ライフサイエンスコーパス: kansasii

1 was greater than smooth "M. canettii" and M. kansasii.

2 as Mycobacterium ulcerans and Mycobacterium kansasii.

3 aded forms of Mycobacterium gordonae from M. kansasii.

4 of differing virulence and by Mycobacterium kansasii.

5 nd 83, 95, 59, and 98%, respectively, for M. kansasii.

6 from three of the patients again revealed M. kansasii.

7 M. avium, 3 M. intracellulare complex, 3 M. kansasii, 4 M. gordonae, and 5 M. chelonae group (all we

8 .35 degrees C (63.27 to 65.42 degrees C); M. kansasii, 59.20 degrees C (58.07 to 60.33 degrees C); M.

9 ium avium-intracellulare complex (83.2%), M. kansasii (7.7%), and M. abscessus (3.4%).

10 evel, with the exception of one strain of M. kansasii (accurately identified but with a low spectral

11 pecies I is the predominant subspecies of M. kansasii among clinical isolates in the United States, a

12 Mycobacterium kansasii, an unusual pathogen in the pre-AIDS era, is in

13 One patient's culture contained both M. kansasii and M. avium, but none of the initial or follow

14 M. kansasii and M. smegmatis also grew very poorly in acidi

15 rarely occur, are genetically related to M. kansasii and morphologically difficult to distinguish.

16 All five isolates of M. kansasii and three of three isolates of M. simiae yielde

17 owing mycobacteria, 100% of patients with M. kansasii, and 0% of patients with M. gordonae.

18 Fourteen patients were treated for M kansasii, and 10 (71%) showed clinical and radiographic

19 acterium avium complex, 4 grew Mycobacterium kansasii, and 2 grew Mycobacterium tuberculosis); 42 iso

20 8% were M. abscessus), 78% (29 of 37) for M. kansasii, and 26% (9 of 35) for M. gordonae.

21 rowing NTM, including 7/7 M. marinum, 7/7 M. kansasii, and 7/11 of other less commonly isolated speci

22 ycobacterial species, M. scrofulaceum and M. kansasii, and eight of the environmental mycobacterial i

23 s Mycobacterium avium complex, Mycobacterium kansasii, and Mycobacterium xenopi among the slowly grow

24 with little to no seroreactivity against M. kansasii- and M. avium subsp. paratuberculosis-infected

25 gainst Mycobacterium avium and Mycobacterium kansasii as well as Mycobacterium tuberculosis.

26 ing three of these patients' isolates and M. kansasii ATCC 12478), and cultures of several other spec

27 rown in liquid medium, whereas Mycobacterium kansasii can be larger and cross-barred.

28 An M. kansasii, DeltaMKAN27435 partially lipooligosaccharide-d

29 ycobacterium tuberculosis, and Mycobacterium kansasii enter macrophages, using the complement recepto

30 On macrophage depletion, we identified M. kansasii forms extracellular cords, resulting in acute i

31 Patients with AIDS and pulmonary M kansasii frequently demonstrate focal alveolar opacities

32 y also distinguished the ESAT-6-secreting M. kansasii from the non-ESAT-6-secreting M. simiae.

33 of 76 (83%) of the specimens positive for M. kansasii, giving sensitivities specificities, positive p

34 Mycobacterium avium complex or Mycobacterium kansasii, half of which were detected by Direct LPA.

35 Persons infected with HIV and M. kansasii have a higher rate of hospitalization and a gre

36 llographic characterization of Mycobacterium kansasii HLP (Mka-HLP) revealed the unexpected presence

37 s, Histoplasma capsulatum, and Mycobacterium kansasii impairs the constitutive production of IL-12 fr

38 Five cultures of M. kansasii (including three of these patients' isolates an

39 erium bovis bacillus Calmette-Guerin, and M. kansasii) induced significantly more AMphi apoptosis tha

40 To date, our knowledge of M. kansasii infection has been hampered owing to the lack o

41 -positive persons, but most patients with M. kansasii infection have clinical and radiologic evidence

42 epidemiology and clinical implications of M. kansasii infection in the AIDS era.

43 hat the zebrafish embryo is permissive to M. kansasii infection, resulting in chronic infection and f

44 Mycobacterium avium complex or Mycobacterium kansasii infection.

45 PIPS from Mycobacterium kansasii is 86% identical to the ortholog from M. tuberc

46 Mycobacterium kansasii is a slow-growing nontuberculous mycobacteria r

47 The fact that a single clone of M. kansasii is responsible for most cases of human disease

48 ied the population genetics of Mycobacterium kansasii isolates from the United States by PCR restrict

49 resistant M. tuberculosis and Mycobacterium kansasii isolates mapped to hadC.

50 One M. fortuitum isolate and one of five M. kansasii isolates were recovered only by the BACTEC 460.

51 Mycobacterium kansasii isolation is more common in HIV-positive person

52 NTM-LD by ICS was highest for Mycobacterium kansasii lung disease.

53 results can occur due to the presence of M. kansasii, M. avium, and possibly other Mycobacterium spe

54 ial species: M. avium, M. intracellulare, M. kansasii, M. chelonae group, M. gordonae, M. xenopi, and

55 of clinically relevant slow growers like M. kansasii, M. szulgai, M. gordonae, and M. asiaticum; how

56 hemerythrin-like protein from Mycobacterium kansasii (Mka HLP) is a member of a distinct class of ox

57 ycobacteria, related to "M. canettii" and M. kansasii, modern M. tuberculosis probably became more hy

58 containing M. bovis (n = 128), Mycobacterium kansasii (n = 10), and Mycobacterium avium subsp. paratu

59 r acid-fast bacilli) that grew Mycobacterium kansasii on culture.

60 with pyrazinamide to include M. avium and M. kansasii, organisms usually not susceptible to pyrazinam

61 the feasibility of zebrafish for studying M. kansasii pathogenesis and for the first time identify ex

62 ients, 16 (17%) satisfied all criteria for M kansasii pulmonary infection.

63 our of seven patients with culture-proven M. kansasii pulmonary infections yielded one or more false-

64 ive identification of M. tuberculosis and M. kansasii, respectively, and as guides for initial probe

65 els a route from environmental Mycobacterium kansasii, through intermediate "Mycobacterium canettii",

66 of M. bovis Bacillus Calmette-Guerin; and M. kansasii to demonstrate detection times greater those ty

67 uld be presumptively treated for pulmonary M kansasii until final culture results are available.

68 Mycobacterium smegmatis and Mycobacterium kansasii were used as models of Mycobacterium tuberculos

69 articipants had pulmonary NTM disease and M. kansasii with a prevalence of 69.2% [95% CI: 63.2-74.7%]

70 resent structures of PIPS from Mycobacterium kansasii with and without evidence of donor and acceptor

71 tients' sputum cultures yielded growth of M. kansasii within 6 to 12 days, and the fifth produced gro