ライフサイエンスコーパス: keratosis

1 9) with a diagnosis of a nevus or seborrheic keratosis.

2 for outpatient encounters related to actinic keratosis.

3 e effects of tirbanibulin therapy on actinic keratosis.

4 ects characteristic of psoriatic and actinic keratosis.

5 racil (5-FU) as an immunotherapy for actinic keratosis.

6 itaria in addition to punctuate palmoplantar keratosis.

7 of this study are not limited to seborrheic keratosis.

8 t of the precancerous skin condition actinic keratosis.

9 inoma (0 vs. 31 [26.1%]; P < .001), verrucal keratosis (0 vs. 79 [66.4%]; P < .001), and Grover disea

10 sis, followed by pterygium (36%) and actinic keratosis (19%).

11 carcinoma, 39 (8.5%) were lichen planus-like keratosis, 21 (4.6%) were melanomas, and 4 (0.9%) were n

12 4 [38.9%], respectively [P = .95]), verrucal keratosis (79 [66.4%] and 26 [72.2%], respectively [P =

13 --changes that resemble hyperplastic actinic keratosis, a commonly observed human precancerous epithe

14 ich is approved for the treatment of actinic keratosis, a precancerous skin condition.

15 in, PTEN levels are reduced in human actinic keratosis, a precancerous skin lesion caused by solar UV

16 In human actinic keratosis, a precursor of squamous cell carcinoma, p16(I

17 estigated as a topical treatment for actinic keratosis, a precursor of squamous-cell carcinoma.

18 0, 2013, and included the search terms solar keratosis, actinic keratosis, photodynamic therapy, and

19 e was no significant association for actinic keratosis (aHR = 0.88, 95% CI = 0.77-1.01).

20 Treatment of actinic keratosis (AK) aims to prevent cutaneous squamous cell c

21 Accurate differentiation between actinic keratosis (AK) and cutaneous squamous cell carcinoma (cS

22 evels, can prevent the occurrence of actinic keratosis (AK) and NMSC.METHODSA human skin/immunodefici

23 Actinic keratosis (AK) are precancerous lesions of the skin whic

24 cinoma (cSCC) after the diagnosis of actinic keratosis (AK) has not been studied during long follow-u

25 Actinic keratosis (AK) is a skin growth induced by UV light expo

26 socioeconomic costs associated with actinic keratosis (AK) management represent major public health

27 ime, 6 months [range, 0-18 months]), actinic keratosis (AK) of the same sun-exposed skin area in the

28 ression from UV-induced precancerous actinic keratosis (AK) to malignant invasive cutaneous squamous

29 lignancies, ranging from a precursor actinic keratosis (AK) to squamous cell carcinoma (SCC) in situ

30 it and harm associated with treating actinic keratosis (AK) with the immune response modifier imiquim

31 Actinic keratosis (AK), a skin growth induced by ultraviolet lig

32 Actinic Keratosis (AK), Intraepidermal Carcinoma (IEC), and Squa

33 s are available for the treatment of actinic keratosis (AK).

34 a distinct precancerous lesion, the actinic keratosis (AK).

35 following: cryotherapy for scattered actinic keratosis (AK); field therapy for AK when grouped in 1 a

36 us cell carcinoma (SCC; n = 28), and actinic keratosis [AK (a precursor to SCC); n = 38].

37 Keratosis also affects other sites such as elbows and kn

38 ly, we evaluated the Raman signal in actinic keratosis and basal cell carcinoma lesions and perilesio

39 es of epidermal carcinogenesis; late actinic keratosis and cSCC.

40 PV viral replication is associated with skin keratosis and cutaneous squamous cell carcinoma.

41 Epidermodysplasia verruciformis keratosis and in some cases actinic keratoses demonstrat

42 lesions including solar lentigo, seborrheic keratosis and lichen planus-like keratosis; dermatofibro

43 in PTEN levels in human premalignant actinic keratosis and malignant SCCs, supporting a key role for

44 ridization; and the expression in seborrheic keratosis and normal skin was very low.

45 The mice exhibited palmoplantar keratosis and progressive alopecia, leading to alopecia

46 n analysis of shotgun metagenomes of actinic keratosis and SCC in healthy skin, revealing the microbi

47 crobial community shifts specific to actinic keratosis and SCC.

48 expression is up-regulated in human actinic keratosis and squamous cell carcinoma.

49 nd show promise for the treatment of actinic keratosis and squamous cell carcinoma.

50 , XPC p53 mutant mice have more severe solar keratosis and suffer accelerated skin cancer compared wi

51 lesions resulting from sun exposure (actinic keratosis), and individual Neandertal alleles were signi

52 nt of a precancerous skin condition (actinic keratosis), and phorbol derivatives such as resiniferato

53 biopsies from 40 healthy donors, 13 actinic keratosis, and 74 cSCC patients.

54 c dysfunction or fibroadiposis, palmoplantar keratosis, and alopecia, resembling the human cardiocuta

55 ectious skin diseases such as genital warts, keratosis, and basal cell carcinoma.

56 ferative diseases such as psoriasis, actinic keratosis, and certain neoplasias.

57 Basal cell carcinoma, solar keratosis, and colorectal cancer were investigated as ne

58 ures include oral leukokeratosis, follicular keratosis, and cysts (steatocysts and pilosebaceous cyst

59 ac arrhythmias and dysfunction, palmoplanter keratosis, and hair abnormalities (cardiocutaneous syndr

60 , which results in skin thinning, wrinkling, keratosis, and malignancy.

61 ronan inhibits basal cell carcinoma, actinic keratosis, and murine colon-26 growth in vivo.

62 or eradicating basal cell carcinoma, actinic keratosis, and squamous cell carcinoma (SCC) in situ by

63 for the treatment of genital warts, actinic keratosis, and superficial basal cell carcinoma.

64 entrations in basal cell carcinomas, actinic keratosis, and their perilesional skin demonstrate a sig

65 osteoarthritis, osteoporosis, and seborrheic keratosis are strongly associated with aging, implying a

66 -induced skin lesions, in particular actinic keratosis, are generally considered as premalignant skin

67 rior to vehicle for the treatment of actinic keratosis at 2 months but was associated with transient

68 eg, seborrheic keratosis, lichen planus-like keratosis, basal cell carcinomas) misclassified as melan

69 nocytic nevus, Basal cell carcinoma, Actinic Keratosis, Benign keratosis, Dermatofibroma, Vascular le

70 1.21, 95% CI 1.17-1.25, p < 0.001, for solar keratosis), but there was no increased risk for colorect

71 , as compared with cSCC in situ, and actinic keratosis by RNA in situ hybridization; and the expressi

72 ell carcinoma (SCC) and premalignant actinic keratosis compared with that in healthy skin to identify

73 to treat (NNT) for one patient to have their keratosis completely cleared after 12-16 weeks was 2.2 (

74 ell and basal-cell carcinoma counts, actinic keratosis counts, or quality-of-life scores were observe

75 any cancer-related end points or in actinic keratosis counts.

76 on; it is an effective treatment for actinic keratosis, cutaneous squamous cell carcinoma in situ, ac

77 al cell carcinoma, Actinic Keratosis, Benign keratosis, Dermatofibroma, Vascular lesion including Squ

78 seborrheic keratosis and lichen planus-like keratosis; dermatofibroma; melanoma; melanocytic nevus;

79 plus 5-FU for the field treatment of actinic keratosis in a randomized, double-blind clinical trial i

80 story of skin cancer, and history of actinic keratosis in both data sets, and male sex and thoracic t

81 PC1 could potentially contribute to abnormal keratosis in DD.

82 rrent knowledge of interventions for actinic keratosis in nonscalp and nonface localizations.

83 y of a new topical field therapy for actinic keratosis, ingenol mebutate gel (0.015% for face and sca

84 Actinic keratosis is a common precursor to sun-related squamous-

85 Actinic keratosis is a precursor to cutaneous squamous cell carc

86 Actinic keratosis is the most frequent premalignant skin disease

87 : Normal epidermis, solar elastosis, actinic keratosis KIN1-2, advanced actinic keratosis KIN3 and we

88 , actinic keratosis KIN1-2, advanced actinic keratosis KIN3 and well-differentiated cSCC.

89 eeth adjacent to the subject's unilateral ST keratosis lesion) to NST-site teeth (contralateral corre

90 Teeth present, ST keratosis lesion, plaque and gingival index, probing dep

91 users, with a unilateral mandibular oral ST keratosis lesion, were recruited.

92 tion of 75% or more in the number of actinic keratosis lesions from baseline to 12 months after the e

93 treatment in patients with multiple actinic keratosis lesions on the head, 5% fluorouracil cream was

94 a clinical diagnosis of five or more actinic keratosis lesions on the head, involving one continuous

95 a and nonmelanocytic lesions (eg, seborrheic keratosis, lichen planus-like keratosis, basal cell carc

96 g palmoplantar carcinoma (MSPC) and familial keratosis lichenoides chronica (FKLC).

97 Seborrheic keratosis-like melanomas can be dermoscopically challeng

98 s are available for the treatment of actinic keratosis located on the face or scalp.

99 carcinoma, squamous cell carcinoma, actinic keratosis, melanocytic nevi, angiokeratoma, dermatofibro

100 Persons who were diagnosed with keratosis, melanosis, Bowen's disease, or squamous cell

101 Nagashima-type palmoplantar keratosis (NPPK) is an autosomal recessive skin disorder

102 ly in invasive SCC nests, but not in actinic keratosis or in situ SCC, compared with normal epidermis

103 Some patients have only palmoplantar keratosis or periodontitis, and in rare individuals the

104 1.7-2.8), and those with a history of solar keratosis (OR, 1.3; 95% CI, 1.1-1.6).

105 The presence of wild type p53 (seborrheic keratosis) or mutant p53 (cutaneous squamous cell carcin

106 ught clinically to be pingueculitis, actinic keratosis, or ocular surface squamous neoplasia (OSSN),

107 r, dermatophytosis, acne rosacea, seborrheic keratosis, or warts; 74.1% of the subjects responded to

108 Papillon-Lefevre syndrome, or keratosis palmoplantaris with periodontopathia (PLS, MIM

109 ed the search terms solar keratosis, actinic keratosis, photodynamic therapy, and photochemotherapy.

110 Keratosis pilaris (KP) is a common skin disorder of foll

111 Although keratosis pilaris (KP) is common, its etiopathogenesis r

112 inearity and in some cases fine-scale and/or keratosis pilaris, whereas homozygotes or compound heter

113 gh arched palate, strabismus, hypotonia, and keratosis pilaris.

114 rised by dry skin, palmar hyperlinearity and keratosis pilaris.

115 he arms and legs, palmar hyperlinearity, and keratosis pilaris.

116 Over 80% had actinic keratosis present, with approximately 30% having 5 or mo

117 Melanomas that clinically mimic seborrheic keratosis (SK) can delay diagnosis and adequate treatmen

118 ified as malignant melanoma (MM), seborrheic keratosis (SK), and benign nevi by a consultant dermatol

119 roliferative skin diseases including actinic keratosis, squamous and basal cell carcinoma as well as

120 oma, and elongated rete ridges in seborrheic keratosis, supporting cross-modal's potential to deliver

121 eel et al. have demonstrated that seborrheic keratosis, the most common of all skin tumors, is depend

122 Evaluation of actinic keratosis therapy depends on reliable measures of the le

123 ogression sequence from precancerous actinic keratosis to invasive cSCC.

124 obial dysbiosis from healthy skin to actinic keratosis to SCC, supporting further investigation of th

125 epidermis, solar elastosis and early actinic keratosis to the 'late' stages of epidermal carcinogenes

126 have limited the efficacy of current actinic keratosis treatments.

127 Palmoplantar keratosis, varying from mild psoriasiform scaly skin to

128 xpression of CD200 in normal skin vs actinic keratosis vs SCC in situ vs invasive SCC.

129 alysis, there was strong evidence that solar keratosis was associated with future PDE5 inhibitor use

130 Actinic keratosis was the most common diagnosis associated with

131 ermatofibroma, solar lentigo, and seborrheic keratosis were measured.