrome (IBS), but little is known abou
99 bnormal gut-brain interactions are common in irritable bowel syndrome (IBS), but the associations bet
100 Peppermint oil is frequently used to treat irritable bowel syndrome (IBS), despite a lack of eviden
101 iated with three other functional disorders; irritable bowel syndrome (IBS), functional dyspepsia (FD
102 the area of functional GI syndromes such as irritable bowel syndrome (IBS), functional dyspepsia, an
103 ose was to evaluate the overlap frequency of irritable bowel syndrome (IBS), gastroesophageal reflux
104 on ultimate diagnosis: Crohn's disease (CD), Irritable bowel syndrome (IBS), NSAIDs enteritis (NSAIDs
105 een pathophysiologic factors and symptoms of irritable bowel syndrome (IBS), or whether these factors
107 patients with functional dyspepsia (FD) and irritable bowel syndrome (IBS), respectively, as defined
110 l gastrointestinal symptoms in patients with irritable bowel syndrome (IBS), yet there is limited evi
111 atory bowel diseases (IBD), the reporting of irritable bowel syndrome (IBS)-type symptoms by patients
138 in, constipation, and bloating; diagnoses of irritable bowel syndrome (IBS); and tegaserod prescripti
139 alence of celiac disease among patients with irritable bowel syndrome (IBS); few data are available w
140 fies the FBDs into five distinct categories: irritable bowel syndrome (IBS); functional constipation
141 shown promise in alleviating the symptoms of irritable bowel syndrome (IBS); however, controlled data
142 and polyols (FODMAPs) exacerbate symptoms of irritable bowel syndrome (IBS); however, their mechanism
144 in the treatment of constipation predominant irritable bowel syndrome (IBS-C), a highly prevalent dis
150 hat patients with symptoms of nonconstipated irritable bowel syndrome (NC-IBS) undergo testing for ce
151 04-1.1), diarrhea (OR, 53; 95% CI, 6.1-471), irritable bowel syndrome (OR, 4.8; 95% CI, 1.6-14), chol
157 ith increased rate ratios (RRs) for incident irritable bowel syndrome (RR, 6.1; 95% confidence interv
160 who are in remission and those who developed irritable bowel syndrome after enteric infection continu
162 ate the persistence, prevalence, and risk of irritable bowel syndrome and chronic fatigue 6 years aft
163 ing is associated with an increased risk for irritable bowel syndrome and chronic fatigue 6 years lat
166 ter, are at increased risk of postinfectious irritable bowel syndrome and inflammatory bowel disease
168 sit and gut microbial communities, including irritable bowel syndrome and inflammatory bowel disease.
169 long-term effects, including postinfectious irritable bowel syndrome and inflammatory bowel disease.
170 , appeared to be most genetically similar to irritable bowel syndrome and most environmentally simila
171 stion that SIBO may be a causative factor in irritable bowel syndrome and of its constituent symptoms
172 successfully treat constipation-predominant irritable bowel syndrome and recent studies show that ex
173 (EPI) confound interpretation of findings in irritable bowel syndrome and severe renal insufficiency.
176 al gastrointestinal disorders, most commonly irritable bowel syndrome but also other functional and o
177 nalities with inflammatory bowel disease and irritable bowel syndrome but were focused on associative
178 In the exposed group, the prevalence of irritable bowel syndrome decreased by 6.7% (RR, 0.85 [95
179 opsies were also taken from 16 patients with irritable bowel syndrome diarrhea who comprised the cont
180 antibiotics and concurrently, postinfectious irritable bowel syndrome has been associated with a long
183 treatment options for diarrhoea-predominant irritable bowel syndrome have had not very promising res
184 microbiota transplantation for patients with irritable bowel syndrome in a randomised, double-blind,
185 valuate the effectiveness of acupuncture for irritable bowel syndrome in primary care when provided a
186 ased understanding of the pathophysiology of irritable bowel syndrome in the past 10 years has led to
187 Underlying mechanisms that could lead to irritable bowel syndrome include genetic factors (most n
189 risk factor is acute enteric infection, but irritable bowel syndrome is also more common in people w
190 w we challenge the widely accepted view that irritable bowel syndrome is an unexplained brain-gut dis
196 n, Latino individuals, and participants with irritable bowel syndrome or Crohn's disease were more li
197 een the control group and Crohn's disease or irritable bowel syndrome patients in terms Blastocystis
199 the gut microbiota and the immune system", "irritable bowel syndrome related to gut microbiota", and
201 ficant difference after 4 weeks in change in irritable bowel syndrome severity scores, but significan
205 ion, 15 with functional bloating, and 3 with irritable bowel syndrome with alternating bowel habits)
209 C) includes functional constipation (FC) and irritable bowel syndrome with constipation (IBS-C).
210 ipation, chronic idiopathic constipation, or irritable bowel syndrome with constipation is often made
211 ith functional intestinal disorders (27 with irritable bowel syndrome with constipation, 15 with func
212 ed by >2 symptoms of chronic constipation or irritable bowel syndrome with constipation, and with >2
215 abdominal pain and diarrhea in patients with irritable bowel syndrome with diarrhea (IBS-D) without c
216 a large proportion of people diagnosed with irritable bowel syndrome with diarrhea, a common functio
218 Chronic idiopathic constipation (CC) and irritable bowel syndrome with predominant constipation (
221 chronic fatigue syndrome' and 'Dopamine and irritable bowel syndrome' was carried out until April 20
222 tic syndromes', 'Chronic fatigue syndrome', 'Irritable bowel syndrome', 'Fibromyalgia', 'Dopamine and
223 titial cystitis/painful bladder syndrome and irritable bowel syndrome) are associated with hyperexcit
224 rome, reactive arthritis, and postinfectious irritable bowel syndrome) contribute considerably to the
226 broid food poisoning, histamine intolerance, irritable bowel syndrome, and inflammatory bowel disease
227 These include inflammatory bowel diseases, irritable bowel syndrome, and metabolic (i.e. obesity, n
228 robiota, such as inflammatory bowel disease, irritable bowel syndrome, and metabolic syndrome, to nam
229 of work on MGBA focused on immunomodulation, irritable bowel syndrome, and neurodevelopmental disorde
230 owel diseases, celiac disease, food allergy, irritable bowel syndrome, and--more recently recognized-
231 ders, such as inflammatory bowel disease and irritable bowel syndrome, are associated with exaggerate
234 to human physiology and to diseases such as irritable bowel syndrome, autism, anxiety, depression, a
235 , management, and role in conditions such as irritable bowel syndrome, chronic fatigue, and autoimmun
236 d costs of ambulatory visits for symptomatic irritable bowel syndrome, chronic functional abdominal p
238 pain - is common in some disorders, such as irritable bowel syndrome, Crohn's disease and pancreatit
239 ur findings link duplications in TPSAB1 with irritable bowel syndrome, cutaneous complaints, connecti
240 expanded from 1946 to December 2014 for IBS, irritable bowel syndrome, diet, treatment, and therapy.
241 ted with chronic prostatitis/CPPS, including irritable bowel syndrome, fibromyalgia, and chronic fati
242 r directly co-morbid somatic disorders, e.g. irritable bowel syndrome, fibromyalgia, or migraine.
243 This issue provides a clinical overview of irritable bowel syndrome, focusing on diagnosis, treatme
244 d in the setting of differentiating IBD from irritable bowel syndrome, for grading inflammation, to d
245 nter placebo-controlled trial, children with irritable bowel syndrome, functional abdominal pain, or
246 disorders of gut-brain interaction, comprise irritable bowel syndrome, functional dyspepsia, abdomina
247 unctional gastrointestinal disorder, such as irritable bowel syndrome, functional dyspepsia, or funct
248 mechanisms may predispose the individual to irritable bowel syndrome, gastroesophageal reflux diseas
249 orders including inflammatory bowel disease, irritable bowel syndrome, infectious and antibiotic-asso
250 various gastrointestinal diseases, including irritable bowel syndrome, inflammatory bowel disease, an
251 , including Clostridium difficile infection, irritable bowel syndrome, inflammatory bowel diseases, i
252 hea and colitis, inflammatory bowel disease, irritable bowel syndrome, necrotizing enterocolitis, and
253 vergrowth is one of the causes suggested for irritable bowel syndrome, particularly for the diarrhoea
254 mens from patients with UC, Crohn's disease, irritable bowel syndrome, sporadic colorectal cancer, or
255 liaison psychiatric setting in patients with irritable bowel syndrome, where positive benefits have b
257 ctional bowel disorders (FBDs), particularly irritable bowel syndrome, with the objective of elucidat
258 est drug to be approved for the treatment of irritable bowel syndrome-diarrhoea is rifaximin, which w
285 ion (95% CI, 2.3-3.2) visits for symptomatic irritable bowel syndrome/chronic abdominal pain, 1.0 mil
286 ed to control (P<.001) and disease controls (irritable bowel syndrome; P<.001; rheumatoid arthritis;
287 searched by using medical subject headings ("irritable bowel syndrome;" "colonic diseases, functional
288 n who do not fit a specific disorder such as irritable bowel, functional dyspepsia, or abdominal migr
289 ior, and neural activity between 19 severely irritable children (operationalized using criteria for s
290 suggest that, relative to healthy children, irritable children have deficient reward learning and el
291 ention flexibility, particularly in severely irritable children, which may contribute to emotion regu
292 levels of positive approach-motivation), and irritable (extreme levels of negative emotionality, ange
293 A greater proportion of patients with the irritable (IR) nociceptor phenotype were responders to i
294 lly and persistently elevated, expansive, or irritable mood and abnormally and persistently increased
297 EMA3F), number of children (CADM2 and ESR1), irritable temperament (MSRA) and risk-taking propensity