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1 s highly expressed in the BCM (mainly in the islets of Langerhans).
2 s and in insulin-producing beta cells of the islets of Langerhans.
3 e secreted with insulin by beta-cells in the islets of Langerhans.
4  insulin by the beta cells of the pancreatic islets of Langerhans.
5 sulin-producing beta cells in the pancreatic islets of Langerhans.
6 rough the analysis of insulin secretion from islets of Langerhans.
7 lls, the largest cellular constituent of the islets of Langerhans.
8 n of the insulin-producing beta cells in the islets of Langerhans.
9 ulin-secreting beta-cell found in pancreatic islets of Langerhans.
10 , eIF2-GTP.Met-tRNAi, in both MIN6 cells and islets of Langerhans.
11 resence of cytotoxic amyloid deposits in the islets of Langerhans.
12  cells, endocrine progenitor cells and adult islets of Langerhans.
13  factor (TNF)-alpha in the beta-cells of the islets of Langerhans.
14 persed in multiple micro-organs known as the islets of Langerhans.
15 one marrow-derived cells populate pancreatic islets of Langerhans.
16 gates, which subsequently differentiate into islets of Langerhans.
17 that STAT3 is deleted from beta cells in the islets of Langerhans.
18 tion following localized inflammation in the islets of Langerhans.
19 derstanding the developmental biology of the islets of Langerhans.
20  secretion from beta-cells in the pancreatic islets of Langerhans.
21 ion process by beta -cells in the pancreatic islets of Langerhans.
22 sponse and insulin secretion dynamics of the islets of Langerhans.
23 uced and secreted by specialized structures, islets of Langerhans.
24 maintaining the identity and function of the islets of Langerhans.
25 ly expressed in pancreatic insulin-producing islets of Langerhans.
26 s contain stem cells that differentiate into islets of Langerhans.
27 lective destruction of the beta cells of the islets of Langerhans.
28 ping and mature beta-cells in the pancreatic islets of Langerhans.
29 uitment of autoaggressive lymphocytes to the islets of Langerhans.
30 ells, while weaker ones were detected in the islets of Langerhans.
31 ggregating in the extracellular space of the islets of Langerhans.
32 m apoptotic destruction of beta cells in the islets of Langerhans.
33 mulated insulin release from perifused human islets of Langerhans.
34 mes restricted to the endocrine cells of the islets of Langerhans.
35 nitrite production, and insulin secretion by islets of Langerhans.
36 e genesis of the inflammatory lesions of the islets of Langerhans.
37 sult of a T cell-mediated destruction of the islets of Langerhans.
38 n of insulin by beta cells in the pancreatic islets of Langerhans.
39  well as low Km hexokinase I in isolated rat islets of Langerhans.
40 sulin-producing beta-cells in the pancreatic islets of Langerhans.
41 on of the costimulatory molecule B7.1 in the islets of Langerhans.
42 possesses multiple peptide hormones from the islets of Langerhans.
43 play a critical role in morphogenesis of the islets of Langerhans.
44 e regulation of hormone secretion within the islets of Langerhans.
45 e regulation of hormone secretion within the islets of Langerhans.
46 w blood-borne factors dynamically access the islets of Langerhans.
47 hypophysis, and in glucose-stimulated murine islets of Langerhans.
48 sulin-producing beta cells in the pancreatic islets of Langerhans.
49 INS isoform expression in postmortem ChP and islets of Langerhans.
50  within inflammatory cytokine-stressed human islets of Langerhans.
51 ction of insulin-producing beta cells in the islets of Langerhans.
52 he volume-restricted microenvironment of the islets of Langerhans.
53 bundance of the transporter localized in the islets of Langerhans.
54 d cystically dilated secretory ducts without islets of Langerhans.
55 ne cell clusters of the type found in normal islets of Langerhans.
56  insulin-secreting cells and isolated rodent islets of Langerhans.
57 d by a subpopulation of nonbeta cells in the islets of Langerhans.
58 ng endocrine cell activity in the pancreatic islets of Langerhans.
59 ion of the pancreas and complete loss of the islets of Langerhans.
60 widely expressed in adult tissues, including islets of Langerhans.
61 umber of insulin-secreting beta-cells in the islets of Langerhans.
62 eleted in the thymus but is activated in the islets of Langerhans.
63  epithelial lining of the ducts and form the islets of Langerhans.
64 in oscillations of intracellular [Ca(2+)] in islets of Langerhans.
65 struction of insulin-producing beta cells in islets of Langerhans.
66  by leukocyte infiltration of the pancreatic islets of Langerhans.
67 lucose waves that were needed to entrain the islets of Langerhans.
68 ere applied in this work to stimulate single islets of Langerhans.
69 sions, 76%), or with stromal fibroblasts and islets of Langerhans (20 of 82 lesions, 24%).
70 ulates protein synthesis in freshly isolated islets of Langerhans, across a range of glucose concentr
71                        SIRT4 is expressed in islets of Langerhans and colocalizes with insulin-expres
72                               Metabolites in islets of Langerhans and Escherichia coli strain DH5-alp
73                               Macrophages in islets of Langerhans and in the interacinar stroma are d
74 a(2+)](i)) in isolated mouse, rat, and human islets of Langerhans and in the MIN6 insulin-secreting m
75 truction of the beta cells of the pancreatic islets of Langerhans and is recapitulated in the nonobes
76 1R) is mainly expressed on beta-cells in the islets of Langerhans and is therefore an attractive targ
77 and ultrastructural changes were observed in islets of Langerhans and livers of mutant animals, as we
78 PC1/3 are expressed in the beta cells of the islets of Langerhans and participate in the processing o
79 tion, the virtual absence of CPE activity in islets of Langerhans and pituitary was associated with a
80                      Western blotting of rat islets of Langerhans and primary beta cells showed 33- a
81  Herein, we focus on the resident APC of the islets of Langerhans and their role in autoimmune diabet
82 d islet-specific T cells from reentering the islets of Langerhans and thereby blocked the autodestruc
83 ed to measure glucagon secretion from single islets of Langerhans and to differentiate cross-reactive
84 s from the rat pituitary, the rat pancreatic islets of Langerhans, and from the Aplysia californica n
85 lood glucose levels, preserves beta-cells in islets of Langerhans, and improves insulin action.
86 rown on nontransparent nanoneedle arrays, of islets of Langerhans, and of hippocampal neurons under u
87  phospholipase C- (PLC-) is expressed in the islets of Langerhans, and that the knockout (KO) of PLC-
88 The availability of paracrine factors in the islets of Langerhans, and the constitution of the beta c
89                                              Islets of Langerhans are a therapeutic target for diabet
90 e human body mass, pancreatic B-cells of the islets of Langerhans are a vulnerable link in metabolism
91                               The pancreatic islets of Langerhans are central to fine-tuning metaboli
92                                          The islets of Langerhans are critical endocrine micro-organs
93                                          The islets of Langerhans are endocrine organs characteristic
94        Endocrine cells within the pancreatic islets of Langerhans are heterogeneous in terms of trans
95                               The pancreatic islets of Langerhans are highly vascularized micro-organ
96                                  Because the islets of Langerhans are insulated from blood vessels by
97                 Pancreatic beta-cells in the islets of Langerhans are key to maintaining glucose home
98                               The pancreatic islets of Langerhans are multicellular micro-organs inte
99                 Thus, the macrophages of the islets of Langerhans are poised to mount an immune respo
100                                              Islets of Langerhans are the regulators of in vivo blood
101                        The beta-cells of the islets of Langerhans are the sole producers of insulin i
102             The beta-cells in the pancreatic islets of Langerhans are the targets of autoreactive T-c
103 protein deposits (amyloid) in the pancreatic islets of Langerhans are thought to be involved in death
104 ecreting beta-cells, found in the pancreatic islets of Langerhans, are destroyed by infiltrating T ce
105 es in microscale tissue samples using single islets of Langerhans as a model system.
106 activates transcription in beta-cells of the islets of Langerhans as well as in acinar cells.
107  10 mum pixel size allows us to identify the islets of Langerhans associated with lipid isomer upregu
108                                    Also, the islets of Langerhans attracted polymorphonuclear cells,
109 ity extends beyond the pancreatic lymph node-islets of Langerhans axis and indicates that circulating
110 localization of diabetogenic T cells only to islets of Langerhans bearing the specific antigen.
111                                   Within the islets of Langerhans, beta cells orchestrate synchronize
112 en, Peyer's patches, lymph nodes, pancreatic islets of Langerhans, bone marrow, and salivary glands o
113 APCs in close contact with beta cells in the islets of Langerhans bore vesicles with the antigenic in
114 r the determination of insulin secreted from islets of Langerhans by a capillary electrophoresis comp
115 and release of interleukin 1 (IL-1) in human islets of Langerhans by resident macrophages results in
116     Insulin secretion from beta cells in the islets of Langerhans can be stimulated by a number of me
117 s is organized into clusters of cells called islets of Langerhans comprising four well-defined cell t
118                                   Pancreatic islets of Langerhans consist of endocrine cells, primari
119                                          The islets of Langerhans constitute the endocrine part of th
120                                              Islets of Langerhans contain antigen-presenting cells th
121                             Human pancreatic islets of Langerhans contain five distinct endocrine cel
122 nsulin from the beta-cells of the pancreatic islets of Langerhans controls energy homeostasis in vert
123  secretion from beta cells of the pancreatic islets of Langerhans controls metabolic homeostasis and
124               In vitro studies of pancreatic islets of Langerhans demonstrated that GLP-1 interacts w
125 o the endocrine cells forming the pancreatic islets of Langerhans depends on a cascade of gene activa
126 es (T1D) autoreactive T-cells infiltrate the islets of Langerhans, depleting insulin-secreting beta-c
127                                   Pancreatic islets of Langerhans display complex intracellular calci
128 s caused by dysfunction to beta-cells in the islets of Langerhans, disrupting insulin secretion and g
129 y the alpha-cells of the endocrine pancreas (islets of Langerhans) during fasting and is essential fo
130 tion and hypertrophy are key determinants of islets of Langerhans "dysfunctional remodeling" and hype
131                      The gut, the pancreatic islets of Langerhans, elements in the portal vasculature
132                                       In the islets of Langerhans, especially in the alpha-cells, int
133                                          The islets of Langerhans exist as multicellular networks tha
134 pancreatic buds and mature beta-cells in the islets of Langerhans express Hlxb9.
135 mulation, nephropathy, atrophy of pancreatic islets of Langerhans, fatty streaks in the aorta, and hy
136 sed to monitor insulin secretion from single islets of Langerhans for 6-39 h.
137 are the major barriers to transplantation of islets of Langerhans for the cure of type 1 diabetes in
138 ate immune cell infiltration into pancreatic islets of Langerhans for the study of disease progressio
139  (MALDI MS) peptide and protein profiling of Islets of Langerhans, for future type 2 diabetes (T2D) s
140                Insulin secretion by isolated islets of Langerhans from 19 human donors (9 women and 1
141 pression of CD44 isoforms in highly purified islets of Langerhans from 4- and 10-week-old NOD mice.
142 is upregulated by insulin and upregulated in islets of Langerhans from mice exposed to a high-fat die
143                                              Islets of Langerhans from normal mice contained dendriti
144 ression of insulin pathway genes in isolated islets of Langerhans from these patients.
145 together with intraportal transplantation of islets of Langerhans, GVHR might damage the implanted is
146 te of success in clinical transplantation of islets of Langerhans has dramatically improved with pers
147 c innervation of the endocrine pancreas, the islets of Langerhans, has been shown to provide choliner
148 lls in adult pancreatic ducts or in isolated islets of Langerhans have been induced to grow in cultur
149  not seem to adversely influence function of islets of Langerhans implanted intrahepatically in this
150 tion by glucose-stimulated beta-cells of the islets of Langerhans in a dose- and time-dependent manne
151 secretory dynamics of multiple peptides from islets of Langerhans in a highly automated fashion is ex
152 anded human Treg to prevent the rejection of islets of Langerhans in a humanized mouse model and exam
153 tense radioactive spots colocalized with the islets of Langerhans in clorgyline-treated animals.
154                 Insulin is secreted from the islets of Langerhans in coordinated pulses.
155                 Insulin is released from the islets of Langerhans in discrete pulses that are linked
156 d insulin secretion (GSIS) from live, murine islets of Langerhans in microfluidic devices by the down
157 n of the insulin-producing beta cells in the islets of Langerhans in pancreas.
158               To investigate the role of the islets of Langerhans in pancreatic carcinogenesis, fresh
159 ol of diabetes, suggesting that the inflamed islets of Langerhans in prediabetic NOD mice are under p
160  levels in the extracellular space of single islets of Langerhans in real-time.
161 ailable concerning the lymph drainage of the islets of Langerhans in the human pancreas.
162 eptide ligands for vascular receptors in the islets of Langerhans in the murine pancreas.
163 troy insulin-producing beta-cells within the islets of Langerhans in the pancreas(1).
164 he somatostatin-producing delta cells in the islets of Langerhans in the pancreas.
165 d in the emergence of hepatocytes within the islets of Langerhans in the pancreas.
166 oendocrine cells in the intestine and in the islets of Langerhans in the pancreas.
167 mbohedral of Zn-insulin hexamers form in the islets of Langerhans in the pancreases of many mammals.
168 ction of insulin-producing beta cells in the islets of Langerhans in type 1 diabetes.
169                            We found that the islets of Langerhans in young nonobese diabetic (NOD) mi
170 f the alginate-based capsules that sequester Islets of Langerhans include fabrication and implantatio
171 ty among insulin-secreting beta-cells within islets of Langerhans, including their insulin secretory
172 ulin-producing B cells within the pancreatic islets of Langerhans (insulitis).
173                We engrafted human pancreatic islets of Langerhans into the renal subcapsular space of
174 riggers a cascade of stressful events in the islets of Langerhans involving activation of apoptosis a
175 cal activity of insulin-secreting pancreatic islets of Langerhans is characterized by bursts of actio
176 ory framework for clinical use of allogeneic islets of Langerhans is described.
177 mulate insulin secretion from the pancreatic islets of Langerhans is enhanced by the intestinal hormo
178              The spatial architecture of the islets of Langerhans is hypothesized to facilitate synch
179 ulates insulin secretion from the pancreatic islets of Langerhans is incompletely understood.
180 ification of insulin release from pancreatic islets of Langerhans is of interest for diabetes researc
181 esis in the endocrine pancreas, of which the islets of Langerhans is the major constituent, has been
182              The spatial architecture of the islets of Langerhans is vitally important for their corr
183 icted manner in the mature beta cells in the islets of Langerhans, is essential both for organ format
184 1D), immune cells infiltrate and destroy the islets of Langerhans - islands of endocrine tissue dispe
185 on secretory responses within the pancreatic islets of Langerhans (islets).
186 coupling mechanisms in beta-cells and intact islets of Langerhans isolated from three patients with a
187              Intrahepatic transplantation of islets of Langerhans (ITx) is a treatment option for ind
188 er into NOD.scid mice induced destruction of islets of Langerhans leading to diabetes.
189           A primary insult to the pancreatic islets of Langerhans, leading to the activation of innat
190 ne mediated destruction of beta cells of the islets of Langerhans leads to insulin-dependent diabetes
191                               The pancreatic islets of Langerhans maintain glucose homeostasis throug
192                               The pancreatic islets of Langerhans maintain glucose homeostasis.
193                             Encapsulation of islets of Langerhans may represent a way to transplant i
194                                          The islets of Langerhans, micro-organs for maintaining gluco
195                                          The islets of Langerhans normally contain resident antigen p
196 l profiles of macrophages that reside in the islets of Langerhans of 3-wk-old non-obese diabetic (NOD
197 rogenitor cells isolated from the pancreatic islets of Langerhans of adult human donor pancreata.
198 constituent of amyloid deposits found in the islets of Langerhans of patients with type II diabetes.
199  have identified a CD4high population in the islets of Langerhans of prediabetic nonobese diabetic (N
200 specifically expressed at high levels in the islets of Langerhans of the endocrine pancreas.
201 and FAM3B protein were both localized to the islets of Langerhans of the endocrine pancreas.
202 nd smoothened are expressed in the endocrine islets of Langerhans of the fully developed rat pancreas
203 d the antigen presenting cells (APCs) in the islets of Langerhans of the non-obese diabetic (NOD) mou
204 rated from the lymphocytic infiltrate in the islets of Langerhans of young (7-wk-old).
205  secretion from pancreatic beta-cells within islets of Langerhans plays a critical role in maintainin
206                                   Pancreatic islets of Langerhans produce bursts of electrical activi
207 es similar, but not identical, to pancreatic islets of Langerhans, produced insulin at levels far gre
208 le the endocrine compartment, organized into islets of Langerhans, produces hormones that regulate bl
209 ly and functionally normal at birth, but the islets of Langerhans progressively degenerate, resulting
210         Moreover, in MIN6 cells and isolated islets of Langerhans, rapamycin, an inhibitor of the mam
211                                   Pancreatic islets of Langerhans regulate blood glucose homeostasis
212                               The pancreatic islets of Langerhans regulate glucose homeostasis.
213  with metabolic parameters and its effect on islets of Langerhans remodeling and relative endocrine-c
214                           Transplantation of islets of Langerhans represents a viable therapeutic app
215                                          The islets of Langerhans reside within the endocrine pancrea
216 ocalization of diabetogenic CD4 T cells into islets of Langerhans resulting in diabetes were examined
217 ocytes to the endothelial venules inside the islets of Langerhans seems to initiate the infiltration
218 ded that the intercellular communications in islets of Langerhans should contribute to the effective
219 tro upon external glucose application to the islets of Langerhans, such as usual bursting oscillation
220 line MIN6 (mouse insulinoma cell line 6) and islets of Langerhans that agents which stimulate increas
221 on of the resident macrophages of pancreatic islets of Langerhans that lasted for several weeks.
222 er a few seconds or a few minutes, in intact islets of Langerhans they are intermediate (10-60 s).
223 cium dynamics and islet mass of transplanted islets of Langerhans throughout diet-induced progression
224                    What coordinates the many islets of Langerhans throughout the pancreas to produce
225 e expressing CXCL10 in the beta cells of the islets of Langerhans to evaluate how bystander inflammat
226 hown that prolonged exposure of isolated rat islets of Langerhans to excessive fatty acid levels impa
227 e hindered the successful transplantation of islets of Langerhans to human patients in efforts to cur
228         Here, we use intact mouse pancreatic islets of Langerhans to investigate signaling properties
229  Multiple peptide hormones are secreted from islets of Langerhans to maintain blood glucose homeostas
230 d clinical sections of an IND for allogeneic islets of Langerhans to treat type 1 diabetes mellitus a
231 (IND) applications for the use of allogeneic islets of Langerhans to treat type 1 diabetes mellitus.
232                                   Allogeneic islets of Langerhans transplantation is hampered in its
233    Transplantation of an excessive number of islets of Langerhans (two to four pancreata per recipien
234                We now demonstrate that human islets of Langerhans undergo apoptosis upon exposure to
235 mmatory mediators that are secreted by human islets of Langerhans upon CVB infection and may shed lig
236 o this end, we studied the response of human islets of Langerhans upon mock or CVB3 infection.
237 in and islet amyloid polypeptide (IAPP) from islets of Langerhans using a microfluidic system with tw
238  has shown that homing of these cells to the islets of Langerhans was affected by the lack of IFN-gam
239 e immunoassay, the insulin content of single islets of Langerhans was determined by flow injection an
240                Allogeneic transplantation of islets of Langerhans was facilitated by the cotransplant
241 ther accumulation of pathogenic Th1 cells to islets of Langerhans, we conclude that CD40L-CD40 costim
242 d beta-cells purified from rodent pancreatic islets of Langerhans, we studied the expression and role
243                                              Islets of Langerhans were isolated from genetically iden
244     The T cells were directly recruited into islets of Langerhans, where they established contact wit
245     Glucose homeostasis is controlled by the islets of Langerhans which are equipped with alpha-cells
246 ed proteins from small tissue depots such as islets of Langerhans, which are required for the proper
247 human insulin promoter in cadaver pancreatic islets of Langerhans, which resulted in insulin promoter
248      The remnant Hlxb9-/- pancreas has small islets of Langerhans with reduced numbers of insulin-pro
249         One problem is the reinfiltration of islets of Langerhans with regenerated, autoaggressive ly
250 ction of insulin-producing beta cells of the islets of Langerhans within the pancreas.

 
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