ライフサイエンスコーパス: kPa

1 creased by 6.4 degrees C, coupled with a 1.0 kPa drop in vapor pressure deficit having a 9-minute lag

2 VCTE cutoffs of 5.0 kPa or less (to exclude) and 8.8 kPa or greater (to conf

3 A cutoff value of 51.0 kPa at 4 cm proximal to the medial malleolus revealed a

4 had the highest probabilities of LSM >/=8.0 kPa (overall probability: 17.2% [12.5-23.4]; this probab

5 decreased from 19.2 +/- 15.3 to 11.7 +/- 8.0 kPa at 48 weeks (p < 0.001).

6 LSM >8.0 kPa but not metabolic syndrome was an independent risk f

7 Patients with baseline LSM >8.0 kPa had higher cumulative probability of hepatic events

8 +/- 14.2 kPa in F4 disease and 14.6 +/- 8.0 kPa in F3 disease, baseline mean Enhanced Liver Fibrosis

9 ed presence) were associated with LSM >/=8.0 kPa in multivariable analyses.

10 adjusted predicted probability of LSM >/=8.0 kPa increased per age decade, with probabilities ranging

11 tion-based study of older adults, LSM >/=8.0 kPa, suggestive of clinically relevant fibrosis, was pre

12 LSM, 169 (5.6%) participants had LSM >/=8.0 kPa.

13 - 0.017 for cross-fiber, and 0.039 +/- 0.011 kPa for shear stress.

14 sor with a sensitivity ( approximately 0.018 kPa(-1) ), response time ( approximately 60 ms), and goo

15 a thickness of 110 nm and a modulus of 0.025 kPa.

16 ressive stiffness of O(1) kPa (0.49 +/- 0.04 kPa stress at 30% compressive strain) and therefore mimi

17 firm q-DC measurements of Ea = 0.53 +/- 0.04 kPa.

18 Average patient level variance was 0.042 kPa, with a coefficient of variation of 10.7%.

19 c airway resistance (abeta coefficient, 0.06 kPa/s; 95% CI, 0.00-0.11 kPa/s; P = .05).

20 .47 kPa, 0.53 +/- 0.12 kPa and 7.06 +/- 4.07 kPa respectively.

21 s, leading to high sensitivities of a 0.0835 kPa(-1) change in output voltage.

22 G (d) = 3.7 +/- 0.2 kPa, G (l) = 2.2 +/- 0.1 kPa, n = 7) breast cancer xenografts, and Th-MYCN neurob

23 vs 54 +/- 1 torr; 7.6 +/- 0.4 vs 7.1 +/- 0.1 kPa; p = 0.37) with a lower minute volume (6.4 +/- 0.5 v

24 us grade 1, 90.4 kPa +/- 9.3; grade 5, 120.1 kPa +/- 15.4), and there were weak correlations between

25 Mean liver stiffness was 2.1 kPa (95% CI: 2.0, 2.2 kPa) with mean +/- 1.96 kPa standa

26 hy in children without liver disease was 2.1 kPa (similar to that in adults).

27 (20 +/- 7 kPa), PaCO2 40 +/- 5 torr (5 +/- 1 kPa), and core temperature 33 degrees C +/- 1 degrees C.

28 important factors associated with LSMs >=7.1 kPa in subjects with the Pi*MZ genotype.

29 enotype vs 4% of noncarriers had LSMs of 7.1 kPa or more (adjusted odds ratio, 4.8; 95% confidence in

30 icant liver fibrosis was defined as TE >=7.1 kPa.

31 rain neurons (ECFNs; optimum approximately 1 kPa) and U251 glioma cells (optimum approximately 100 kP

32 .70]), and LS at SVR (1.05 [1.02-1.08] for 1 kPa increase).

33 1.70]) and LS at SVR (1.05 [1.02-1.08] for 1 kPa increase).

34 LS value at SVR [sHR=1.03 (1.01-1.06) for 1 kPa increase; p=0.011].

35 ection and localized retention of a soft (<1 kPa) hydrogel.

36 Cells on soft (1 kPa) gels formed fewer stress fibers and retained a more

37 e were 249.7 +/- 58.1 dB/m and 11.9 (6-18.1) kPa.

38 ility from 5.82 to 2.51 cm(3) um m(-2) d(-1) kPa(-1), water vapor permeability from 1.89 to 1.38 g mm

39 meability from 1.89 to 1.38 g mm m(-2) h(-1) kPa(-1), solubility from 0.65 to 0.55 g soluble solid/to

40 ith an average compressive stiffness of O(1) kPa (0.49 +/- 0.04 kPa stress at 30% compressive strain)

41 ated advanced chronic liver disease (LSM >10 kPa); (2) availability of EGD within 6 months of reliabl

42 ated advanced chronic liver disease (LSM >10 kPa); 2) availability of EGD within 6 months of reliable

43 antable soft electronics (on the order of 10 kPa in modulus) remains a challenge because of the limit

44 Keratocytes cultured on stiff (10 kPa) gels or collagen-coated glass coverslips, however,

45 ctable low-frequency shear moduli ca. 10-100 kPa, well below values typical of linear polymer CANs (1

46 hanical properties (typically between 10-100 kPa/mm) on small scales, which will be of significant us

47 de gels of three stiffnesses (1, 30, and 100 kPa) and treated with a range of epidermal growth factor

48 U251 glioma cells (optimum approximately 100 kPa).

49 At 100 kPa and 298 K Azole-Th-1 performs excellent separation o

50 c materials with a Young's modulus below 100 kPa conforming to biological applications, yet those gel

51 d in a fixed-bed reactor (550 degrees C, 100 kPa, space velocity 4650 mL g(cat)(-1) h(-1)).

52 oft biological tissue (Young's modulus < 100 kPa), and the capability to undergo extreme deformations

53 s compliant, with an elastic modulus G < 100 kPa, the droplet will markedly slow as a consequence of

54 h strength (1 MPa), low Young's modulus (100 kPa), and high water content (70 to 80 wt %), which have

55 rapidly generated in presence of oxygen (100 kPa), which remained stable up to 22 h.

56 creased by the highest stiffness tested (100 kPa).

57 ical Young's modulus range of 0.5 kPa to 100 kPa.

58 airway resistance (abeta coefficient, -0.11 kPa/s; 95% CI, -0.21 to -0.02; P = .02), decreased bronc

59 a coefficient, 0.06 kPa/s; 95% CI, 0.00-0.11 kPa/s; P = .05).

60 outer boundaries of the vessel wall (from 11 kPa to zero).

61 For small effective overburden loads (<=11 kPa), stable ascent is followed by dynamic ascent (which

62 nonpolar (e.g., vitamin D2; VP 8.5 x 10(-11) kPa) compounds were successfully ionized by the containe

63 (day 14) of 1.04 +/- 0.47 kPa, 0.53 +/- 0.12 kPa and 7.06 +/- 4.07 kPa respectively.

64 , lower platelets, and liver stiffness >/=12 kPa at year 5 represent the main risk factors for late H

65 seline and year 5, and liver stiffness >/=12 kPa at year 5 were independently associated with more fr

66 histologically or when liver stiffness >=12 kPa) treated with DAAs.

67 rm (25 degrees C) normoxia, warm hypoxia (12 kPa O(2) ), cold (5 degrees C) normoxia, or cold hypoxia

68 ge of 67 kPa on the volcano surface and ~120 kPa in the spherical magma chamber.

69 tion and were classified as (1) OcC (TE >=13 kPa with no sign of cirrhosis, including absence of thro

70 roscopy); (2) overt cirrhosis (OvC) (TE >=13 kPa with signs of cirrhosis); or (3) noncirrhotic patien

71 hosis); or (3) noncirrhotic patients (TE <13 kPa).

72 corresponding to adipose (4 kPa), muscle (13 kPa) and osteoid (30 kPa) tissues respectively by exposi

73 orresponding to a shear modulus of 72 +/- 14 kPa.

74 cifically investigated mild TBI range (0-145 kPa) using physiological (heart rate), pathological (lun

75 rats when the BOP is in the range of 85-145 kPa.

76 We determined a BOP of 145 kPa as upper mild TBI threshold (5% PMR).

77 timate a cortex elastic modulus of E(c) ~ 15 kPa, an effective cortex thickness of t(c) ~ 0.3 mum, an

78 52 patients had mean liver stiffness >= 15 kPa.

79 a moderate overpressure ( approximately 150 kPa) and stirring the sample.

80 rom the computer simulation ranged from 3-16 kPa at the vitreoretinal interface through a cycle of sh

81 media and adventitia layers (from 80 to 160 kPa), dropped abruptly at the interface (from 160 to <5

82 nsion of 0.44 +/- 0.25 kPa and 2.53 +/- 1.17 kPa, respectively.

83 equilibrium Young's moduli of 37 kPa and 17 kPa, respectively.

84 rogels with an initial elastic modulus of 17 kPa.

85 as the difference between 5 and 20 Hz, 0.18 kPa.L(-1).s; Asthma Control Questionnaire score, 0.76; a

86 y AFM passing from 17 +/- 6 kPa to 56 +/- 18 kPa.

87 elastic modulus of the zebra finch ML is 18 kPa at 5% strain, which is comparable to elastic moduli

88 in a compressed gas-driven shock tube at 180 kPa and the BBB integrity was assessed by extravasation

89 te MPs, those exposed to air pressure at 180 kPa for 1 hour generated 1.4 +/- 0.1 MPs/cell (n = 8, p

90 ll lines (Ea = 2.1 +/- 0.1 and 0.80 +/- 0.19 kPa for MCF-7 and MDA-MB-231 cells).

91 ed luc-MDA-MB-231-LM2-4 (G (d) = 3.7 +/- 0.2 kPa, G (l) = 2.2 +/- 0.1 kPa, n = 7) breast cancer xenog

92 .2 kPa, n = 7), luc-RG2 (G (d) = 3.5 +/- 0.2 kPa, G (l) = 2.3 +/- 0.2 kPa, n = 5), and luc-U-87-MG (G

93 Th-MYCN neuroblastomas (G (d) = 3.5 +/- 0.2 kPa, G (l) = 2.3 +/- 0.2 kPa, n = 5).

94 orthotopic luc-D-212-MG (G (d) = 3.5 +/- 0.2 kPa, G (l) = 2.3 +/- 0.2 kPa, n = 7), luc-RG2 (G (d) = 3

95 (KI/KI) medulloblastoma (G (d) = 3.5 +/- 0.2 kPa, G (l) = 2.3 +/- 0.2 kPa, n = 7), orthotopic luc-D-2

96 n = 5), and luc-U-87-MG (G (d) = 3.5 +/- 0.2 kPa, G (l) = 2.3 +/- 0.2 kPa, n = 8) glioblastoma xenogr

97 r for orthotopic BT-474 (G (d) = 5.9 +/- 0.2 kPa, G (l) = 4.7 +/- 0.2 kPa, n = 7) and luc-MDA-MB-231-

98 G (d) = 3.5 +/- 0.2 kPa, G (l) = 2.3 +/- 0.2 kPa, n = 5), and luc-U-87-MG (G (d) = 3.5 +/- 0.2 kPa, G

99 G (d) = 3.5 +/- 0.2 kPa, G (l) = 2.3 +/- 0.2 kPa, n = 5).

100 G (d) = 7.9 +/- 0.4 kPa, G (l) = 6.0 +/- 0.2 kPa, n = 6) breast cancer xenografts, and luc-PANC1 (G (

101 G (d) = 5.9 +/- 0.2 kPa, G (l) = 4.7 +/- 0.2 kPa, n = 7) and luc-MDA-MB-231-LM2-4 (G (d) = 7.9 +/- 0.

102 G (d) = 6.9 +/- 0.3 kPa, G (l) = 6.2 +/- 0.2 kPa, n = 7) pancreatic cancer xenografts, compared with

103 G (d) = 3.5 +/- 0.2 kPa, G (l) = 2.3 +/- 0.2 kPa, n = 7), luc-RG2 (G (d) = 3.5 +/- 0.2 kPa, G (l) = 2

104 G (d) = 3.5 +/- 0.2 kPa, G (l) = 2.3 +/- 0.2 kPa, n = 7), orthotopic luc-D-212-MG (G (d) = 3.5 +/- 0.

105 G (d) = 3.5 +/- 0.2 kPa, G (l) = 2.3 +/- 0.2 kPa, n = 8) glioblastoma xenografts, intracranially prop

106 I argue for a low value, 1-2 kPa, in frog eggs and perhaps more generally.

107 , P = .0003) but not at 28 Hz (children, 1.2 kPa +/- 0.2; adolescents, 1.3 kPa +/- 0.3; adults, 1.2 k

108 2; adolescents, 1.3 kPa +/- 0.3; adults, 1.2 kPa +/- 0.2; analysis of variance, P = .40).

109 105 +/- 9 torr; 20.5 +/- 2.8 vs 14.0 +/- 1.2 kPa; p = 0.035) and achieved comparable PaCO2 (57 +/- 3

110 hear wave elastography were 9.6 kPa and 10.2 kPa, and for cirrhosis 19.7 kPa and 16.4 kPa.

111 eets of different stiffnesses (50.0 +/- 11.2 kPa and 90.0 +/- 16.4 kPa) to engineer biomimetic models

112 s, baseline liver stiffness of 24.1 +/- 14.2 kPa in F4 disease and 14.6 +/- 8.0 kPa in F3 disease, ba

113 (children, 2.2 kPa +/- 0.3; adolescents, 2.2 kPa +/- 0.2; adults, 2.6 kPa +/- 0.3; analysis of varian

114 cents than in adults at 56 Hz (children, 2.2 kPa +/- 0.3; adolescents, 2.2 kPa +/- 0.2; adults, 2.6 k

115 iver stiffness was 2.1 kPa (95% CI: 2.0, 2.2 kPa) with mean +/- 1.96 kPa standard deviation of 1.5-2.

116 ctuators that can generate up to 1.7 mN (3.2 kPa) of passive tension force and 300 muN (0.56 kPa) of

117 r than that for P7 shams (691.6 kPa vs 429.2 kPa, p = 0.0194).

118 achieved HBsAg seroclearance (5.5 versus 5.2 kPa, respectively; P = 0.52).

119 sensitivity >/=90% were very close: 6.3/6.2 kPa for >/=F2, 8.3/8.2 kPa for >/=F3, and 10.5/9.5 kPa f

120 vs 57.9 kPa [interquartile range, 45.1-64.2 kPa]; p = 0.019].

121 e very close: 6.3/6.2 kPa for >/=F2, 8.3/8.2 kPa for >/=F3, and 10.5/9.5 kPa for F4.

122 f values for F>=F2, F>=F3, and F=F4 were 8.2 kPa, 9.7 kPa, and 13.6 kPa, respectively.

123 radients of 0.5, 1.7, 2.9, 4.5, 6.8, and 8.2 kPa/mm, spanning the in vivo physiological and pathologi

124 gher forces of ~360 nN and pressures of ~9.2 kPa.

125 1 increased linearly with VPD, up to about 2 kPa.

126 +/- 1.37%) over a wide pressure range (0-20 kPa), and fast response (400 Hz).

127 peptides forms robust hydrogels from 0.2-20 kPa at low weight percent (less than 3 wt %) in cell cul

128 s with liver stiffness measurements below 20 kPa and platelet counts above 150,000 (favorable Baveno

129 and a liver stiffness measurement (LSM) <20 kPa (Baveno VI criteria).

130 Baveno VI (LSM <20 kPa and platelets >150 000/muL), expanded Baveno VI (LSM

131 chanical thresholds of contact pressure (~20 kPa) and sliding velocity (~0.3 m s(-1) ).

132 which return a range of values from 1 to 20 kPa.

133 s over an order of magnitude range of 10-200 kPa, which is in the region of many soft tissues.

134 er, the operating pressure was less than 200 kPa, which eliminated the need for an expensive high-pre

135 resulting in an adhesive strength up to 204 kPa.

136 Rats received two 30-psi (~ 207-kPa) blasts 24 h apart or were handled identically witho

137 epatitis Viricas (HEPAVIR) criteria (LSM <21 kPa) were applied to identify patients not requiring EGD

138 Also we determined 146-220 kPa and 221-290 kPa levels as moderate and severe TBI ba

139 precedentedly high sensitivity (S(min) > 220 kPa(-1)) over a broad pressure regime (0.08 Pa-360 kPa),

140 ure (fracture strengths of 41-157 vs. 48-226 kPa for fine powders).

141 corresponding to a tension of 0.44 +/- 0.25 kPa and 2.53 +/- 1.17 kPa, respectively.

142 ygen in inhaled air stepwisely (pO(2); 21.25 kPa (0 k), 16.42 kPa (2 k), 12.63 kPa (4 k) and 9.64 kPa

143 s >150 000/muL), expanded Baveno VI (LSM <25 kPa and platelets >110 000/muL), and Estudio de las Hepa

144 telet count >110 x 10(9) cells/L and LSM <25 kPa.

145 ., carminic acid; estimated VP 5.1 x 10(-25) kPa) and nonpolar (e.g., vitamin D2; VP 8.5 x 10(-11) kP

146 RI-PDFF was 16.3% and MRE-stiffness was 3.27 kPa.

147 of oxygen to fractional inspired oxygen <27 kPa and positive end-expiratory pressure [PEEP] >=8 cm H

148 nd subsequent stiffening (from ca. 3.5 to 28 kPa) increased the cell area and nuclear localization of

149 7 (20-37) kPa before treatment to 18 (14-28) kPa after SVR (P < .05).

150 /- 0.12 in the steatohepatitis group vs 0.29 kPa +/- 0.01 in the control group; P = .02).

151 logical range of stiffness (E = 1.85 to 5.29 kPa), with and without RGD binding sites or collagen fib

152 Also we determined 146-220 kPa and 221-290 kPa levels as moderate and severe TBI based on 35%, and

153 d 70% PMR, respectively, while BOP above 290 kPa is lethal.

154 nografts, and luc-PANC1 (G (d) = 6.9 +/- 0.3 kPa, G (l) = 6.2 +/- 0.2 kPa, n = 7) pancreatic cancer x

155 (children, 1.2 kPa +/- 0.2; adolescents, 1.3 kPa +/- 0.3; adults, 1.2 kPa +/- 0.2; analysis of varian

156 ated cutoffs for moderate fibrosis (8.0-12.3 kPa) and severe fibrosis/cirrhosis (>12.3 kPa).

157 .3 kPa) and severe fibrosis/cirrhosis (>12.3 kPa).

158 e Pa(O(2)) time curve and a boundary of 13.3 kPa (100 mm Hg) divided by the hours of potential exposu

159 patients with score 0 (baseline LSM <= 17.3 kPa, albumin >4.2 g/dL, and 1-year DeltaLSM > 25.5%) ver

160 slinking) stiffened the hydrogel (41.4+/-4.3 kPa) after injection.

161 (4.7-5.3, 5.3-6.4, 21.2-21.3, and 50.7-53.3 kPa).

162 ed a range of elastic moduli from 0.4 to 9.3 kPa.

163 n silt-loam under high evaporative demand (3 kPa VPD, 38 degrees C).

164 brosis (magnetic resonance elastography >/=3 kPa).

165 Hypoxic incubation (3 kPa) caused (i) stabilization of HIF-2alpha and up-regul

166 in paraspeckles in cells cultured on soft (3 kPa) hydrogels compared with stiffer (40 kPa) hydrogels.

167 have obtained can sustain up to 4.8 x 10(3) kPa pressure, a level comparable to cementitious materia

168 P)) are 0.038 degrees C(-1) and 5.3 x 10(-3) kPa(-1), respectively.

169 ns by a factor of approximately 360, from 30 kPa to 11 MPa along the first millimeter of the follicle

170 out EVNT were platelets >110,000 and LSM <30 kPa (HIV cirrhosis criteria), with 34.6% EGD spared and

171 EVNT were platelets >110 000/muL and LSM <30 kPa (HIV cirrhosis criteria), with 34.6% of EGDs spared

172 t tissue-like softness (Young's modulus ~ 30 kPa), have uniform and tunable thickness in the range of

173 ose (4 kPa), muscle (13 kPa) and osteoid (30 kPa) tissues respectively by exposing them to 2 W/cm(2)

174 Cell adhesion to a 30-kPa Ecad-Fc PA gel required Cdc42- and formin-dependent

175 e were exposed to blast wave pressure of 300 kPa (43.5 psi) per day for 3 successive days, and euthan

176 re (100 degrees C) and applied pressure (300 kPa), ground TBEU thermoset powder can be remolded to bu

177 0.05 versus ambient air) when exposed to 300 kPa for 35 minutes.

178 We found that 300-kPa blasts yielded no detectable cognitive or motor defi

179 2.4 years, median hepatic stiffness was 2.35 kPa.

180 93 kPa +/- 2.31 [standard deviation] vs 3.35 kPa +/- 1.44, P < .0001).

181 al (p > 0.08) over a large range (47 Pa - 36 kPa).

182 )) over a broad pressure regime (0.08 Pa-360 kPa), and an ultrahigh pressure resolution (18 Pa or 0.0

183 ressive and equilibrium Young's moduli of 37 kPa and 17 kPa, respectively.

184 LSM decreased from 27 (20-37) kPa before treatment to 18 (14-28) kPa after SVR (P < .0

185 fined compressive strengths of 5171 and 3848 kPa were obtained, significantly higher than those for t

186 nd luc-MDA-MB-231-LM2-4 (G (d) = 7.9 +/- 0.4 kPa, G (l) = 6.0 +/- 0.2 kPa, n = 6) breast cancer xenog

187 ly decreased from 5.4 +/- 0.9 to 4.8 +/- 1.4 kPa (p = 0.001).

188 ng alternating traction forces, of up to 1.4 kPa, at each flank of the lamellipodium, ColXVII knockdo

189 sensor unit achieves high sensitivity (14.4 kPa(-1) ), low detection limit (2 Pa), fast response ( a

190 fnesses (50.0 +/- 11.2 kPa and 90.0 +/- 16.4 kPa) to engineer biomimetic models of vascularized cardi

191 0.2 kPa, and for cirrhosis 19.7 kPa and 16.4 kPa.

192 and the mean post-meal delta increase of 2.4 kPa.

193 otest score, >=0.54; liver stiffness, >=23.4 kPa.

194 d sham mice were similar (341.9 kPa vs 363.4 kPa, p = 0.6140), the modulus for P7 MI mice was signifi

195 kPa +/- 2.1; annulus fibrosus grade 1, 90.4 kPa +/- 9.3; grade 5, 120.1 kPa +/- 15.4), and there wer

196 stiffness levels corresponding to adipose (4 kPa), muscle (13 kPa) and osteoid (30 kPa) tissues respe

197 mechanical resistance (Young's modulus of 4 kPa).

198 rosis) (WMD: -0.70 kPa; 95% CI: -1.00, -0.40 kPa), although analyses showed heterogeneity (I2 = 90.6%

199 e pain threshold on the hand (mean change 40 kPa, 95% CI 0.7 to 80, p=0.046) and myometric measuremen

200 re was defined as PO(2)/FiO(2) ratio of <=40 kPa.

201 (3 kPa) hydrogels compared with stiffer (40 kPa) hydrogels.

202 ir stepwisely (pO(2); 21.25 kPa (0 k), 16.42 kPa (2 k), 12.63 kPa (4 k) and 9.64 kPa (6 k)).

203 0, 130, 160, 190, 230, 250, 290, 350 and 420 kPa) and determines the mortality rate as a non-linear f

204 ed a compressive modulus of 499.18 +/- 86.45 kPa.

205 monstrates 24 hour survival of rats in 0-450 kPa (0-800 Pas impulse) range at 10 discrete levels (60,

206 d cells and nuclei (day 14) of 1.04 +/- 0.47 kPa, 0.53 +/- 0.12 kPa and 7.06 +/- 4.07 kPa respectivel

207 at of NDM adipose tissue (11.50 kPa vs. 4.48 kPa) to a 95% confidence level, with significant variabi

208 mal fouling of the MnO2 coated membrane (0.5 kPa for 70 days), while the uncoated membrane experience

209 e physiological Young's modulus range of 0.5 kPa to 100 kPa.

210 , APRI>=1.5, or Fibroscan measurement >=10.5 kPa.

211 ration grade (nucleus pulposus grade 1, 12.5 kPa +/- 1.3; grade 5, 16.5 kPa +/- 2.1; annulus fibrosus

212 sus grade 1, 12.5 kPa +/- 1.3; grade 5, 16.5 kPa +/- 2.1; annulus fibrosus grade 1, 90.4 kPa +/- 9.3;

213 dry/wet scribed tape peel adhesion and 17.5 kPa Taber linear abraser tests.

214 on (MRI-PDFF) >=8% and liver stiffness >=2.5 kPa by magnetic resonance elastography (MRE) or historic

215 ll was determined to be 3.0-6.5 kPa (1.5-2.5 kPa for the brush model), with a significant increase in

216 situ hydrogel softening (from ca. 14 to 3.5 kPa) led to a decrease in the cell area and nuclear loca

217 my (45.8 kPa [interquartile range, 36.9-56.5 kPa] vs 57.9 kPa [interquartile range, 45.1-64.2 kPa]; p

218 ndothelial cell was determined to be 3.0-6.5 kPa (1.5-2.5 kPa for the brush model), with a significan

219 (children, 5.6 kPa +/- 0.8; adolescents, 6.5 kPa +/- 1.2; adults, 7.8 kPa +/- 1.2; analysis of varian

220 Samples were fried under vacuum (6.5 kPa, Twater-boiling-point=38 degrees C) or atmospheric c

221 h cell pre-stress with culture on stiff (7.5 kPa) polyacrylamide gels (with or without transforming g

222 TE examination with mean liver stiffness 8.5 kPa (SD 9.2).

223 SVR with DAA-including regimen; ii) LS >=9.5 kPa before therapy and; iii) LS measurement available at

224 R with DAA-including regimen; (ii) LS >= 9.5 kPa before therapy; and (iii) LS measurement available a

225 r >/=F2, 8.3/8.2 kPa for >/=F3, and 10.5/9.5 kPa for F4.

226 d combination; 2) Liver stiffness (LS) >=9.5 kPa previous to treatment; 3) LS measurement at the SVR

227 e (PA) gels with varying stiffness (6.5-92.5 kPa) as the substrate to construct a mechanical microenv

228 ed abruptly at the interface (from 160 to <5 kPa), and increased slightly towards the outer boundary

229 was twice that of NDM adipose tissue (11.50 kPa vs. 4.48 kPa) to a 95% confidence level, with signif

230 hen the CFbs are cultured on either 10 or 50 kPa static hydrogels.

231 l formulation that can stiffen from 10 to 50 kPa, corresponding to the stiffness of a healthy and fib

232 ge of stiffness from approximately 0.5 to 50 kPa, stemness maintenance did not correlate with initial

233 stiffness at 80 Hz and 48-week feeding, 0.51 kPa +/- 0.12 in the steatohepatitis group vs 0.29 kPa +/

234 tor that can generate up to 300 microN (0.56 kPa) of active tension force in response to a noninvasiv

235 ) of passive tension force and 300 muN (0.56 kPa) of active tension force in response to external sti

236 ife scratch and sandpaper abrasion under 570 kPa.

237 th blebbistatin or culture on soft gels (0.6 kPa).

238 fness was around 150 Pa and increased to 1-6 kPa in areas near fibrillar collagen deposition in fibro

239 tients with a reduction in LSM to below 13.6 kPa after SVR still had CSPH.

240 F3, and F=F4 were 8.2 kPa, 9.7 kPa, and 13.6 kPa, respectively.

241 odulus measured by AFM passing from 17 +/- 6 kPa to 56 +/- 18 kPa.

242 3; adolescents, 2.2 kPa +/- 0.2; adults, 2.6 kPa +/- 0.3; analysis of variance, P = .009) and 84 Hz (

243 ) and oxygen saturation (17-206% or 3.6-43.6 kPa).

244 When LS values >5-6 kPa are detected, several aspects should be taken into a

245 variance, P = .009) and 84 Hz (children, 5.6 kPa +/- 0.8; adolescents, 6.5 kPa +/- 1.2; adults, 7.8 k

246 low forces of ~220 nN and pressures of ~5.6 kPa and sustained responses at higher forces of ~360 nN

247 cantly greater than that for P7 shams (691.6 kPa vs 429.2 kPa, p = 0.0194).

248 dimensional shear wave elastography were 9.6 kPa and 10.2 kPa, and for cirrhosis 19.7 kPa and 16.4 kP

249 f unsaturation of e (i), even at VPD above 6 kPa.

250 ves were approximately 3 and approximately 6 kPa, respectively.

251 e fibers with Young's moduli between 40 - 60 kPa was found, compatible with reported physiological ra

252 ilopodia formation, whereas adhesion to a 60-kPa Ecad-Fc PA gel induced Arp2/3-dependent lamellipodia

253 gths, generate stresses of approximately 600 kPa, and produce peak power densities over 2 kW/kg-all e

254 (2); 21.25 kPa (0 k), 16.42 kPa (2 k), 12.63 kPa (4 k) and 9.64 kPa (6 k)).

255 agnosed by liver stiffness greater than 3.63 kPa using magnetic resonance elastography (MRE).

256 with higher pressure (777 kPa +/- 308 vs 630 kPa +/- 252, respectively; P < .001).

257 th tunable wide-range stiffness, from ~10-64 kPa, without affecting the protein mechanics and nanostr

258 ), 16.42 kPa (2 k), 12.63 kPa (4 k) and 9.64 kPa (6 k)).

259 es C) and vapor pressure deficit (VPD, +0.66 kPa), annual percent deviation in precipitation (-38%),

260 o, inducing peak dynamic stress change of 67 kPa on the volcano surface and ~120 kPa in the spherical

261 9.6 kPa and 10.2 kPa, and for cirrhosis 19.7 kPa and 16.4 kPa.

262 softest regions of stiffness gradients (2-7 kPa), with decreased responsiveness on stiff regions of

263 han 50 mm Hg, PaO2 150 +/- 50 torr (20 +/- 7 kPa), PaCO2 40 +/- 5 torr (5 +/- 1 kPa), and core temper

264 for F>=F2, F>=F3, and F=F4 were 8.2 kPa, 9.7 kPa, and 13.6 kPa, respectively.

265 ing transpiration rate up to VPD of nearly 7 kPa, and fully recovered photosynthetic function afterwa

266 as 16.4 (95% confidence interval, 14.4-18.7) kPa (21.5% predicted).

267 The median LSM change was -0.70 kPa (95% confidence interval, -3.0 to +1.7) and was simi

268 cting inflammation and fibrosis) (WMD: -0.70 kPa; 95% CI: -1.00, -0.40 kPa), although analyses showed

269 or compensated cirrhosis (FibroScan, 9.6-70 kPa; cirrhosis, n = 9); median baseline HCV-RNA level wa

270 .29 measured at GRE MR elastography and 2.76 kPa +/- 1.39 at SE-EPI MR elastography (n = 290; P = .15

271 ate was associated with higher pressure (777 kPa +/- 308 vs 630 kPa +/- 252, respectively; P < .001).

272 profiling revealed severe hypoxia and c. 0.8 kPa CO(2) within the root vascular cylinder.

273 percentile of normal liver stiffness was 2.8 kPa.

274 n +/- 1.96 kPa standard deviation of 1.5-2.8 kPa.

275 percutaneous dilatational tracheostomy (45.8 kPa [interquartile range, 36.9-56.5 kPa] vs 57.9 kPa [in

276 Subjects with a FibroScan >6.8 kPa had higher alanine aminotransferase (42 versus 28U/L

277 8; adolescents, 6.5 kPa +/- 1.2; adults, 7.8 kPa +/- 1.2; analysis of variance, P = .0003) but not at

278 offs of 5.0 kPa or less (to exclude) and 8.8 kPa or greater (to confirm) advanced fibrosis had a sens

279 on (3) hepatology consultation for VCTE >= 8 kPa and (4) referral to weight management (WM).

280 6-0.56), 1.35 (0.99-1.89), and 4.9 (3.8-6.8) kPa, respectively.

281 regions of different stiffness (200 and 800 kPa).

282 for P1 MI and sham mice were similar (341.9 kPa vs 363.4 kPa, p = 0.6140), the modulus for P7 MI mic

283 [interquartile range, 36.9-56.5 kPa] vs 57.9 kPa [interquartile range, 45.1-64.2 kPa]; p = 0.019].

284 Tri-cultures at this ratio on 90 kPa substrates exhibited average CVs of 14 +/- 0.6 cm/s,

285 Mean liver stiffness was 2.92 kPa +/- 1.29 measured at GRE MR elastography and 2.76 kP

286 moderate fibrosis (stages F0, F1, F2) (5.93 kPa +/- 2.31 [standard deviation] vs 3.35 kPa +/- 1.44,

287 was used (mean +/- standard deviation, 6.94 kPa +/- 1.42 vs 5.96 kPa +/- 1.31; P = .006).

288 Pa (95% CI: 2.0, 2.2 kPa) with mean +/- 1.96 kPa standard deviation of 1.5-2.8 kPa.

289 tandard deviation, 6.94 kPa +/- 1.42 vs 5.96 kPa +/- 1.31; P = .006).

290 ermeability (from 3.62 to 4.60 g.mm/m(2).day.kPa) and solubility in water (from 14.18 to 25.46%).

291 resolving small pressure changes in the few kPa range, making them unsuitable for applications such

292 ffness measurement (LSM) >/=8.0 kilopascals (kPa) was used as a cutoff suggesting clinically relevant

293 er stiffness measurements (LSM; kiloPascals [kPa]) at ART initiation were categorized as no or minima

294 pressure-to-capacitance sensitivity (114 nF kPa(-1) ), which is at least 1000 times higher than any

295 ting in an extraordinary sensitivity (217 nF kPa(-1) or 24 muC N(-1) at a bias of 1.0 V) without usin

296 tiffness, from hundreds of Pa to hundreds of kPa, T cell metabolic properties and cell cycle progress

297 studies report corneal moduli around tens of kPa, orders-of-magnitude less than those (few MPa) obtai

298 possess high sensitivities (> 10(7) Omega . kPa(-1)), low hysteresis (2.99 +/- 1.37%) over a wide pr

299 p to six orders of magnitude from the GPa to kPa level at a controlled temperature within 28-43 degre

300 pressure with a sensitivity of about 0.044 V kPa(-1) .