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1 tial intervention to fragment or remove or a kidney stone.
2 trolling an ingestible camera or expelling a kidney stone.
3 associated with a lower risk of developing a kidney stone.
4 OM), the most prominent constituent of human kidney stones.
5 tly associated with a lower risk of incident kidney stones.
6 5 participants, 19,678 reported a history of kidney stones.
7 critical step in the pathogenesis of cystine kidney stones.
8 M crystal attachment and the pathogenesis of kidney stones.
9 roduced in soil and is a common component of kidney stones.
10 cognized as the cause of gouty arthritis and kidney stones.
11 yr of follow-up, we documented 5645 incident kidney stones.
12 ction of oxalate results in the formation of kidney stones.
13 on pathophysiology and medical treatment of kidney stones.
14 duce hip fracture, and increased the risk of kidney stones.
15 leads to formation of calcium oxalate (CaOx) kidney stones.
16 se, forming the major inorganic component of kidney stones.
17 ays an important role in the pathogenesis of kidney stones.
18 ion of crystals that might eventually become kidney stones.
19 d between menopause and PMH use and incident kidney stones.
20 n menopause and PMH use and risk of incident kidney stones.
21 to be a critical step in the development of kidney stones.
22 sk for the development of calcium-containing kidney stones.
23 terial plaque formation and the formation of kidney stones.
24 a cohort of 85,557 women with no history of kidney stones.
25 All 20 patients with symptoms had kidney stones.
26 5 years of age in 1986 and had no history of kidney stones.
27 es including myotonias, cystic fibrosis, and kidney stones.
28 ain outcome measure was incident symptomatic kidney stones.
29 n among individuals with a family history of kidney stones.
30 9 years of age in 1980 and had no history of kidney stones.
31 n, 40-75 years of age, who had no history of kidney stones.
32 ther renal tubular disorders associated with kidney stones.
33 s, who account for > 80% of new diagnoses of kidney stones.
34 d have been warranted because of undiagnosed kidney stones.
35 of thiazide diuretics for the prevention of kidney stones.
36 or clinical diagnosis and early screening of kidney stones.
37 ations with CKD stages and complications and kidney stones.
38 formigenes colonization reduces the risk for kidney stones.
39 linical importance in the absence of gout or kidney stones.
40 sociate significantly with increased risk of kidney stones.
41 tribution to protection from calcium oxalate kidney stones.
42 gut and degrade oxalate, a component of most kidney stones.
43 ed regularly, can lead to the development of kidney stones.
44 come symptomatic, resulting in bone loss and kidney stones.
45 s may be useful to prevent the recurrence of kidney stones.
46 n D supplementation did not increase risk of kidney stones.
47 d prevalence of calcium phosphate-containing kidney stones.
48 pected focal liver lesions, lung nodules, or kidney stones.
49 date genes in 348 unrelated individuals with kidney stones.
50 epair extends the lifespan of flies carrying kidney stones.
51 e effects: hypercalcemia, hypercalciuria, or kidney stones.
52 o unrelated individuals with calcium oxalate kidney stones.
53 , P=4.1 x 10(-5)) associating with recurrent kidney stones.
54 ehensively evaluate patients presenting with kidney stones.
56 ithotripsy for children and adolescents with kidney stones 20 mm or larger, without mention of ureter
59 were used to estimate the odds ratio (OR) of kidney stones adjusted for body mass index; hypertension
63 tus include malabsorption, dumping syndrome, kidney stones, altered intestinal bile acid availability
67 single-phase unenhanced CT for evaluation of kidney stones and associated RadLex(R) Playbook identifi
68 Previous studies of the association between kidney stones and CHD have often not controlled for impo
70 T) deficiency is a rare, hereditary cause of kidney stones and chronic kidney disease (CKD) which is
71 T) deficiency is a rare, hereditary cause of kidney stones and chronic kidney disease (CKD), characte
72 calciuria is the most common risk factor for kidney stones and has a recognized familial component.
73 calciuria is the most common risk factor for kidney stones and has a substantial genetic component.
74 Hyperoxaluria is a major risk factor for kidney stones and has no specific therapy, although Oxal
75 s uniformly form calcium phosphate (apatite) kidney stones and have been termed genetic hypercalciuri
77 lcium was inversely associated with risk for kidney stones and intake of supplemental calcium was pos
78 d diagnostic codes to determine incidence of kidney stones and presence of comorbidities (CKD, hypert
79 al treatments are to eliminate the burden of kidney stones and prevent recurrence while simultaneousl
81 le to the clinician caring for patients with kidney stones and to the scientist interested in their c
84 ry citrate increases the risk for developing kidney stones, and elevation of luminal succinate in the
85 te is the predominant component in 70-80% of kidney stones, and small changes in urinary oxalate conc
86 se in total fluid intake can reduce risk for kidney stones, and the choice of beverage may be meaning
96 long-term catheterization, forms bladder and kidney stones as a consequence of urease-mediated urea h
97 ctive of this study was to determine whether kidney stones associate with an increased risk for MI.
98 le clinical presentation, high recurrence of kidney stones associated with abnormalities of metabolis
99 American women and 12% of men will develop a kidney stone at some time in their life, and prevalence
100 identify sequence variants associating with kidney stones at ALPL (rs1256328[T], odds ratio (OR)=1.2
102 ions that are caused by biofilms--infectious kidney stones, bacterial endocarditis, and cystic fibros
104 pansion of the present-day southeastern U.S. kidney stone "belt." The fraction of the U.S. population
105 ul interpretation of changes in radiographic kidney stone burden requires understanding how radiograp
106 o play an important role in the formation of kidney stones, but data on the risk factors for stone fo
107 lays an essential role in the development of kidney stones by allowing small crystals to be retained
108 average radiation dose for CT evaluation for kidney stones by querying a national dose registry.
110 ,636 Icelanders that were imputed into 5,419 kidney stone cases, including 2,172 cases with a history
111 is, and these mutations explain about 15% of kidney stone cases, suggesting that additional nephrolit
115 at high risk may benefit from a specialized kidney stone clinic staffed by a pediatric nephrologist,
118 mation on self-reported, physician-diagnosed kidney stones collected from 1,167,009 men and women, ag
121 ype to phenotype in 355 patients in the Rare Kidney Stone Consortium PH registry and calculated preva
122 in the APRT Deficiency Registry of the Rare Kidney Stone Consortium, 2 from Westmead Hospital in Syd
123 18 years, which included 44 individuals with kidney stones containing >=50% calcium oxalate and 44 co
124 lts Three hundred four study descriptors for kidney stone CT corresponding to data from 328 facilitie
127 ake, and body mass index (BMI) with incident kidney stone development was evaluated after adjustment
128 dy size affect the relation between diet and kidney stones, dietary recommendations for stone prevent
131 ting a path toward a better understanding of kidney stone disease and the eventual design of therapeu
132 the microbiomes of the host could influence kidney stone disease at multiple levels, including intes
133 To emphasize an exploration of mechanisms of kidney stone disease based on a molecular understanding
134 a Type 1 (PH1) is a rare autosomal recessive kidney stone disease caused by deficiency of the peroxis
135 Primary hyperoxaluria type I is a severe kidney stone disease caused by mutations in the protein
142 ich occurs in the hereditary calcium oxalate kidney stone disease primary hyperoxaluria type 1 (PH1).
143 nsible for the potentially lethal hereditary kidney stone disease primary hyperoxaluria type 1 (PH1).
145 d information on diet, menopause status, and kidney stone disease were used to examine the independen
146 ses the risk for hyperoxaluria and recurrent kidney stone disease, and that replacement therapy is an
147 s of AGT, deficiency of which results in the kidney stone disease, primary hyperoxaluria type I, iden
153 course, and prognosis for genetic causes of kidney stone diseases has been made available to the cli
154 t, and the possible presence of rare genetic kidney stone diseases would require physicians to compre
155 ies suggest that the prevalence of monogenic kidney stone disorders, including renal tubular acidosis
156 there was no increased risk of a symptomatic kidney stone during the 1-year period after antibiotic u
157 ing kidney stones are by far the most common kidney stones encountered in clinical practice, and thus
158 A prediction tool for the risk of a second kidney stone episode is needed to optimize treatment str
160 correlation between the number of recurrent kidney stone episodes and the lack of O. formigenes colo
164 rticipants provided information about recent kidney stone events in regular questionnaires sent to th
166 al metabolites were associated with incident kidney stone formation at prespecified levels of metabol
167 ex hormones on urinary oxalate excretion and kidney stone formation in an experimental model of uroli
171 FINDINGS: There is increasing evidence that kidney stone formation is associated with a number of sy
173 lleles had a significantly increased risk of kidney stone formation or medullary nephrocalcinosis, na
175 relation between family history and risk of kidney stone formation was studied in a cohort of 37,999
176 weight, and body mass index) and the risk of kidney stone formation was studied in two large cohorts:
179 onents have been associated with the risk of kidney stone formation, but there is limited evidence re
180 s of eGFR on overall and cause-specific CKD, kidney stone formation, diastolic blood pressure and hyp
194 currence of Kidney Stone nomogram identifies kidney stone formers at greatest risk for a second sympt
195 oluble oxalate in foods is major concern for kidney stone formers due to its tendency to increase uri
197 xa identified as decreased in those who were kidney stone formers were components of a larger abundan
198 1247 chart-validated first-time symptomatic kidney stone formers with a documented obstructing or pa
202 eteroscopy or pyeloscopy can safely render a kidney-stone free prior to transplantation and in living
203 ho sought medical evaluation or treatment of kidney stones from 2005-2011 in the U.S. cities of Atlan
204 arly explained by mutations in 1 of 30 known kidney stone genes, we conducted a high-throughput mutat
205 mong women, those with a reported history of kidney stones had an increased risk of CHD than those wi
206 reduced-radiation dose CT for evaluation of kidney stones has increased since 2011-2012, but remains
208 cant difference was shown for conspicuity of kidney stones in 22 patients who underwent CT with z-axi
210 ferent beverages and the risk of symptomatic kidney stones in a cohort of 45,289 men, 40-75 years of
211 ion was found between menopause and incident kidney stones in age-adjusted (relative risk [RR], 1.07;
213 ium thiosulfate reduces formation of calcium kidney stones in humans, but this has not been establish
214 actors and the risk of incident, symptomatic kidney stones in men and to determine whether these asso
216 risk of CHD than those without a history of kidney stones in NHS I (incidence rate [IR], 754 vs 514
217 cal activity may reduce the risk of incident kidney stones in postmenopausal women independent of cal
219 ation between a DASH-style diet and incident kidney stones in the Health Professionals Follow-up Stud
220 e whether geographic variability in rates of kidney stones in the United States was attributable to d
221 ociated with hypercalciuric nephrolithiasis (kidney stones) in the Northern European and Japanese pop
222 cifically hypercalcemia, hypercalciuria, and kidney stones, in participants who were given vitamin D
223 s influence the formation of calcium oxalate kidney stones, including gender, diet, and urinary excre
224 e computed tomography (CT) for evaluation of kidney stones increased in 2015-2016 compared with that
227 the hypothesis that the rising incidence of kidney stones is associated with the progressive loss of
234 calcium-oxalate crystal formation leading to kidney stones, nephrocalcinosis, and ultimately kidney f
238 mics and SCFAs in 153 fecal samples from non-kidney stone (NS) controls, patients with occasional ren
246 erably lower in the gut microbiota among the kidney stone patients compared with the NS controls.
247 ociations between mean daily temperature and kidney stone presentation according to lag time and temp
248 ur cities, the strongest association between kidney stone presentation and a daily mean temperature o
249 dels, we estimated the relative risk (RR) of kidney stone presentation associated with mean daily tem
251 ociations between mean daily temperature and kidney stone presentation were not monotonic, and there
256 eported isolation of nanobacteria from human kidney stones raises the intriguing possibility that the
257 des should be useful in reducing the risk of kidney stone recurrence in patients with Dent's disease.
265 ons of recurrence, we used the Recurrence of Kidney Stone (ROKS) score, which sums multiple baseline
267 ese results suggest that a family history of kidney stones substantially increases the risk of stone
268 ntly in individuals with a family history of kidney stones than in those without a family history; ho
269 ectopic biomineralization of calcium oxalate kidney stones, the competition between calcium oxalate m
271 ed initial solid phase in patients with CaOx kidney stones, the reduction in supersaturation with res
272 tinely recommended for patients who have had kidney stones to decrease the likelihood of recurrence.
274 ing have important roles in the aetiology of kidney stones: transporters and channels; ions, protons
276 also observe associations of the identified kidney stone variants with biochemical traits in a large
277 MED score category, the risk of developing a kidney stone was between 13% and 41% lower compared with
278 Among the 2 cohorts of women, a history of kidney stones was associated with a modest but statistic
279 consumption of caffeine and the incidence of kidney stones was collected by validated questionnaires.
280 pleted a 24-h urine collection, the risk for kidney stones was directly proportional to urinary oxala
282 n the type of menopause and risk of incident kidney stones was examined, surgical menopause was assoc
284 on risk of hypercalcemia, hypercalciuria, or kidney stones was not modified by baseline 25-hydroxyvit
285 H score, the multivariate relative risks for kidney stones were 0.55 (95% CI, 0.46 to 0.65) for men,
294 itamins B6 and C and the risk of symptomatic kidney stones were prospectively studied in a cohort of
296 ho underwent parathyroidectomy had recurrent kidney stones, whereas 6 of the 8 patients who did not u
297 ium appears to decrease risk for symptomatic kidney stones, whereas intake of supplemental calcium ma
298 ore than 90% of procedural interventions for kidney stones, which affect 1 in 11 persons in the Unite
299 g of numerous human renal conditions such as kidney stones, while the hindgut provides an outstanding