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1  cytoplasmic inclusions appear as a distinct juxtanuclear accumulation at the centrosome and this req
2 unication, daughter cell orientation and the juxtanuclear accumulation of actin, but only modest defe
3 y, Anxa6 knockdown also abrogated PE-induced juxtanuclear accumulation of secretory granules (SG) con
4 ts led to increased superoxide anion levels, juxtanuclear accumulation of ubiquitin- and p62/SQSTM1-p
5 e that undergoes a rapid condensation into a juxtanuclear aggregate during chemokine-induced polariza
6                            Of note, abnormal juxtanuclear aggregates of mitochondria were observed, a
7 roteasome activity elicited the formation of juxtanuclear aggregates with characteristics of aggresom
8 , trafficking of a folding mutant that forms juxtanuclear aggregates, EGFP/SP-C(C122/186G), was not c
9 ese severe defects developed gradually after juxtanuclear aggresome formation and were not associated
10 /-) cells with prosaposin localized to large juxtanuclear aggresome-like inclusions, which is indicat
11 racellular degradative capacity is exceeded, juxtanuclear aggresomes are formed to sequester misfolde
12                                              Juxtanuclear aggresomes form in cells when levels of agg
13 nd caused redistribution of the receptors to juxtanuclear aggresomes, significantly more so for TPbet
14 ubiquitinated (K48/K63-linked) proteins into juxtanuclear aggresomes, without affecting 20S proteasom
15  of microtubules eliminated the formation of juxtanuclear and intranuclear inclusion bodies by HtEx1.
16 osomes and mitochondria were enriched in the juxtanuclear area and co-aggregate into a compact inclus
17 romote concentration of endolysosomes in the juxtanuclear area of non-neuronal cells, and drive redis
18 n also causes clustering of lysosomes in the juxtanuclear area of the cell, but the mechanisms respon
19 ining the redistribution of lysosomes to the juxtanuclear area.
20 ts and HeLa cells result in the formation of juxtanuclear bodies containing SSBP2.
21 ogenous SSBP2 protein and sequesters it into juxtanuclear bodies in adenovirally transformed human em
22  microtubules to retain the integrity of the juxtanuclear bodies suggests them to be E1B55K containin
23 ower rate and did not rapidly concentrate in juxtanuclear bodies.
24 t polymerizes into networks extending from a juxtanuclear cage to the cell periphery.
25   When first expressed, vhs-GFP localized to juxtanuclear clusters, and later it colocalized and inte
26 en fluorescent protein, hLnk is found at the juxtanuclear compartment and also appears to be localize
27 s, while the 75-kDa protein localized to the juxtanuclear compartment and was packaged into virion pa
28  betaII (PKC betaII) translocated to a novel juxtanuclear compartment as observed in several cell typ
29 n the plasma membrane, but not in a distinct juxtanuclear compartment in which NHE3 is predominantly
30        In addition, the SDYQRL-TR-containing juxtanuclear compartment is more acidic than the recycli
31 zed together with other virion proteins in a juxtanuclear compartment termed the assembly compartment
32 rane, while E3-7.7K localized primarily to a juxtanuclear compartment that could not be identified.
33                                         This juxtanuclear compartment was localized close to the Golg
34 ation, and ErbB2 was observed to move into a juxtanuclear compartment where it colocalized with PKC-a
35 uitinated misfolded proteins accumulate in a juxtanuclear compartment where proteasomes are concentra
36                                          The juxtanuclear compartment, however, is a bona fide recycl
37 umulation of internalized cargo in a compact juxtanuclear compartment, Rabenosyn-5-RNAi caused its re
38 ate the targeting of misfolded proteins to a juxtanuclear compartment.
39  THP-1 cells at the plasma membrane and in a juxtanuclear compartment.
40 -HT receptor (5-HT2AR) into a Rab11-positive juxtanuclear compartment.
41 sical PKC isoenzymes, alpha and betaII, to a juxtanuclear compartment.
42 egress from the nucleus and associate with a juxtanuclear cytoplasmic assembly compartment, where vir
43  one (perivacuolar) or two (perivacuolar and juxtanuclear) dot-like aggregates per cell.
44 such membrane interactions are identified at juxtanuclear endocytic recycling compartments.
45 ly causing the observed retention of BST2 in juxtanuclear endosomes and stimulating its degradation i
46        We found that kazrin depletion delays juxtanuclear enrichment of internalized material, indica
47 ible and only partially affects STIM1 in the juxtanuclear ER compartment.
48 of the COPII transport protein, Sec24C, from juxtanuclear ER exit sites (ERES) into a diffusely cytos
49 g microtubule depolymerization, the central, juxtanuclear Golgi apparatus scatters to multiple periph
50      All Golgi region proteins examined lost juxtanuclear Golgi apparatus-like distribution as scored
51 nhibited the transport of Shiga toxin to the juxtanuclear Golgi apparatus.
52 d dynein for its retrograde transport to the juxtanuclear Golgi complex and that STB increases MT ass
53 with antimannosidase II and anti-p200 in the juxtanuclear Golgi complex.
54  fragments tracked outward from the compact, juxtanuclear Golgi complex.
55 lgi enzymes gradually redistributed from the juxtanuclear Golgi or Golgi ministacks to the ER in cell
56  gp75 associates with GIPC, primarily in the juxtanuclear Golgi region.
57 al markers accumulated at the same rate in a juxtanuclear Golgi.
58  aggresome-like organelles we identified are juxtanuclear, HttPolyQ aggregate-enriched, and dependent
59 duce two distinct types of aggregates: large juxtanuclear inclusion bodies and small punctate aggrega
60  S reactivity of the fractions show that the juxtanuclear inclusion bodies are filled with amyloid-li
61 d cells and prevented the formation of large juxtanuclear inclusion bodies.
62 tein suffices to promote its delivery to the juxtanuclear inclusion.
63 d cells frequently leads to the formation of juxtanuclear inclusions that have been termed 'aggresome
64 sequently found to be enclosed within large, juxtanuclear, LAMP-1-positive vacuoles called Francisell
65 er, a large fraction of Hook3 maintained its juxtanuclear localization after Brefeldin A treatment, i
66             PKC-alpha knockdown impaired the juxtanuclear localization of ErbB2.
67  Fragmented Golgi membranes maintained their juxtanuclear localization, cisternal organization and ar
68 ding to IEV in size and shape, moving from a juxtanuclear location to the periphery of the cell, wher
69 o one in which filaments are aggregated in a juxtanuclear location, opposite to the direction of cell
70 lpha1, or N-cadherin, generate intracellular juxtanuclear membrane tubules when expressed in cells.
71 o focal adhesions in addition to cytoplasmic juxtanuclear membranes within infected cells.
72                          During the process, juxtanuclear mitochondrial aggregates resembling a prote
73 y: peripheral lysosomes are less acidic than juxtanuclear ones despite their comparable buffering cap
74 lized SV5 HN in vesicle-like structures in a juxtanuclear pattern coincident with the localization of
75 form an elaborately intertwined network with juxtanuclear PKA stores bound to Golgi membranes.
76 isternae were still stacked and located in a juxtanuclear position.
77 G132-treated quiescent cells displayed fewer juxtanuclear protein aggregates, less apoptosis, and hig
78 the insoluble protein deposit (IPOD) and the juxtanuclear quality control (JUNQ).
79 s localize to one of these compartments, the juxtanuclear quality control compartment (JUNQ), and int
80   Particle tracking reveals that INQ and the juxtanuclear quality control compartment converge to fac
81 folded proteins concentrate in a cytoplasmic juxtanuclear quality control compartment, while nuclear
82 3 in retrograde transport contributes to the juxtanuclear redistribution of endolysosomes upon cytoso
83                 FRM2 localizes to the apical juxtanuclear region and participates in apicoplast inher
84 vented the translocation of PKCbetaII to the juxtanuclear region but not to the plasma membrane, thus
85 dosomes, which were less concentrated at the juxtanuclear region in mutant cells than in control fibr
86 at the deposition of the protofibrils in the juxtanuclear region is important in fibril formation.
87 tankyrase with GLUT4 storage vesicles in the juxtanuclear region of adipocytes.
88 ular membrane compartment emanating from the juxtanuclear region of cells, which resembled the compar
89 olgi apparatus and recycling endosome in the juxtanuclear region of resting peritoneal macrophages.
90 ular distribution of the gene product in the juxtanuclear region of the cells.
91 istically, translocation of PKCbetaII to the juxtanuclear region required kinase activity.
92 oproteins results in their localization to a juxtanuclear region that is presumably the site of intra
93 y demonstrated that virion movement from the juxtanuclear region to the periphery was saltatory with
94 es a dramatic redistribution of CAL from the juxtanuclear region to the plasma membrane where the two
95 FP-enriched vesicles rapidly travel from the juxtanuclear region to the plasma membrane.
96 C10 directs the trafficking of CFTR from the juxtanuclear region to the secretory pathway toward the
97  infection results in WDR5 localization in a juxtanuclear region, and that its localization to this c
98 ER stress conditions, the QCVs converge in a juxtanuclear region, at the ERQC, as previously reported
99 ng between the two, concentrated ERES in the juxtanuclear region, blocked cargo export and relocated
100 KC) alpha and betaII become sequestered in a juxtanuclear region, the pericentrion.
101  AGS3 and AGS3-GFP from the cell cortex to a juxtanuclear region, where it co-localized with markers
102 r trafficking from the cell periphery to the juxtanuclear region, where they acquire EEA1.
103 to sequestration of recycling endosomes in a juxtanuclear region.
104 ing of B5-GFP to the site of wrapping in the juxtanuclear region.
105  and APPL to the early EEA1 endosomes in the juxtanuclear region.
106 tivity within HSFs was mostly present in the juxtanuclear region.
107 g COPII-associated membranes to cluster to a juxtanuclear region.
108 tacks in the pericentriolar and often in the juxtanuclear regions of the cell.
109 teract with SEC16A and could localize to the juxtanuclear secretory compartment.
110 KCbetaI translocates to a recently described juxtanuclear site of localization for PKCalpha and PKCbe
111 ible fragmentation of the Golgi complex into juxtanuclear, stacked cisternal elements.
112  Immunofluorescent staining reveals striking juxtanuclear staining characteristic of the Golgi appara
113 e cycle of HCMV and accumulated in a stable, juxtanuclear structure late in infection.
114 essed, fluorescence began to accumulate in a juxtanuclear structure.
115   The morphology of the SDYQRL-TR-containing juxtanuclear structures is different from the recycling
116 ng pathway, colocalize with SDYQRL-TR in the juxtanuclear structures.
117 s and late endosomes into large (2-3 microm) juxtanuclear structures.
118 alization in corticotropes, SSTR2 moves to a juxtanuclear syntaxin-6-positive compartment, where it r
119 both necessary and sufficient for preventing juxtanuclear translocation of PKC betaII in response to
120 define a role for this pathway in regulating juxtanuclear translocation of PKC betaII.
121 F-7 cells but not in those cells that showed juxtanuclear translocation of PKC betaII.
122                                              Juxtanuclear translocation of PKC required an intact C1
123 onstrating a role for phospholipase D in the juxtanuclear translocation of PKCbetaII.
124  NFlank, shifting NFlank immunostaining to a juxtanuclear tubular array.
125 ells, the SDYQRL-TR construct accumulated in juxtanuclear tubules and vesicles that are in the vicini
126 GF receptors from peripheral compartments to juxtanuclear vesicles, and their subsequent degradation.
127 oprecipitate and colocalize with Dlg1 in the juxtanuclear zone.

 
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