ライフサイエンスコーパス: jasmonate

1 ic acid and jasmonic acid (applied as methyl jasmonate).

2 y exogenous applications of the phytohormone jasmonate.

3 (1)O2-induced PCD, likely acting upstream of jasmonate.

4 as not modified by the application of methyl jasmonate.

5 ic biosynthetic pathways by the phytohormone jasmonate.

6 ) which was specifically activated by methyl jasmonate.

7 he gene can be induced in leaves with methyl jasmonate.

8 ns aphid resistance when treated with methyl jasmonate.

9 te plays a crucial role in PCD downstream of jasmonate.

10 investigate radial cell-to-cell transport of jasmonates.

11 scular cells, revealing a radial movement of jasmonates.

12 under normal conditions, addition of methyl jasmonate, a biotic stress hormone, induced expression i

13 Interestingly, methyl jasmonate, a repellent derived from the nonvolatile jasm

14 anceolata shoot cultures treated with methyl jasmonate, a well-known elicitor of plant specialized me

15 responsible and essential for stress-induced jasmonate accumulation in roots.

16 hat surprisingly only partly overlapped with jasmonate accumulation polymorphisms and deviated from c

17 ed the extent of the transport of endogenous jasmonates across the plant vegetative growth phase.

18 The findings indicate that jasmonates acting synergistically with ethylene are the

19 es involved in auxin synthesis/transport and jasmonate activity were differentially expressed, indica

20 nse to wounding followed by ethylene, methyl jasmonate and ABA treatment.

21 ids and lipophilic hormone precursors of the jasmonate and auxin biosynthetic pathways.

22 F-box proteins COI1 and TIR1, receptors for jasmonate and auxin, respectively.

23 induction of inositol pyrophosphate InsP8 by jasmonate and demonstrate that steady state and jasmonat

24 and Eui1-OX mutants combined with nutrient, jasmonate and gene expression analyses were used to test

25 ex and suggest that coincidence detection of jasmonate and InsP8 by COI1-JAZ is a critical component

26 ession was observed with the hormones methyl jasmonate and naphthalene acetic acid and diterpenes.

27 regulators of cell death through modulating jasmonate and salicylate levels.

28 h phenomena rely on a marked accumulation of jasmonate and salicylate.

29 elevated expression of genes associated with jasmonate and stress responses.

30 The pathway is coordinately induced by jasmonate and the key enzyme, adenosine 5'-phosphosulfat

31 at foliar treatments carried out with methyl jasmonate and yeast extract achieved the best results, i

32 e results of this study indicate that methyl jasmonate and yeast extract applications could be a simp

33 ncreasing CO(2) suppresses the production of jasmonates and ethylene and increases the production of

34 olution and collection of both major (methyl jasmonates) and minor (epi-methyl jasmonates) stereoisom

35 mones (auxins, cytokinins, abscisic acid and jasmonates), and in the nutrient composition of the leav

36 response to UV-B, dehydration, NaCl, methyl jasmonate, and abscisic acid treatments indicating its p

37 NT1 (BZR1)-that are key regulators in light, jasmonate, and brassinosteroid signaling pathways, respe

38 al defense hormone pathways (salicylic acid, jasmonate, and jasmonate/ethylene pathways) were up-regu

39 ry hub, integrating ethylene, abscisic acid, jasmonate, and redox signaling in the plant response to

40 and other plant hormones, including auxins, jasmonates, and gibberellins.

41 ion increased accumulation of abscisic acid, jasmonates, and salicylic acid in wild type; in irAGO4 p

42 s and exogenous methyl salicylate and methyl jasmonate applications showed that plant defense against

43 th-defense tradeoffs mediated by the hormone jasmonate are uncoupled in an Arabidopsis mutant (jazQ p

44 Jasmonates are important phytohormones regulating reprod

45 Endogenous jasmonates are important regulators of plant defenses.

46 Jasmonates are key regulators of the balance between def

47 Exogenous jasmonates are known to trigger ISR by activating the pl

48 Jasmonates are oxygenated lipids (oxylipins) that contro

49 Jasmonates are oxylipin signals that play important role

50 Jasmonates are vital plant hormones that not only act in

51 essors, and SA/JA cross talk did not involve JASMONATE ASSOCIATED MYC2-LIKEs, which are negative regu

52 GT1b impairs responses to the plant hormones jasmonate, auxin and gibberellic acid, but not brassinol

53 for the classical defense hormones, with the jasmonate-based signaling being more critical than other

54 Plastid jasmonate biosynthesis enzymes were recruited to the k1

55 Also jasmonate biosynthesis is induced, pointing to the need

56 Jasmonate biosynthesis was strongly induced in ch1 mutan

57 that mutant cgi-58 plants display changes in jasmonate biosynthesis, auxin signaling, and lipid metab

58 on of genes related to mechanical stress and jasmonate biosynthesis/signaling during wood formation i

59 s including organelle-specific regulation of jasmonate biosynthesis; simultaneous induction of synthe

60 Jasmonate biosynthetic genes were highly represented amo

61 Methyl jasmonate, chitosan, and a commercial yeast extract were

62 f active jasmonate, JAZ proteins function as jasmonate co-receptors by forming a hormone-dependent co

63 ibberellins, cytokinins, salicylic acid, and jasmonate compared with diploid individuals.

64 ecies other than Arabidopsis thaliana, and a jasmonate-containing galactolipid.

65 r and redefine the dynamic metabolic grid of jasmonate conversion in the wound response.

66 pyrophosphates to the F-box protein COI1-JAZ jasmonate coreceptor complex and suggest that coincidenc

67 All of the data demonstrate that the jasmonate defense signal pathway is a major defense sign

68 /AN1 were found to almost all be involved in jasmonate defense signaling pathways.

69 plants and by the increased tolerance of the jasmonate-deficient mutant delayed-dehiscence2.

70 Grafting experiments with a jasmonate-deficient mutant demonstrated that roots produ

71 We demonstrate that jasmonate-deficient Nicotiana attenuata plants suffer mo

72 but was not significantly affected by methyl jasmonate, dehydration or heat shock stress.

73 different types of herbivores in nature, and jasmonate-dependent defenses are important for plants to

74 ase CYP705A1 and is transiently induced in a jasmonate-dependent manner by infection with the root-ro

75 Stems are defended by jasmonate-dependent nicotine, and the native cottontail

76 and neither the salicylic acid-dependent nor jasmonate-dependent pathways were induced.

77 1, the levels of metabolites, ABA, auxin and jasmonate derivatives did not change significantly in de

78 Treatment with herbivore or jasmonate elicitors induces emission of (E)-alpha-bergam

79 Thus, endogenous jasmonates enable plants to resist different types of he

80 Several phytohormones, including methyl jasmonate, ethylene, and abscisic acid, regulated RIP2 p

81 tial components in the signaling pathways of jasmonate, ethylene, and salicylate (classic defense hor

82 ween extracellular ATP signaling and that of jasmonate, ethylene, and salicylate.

83 four major sectors of the signaling network, jasmonate, ethylene, PAD4, and salicylate, are disabled,

84 containing four major signaling sectors, the jasmonate, ethylene, phytoalexin-deficient 4, and salicy

85 one pathways (salicylic acid, jasmonate, and jasmonate/ethylene pathways) were up-regulated in ago1 m

86 respond accordingly with salicylate-based or jasmonate/ethylene-based defensive signaling, respective

87 Here we describe a jasmonate family binding protein, cyclophilin 20-3 (CYP2

88 The jasmonate family of growth regulators includes the isole

89 The jasmonate family of phytohormones plays central roles in

90 acids was increased as consequence of methyl jasmonate foliar application, i.e., histidine, serine, t

91 Further, transcript profiling under methyl jasmonate, gibberellic acid, and yeast extract elicitati

92 Jasmonate hormone (JA) plays critical roles in both plan

93 for the regulation of antiviral RNAi by the jasmonate hormone signaling in plants.

94 This system harnesses the plant auxin and jasmonate hormone-induced degradation pathways, and is d

95 ed in the plant immune response dependent on jasmonate hormones.

96 In conclusion, foliar application of methyl jasmonate improved must nitrogen composition.

97 formation available on the multiple roles of jasmonates in plant development and defense, knowledge a

98 sjar1-3, to study the biological function of jasmonates in rice anthesis.

99 to understand the synthesis and function of jasmonates in roots.

100 , including a lower but more potent burst of jasmonates in several plant species.

101 Jasmonates, including jasmonic acid (JA) and methyl jasm

102 cient mutant demonstrated that roots produce jasmonates independently of leaves, despite low expressi

103 tes, including jasmonic acid (JA) and methyl jasmonate, induced the formation of tyloses, whereas tre

104 Because they are induced by JA we named them JASMONATE-INDUCED OXYGENASES (JOXs).

105 monate and demonstrate that steady state and jasmonate-induced pools of InsP8 in Arabidopsis seedling

106 Jasmonate-induced protein 60 (JIP60) is a ribosome-inact

107 Two closely related genes encoding the jasmonate-induced protein 60 (JIP60) were identified in

108 Here, we report on two homologous jasmonate-inducible transcription factors of the basic h

109 ine insensitive1 (coi1) and myc2 (allelic to jasmonate insensitive1) mutants, suggesting LBD20 may fu

110 iverse developmental and environmental cues, jasmonate is produced, conjugated to the amino acid isol

111 We found that the COR and jasmonate isoleucine (JA-Ile) co-receptor JAZ2 is consti

112 re-induced production of the stress hormones jasmonate-isoleucine conjugate and abscisic acid, which

113 The plant hormone jasmonate (JA) activates gene expression by promoting ub

114 TOR1 (ERF1) is an upstream component in both jasmonate (JA) and ethylene (ET) signaling and is involv

115 upon interactions between the plant hormones jasmonate (JA) and ethylene (ET).

116 strate for the green leaf volatile (GLV) and jasmonate (JA) biosynthesis pathways, respectively.

117 5 min after induction, thereby preceding the jasmonate (JA) burst.

118 d membrane depolarizations that activate the jasmonate (JA) defense pathway in leaves distal to wound

119 vity of the wound-inducible defence mediator jasmonate (JA) in undamaged tissues.

120 determine the effect of the defense hormone jasmonate (JA) on the growth, photosynthetic efficiency,

121 n of defence-related genes and activation of jasmonate (JA) pathway in the 'receiver' plant.

122 The plant hormone jasmonate (JA) plays an important role in regulating gro

123 Jasmonate (JA) promotes AAL toxin induced PCD in a COI1

124 The plant hormone jasmonate (JA) promotes resilience to many environmental

125 The plant hormone jasmonate (JA) promotes resistance to biotic stress by s

126 The plant hormone jasmonate (JA) promotes the degradation of JASMONATE ZIM

127 nt studies have identified the plant hormone jasmonate (JA) receptor as one of the common targets of

128 of transcriptional repressors that modulate jasmonate (JA) responses.

129 affected in the organ-specific activation of jasmonate (JA) signaling in Arabidopsis thaliana seedlin

130 Jasmonate (JA) signaling in plants is mediated by the JA

131 Jasmonate (JA) signaling is essential for several enviro

132 Far-red (FR) light-coupled jasmonate (JA) signaling is necessary for plant defense

133 tric stimulation activates the touch hormone jasmonate (JA) signaling pathway, which initiates secret

134 1) mutants, suggesting LBD20 may function in jasmonate (JA) signaling.

135 indole and iridoid pathways that respond to jasmonate (JA) signaling.

136 ysis into acetate synthesis to stimulate the jasmonate (JA) signalling pathway to confer drought tole

137 activation of the intracellular signaling of jasmonate (JA), a well-characterized defense hormone.

138 ction of FAD7 also inhibits the synthesis of jasmonate (JA), the effects of this desaturase on aphid

139 ete neighbors but has a repressive effect on jasmonate (JA)-dependent defenses.

140 otic gut bacteria from CPB larvae suppressed jasmonate (JA)-induced defenses in tomato.

141 s varies greatly and leaves are protected by jasmonate (JA)-inducible defenses.

142 The jasmonate (JA)-pathway regulators MYC2, MYC3, and MYC4 a

143 which structurally mimics the plant hormone jasmonate (JA).

144 g deficiencies were complemented with methyl jasmonate, JA-Ile, and its functional homolog, coronatin

145 Jasmonates (JAs) are a class of signaling compounds that

146 Jasmonates (JAs) are signaling molecules that have been

147 The plant hormones jasmonates (JAs) control the synthesis of specialized me

148 ely to be caused by the over-accumulation of jasmonates (JAs) in the llb mutant including the JA prec

149 esponsible for wound-inducible production of jasmonates (JAs), and green leafy volatiles (GLVs) respe

150 However, only endogenous levels of jasmonates (JAs), but not salicylic acid (SA) and abscis

151 ots and roots that controls the synthesis of jasmonates (JAs), in order to enhance defense responses

152 rough mimicking defense signaling molecules, jasmonates (JAs).

153 res the synthesis of potent mediators called jasmonates (JAs).

154 Jasmonic acid and its derivatives (jasmonates [JAs]) play central roles in floral developme

155 In the presence of active jasmonate, JAZ proteins function as jasmonate co-recepto

156 n irAGO4 plants, infection accumulated lower jasmonate levels and lower transcripts of jasmonic acid

157 he oxi1 mutation was associated with reduced jasmonate levels and with the up-regulation of genes enc

158 In addition, jasmonate levels in the fsh suppressor are significantly

159 D2 overexpression decreased OXI1 expression, jasmonate levels, and sensitivity to photooxidative stre

160 include the use of elicitors such as methyl jasmonate (MeJ) and benzothiadiazole (BTH).

161 pplications of phenylalanine (Phe) or methyl jasmonate (MeJ) could improve the synthesis of secondary

162 tudy the effect of the elicitation of methyl jasmonate (MeJ) supported by phenylalanine (Phe) as a pr

163 e, this work studied elicitation with methyl jasmonate (MeJ), supported by precursor feeding with phe

164 of preharvest treatments with 0.1 mM methyl jasmonate (MeJA) and 0.5 mM salicylic acid (SA) on quali

165 vest life, preharvest applications of methyl jasmonate (MeJA) and chitosan were evaluated during post

166 arried out to determine the effect of methyl jasmonate (MeJA) and different storage temperatures on t

167 Exposure to methyl jasmonate (MeJA) and methyl salicylate (MeSA) vapours at

168 Methyl jasmonate (MeJA) elicited the accumulation of levopimara

169 Methyl jasmonate (MeJA) elicits stomatal closure in many plant

170 Methyl jasmonate (MeJA) induced AtAO1 gene expression in vascul

171 Methyl jasmonate (MeJA) is commonly used to elicit plant stress

172 The effects of methyl jasmonate (MeJA) on leaf growth regulation were investig

173 Foliar application of methyl jasmonate (MeJA) or brassinazole (BRZ) resulted in a sig

174 with gamma-aminobutyric acid (GABA), methyl jasmonate (MeJA) or methyl salicylate (MeSA) on antioxid

175 We further show that applying methyl jasmonate (MeJA) phenocopied the stem elongation of Zeo1

176 with the well-known defense elicitor methyl jasmonate (MeJA) to young leaves of Arabidopsis (Arabido

177 pine needle transcriptomes, following methyl jasmonate (MeJA) treatment using RNA-seq.

178 iptional changes in sweet basil after methyl jasmonate (MeJA) treatment, which is considered an elici

179 Methyl jasmonate (MeJA) was applied in a vineyard on leaves and

180 in response to abscisic acid (ABA) or methyl jasmonate (MeJA) were identified by complementary proteo

181 the treatment doses of the elicitors: methyl jasmonate (MeJA), jasmonic acid (JA) and DL-methionine (

182 s of (3)C* and (1)O2* with five GLVs: methyl jasmonate (MeJa), methyl salicylate (MeSa), cis-3-hexeny

183 of phytohormone salicylic acid (SA), methyl jasmonate (MeJA), phytopathogenic bacteria, and flagelli

184 The effects of methyl jasmonate (MeJA), spermine (Spm), epibrassinolide (EBL)

185 onally characterized a leaf-specific, methyl jasmonate (MeJA)-responsive monoterpene synthase (Li3CAR

186 tebark pine needles demonstrated that methyl jasmonate (MeJA)-triggered transcriptome re-programming

187 es occur when the plant is exposed to methyl jasmonate (MeJA).

188 fferentially regulated in response to methyl jasmonate (MeJA).

189 The jasmonate-mimicking coronatine (COR) toxin produced by P

190 rant cultivars after elicitation with methyl jasmonate (MJ) were examined.

191 ers present in a commercial sample of methyl jasmonate (MJ) were isolated at semi-preparative scale b

192 ed with the plant-derived JA molecule methyl jasmonate (MJ).

193 he influence of foliar application of methyl jasmonate on must amino acid content.

194 L-carboxylic acid, salicylic acid and methyl jasmonate) on the phytochemical composition of broccoli

195 n Arabidopsis wild-type and two mutants with jasmonate or salicylic acid compromised immunities.

196 In seedlings, we found that jasmonate (or JA precursors) could translocate axially f

197 Treating irAGO4 plants with JA, methyl jasmonate, or cis-(+)-12-oxo-phytodienoic acid restored

198 caterpillar herbivory, application of methyl jasmonate, or mechanical damage during vegetative growth

199 Jasmonates, oxylipin-type plant hormones, are implicated

200 tes the evolutionary appearance of any other jasmonate pathway component.

201 of oxidative and hydrolytic branches in the jasmonate pathway highlight novel mechanisms of JA-Ile h

202 necessary and sufficient for activating the jasmonate pathway in M. polymorpha, but unlike their Ara

203 mediated restriction of AP2 may modulate the jasmonate pathway to facilitate gibberellin-promoted ste

204 vary incrementally in the expression of the jasmonate pathway, which mediates induced resistance to

205 vores induce plant resistance mainly via the jasmonate pathway.

206 nd contrasting effects were observed for the jasmonate pathway.

207 hesis pathway of salicylic acid (sid2-2) and jasmonate perception (coi1).

208 ll discuss how several pathogens exploit the jasmonate perception and early signalling machinery to d

209 We identify a role of VIH2 in regulating jasmonate perception and plant defenses against herbivor

210 gulated in ago1 mutants, we demonstrate that jasmonate perception drives the lesion phenotype.

211 Genes associated with the metabolism of jasmonates, phenylpropanoids, terpenoids and L-phenylala

212 The plant hormone jasmonate plays crucial roles in regulating plant respon

213 t, where several enzymatic steps produce the jasmonate precursor 12-oxophytodienoic acid (OPDA) from

214 ottontail rabbit Sylvilagus nuttallii avoids jasmonate-producing N. attenuata shoots because of their

215 by arthropod and vertebrate herbivores than jasmonate-producing plants in nature.

216 the first step of lipid oxidation leading to jasmonate production.

217 and biosynthesis of distinct MIAs following jasmonate production.

218 By contrast, treatment with methyl jasmonate promoted an increase of all analyzed AtPep-tri

219 nstrating that MYCs acting downstream of the jasmonate receptor complex and calmodulin-binding transc

220 nts of ago1 and coronatine insensitive1, the jasmonate receptor, showed greatly decreased frequency o

221 vincristine and vinblastine, we identified a jasmonate-regulated basic helix-loop-helix (bHLH) transc

222 InsP8 by COI1-JAZ is a critical component in jasmonate-regulated defenses.

223 ylate-based immunity and the repression of a jasmonate-related branch.

224 factors that are known to additively control jasmonate-related defense responses, was shown to have a

225 ranscription factors that additively control jasmonate-related defense responses, we found that egg e

226 SA, JA and COR and co-operation between JAZ jasmonate repressor proteins during DC3000 infection.

227 d that NaJAZi functions as a flower-specific jasmonate repressor that regulates JAs, (E)-alpha-bergam

228 death phenotype was strongly reduced in the jasmonate resistant1 mutant background.

229 SA hydroxylase NahG transgene, but not in a jasmonate resistant1-1 mutant, after B. cinerea infectio

230 response and a K(+) -dependent Vm-activated jasmonate response associated with the release of VOCs.

231 fy ILL6 as a new regulatory component of the jasmonate response pathway.

232 nregulation of photosynthesis and attenuated jasmonate responses systemically within the plant.

233 Jasmonate responses were implicated in resistance to GLS

234 KEY MESSAGE: A methyl jasmonate responsive 3-carene synthase (Li3CARS) gene wa

235 Consequently, the expression of jasmonate-responsive genes was reduced as well.

236 Against this backdrop, a jasmonate-responsive MYC2 transcription factor was ident

237 Last but not least, together with jasmonates, salicylate, and abscisic acid, ethylene is i

238 more, the elicitations in the form of methyl jasmonate, salicylic acid, ultraviolet B light, and woun

239 egulator of downstream responses mediated by jasmonate-salicylic acid signaling cross talk, is involv

240 network robustness via its inhibition of the jasmonate sector.

241 s monocot crop against root nematodes, where jasmonate seems to play a key role.

242 sic acid sensing/signaling with ethylene and jasmonate sensing/signaling in RNAi compared to WT roots

243 In parallel to impaired jasmonate signaling and metabolism, irHER1 plants were m

244 ion of genes encoding negative regulators of jasmonate signaling and PCD.

245 ion has been shown previously to up-regulate jasmonate signaling and to increase plant resistance to

246 gulation of nine hub regulators, including a jasmonate signaling pathway gene, PuMYC2, and an auxin s

247 illar feeding induces resistance through the jasmonate signaling pathway that is associated with the

248 and highlight a novel connection between the jasmonate signaling pathway, cell death, and sphingolipi

249 obal defense response is triggered involving jasmonate signaling, PR proteins and stilbenoid metaboli

250 on polymorphisms and deviated from canonical jasmonate signaling.

251 ling during herbivory, connecting Ca(2+) and jasmonate signaling.

252 Key mediators of jasmonate signalling include MYC transcription factors,

253 Loss of dormancy was dependent on an intact jasmonate signalling pathway and was associated with inc

254 In addition, jasmonate signalling plays a crucial role in the rhythmi

255 nvironment, illustrating the significance of jasmonate signalling, and of the proteins involved, for

256 thogens antagonize SA immunity by activating jasmonate signalling, for example Pseudomonas syringae p

257 wired to attenuate growth upon activation of jasmonate signalling.

258 The content of jasmonates, signalling molecules involved in glucosinola

259 The biogenesis of jasmonates starts in the chloroplast, where several enzy

260 of red raspberry fruits with the pure methyl jasmonate stereoisomers isolated proved that (-)-epi-MJ

261 or (methyl jasmonates) and minor (epi-methyl jasmonates) stereoisomers.

262 ids, which results in impaired expression of jasmonate target genes on exposure to various stresses.

263 onsive to treatment with the defense hormone jasmonate, there are no significant changes in nucleosom

264 e tools with which to identify intercellular jasmonate transport routes.

265 reased tolerance to photooxidative damage of jasmonate-treated ch1 plants and by the increased tolera

266 induction of MusaSAP1 by wounding and methyl jasmonate treatment indicated possible involvement of Mu

267 This finding suggests that methyl jasmonate treatment might be conducive to obtain wines o

268 In response to methyl jasmonate treatment, LIF2 was rapidly recruited to chrom

269 We found that upon wounding and methyl jasmonate treatments, MYB11 and ZML2 proteins are degrad

270 fection or after benzothiadiazole and methyl jasmonate treatments.

271 induced in leaves by alamethicin and methyl jasmonate treatments.

272 Hormonal elicitors, such as jasmonates, trigger a complex signaling circuit leading

273 Exogenous applications of jasmonate upregulated salicylate biosynthesis genes and

274 ponse to the exogenous application of methyl jasmonate was associated with increased bark concentrati

275 opening induced by exogenous applied methyl jasmonate was impaired in osjar1 plants and was restored

276 significant reduction in the accumulation of jasmonates was observed, due to reduced expression of JA

277 ome data from seedlings elicited with methyl jasmonate were also obtained, which enabled the identifi

278 e cytosol for further conversion into active jasmonates, which subsequently induces the expression of

279 .9%, and 0.18 mg for (-)- and (+)-epi-methyl jasmonates, with 98.6% and 91.6% respective purities.

280 amounts were 3.56 mg for (-) and (+)-methyl jasmonates, with respective purities of 96.1% and 99.9%,

281 ncluding CORONATINE INSENSITIVE 1 (COI1) and JASMONATE ZIM domain (JAZ) proteins.

282 promoting ubiquitin-dependent degradation of jasmonate ZIM domain (JAZ) transcriptional repressor pro

283 We used a JASMONATE ZIM DOMAIN 10 (JAZ10) reporter to screen for m

284 the SA effect, requires a functional SCFCOI1-JASMONATE ZIM-DOMAIN (JAZ) JA receptor module.

285 Jasmonate zim-domain (JAZ) proteins comprise a family of

286 JAsmonate ZIM-domain (JAZ) proteins repress the activity

287 (JA) signaling in plants is mediated by the JASMONATE ZIM-DOMAIN (JAZ) proteins that repress the act

288 e jasmonate (JA) promotes the degradation of JASMONATE ZIM-DOMAIN (JAZ) proteins to relieve repressio

289 As with other characterized JASMONATE ZIM-DOMAIN (JAZ) proteins, PtJAZ6 interacts wi

290 ense-related genes, including those encoding jasmonate ZIM-domain (JAZ) proteins, which play key role

291 tic stress by stimulating the degradation of JASMONATE ZIM-DOMAIN (JAZ) proteins, which relieves repr

292 ved by a co-receptor complex composed of the Jasmonate ZIM-domain (JAZ) repressor proteins and an E3

293 ranscription factors, which are repressed by jasmonate ZIM-domain (JAZ) transcriptional repressors in

294 8) that was induced by salicylic acid; and a jasmonate ZIM-domain protein 1 (DMG400002930) which was

295 ullin-F-box complex SCF(COI1), which targets JASMONATE ZIM-domain transcriptional repressor proteins

296 psis mutant (jazQ phyB) lacking a quintet of Jasmonate ZIM-domain transcriptional repressors and the

297 t did not enhance expression or stability of JASMONATE ZIM-domain transcriptional repressors, and SA/

298 n and removal of transcriptional repressors (JASMONATE-ZIM DOMAIN [JAZ] proteins) by an SCF receptor

299 D94N negatively affects the interaction with JASMONATE-ZIM DOMAIN protein, thereby resulting in const

300 HYTOCHROME-INTERACTING FACTOR3 (PIF3), PIF4, JASMONATE-ZIM DOMAIN1, and BRASSINAZOLE-RESISTANT1 (BZR1

301 howed nuclear localization, interaction with JASMONATE-ZIM-DOMAIN PROTEIN repressors, and regulation