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1 soactive mediator within the confines of the juxtaglomerular apparatus.
2 s confirmed that BNZ affected TGF within the juxtaglomerular apparatus.
3 tive renin in afferent arterioles and in the juxtaglomerular apparatus.
4 populations of fibroblasts and cells of the juxtaglomerular apparatus.
5 cells that constitute a key component of the juxtaglomerular apparatus.
6 ad fewer or no renin-expressing cells in the juxtaglomerular apparatus.
7 of renal glomeruli producing renin in their juxtaglomerular apparatus and by four times wild-type nu
10 o signal the release of renin from the renal juxtaglomerular apparatus, especially during volume depl
11 and provide evidence for a new role for the juxtaglomerular apparatus in the maintenance of the mesa
12 cated techniques including microperfusion of juxtaglomerular apparatus in vitro, micropuncture of kid
13 ated techniques, including microperfusion of juxtaglomerular apparatus in vitro, micropuncture of ren
14 s, and elevation of luminal succinate in the juxtaglomerular apparatus increases renin secretion, cau
16 t 20-28 h, while Thy 1-positive cells in the juxtaglomerular apparatus (JGA) were sequestered from th
17 erized by hypertrophy and hyperplasia of the juxtaglomerular apparatus of the kidneys, aldosteronism
18 duction was similar in the isolated perfused juxtaglomerular apparatus of wild-type (WT) and nitric o
19 buloglomerular feedback in isolated perfused juxtaglomerular apparatus preparations, although minor d
21 t generate paracrine chemical signals in the juxtaglomerular apparatus to control vital kidney functi