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1 f therapeutic response for many disorders of keratinization.
2 eus (nucleophagy) during the final stages of keratinization.
3 biological and structural events during hair keratinization.
4 efore the onset of corneal opacification and keratinization.
5 13, and 14, to detect presence or absence of keratinization.
6 mately involved in the program of epithelial keratinization.
7 nt recipients caused advanced ocular surface keratinization.
8 V), the most prevalent inherited disorder of keratinization.
9 protein leads to varying degrees of impaired keratinization.
10 dhesion between epidermal cells and abnormal keratinization.
11 of desmosome-keratin filaments, and abnormal keratinization.
12 moglein 3 correlated with different types of keratinization.
13 epithelium with occasional vacuolization and keratinization.
14  epidermal cells (acantholysis) and abnormal keratinization.
15  epidermal cells (acantholysis) and abnormal keratinization.
16 idermal cells (acantholysis) and by abnormal keratinization.
17  morphology, skin and hair pigmentation, and keratinization.
18 evated in patients with severe eyelid margin keratinization.
19 h genes associated with skin development and keratinization.
20 uciformis of Hopf is a localized disorder of keratinization affecting the distal extremities.
21 nce and migration of neutrophils, epithelial keratinization, alterations in corneal wound healing, an
22 n implicated in a few Mendelian disorders of keratinization, although ceramides are known to have key
23 ermis and implicated in several disorders of keratinization and cancer.
24 ostmitotic, maturing cells prior to terminal keratinization and death suggests that (i) PAI-2 may be
25      Functional enrichment analysis revealed keratinization and differentiation as the most affected
26                   In addition, DX314-induced keratinization and epidermal proliferation effects are o
27                             The palmoplantar keratinization and esophageal cancer syndrome, tylosis w
28  and/or IL-36gamma in reducing eyelid margin keratinization and its associated ocular complications i
29 testosterone upregulated pathways related to keratinization and laminin formation on pathway analysis
30 00) is the most common inherited disorder of keratinization and one of the most frequent single-gene
31 -gamma are potent inducers of ocular surface keratinization and SPRR1B expression.
32  demonstrate that inhibition of NRG1 induces keratinization and terminal squamous differentiation of
33  inflammation is a key inducer of pathologic keratinization and that SPRR1B represents an analytical
34 te a clear correlation between eyelid margin keratinization and the expression of IL-36gamma, potenti
35 sses involved in epithelial differentiation, keratinization, and anion exchange.
36 eutrophil activation, cellular cornification/keratinization, and antimicrobial peptide production.
37 ionally enriched for inflammatory responses, keratinization, and cornified envelope formation.
38  proliferation, cell death, differentiation, keratinization, and inflammation, in human meibomian gla
39 ism, cellular response to hydrogen peroxide, keratinization, and keratinocyte differentiation in OTSC
40 raction sockets, wound closure, socket fill, keratinization, and proliferative activities were accele
41 of EMT-like keratinocyte activation, limited keratinization, and slow reepithelialization.
42 thyosis vulgaris-the most common disorder of keratinization-and also a strong genetic risk factor for
43 SAP) is an inherited or sporadic disorder of keratinization associated with germline variations.
44 ds induced extensive squamous metaplasia and keratinization associated with terminal differentiation
45 h poor prognosis of oral cancer patients and keratinization-associated miRNAs mediate deregulation of
46 rich protein with kazal motifs (RECK) by two keratinization-associated miRNAs, miR-7 and miR-21.
47 e directed activated HFSCs towards epidermal keratinization by generating excess reactive oxygen spec
48 y alterations in many gene ontologies (e.g., keratinization, cell cycle, and DNA repair).
49 ted gene clusters included those involved in keratinization, cell division, and metabolism.
50 yosis (BSI) is a rare congenital disorder of keratinization characterized by restriction of scale to
51 ed superficial porokeratosis (DSP) is a rare keratinization disorder of the epidermis.
52 a redefinition of PRP as an autoinflammatory keratinization disorder.
53 d may offer an improved treatment option for keratinization disorders such as congenital ichthyosis a
54 ther study and development in the context of keratinization disorders.
55 erapeutic benefit in the local management of keratinization disorders.
56 pand the heterogeneous spectrum of inherited keratinization disorders.
57                                 Disorders of keratinization (DOK) show marked genotypic and phenotypi
58 files depending on anatomical location, with keratinization dominating the transcriptomic signatures
59 mach has an abnormal phenotype, with reduced keratinization, ectopic mucus cells and columnar epithel
60 ional DD skin shows the presence of abnormal keratinization (epidermal differentiation) and acantholy
61 in-associated proteins [KRTAPs]) involved in keratinization, epidermis development, and the hair cycl
62 eye, it significantly reduced ocular surface keratinization, epitheliopathy, and muco-glycoconjugate
63  1E-8) and were enriched in pathways such as keratinization (FDR p-value = 7.37E-14), chemokine activ
64 nes included epithelial cell differentiation/keratinization genes (Sprr2h, Tgm1) and proinflammatory
65 ay in expression of adhesion/differentiation/keratinization genes at E17.5, a subset of which recover
66 smitter activity, hypoxia-induced processes, keratinization, hormone, thermogenic and immune pathways
67 ovascularization, keratitis, ulceration, and keratinization identifiable from 9 weeks of age; and 3)
68 ng factor to the development of inflammatory keratinization in Notch1(-/-) mice.
69 nce of Dscl and Dsgl correlates with lack of keratinization in ocular epithelia.
70  and telomerase activity, except for foci of keratinization in squamous cell carcinomas.
71                     In areas of trichilemmal keratinization in the follicle, and in cysts arising fro
72  interference with the process of follicular keratinization in the pilosebaceous unit leading to infl
73 ated in an extended spectrum of disorders of keratinization in which thrombocytopenia is also part of
74           Taken together, our data show that keratinization is associated with poor prognosis of oral
75  expression patterns related to processes of keratinization, keratinocyte differentiation, cell proli
76                                 In contrast, keratinization, lipid metabolic processes, and epidermal
77  surface was found to cause inflammation and keratinization of the corneal epithelium.
78 placement, and having adequate thickness and keratinization of the soft tissue were protective factor
79                                              Keratinization of the stratum corneum involves a highly
80 r that the virally transformed cells inhibit keratinization of the tumor cells.
81 her aim was to evaluate if EMD can influence keratinization of tissues utilizing these procedures.
82                                              Keratinization of tumors and the high expression of miR-
83                             No effect on the keratinization or connective tissue organization of the
84 ntages of adnexal structures and a different keratinization pattern than oral mucosa.
85 rgeted preprocessing to enhance vascular and keratinization patterns.
86  S100 calcium-binding proteins, suggest that keratinization plays an important role in the pathogenes
87 n nonkeratinized OCP epithelia is reduced as keratinization proceeds in the disease.
88 of GalNAc-Ts observed in early stages of the keratinization process in patients with OCP suggests a c
89 networks involved in the immune response and keratinization processes revealed potential novel mediat
90 loss of sebaceous glands, and changes to the keratinization program occurred ~16 Ma earlier in cetace
91                                        While keratinization-related processes in glomeruli were sensi
92 atinized epithelia (K1, K2, and K10) and the keratinization-related proteins filaggrin and involucrin
93 ned squamous metaplasia using a biomarker of keratinization, small proline-rich protein 1B.
94                                     At later keratinization stages, the inner mechanical properties o
95                   In areas of epidermal-like keratinization, such as in the infundibulum and in epide
96                           Due to its lack of keratinization, teleost skin possesses living epithelial
97 ontrast, downregulation of genes involved in keratinization was observed throughout treatment in pati