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1                                              Isocapnic acidosis (5 % CO(2), 7 mM HCO(3)(-), pH(o) 6.8
2 hypercapnic acidosis also had no response to isocapnic acidosis (n = 12).
3                                              Isocapnic acidosis (pH(o) 7.16; [NaHCO(3)](o) 15 mM) ind
4 ones tested (n = 15) were also stimulated by isocapnic acidosis, and all CO2-inhibited neurones teste
5 d neurones tested (n = 19) were inhibited by isocapnic acidosis.
6                Neurones were also exposed to isocapnic acidosis.
7 quencies than during NREM sleep (p < 0.001); isocapnic and hypercapnic conditions did not differ (p =
8  machine rates during wakefulness and during isocapnic and hypercapnic NREM sleep.
9 rved during wakefulness and sleep, and under isocapnic as well as hypocapnic conditions.
10 ied out by three different levels of hypoxic isocapnic blood (PO2 approximately 58, 40 and 22 mmHg) o
11 higher following NaHCO(3)(-) infusion during isocapnic breathing providing initial evidence for a dir
12 12%; P = 0.047) following NaHCO(3)(-) during isocapnic breathing providing support for a direct influ
13       Single-unit chemoreceptor responses to isocapnic changes in oxygenation within the arterial oxy
14         The larger the VT applied during the isocapnic CMV (120-200% of eupnea) and the longer the du
15                                              Isocapnic cold air hyperventilation (ISH) is believed to
16 hty volunteers individually underwent an 8-h isocapnic exposure to hypoxia (end-tidal P(O2)=55 Torr)
17 measured while breathing room air and during isocapnic hyperoxia (100% O2 breathing) and isoxic hyper
18 le pump alone), during unloaded cycling with isocapnic hyperpnea (muscle and ventilatory pump), durin
19 d that this resetting would not occur during isocapnic hyperpnoea at the same breathing rate and dept
20                                        While isocapnic hyperpnoea did not alter heart rate, arterial
21 e modified Oxford technique during normoxia, isocapnic hyperpnoea, and isocapnic hypoxia (85 % arteri
22 atory tract by having eight subjects perform isocapnic hyperventilation for 1, 2, 4, and 8 min at a c
23                                              Isocapnic hyperventilation is a promising therapy for CO
24 aluations including bronchoprovocations with isocapnic hyperventilation of frigid air, methacholine a
25 easured to determine the airway responses to isocapnic hyperventilation of humidified air at hot (49
26                    We studied the effects of isocapnic hyperventilation on five adult mechanically ve
27                                              Isocapnic hyperventilation utilizing mechanical ventilat
28 0 normal and 13 asthmatic subjects performed isocapnic hyperventilation with frigid air while the fra
29                                              Isocapnic hyperventilation, in our animal model, did not
30   At 130 +/- 4 days, a 1 h episode of acute, isocapnic hypoxaemia (9 % O(2) in N(2), to reduce caroti
31 station) we investigated the effect of acute isocapnic hypoxaemia (arterial Po2, 12.5 +/- 0.6 mmHg) o
32      Seven animals were given repeated acute isocapnic hypoxaemia (Pa,O2 reduced to ca. 13 mmHg) for
33   At 130 +/- 2 days, a 1 h episode of acute, isocapnic hypoxaemia (to reduce carotid P(O(2)) to 12 +/
34  the mechanism producing a fall in RO during isocapnic hypoxaemia in the neonate.
35 ) and [ATP](V) were measured at rest, during isocapnic hypoxia (80% SpO2 ), and during graded rhythmi
36 e during normoxia, isocapnic hyperpnoea, and isocapnic hypoxia (85 % arterial O2 saturation).
37 ity frequently induced by acute intermittent isocapnic hypoxia (AIH, three 5 min isocapnic hypoxic ep
38 hrenic nerve amplitude (integral Phr) during isocapnic hypoxia (arterial partial pressures of O2, 60,
39 rm blood flow responses (plethysmography) to isocapnic hypoxia (arterial saturation approximately 85%
40 esented with two or three, 5 min episodes of isocapnic hypoxia (FIO2 approximately 0.11), separated b
41 de of the ventilatory sensitivities to acute isocapnic hypoxia (G(pO2)) and hyperoxic hypercapnia, th
42 luntary contraction in normoxia (NormEx) and isocapnic hypoxia (HypEx; O2 saturation approximately 85
43                  The respiratory response to isocapnic hypoxia (inspired O2 fraction (FI,O1), 0.1-0.1
44 uctance (FVC) responses to steady-state (SS) isocapnic hypoxia (O(2) saturation ~85%).
45 re, during and after three episodes of 5 min isocapnic hypoxia (P(a,O2) = 30-45 mmHg), separated by 5
46                         Phrenic responses to isocapnic hypoxia (P(a,O2) = 60, 50 and 40 +/- 2 mmHg) w
47 ition (Pa(O2), 160 +/- 12 mm Hg) and in mild isocapnic hypoxia (Pa(O2), 69 +/- 7.2 mm Hg), with and w
48 and for 60 min after three 5 min episodes of isocapnic hypoxia (Pa,O2 35-45 mmHg) separated by 5 min
49  successive days: normoxia followed by acute isocapnic hypoxia (Pa,O2 to ca 12 mmHg) with infusion of
50 station) we investigated the effect of acute isocapnic hypoxia (Pa,O2, 12 +/- 0.6 mmHg) on the fetal
51 d eight healthy human participants to 8 h of isocapnic hypoxia and 8 h of air as a control.
52                                              Isocapnic hypoxia decreased the autocorrelation coeffici
53 ted phrenic responses to strictly controlled isocapnic hypoxia in urethane-anaesthetized, vagotomized
54                           Compared with air, isocapnic hypoxia increased the gross variability of min
55 activation of peripheral chemoreceptors with isocapnic hypoxia resets arterial baroreflex control of
56 tion of peripheral chemoreceptors with acute isocapnic hypoxia resets arterial baroreflex control of
57 activation of peripheral chemoreceptors with isocapnic hypoxia resets baroreflex control of both hear
58 ncrease in total variational activity during isocapnic hypoxia was found to result from increases in
59                  The respiratory response to isocapnic hypoxia was measured again post-lesioning.
60                                              Isocapnic hypoxia was repeated 10 min later.
61  and was identical to the first, except that isocapnic hypoxia was replaced with normoxia.
62 essive hyperoxic hypercapnia and progressive isocapnic hypoxia were associated with recruitment of ph
63 nt activity, integrated phrenic responses to isocapnic hypoxia were investigated in urethane-anaesthe
64 tery flow velocity (MCFV) during progressive isocapnic hypoxia, progressive hyperoxic hypercapnia, an
65 frequency-90 (SEF(90)) in response to 40 min isocapnic hypoxia.
66 e during euoxia and during a short period of isocapnic hypoxia.
67  group were exposed to two 6-hour periods of isocapnic hypoxia.
68 and 60 min following three 5-min episodes of isocapnic hypoxia.
69  the same breathing rate and depth as during isocapnic hypoxia.
70 ram during and after three 5 min episodes of isocapnic hypoxia.
71 jects, we described ventilatory responses to isocapnic-hypoxia, hyperoxic-hypercapnia, and exercise;
72  ventilation (VI) increased during the first isocapnic hypoxic episode and reached progressively high
73 e of phrenic nerve activity following brief, isocapnic hypoxic episodes in rats is diminished by prio
74 rmittent isocapnic hypoxia (AIH, three 5 min isocapnic hypoxic episodes).
75 od was drawn for hormone measurement and the isocapnic hypoxic ventilatory response was measured.
76  ventilatory responsiveness being normal and isocapnic-hypoxic ventilatory responsiveness being low r
77 nd-tidal CO2 was monitored and maintained at isocapnic levels; arterial blood gases were determined.
78 nspired gas mixtures were alternated between isocapnic normoxia and hypoxia (arterial partial pressur
79  contributed to the augmented VI during both isocapnic normoxia and hypoxia.
80 se to mild-to-moderate exercise in humans is isocapnic, or 'error-free'.
81 capnic level that existed prior to exercise (isocapnic post-exercise protocol, IPE).
82 icant impact on average HVR values, but that isocapnic protocols produced higher HVRs than poikilocap
83                                    Two 5 min isocapnic PTL tasks targeting a transdiaphragmatic press
84                                    Two 5 min isocapnic PTL tasks targeting a transdiaphragmatic press
85 n V I, VT, and TE despite wakefulness and an isocapnic state, suggesting that neural responses may ha
86 P ET , CO 2) was clamped at baseline levels (isocapnic trial) or uncontrolled.
87                       In study 1, during the isocapnic trial, P ET , CO 2 was successfully clamped at
88                              Compared to the isocapnic trial, the decline in mean MCAv was 15 +/- 4 c
89 unter-regulatory hormones, a 54% increase in isocapnic ventilation, and a 108% increase in the hypoxi
90 induce hypoxia, although HVR is greater with isocapnic versus poikilocapnic methods.