ライフサイエンスコーパス: kidney cell

1 not have been studied using human embryonic kidney cells.

2 sma membrane localization in human embryonic kidney cells.

3 me PCR in BoHV-1 infected Madin-darby bovine kidney cells.

4 ound no definitive evidence of SARS-CoV-2 in kidney cells.

5 in cultured and primary VHL loss-of-function kidney cells.

6 ase I hypersensitivity sites (DHSs) in human kidney cells.

7 alance, particularly for mammalian brain and kidney cells.

8 obtain pure populations of specific types of kidney cells.

9 id cell line derived from rainbow trout head kidney cells.

10 ication machinery in HEK293T human embryonic kidney cells.

11 y of Sindbis virus particles in baby hamster kidney cells.

12 hil-attracting chemokines CXCL1 and CXCL5 in kidney cells.

13 ructures in combination with mouse embryonic kidney cells.

14 inst mammalian HepG2 cells and human primary kidney cells.

15 eys and can be cultivated in vitro in monkey kidney cells.

16 kidney, with much lower expression in other kidney cells.

17 y acquires properties of a sub-population of kidney cells.

18 odulation of cytokine production by resident kidney cells.

19 otein synthesis, uptake, and localization in kidney cells.

20 elial cell colonies using Madin-Darby canine kidney cells.

21 n green monkey kidney and Madin-Darby canine kidney cells.

22 cortex, hippocampus, and cultured embryonic kidney cells.

23 of FH loss in immortalized and primary mouse kidney cells.

24 vated simultaneously in large percentages of kidney cells.

25 lyl cyclase activity by edema toxin in human kidney cells.

26 wth in either immortalized or primary monkey kidney cells.

27 We validated this sensor in human embryonic kidney cells.

28 in vitro were transfected into baby hamster kidney cells.

29 ial cell-cell contacts in Madin-Darby canine kidney cells.

30 nced in CXCR7-transfected Madin-Darby canine kidney cells.

31 ential tropism for salivary gland cells over kidney cells.

32 r-suppressive potential in tumorigenic human kidney cells.

33 rom chickens and mice and Madin-Darby canine kidney cells.

34 at the apical surface of Madin-Darby canine kidney cells.

35 in TLR2- or TLR4-transformed human embryonic kidney cells.

36 -sorting of E-cadherin in Madin-Darby canine kidney cells.

37 a characteristic cytopathic effect in feline kidney cells.

38 s in vivo and of transfected human embryonic kidney cells.

39 ) in induced inflammatory responses of human kidney cells.

40 well as hESC-CMs, but not in human embryonic kidney cells.

41 eral surface of polarized Madin-Darby canine kidney cells.

42 junctional actin ring in Madin-Darby canine kidney cells.

43 s lost in HGPRT-deficient Madin-Darby canine kidney cells.

44 eased ciliary PC2 expression levels in mouse kidney cells.

45 pid remodeling process in Madin-Darby canine kidney cells.

46 active oxygen species (ROS)/PKC signaling in kidney cells.

47 xpression at the gene transcription level in kidney cells.

48 surface of live polarized Madin-Darby canine kidney cells.

49 an epithelial monolayer, Madin-Darby canine kidney cells.

50 the expression of stably integrated genes in kidney cells.

51 cell contact formation in Madin-Darby canine kidney cells.

52 creases ciliary length in Madin-Darby canine kidney cells.

53 phorylated proteins in WT and Itgalpha1-null kidney cells.

54 ese cells comprise a small fraction of total kidney cells.

55 e clinical application of in vitro generated kidney cells.

56 ectly stimulated EGFR expression in cultured kidney cells.

57 were involved in metabolic processes of the kidney cells.

58 .11 muM, respectively, in Madin-Darby canine kidney cells.

59 eractions between epithelial cells and other kidney cells.

60 regulation of immune response genes in human kidney cells.

61 ly, but does prevent virus spread, in feline kidney cells.

62 conductance and membrane abundance in human kidney cells.

63 rfusion injury, as well as in cultured human kidney cells.

64 ctivity in transiently transfected liver and kidney cells.

65 d ability to homodimerize in human embryonic kidney cells.

66 f DNA replication in HEK293T human embryonic kidney cells.

67 a synthetic gene circuit integrated in human kidney cells.

68 ly degraded by proteasome in human embryonic kidney cells.

69 nant in our preparation from human embryonic kidney cells.

70 ithin monolayers of MDCK (Madin Darby canine kidney) cells.

71 GRHL2 in tubulogenesis of Madin-Darby canine kidney cells, a process requiring transient, partial EMT

72 3D cell culture spheroids of primary murine kidney cells after exposure to CDK inhibitors.

73 studied the dynamics of Madine-Darby canine kidney cells after permeabilization by saponin molecules

74 When expressed in human embryonic kidney cells, all three new mutations resulted in a loss

75 her, knockdown of NCX1 in Madin-Darby canine kidney cells alters epithelial morphology and characteri

76 isolated nuclear envelope lipids from human kidney cells, analyzed their composition and determined

77 the autophagy response in different types of kidney cells and across the spectrum of kidney diseases.

78 2XB, P2XC, P2XD, and P2XE in human embryonic kidney cells and altered the ionic and proton environmen

79 This method was applied to human embryonic kidney cells and cardiac tissue lysates to enable the id

80 in A549 cells as well as in human embryonic kidney cells and Chinese hamster ovary cells heterologou

81 d similar growth phenotypes in adult chicken kidney cells and ex vivo tracheal organ cultures.

82 t are capable of differentiation into mature kidney cells and have high potential for regenerative ki

83 ecreased transcriptional activity in patient kidney cells and impaired binding of the transcription f

84 onducted in vitro studies in mouse and human kidney cells and in vivo studies in mice, including wild

85 t HSP90 client proteins in Pkd1(-/-) primary kidney cells and in vivo.

86 eceptor for serum d-binding protein (DBP) in kidney cells and is required for uptake of the 25(OH)D(3

87 sed the rat P2X7 receptor in human embryonic kidney cells and measured membrane currents before and a

88 curs primarily by killing of proximal tubule kidney cells and mechanosensory hair cells, though the m

89 on two different cell lines: human embryonic kidney cells and mouse embryonic fibroblasts.

90 ophotonic assay performed in human embryonic kidney cells and Nicotiana benthamiana leaf cells.

91 t changes in long-term calcium signalling in kidney cells and primary cortical neurons.

92 ay in PKD, transactivated miR-21 promoter in kidney cells and promoted miR-21 expression in cystic ki

93 e recombinantly expressed in human embryonic kidney cells and purified by immobilized metal ion affin

94 ing of protein extracts from human embryonic kidney cells and rat organs reveals that regulatory subu

95 normalized the proliferation rate of primary kidney cells and significantly rescued the disease pheno

96 the cell sources used for the generation of kidney cells and strategies used for transplantation in

97 BK polyomavirus (BKPyV) infection in primary kidney cells and that the upregulated enzyme is active.

98 ds were allowed to adsorb on human embryonic kidney cells and then detached one by one.

99 esis using Tuba knockdown Madin-Darby canine kidney cells and tuba knockdown in zebrafish.

100 ally mutated TRP channels in human embryonic kidney cells and used calcium imaging or whole-cell patc

101 ee cell lines: parental HEK (human embryonic kidney) cells and transfected HEK cells that stably expr

102 Receptors were expressed in human embryonic kidney cells, and disulfide formation was assessed by ob

103 ction in both human salivary gland cells and kidney cells, and expressed viral DNA and Tag protein.

104 xpression systems, COS-7 cells, baby hamster kidney cells, and in VWF-deficient mice through hydrodyn

105 olymorphism was expressed in human embryonic kidney cells, and its effect on phospholemman phosphoryl

106 BCG2, encoding a major urate transporter, in kidney cells, and that HNF4A p.Thr139Ile is a functional

107 opism, including the ability to replicate in kidney cells, and that kidney transduction by SARS-CoV-2

108 n a dual-luciferase assay in human embryonic kidney cells, and they strongly inhibit the infectivity

109 y delayed growth kinetics in primary porcine kidney cells, and they were significantly attenuated in

110 available to diagnose the presence of HIV in kidney cells are complex, the rate of infection is certa

111 In human kidney cells, ARL15 regulated TRPM6-mediated currents.

112 ted with ABCB19 expressed in human embryonic kidney cells as measured by patch-clamp electrophysiolog

113 f cellular CEP290 in primary human and mouse kidney cells as well as in zebrafish embryos leads to en

114 glycine receptor-transfected human embryonic kidney cells at room temperature, and caused internaliza

115 ne as an in vivo readout for ototoxicity and kidney cell-based nephrotoxicity assay, we screened 1280

116 f2 activators were tested in human embryonic kidney cells bearing the Swedish mutation of amyloid pre

117 ied atorvastatin (ATV) to Madin-Darby Canine Kidney cells before infecting them.

118 g to contactin-1-transfected human embryonic kidney cells, binding to paranodes of murine teased fibr

119 ge gaps exist in the roles of these genes in kidney cell biology and renal diseases.

120 ation is present in normal mammary acini and kidney cells but absent in cancerous cells.

121 In WT mouse kidney cells, but not in cells lacking PKA, treatment wi

122 POL1) protein circulates and is localized in kidney cells, but the contribution of APOL1 location to

123 rent has been measured from primary cilia of kidney cells, but the responsible genes have not been id

124 agliflozin prevents ISO-induced apoptosis of kidney cells by inhibiting Bax protein upregulation and

125 study, we isolated primary cilia from mouse kidney cells by using a calcium-shock method and identif

126 The presence of HIV in kidney cells can manifest itself in multiple ways, rangi

127 vein endothelial cells, and human embryonic kidney cells (cell line HEK293).

128 otype 1 virus is severely inhibited in swine kidney cells compared to its translation in rhesus macaq

129 of the putative enhancer element in HEK293T kidney cells; compared to expression of mock-edited cell

130 n of SGLT2 activity also occurred in opossum kidney cells cotransfected with SGLT2 and MAP17.

131 In Madin-Darby canine kidney cell cultures overlaid with human or swine mucus,

132 genesis in Tuba knockdown Madin-Darby canine kidney cell cysts cultured in a collagen gel.

133 f mTORC2, was silenced in Madin-Darby canine kidney cell cysts grown in 3D cultures.

134 term (5.5 days after surgery), Vhl-deficient kidney cells demonstrate both spindle misorientation and

135 tivity of DbpA and YAP in Madin-Darby canine kidney cells depleted either of ZO-1, or one of the rela

136 iminating E1A in transformed human embryonic kidney cells derepressed ~2600 genes, generating a gene

137 Madin-Darby canine kidney cell-derived and Vero cell-derived glycovariants

138 In Myo1c-depleted Madin-Darby canine kidney cells, E-cadherin localization was dis-organized

139 Using human embryonic kidney cells enabled the purification of the TBCD.ARL2.b

140 or formation by mammary epithelial cells and kidney cells engineered to express SV40 early region pro

141 ly higher activation of both human embryonic kidney cells engineered to express TLR2 (HEK-TLR2) and w

142 ats with streptozotocin-induced diabetes and kidney cells exposed to high glucose.

143 brain, cultured neurons, and human embryonic kidney cells expressing GAD65, GAD67, alpha1-subunit of

144 brane currents in individual human embryonic kidney cells expressing rat P2X7 receptors.

145 Kidney cells expressing renin were genetically fate-mapp

146 ssing the receptor and in Madin-Darby canine kidney cells expressing the native receptor.

147 e-cell current recordings in human embryonic kidney cells expressing the wild-type or the mutant Kir2

148 phosphorylated Smad3 and ERK1/2, in primary kidney cells from CDA1 knockout animals.

149 Compared with those from WT mice, primary kidney cells from Cep290-deficient mice exhibited supern

150 During in vitro diabetes, human kidney cells had down-regulation of Tyro3 and Mer mRNA a

151 le galectins expressed in Madin-Darby canine kidney cells had no effect on p75 sorting, suggesting th

152 Mitochondrial dysfunction in kidney cells has been implicated in the pathogenesis of

153 g the actin cytoskeleton in human epithelial kidney cells (HEK 293) and rat vascular smooth muscle ce

154 strategy over an image data set of embryonic kidney cells (HEK 293T) from multiple experiments.

155 al melanoma cells (MEL 270), human embryonic kidney cells (HEK) and breast cancer cells (MCF7) we sho

156 e goal of transfection using human embryonic kidney cells (HEK-293) stained with Calcein as a model.

157 re, lipopeptides DCS1 are not toxic to human kidney cells HEK293 up to a concentration of 250 ug/ml.

158 Monocytes, Leukocytes erythrocytes and human kidney cells HEK293), animal cells (neuroblastoma N115 a

159 n our setup, drug binding to human embryonic kidney cell (HEK293) homogenate was measured in a small-

160 In kidney cell (HEK293-T) culture, the GAP activity toward

161 tantial protein knockdown in human embryonic kidney cells (HEK293) and hard-to-transfect primary huma

162 Rings of human embryonic kidney cells (HEK293) and tracheal smooth muscle cells (

163 ium was detected in cultured human embryonic kidney cells (HEK293) expressing heterologous ZIP8-Ala39

164 expression of QKI mutants in human embryonic kidney cells (HEK293) significantly decreased the abunda

165 uantify TRbeta antagonism in human embryonic kidney cells (HEK293/17) at concentrations ranging from

166 from mitochondria in lysates human embryonic kidney cells HEK293T.

167 n contrast to monolayered Madin-Darby canine kidney cells, hepatocytic epithelial cells, which typica

168 sed whole-cell recordings of human embryonic kidney cells heterologously expressing either wild-type

169 lticellular monolayers of Madin-Darby canine kidney cells, highlighting its acuity in reconstructing

170 Human-pluripotent-stem-cell-derived kidney cells (hPSC-KCs) have important potential for dis

171 lts were also observed in primary rat embryo kidney cells, human fibroblasts, and human respiratory t

172 in situ in murine kidney slices and in 786-O kidney cells in culture as determined by reverse transcr

173 Upregulation of autophagy protected kidney cells in culture from oxidative stress and reduce

174 Kidney cells in HIV transgenic mice and HIV-infected pod

175 idney tumor cells give rise to nonneoplastic kidney cells in mice, proving that they have not undergo

176 g organotypic cultures of Madin-Darby canine kidney cells in reconstituted basement membrane, we show

177 as replenishable sources for replacement of kidney cells in the setting of human disease.

178 altered as part of the adaptive response of kidney cells, in a process that is tightly regulated by

179 al gene expression in infected primary chick kidney cells indicated that the host cell response to IB

180 les derived from transfected human embryonic kidney cells induced a significant neutrophil chemotacti

181 Knockdown of NCX1 in Madin-Darby canine kidney cells induced fibroblastic morphology, increased

182 DSTYK knockdown in human embryonic kidney cells inhibited FGF-stimulated phosphorylation of

183 Diabetes-induced kidney cell injury involves an increase in matrix protei

184 uces the malignant SN12C, but not benign HK2 kidney cell invasion.

185 Basal autophagy in kidney cells is essential for the maintenance of kidney

186 underwent additional testing against primary kidney cells isolated from human kidneys to better predi

187 alcium oxalate binding and toxicity in human kidney cells, it may provide a different therapeutic app

188 In HL-1 cardiac cells and human embryonic kidney cells, KCNE5 and K(V)2.1 colocalized at the cell

189 Using primary kidney cells lacking a catalytic subunit of Cn (CnAalpha

190 sette transporter A1-expressing baby hamster kidney cells leads to formation of two populations of FC

191 r localization in MDCKII (Madin-Darby canine kidney (cell line)) cells and in mouse liver, and tested

192 R-based R2 relaxation measurements of monkey kidney cell line (CV1) fibroblasts that overexpress FHC,

193 s well in the nontumorigenic human embryonic kidney cell line (HEK-293T), showing in some cases impor

194 CF7) and in a nontumorigenic human embryonic kidney cell line (HEK-293T).

195 n hERG channels expressed in human embryonic kidney cell line 293 (HEK293) cells and I(Kr) in isolate

196 y-based safety testing using human embryonic kidney cell line 293 cells expressing human ether-a-go-g

197 ology in Xenopus oocytes and human embryonic kidney cell line 293 cells in which we coexpressed rat C

198 itment of beta-arrestin 2 in human embryonic kidney cell line 293 cells monitored with confocal and t

199 ncer (TOV112D) and noncancer human embryonic kidney cell line 293 to 15.8 and 18.1 nM, respectively,

200 zation via endogenous M3R in human embryonic kidney cell line 293T (HEK293T) or mouse insulinoma (MIN

201 Utilizing the human embryonic kidney cell line 293T, it was possible to demonstrate th

202 ation and infectious viral burdens in a frog kidney cell line and in tadpoles.

203 n of human 293H cells, Crandall Renal Feline Kidney cell line and primary feline peripheral blood mon

204 show low cytotoxicity in the nontumorigenic kidney cell line BGM and therefore high selectivity fact

205 o be optically analyzed in CRL-2794, a human kidney cell line expressing an unstable green fluorescen

206 lobacter strains towards the human embryonic kidney cell line HEK 293 was not impacted by the T6SS.

207 However, by utilizing the human embryonic kidney cell line HEK293T, it was possible to demonstrate

208 homeostasis of hMSH4 in the human embryonic kidney cell line HEK293T.

209 ing the minor allele of rs9315202 in a human kidney cell line HK-2 genomic DNA resulted in a change i

210 demonstrate that overexpression of OCT4 in a kidney cell line is sufficient for signal-dependent acti

211 Overexpression in a human kidney cell line showed that wild-type PCBD1 binds HNF1B

212 al receptor for FMDV, we transduced a bovine kidney cell line to stably express both the alphaV and b

213 from tubulin purified from a human embryonic kidney cell line with isoform composition characteristic

214 lectivity was demonstrated in a green monkey kidney cell line, CV-1, in which CITCO displayed >100-fo

215 ted intracellular vesicle acidification in a kidney cell line, providing validation for the utility o

216 rated a stably transfected BHK (baby hamster kidney) cell line that expresses a moderate level of myc

217 e and 2 pM against HEK 293T (human embryonic kidney) cell line], and a set of valuable structure-acti

218 L, KB, BT-549, SK-OV-3) and two noncancerous kidney cell lines (LLC-PK1 and Vero).

219 blastoma cell line but also affected control kidney cell lines and the sarcoma cells; only podocytes

220 esis, we generated stable Madin-Darby canine kidney cell lines depleted of both ZO-1 and -2.

221 ty studies were performed on human embryonic kidney cell lines expressing human P2X7R (HEK293-hP2X7R)

222 (FRET) in stably expressing human embryonic kidney cell lines.

223 infections and syncytium formation in monkey kidney cell lines.

224 marker genes [TPM threshold =15]), with pig kidney cells (LLC-PK1) close behind (39%).

225 on of NCX1 by KB-R7943 in Madin-Darby canine kidney cells, LLC-PK1, and human primary renal epithelia

226 pproach also identifies native CAII in human kidney cell lysate as an AEBSA target.

227 Lipidomic analysis of Madin-Darby canine kidney cell membranes and of the corresponding detergent

228 this method to measure ECM accumulation in a kidney cell model, we demonstrated good agreement with e

229 ontaneous prion formation in the RK13 rabbit kidney cell model.

230 Tension across Madin-Darby canine kidney cell monolayers was increased by a low level of u

231 nese Hamster Ovary cells and Human Embrionic Kidney cells on two polyelectrolytes that are widely use

232 ructures in combination with embryonic mouse kidney cells over a period of 18 d.

233 y-derived virus had preferential tropism for kidney cells over salivary gland cells.

234 ence for 3 of these sites in human embryonic kidney cells over-expressing BRAF as well as further com

235 ylation arrays, we show that human embryonal kidney cells over-expressing WT1 acquire DNA methylation

236 F4/80(+) and CD11c(+) kidney cells phagocytize uromodulin.

237 Expression of cyclin E1 in human embryonic kidney cells prevents Cdk5-mediated phosphorylation of K

238 insight into the role of FLCN in regulating kidney cell proliferation and facilitate the development

239 the function of wild-type FLCN in regulating kidney cell proliferation and, therefore, act as an onco

240 nd K508R missense mutations promote aberrant kidney cell proliferation leading to pathogenicity, we g

241 nd K508R missense mutations promote aberrant kidney cell proliferation, but to different degrees.

242 ypes observed in Lgr4KO intestines, impaired kidney cell proliferation, reduced epidermal thickness,

243 ophils and 5-LOX-transfected human embryonic kidney cells, propofol attenuated the production of 5-LO

244 overexpression of viperin in human embryonic kidney cells reduces the intracellular rate of accumulat

245 ATR was inhibited in BKPyV-infected primary kidney cells, severe DNA damage occurred due to prematur

246 Complementary in vitro studies in normal rat kidney cells showed that dh404 significantly upregulates

247 hMATE1 double-transfected Madin-Darby canine kidney cells showed that mIBG transport in the basal (B)

248 In human embryonic kidney cells stably expressing a Flag-tagged version of

249 Notably, Madin-Darby canine kidney cells stably expressing apically mistrafficked ER

250 In Madin-Darby canine kidney cells stably expressing epitope-tagged alphabetag

251 5 and basolateral VSVG in Madin-Darby canine kidney cells still undergo progressive sorting after the

252 linked to dysregulated signaling via PKC in kidney cells such as podocytes.

253 , or the APOL1 G2 variant in human embryonic kidney cells (T-REx-293) using a tetracycline-mediated (

254 receptors were expressed in human embryonic kidney cells, taurine and AL34662, a non-specific 5-HT(2

255 ve lipid nanodiscs derived from baby hamster kidney cells, that G12V-KRAS samples three conformationa

256 In human embryonic kidney cells, the R70C mutation prevented phospholemman

257 Of the diversity of ion channels in kidney cells, the transient receptor potential (TRP) sup

258 ic growth of benign MDCK (Madin Darby Canine Kidney) cells through effects on the Rho-like GTPase cdc

259 bserve dynamic variations of human embryonic kidney cells, through a silicon substrate, in response t

260 we reduced the percentage of Pkd1-deficient kidney cells to 8%.

261 sed Biotin Identification in human embryonic kidney cells to identify proximity interaction partners

262 variants proliferated in Madin-Darby canine kidney cells to nearly the degree as WT PR8.

263 al preparations ranging from human embryonic kidney cells to neurons in culture, slices, and in vivo.

264 tory sensory neurones and in human embryonic kidney cells, together with electrophysiological recordi

265 s in polarized epithelial Madin-Darby canine kidney cell transfectants.

266 alysis of RNA extracted from human embryonic kidney cells transfected with exon trapping constructs.

267 ed transport studies with Madin-Darby canine kidney cells transfected with human MDR1 gene (MDCK/MDR1

268 Madin-Darby canine kidney cells transiently transfected with meprin alpha o

269 of PRDX2 is also observed in human embryonic kidney cells treated with TNF-alpha.

270 ional activation of proinflammatory genes in kidney cells treated with TNFalpha or infected by HIV.

271 xpansion, and differentiation of appropriate kidney cell types and the integration of these cells int

272 This process can be applied to other kidney cell types and will enhance our understanding of

273 , enabling isolation and characterization of kidney cell types of interest.

274 all three platforms captured a diversity of kidney cell types that were not represented in the scRNA

275 art, aorta and tibial arteries, and multiple kidney cell types.

276 single-base resolution of Madin-Darby canine kidney cells undergoing EMT and translated the identifie

277 signaling in cilia of mouse fibroblasts and kidney cells upon chemical or mechanical stimulation wit

278 the global kinome of HEK293T human embryonic kidney cells upon treatment with methylglyoxal, a glycol

279 on) and nitric oxide-induced human embryonic kidney cell using 2 labeling reagents: the cysteine-reac

280 dynamics directly in living human embryonic kidney cells using fluorescence fluctuation spectroscopy

281 regation domains, in live Madin-Darby canine kidney cells using spinning disk confocal microscopy.

282 e siRNA internalization into non-tumorigenic kidney cells was negligible with all fatty acyl-peptide

283 moregulation of the mouse Cln3 mRNA level in kidney cells was recently reported.

284 2R and the MC1R variants are present in head kidney cells, we hypothesized that MC2R activity is modu

285 s expression in polarized Madin-Darby canine kidney cells, we show that beta2 is N-glycosylated in vi

286 rsor protein in vitro and in human embryonic kidney cells, we show that gamma-secretase is a very slo

287 tantly, using experiments in human embryonic kidney cells, we show that specific parameters of the sy

288 zation with GluN1-expressing human embryonic kidney cells were confirmed to be against the NMDAR.

289 o-go-related gene expressing human embryonic kidney cells were used as controls.

290 eral surface in polarized Madin-Darby canine kidney cells, whereas in double tyrosine mutant, it was

291 sents a novel function for these specialized kidney cells, which are best known for their role in mod

292 eight concatenated cDNAs in human embryonic kidney cells, which encoded three serially joined, epito

293 contrast, in primary cultures of human fetal kidney cells, which maintain WNT activation and more clo

294 multaneous transfection of HEK-293 embryonic kidney cells with a plasmid vector containing a fluoresc

295 protein (GFP) expression of human embryonic kidney cells with efficiency up to 90%.

296 hat reduced the viability of Pkd1-null mouse kidney cells with minimal effects on wild-type cells.

297 es in cells with hyperactive mTORC1, such as kidney cells with mutations in the tumor suppressor gene

298 Madin-Darby canine kidney cells with Na,K-beta knockdown have reduced NCX1

299 /2007 (H1N1) was grown in Madin-Darby canine kidney cells with or without escalating concentrations o

300 Human kidney cells with Polbeta knockout (KO) had higher endog