ライフサイエンスコーパス: killer

1 s in Alpine silver ants is a maternal effect killer.

2 dification of GluK2/3 with the human natural killer-1 (HNK-1) glycan, a modulator of GluK2/3 function

3 r apoptosis with Bcl-2 homologous antagonist/killer activation in vitro and in vivo in primary sclero

4 t increases in proliferating (Ki67+) natural killer and CD8 T cells.

5 NKG7 function in natural killer and CD8(+) T cells was linked with their ability

6 and functions of tumour-infiltrating natural-killer and CD8(+) T lymphocytes.

7 celerated in SZ related to antitumor natural killer and CD8T cells, which may help explain conflictin

8 al kin as in some whale societies, including killer and sperm whales.

9 of mice show increased lung B cell, natural killer and T cell effector responses in the lung upon in

10 ogether with a loss in B lymphocyte, natural killer and T cell effector responses.

11 chimeric antigen receptor-expressing natural killer and T cells, the overall survival of patients wit

12 ; T), which differentially influence natural killer and T-cell alloresponses.

13 %; P = .03) but similar levels of B, natural killer, and CD4 T lymphocytes.

14 nfiltration of F4/80(+) macrophages, natural killer, and CD8 T cells, displaying a central memory phe

15 promoted the activation of CD8(+) T, natural killer, and dendritic cells in relevant tissues; and fac

16 n G179P was transduced into BAX/BCL2 agonist killer (BAK) double-knockout mouse embryonic fibroblasts

17 apsing bacteremia and found that a persister killer, but not a persister formation inhibitor, could p

18 ocytosis, cellular cytotoxicity, and natural killer cell activation were assessed.

19 gocytosis, complement activation and natural killer cell activation, are substantially enhanced by a

20 Natural killer cell deficiencies (NKDs) are an emerging phenotyp

21 Human natural killer cell deficiency (NKD) arises from inborn errors o

22 aint in macrometastases triggered by natural killer cell depletion suggests a dynamic interplay betwe

23 phenotype with a T-cell, B-cell, and natural killer cell developmental defect and hypogammaglobulinem

24 ules that seek to subvert T cell and natural killer cell function via a remarkable array of mechanism

25 inflammation, T(H)17 signaling, and natural killer cell function.

26 Here, we show that natural killer cell granule protein 7 (NKG7) is a regulator of l

27 ptor genes encoded within the NK complex and killer cell Ig-like receptor genes encoded within the le

28 The killer cell Ig-like receptors (KIR) modulate immune resp

29 cognition of these HLA-B57 allomorphs by the killer cell immunoglobulin-like receptor (KIR) 3DL1 was

30 uch as the human leukocyte antigen (HLA) and killer cell immunoglobulin-like receptor (KIR) regions.

31 gulate immune functions and are modulated by killer cell immunoglobulin-like receptors (KIR).

32 killer (NK) cell reactivity mediated through killer cell immunoglobulin-like receptors (KIRs) could r

33 ward infected or tumor cells is regulated by killer cell immunoglobulin-like receptors (KIRs) that bi

34 adults; for example, increased expression of killer cell lectin-like receptor B1 (KLRB1; CD161) 28 da

35 Further, we examined killer cell lectin-like receptor G1 (KLRG1) as a marker

36 nt CD4+ and CD8+ T cells expressing CD57 and killer cell lectin-like receptor G1 (KLRG1), which was n

37 ontext, supporting the hypothesis of natural killer cell mechanosensitivity.

38 on of innate immunity in infants and natural killer cell networks in children, and additionally demon

39 Furthermore, a shift in the natural killer cell phenotype was observed with features suggest

40 3-like ILCs was not dependent on the natural killer cell receptor (NCR1), since NCR1-deficient mice s

41 Killer cell receptor genes encoded within the NK complex

42 h CMVs evade or reprogram T cell and natural killer cell responses in vivo However, the role of humor

43 137 MAb that can activate T cell and natural killer cell responses to clear tumors.

44 oupled with T-helper cell type 2 and natural killer cell signaling in children.

45 ith an additional role of T(H)17 and natural killer cell signaling.

46 The mechanisms underlying killer cell's navigation are not well understood.

47 lar lavage samples and repression of natural killer cell- and T cell-associated transcripts in periph

48 long with CRISPR/Cas9-KO cell lines, natural killer cell-killing assays, and RNA-Seq experiments, we

49 domain attenuated Necl-5 binding and natural killer cell-mediated cytotoxicity.

50 1 cells than F1 cells was reduced by natural killer-cell depletion.

51 Killer-cell granzyme B also activates caspase-independen

52 cyte immunoglobulin-like receptor (LILR) and killer-cell immunoglobulin-like receptor (KIR).

53 n contrast, dNK1 express receptors including Killer-cell Immunoglobulin-like Receptors (KIR), indicat

54 Additionally, these NKG2C NK cells expressed killer-cell immunoglobulin-like receptors distinct from

55 targets deployed in T cells (CAR-T), natural killer cells (CAR-NK) or mixtures (CAR-NK/T) from over 5

56 eloid DCs; neutrophils; macrophages; natural killer cells (NK); Marginal Zone-like B cells (MZB); gam

57 Cytotoxic T-lymphocytes (CTLs) and natural killer cells (NKs) kill compromised cells to defend agai

58 an increased abundance of activated natural killer cells and a newly identified CD3(-)CD68(+)CD4(+)G

59 gnificantly decreased frequencies of natural killer cells and a trend toward reduced ILC populations,

60 a-induced pathways and activation of natural killer cells and endothelial cells.

61 s and dendritic cells) and lymphoid (natural killer cells and innate lymphoid cells) cell populations

62 ived PGE2 blocks early activation of natural killer cells and interferes with subsequent adaptive imm

63 wed exosomes were mainly taken up by natural killer cells and macrophages in the lung.

64 n HL cells and the CD16A receptor on natural killer cells and macrophages, to induce tumor cell killi

65 receptors (KIRs) which are found on natural killer cells and some T cells; for the CD94:NKG2 family

66 Cytotoxic T lymphocytes (T) and natural killer cells are the main cytotoxic killer cells of the

67 +) cytotoxic T lymphocytes (CTL) and natural killer cells are the main cytotoxic killer cells of the

68 The killer cells bound to infected RBCs and killed intracell

69 Natural killer cells employ a diverse arsenal of effector mechan

70 The antibody-induced killer cells express, Granzyme B and Perforin that assau

71 ells, and CD8 T lymphocytes and CD57 natural killer cells in the ALNs(-) were factors associated with

72 ls via recruitment and activation of natural killer cells in the tumor.

73 NKG2A subset of CD56 natural killer cells increased substantially and persisted follo

74 s, dendritic cells, macrophages, and natural killer cells is crucial for its protection.

75 Our results suggest that natural killer cells modified with glycan ligands to CD22 and se

76 natural killer cells are the main cytotoxic killer cells of the human body to eliminate pathogen-inf

77 natural killer cells are the main cytotoxic killer cells of the human body to eliminate pathogen-inf

78 In contrast to viral evasion of natural killer cells or T cell recognition, the evasion of antib

79 A combination of serum and natural killer cells provided the most effective protection agai

80 Quantification of natural killer cells spreading on surfaces coated with the nanob

81 RNA-seq analysis of natural killer cells subjected to cellular crowding and hypoxia

82 lly changing channel network produced by the killer cells themselves.

83 ds onto NK-92MI and cytokine-induced natural killer cells to achieve tumor-specific CD22 targeting.

84 anced the activity and maturation of natural killer cells to effectively treat anti-PD-1 resistant tu

85 reted by cytotoxic T lymphocytes and natural killer cells to help eliminate virus-infected and transf

86 Additionally, NKG7 expressed by natural killer cells was critical for controlling cancer initiat

87 Moreover, natural killer cells were involved in AS by increasing the acces

88 n in NKG2A and NKG2C subsets of CD56 natural killer cells which might have a pathogenic role in chron

89 es, monocytes, T cells, B cells, and natural killer cells) in patients having undergone HSPC gene the

90 totoxic cells (cytotoxic T cells and natural killer cells), and interleukin 12 production by antigen-

91 multiple human cell types, including natural killer cells, an IL-12 indicator cell line, and primary

92 olled by T cells (T(H)1 and CD8(+)), natural killer cells, and antigen-presenting cells; T2 CRSsNP wa

93 Tregs), gammadelta T cells, B cells, natural killer cells, and primary and induced pluripotent stem c

94 umor, activating dendritic cells and natural killer cells, and recruiting the adaptive immune system.

95 onocytes, CD4(+) and CD8(+) T cells, natural killer cells, conventional and plasmacytoid dendritic ce

96 plied to other immune cells, such as natural killer cells, hematopoietic cells or induced pluripotent

97 macrophages, innate lymphoid cells, natural killer cells, innate gammadelta T cells, and other signa

98 cytes, neutrophils, dendritic cells, natural killer cells, innate lymphoid cells-2, and CD (cluster o

99 cells such as primary T and B cells, natural killer cells, macrophages, and primary microglia.

100 at CD8 T cells, but not CD4 cells or natural killer cells, mediated elimination of KPC-Par-1(KO) tumo

101 + T cells, CD56 + T cells, CD56(dim) natural killer cells, monocytes and dendritic cells were not red

102 r of tumor-infiltrating CD8(+) T and natural killer cells, slowed tumor growth, and improved mouse su

103 okine milieu, macrophages/monocytes, natural killer cells, T cells, and neutrophils in severe COVID-1

104 inical investigation of lymphokine-activated killer cells, tumour-infiltrating lymphocytes, and allog

105 grafts is driven by the recipient's natural killer cells, which overwhelmingly use perforin to kill

106 infection, for example proliferating natural killer cells, which potentially may associate with futur

107 tively recruited and activated human natural killer cells, while vaccine-elicited RM antibodies were

108 + cytotoxic T lymphocytes (CTLs) and natural killer cells-and regulated by Runt-related (Runx) transc

109 in controlling DENV infection using natural killer cells.

110 s well as decreased ICOS+ and 4-1BB+ natural killer cells.

111 xhibits SOX9-dependent resistance to natural killer cells.

112 "), especially cytotoxic T cells and natural killer cells.

113 cells, CD11c(+) dendritic cells and natural killer cells.

114 onocytes, innate lymphoid cells, and natural killer cells.

115 t and the homologous FcgammaRIIIa on natural killer cells.

116 rphic stress molecules recognized by natural killer cells.

117 son and differentiated into cytokine-induced killer (CIK) cells.RESULTSThe cellular product was produ

118 This tumour suppression is mediated by killer cytotoxic lymphocytes: it is abrogated in perfori

119 MCMV-induced natural killer cytotoxicity was dependent on MyD88 and STING.

120 bind the N-terminal four-helix bundle (4HB) "killer" domain and neighboring first brace helix of huma

121 -survival, attenuating the cytosolic L-A and Killer double-stranded RNA mycoviruses and protecting me

122 identify perturbations that enhance natural killer effector functions.

123 approach to redirect TAMs to serve as tumor killers for late-stage or drug-resistant cancers.

124 ters the two-helix 'brace' that connects the killer four-helix bundle and regulatory pseudokinase dom

125 efective expression of the antiviral natural-killer group 2 member D (NKG2D) protein and abnormal Mg(

126 ace expression of immune stimulatory natural killer group 2D (NKG2D) ligands on the monocytes.

127 has hitchhiked to high frequency as the male-killer has spread through the population.

128 This killer hidden in a social supergene shows that large non

129 Variegated expression of killer Ig-like receptors (KIR) in human NK cells is a st

130 Several studies support a role for specific killer immunoglobulin-like receptor (KIR)-HLA combinatio

131 inhibitory receptors, such as the family of killer immunoglobulin-like receptors (KIRs) and the NKG2

132 ortant ligands for inhibitory and activating killer immunoglobulin-like receptors (KIRs) which are fo

133 s urgently required as it is the 2nd largest killer in the world and its incidence is likely to incre

134 Lung cancer is the number one cancer killer in the world with more than 142,670 deaths estima

135 C6 had the lowest natural killer infiltration rate and was represented by copy gai

136 Invariant natural killer (iNKT) cells are among the first innate immune ce

137 rleukin 8 (IL8)-treated lymphokine-activated killer (LAK) cells.

138 perforin-deficient mice or mice depleted of killer lymphocytes.

139 ic reticulum, membrane dynamics, and Natural Killer-mediated cytotoxicity.

140 Killer meiotic drivers are genetic parasites that destro

141 correlated with perforin-expressing natural killer (NK) and CD3+ T cells.

142 Although CXCL14 recruits natural killer (NK) and T cells to the tumor microenvironment, t

143 To analyze human natural killer (NK) cell activation by both species, we performe

144 Differentiation of natural killer (NK) cell activation pathways occurs along a cont

145 We evaluated 2 patients with natural killer (NK) cell CAEBV and studied their NK cell phenoty

146 mplex II upregulation and persistent natural killer (NK) cell cytolytic killing.

147 conducted a systematic evaluation of natural killer (NK) cell cytotoxicity in adult patients with sec

148 Natural killer (NK) cell function is regulated by inhibitory rec

149 Following NACT, increased natural killer (NK) cell infiltration and oligoclonal expansion

150 ssing macrophages, and activation of natural killer (NK) cell killing through TNF-related apoptosis-i

151 demonstrate a systemic reduction in natural killer (NK) cell numbers in SRalpha-tTA/Tet-O-MYC(ON) mi

152 eral studies suggest that harnessing natural killer (NK) cell reactivity mediated through killer cell

153 ow that an endogenous ligand for the natural killer (NK) cell receptor NKG2D, Retinoic Acid Early 1 (

154 Genetic diversity in human natural killer (NK) cell receptors is linked to resistance and s

155 The impact of such HLA variation on natural killer (NK) cell recognition remains unclear.

156 evidence suggests that dysfunctional natural killer (NK) cell responses during hepatitis C virus (HCV

157 y isoflavones and metabolites impact natural killer (NK) cell signaling and function.

158 ncreased microbial translocation and natural killer (NK) cell skewing towards an inflammatory state,

159 In contrast, a CD27(+) Natural killer (NK) cell subset accumulated in the lungs of LTBI

160 Herein, we profiled natural killer (NK) cell subsets in the blood of 72 asymptomatic

161 l antibodies differed, skewed toward natural killer (NK) cell-activating antibodies.

162 e-pemetrexed assemblies that combine natural killer (NK) cell-based cancer immunotherapy with radioth

163 s of cancer by unleashing both T and natural killer (NK) cell-mediated antitumor responses.

164 Natural Killer (NK) cell-mediated early innate defense responses

165 inflammation by protecting them from natural killer (NK) cell-mediated elimination.

166 (ILC2s) orchestrated suppression of natural killer (NK) cell-mediated innate antitumor immunity, lea

167 Natural killer (NK) cell-mediated killing involves the membrane

168 ivation of an immune cell known as a natural killer (NK) cell.

169 Natural killer (NK) cells acquire effector functions through edu

170 displayed increased infiltration by natural killer (NK) cells and CD8alpha(+) T cells, and a decreas

171 These include natural killer (NK) cells and gammadelta T cells of innate immun

172 the brain, unlike in the periphery, natural killer (NK) cells and monocytes are not involved in cont

173 is localised predominantly on human natural killer (NK) cells and monocytes.

174 ), and that this resistance requires natural killer (NK) cells and other immune cells.

175 In natural killer (NK) cells and other innate lymphoid cells, compe

176 to increase IFNgamma(+) T cells and natural killer (NK) cells and reduce the percentage of macrophag

177 els of IFN-gamma production by liver natural killer (NK) cells and stronger resistance to MCMV.

178 1 ILCs consist of circulating mature natural killer (NK) cells and tissue-resident ILC1s, the functio

179 Natural killer (NK) cells are a critical component of the innate

180 Natural killer (NK) cells are a promising cellular immunotherapy

181 Human natural killer (NK) cells are critical for innate defense agains

182 Natural killer (NK) cells are critical mediators of host immunit

183 Natural killer (NK) cells are cytotoxic lymphocytes of the innat

184 Natural killer (NK) cells are cytotoxic lymphocytes targeting vi

185 Natural killer (NK) cells are cytotoxic type 1 innate lymphoid c

186 Human natural killer (NK) cells are defined as CD56(+)CD3(-).

187 ough innate lymphoid cells (ILC) and natural killer (NK) cells are found throughout the oral mucosae,

188 Natural killer (NK) cells are frequent in human liver and infilt

189 Natural killer (NK) cells are important effector cells in the im

190 Natural killer (NK) cells are important effectors of innate and

191 Natural killer (NK) cells are important in the immune defense ag

192 Natural killer (NK) cells are innate cytotoxic lymphocytes invol

193 Natural killer (NK) cells are innate effector lymphocytes that a

194 Natural killer (NK) cells are innate lymphocytes that are consid

195 Natural killer (NK) cells are innate lymphocytes that exhibit ad

196 Natural killer (NK) cells are innate lymphoid cells being explor

197 ogrammed death-ligand 1 (PD-L1), and natural killer (NK) cells are involved in trophoblast immunosurv

198 Natural killer (NK) cells are key effector cells of innate resis

199 Natural killer (NK) cells are key innate immunity effectors that

200 The promise of natural killer (NK) cells as effectors in cancer cellular therap

201 Natural killer (NK) cells belong to the innate immune system and

202 stem, which mimics the activation of natural killer (NK) cells by antibody-dependent cell-mediated cy

203 typic changes induced in CD8(+) T or natural killer (NK) cells by MTEX or non-MTEX were compared.

204 Natural killer (NK) cells control viral infection through the in

205 Natural killer (NK) cells develop from common progenitors but di

206 ently, we and others have shown that natural killer (NK) cells exhibit memory-like recall responses a

207 Natural killer (NK) cells form immune synapses to ascertain the

208 l killer T-like cell (NKT-like), and natural killer (NK) cells from patients with BOS (n = 10), stabl

209 Natural Killer (NK) cells have an important role in immune respo

210 CE Epidemiological studies show that natural killer (NK) cells have anti-HIV activity: they are able

211 Natural killer (NK) cells have potent antitumor and antimetastat

212 rr virus (EBV) DNA in T cells and/or natural killer (NK) cells in blood and skin lesions induced by s

213 tities, activity, and composition of natural killer (NK) cells in blood as well as expression changes

214 CTV; the agent of mousepox) and that natural killer (NK) cells in CL13-infected mice are reduced in n

215 nwhile, the cytotoxic role played by natural killer (NK) cells in cutaneous leishmaniasis (CL) remain

216 eficiencies of B cells, T cells, and natural killer (NK) cells in Rabl3 (xm/xm) mice.

217 itiate the recruitment of protective natural killer (NK) cells in the infected trachea.

218 s and effector T cells and decreased natural killer (NK) cells in these tumors.

219 terferon gamma (IFN-gamma)-producing natural killer (NK) cells induced a profound remodeling of the T

220 Natural killer (NK) cells inhibit tumor development in mouse mod

221 Here we report that natural killer (NK) cells of the innate immune system reside in

222 Natural killer (NK) cells play critical roles in protection agai

223 which has primarily been observed in natural killer (NK) cells responding to cytomegalovirus infectio

224 s DARA-induced depletion of CD38high natural killer (NK) cells resulting in crippled antibody-depende

225 Natural killer (NK) cells that have been modified to express an

226 ADCC) is a key effector mechanism of natural killer (NK) cells that is mediated by therapeutic monocl

227 lity, it engaged macrophages but not natural killer (NK) cells to induce antibody-dependent cellular

228 ificantly higher number of activated natural killer (NK) cells was accumulated in the colonic lamina

229 ssociated with T cells, B cells, and natural killer (NK) cells when matched sibling donors are unavai

230 impairment in T-cell homeostasis and natural killer (NK) cells which leads to autoimmunity, recurrent

231 d for functionalizing the surface of natural killer (NK) cells with a supramolecular aptamer-based po

232 tokines that regulate the biology of natural killer (NK) cells(1).

233 on of CD8-positive (CD8(+)) T cells, natural killer (NK) cells, and NK-T cells and increased accumula

234 imit antiviral responses mediated by natural killer (NK) cells, but molecular mechanisms for these pr

235 y, as mediated by CD8(+) T cells and natural killer (NK) cells, by obstructing contact between immune

236 n into mice activated ILC1s, but not natural killer (NK) cells, in the liver.

237 he development and function of human natural killer (NK) cells, innate lymphocytes important for anti

238 hanges at 48 and 96 hours, including natural killer (NK) cells, macrophage polarization, and T-cell i

239 th unique immune properties, such as natural killer (NK) cells, NKT cells, gammadelta T cells, and ma

240 the frequencies of T cells, B cells, natural killer (NK) cells, polymorphonuclear myeloid-derived sup

241 n of several immune cells, including Natural Killer (NK) cells, which play an important role in the c

242 interleukin-15 (IL-15) signaling in natural killer (NK) cells.

243 s is FcgammaRIIIa/CD16a expressed by natural killer (NK) cells.

244 onal B cells and increased cytolytic natural killer (NK) cells.

245 receptor expression on monocytes and natural killer (NK) cells.

246 or function of cytotoxic T cells and natural killer (NK) cells.

247 lymphoid cell populations including natural killer (NK) cells.

248 on is characterized by activation of natural killer (NK) cells.

249 or the survival and proliferation of natural killer (NK) cells.

250 s are dictated by the status of host natural killer (NK) cells.

251 ein complex containing the agonistic natural killer (NK) receptor NKG2D and the NK adaptor molecule D

252 Extranodal natural killer (NK) T-cell lymphoma (ENKTL) is a unique clinicop

253 f circulating Mycobacterium-reactive natural killer (NK), invariant NKT (iNKT), mucosal-associated in

254 esive phenotypes contribute to human natural killer (NK)- and T-cell development as they undergo deve

255 ystem require abrogating both T- and natural killer (NK)-cell responses, which eliminate foreign cell

256 Despite being prolific innate killers, NK cells are also key helper cells in antiviral

257 TB is the major killer of people living with HIV globally, with suboptim

258 Tuberculosis (TB) is the major killer of people living with human immunodeficiency viru

259 Once a natural killer or T cell has identified a target cell, they form

260 expressed GATA3 and cosecreted IL-13 and the killer protease granzyme B in response to allergen chall

261 tors, such as Toll-like receptors or natural killer receptors, are commonly contrasted with diverse,

262 w, the view of neutrophils as indiscriminate killers seems to be changing as evolving evidence sugges

263 Killer shrimp, Dikerogammarus villosus, a notoriously hi

264 te-like T cells, including invariant natural killer T (iNKT) and gammadelta-T cells, but their contri

265 Innate T cells, including invariant natural killer T (iNKT) and mucosal-associated innate T (MAIT) c

266 Invariant natural killer T (iNKT) cells acquire effector functions during

267 Human invariant natural killer T (iNKT) cells are a rare innate-like lymphocyte

268 Invariant natural killer T (iNKT) cells are innate-like lymphocytes with u

269 Semi-invariant natural killer T (iNKT) cells are self-reactive lymphocytes, yet

270 igated the hypothesis that invariant natural killer T (iNKT) cells contribute to innate immune dysfun

271 CD1d-restricted invariant natural killer T (iNKT) cells represent a heterogeneous populati

272 Invariant natural killer T (iNKT) cells serve as early rapid responders in

273 000 differentiating thymic invariant natural killer T (iNKT) cells using single-cell RNA sequencing t

274 ative regulatory function of BTLA in natural killer T (NKT) cell activation has been reported, whethe

275 Valpha24-invariant natural killer T (NKT) cells have shown potent anti-tumor proper

276 Natural killer T (NKT) cells rapidly respond to antigenic stimul

277 is essential for the development of natural killer T cell (NKT cell) effector functions.

278 ial cells, macrophages, T cells, and natural killer T cells in the CNV.

279 sponsible for processing antigens that allow killer T cells to distinguish between healthy and compro

280 diators in proliferating T cells and natural killer T cells, that also expressed the antimicrobial cy

281 of normal frequencies of circulating natural killer T cells.

282 of tumoricidal immune cells such as natural killer T cells.

283 Extranodal natural killer T-cell lymphoma (NKTCL; nasal type) is an aggress

284 and SIRT1 were measured in blood T, natural killer T-like cell (NKT-like), and natural killer (NK) c

285 mic and transcriptional landscape of natural killer/T cell lymphoma (NKTCL), a rare form of non-Hodgk

286 fferent cancers including extranodal natural killer/T-cell lymphoma (NKTL).

287 and test TF-targeting CAR-engineered natural killer (TF-CAR-NK) cells that co-express CD16, the Fc re

288 pulations of Podospora anserina, seven spore killer types (Psks) have been identified through classic

289 sperm whale (Physeter macrocephalus) and/or killer whale (Orcinus orca) interactions as proportions

290 l, and old post-reproductive female resident killer whales (Orcinus orca) lead collective movements i

291 phala macrorhynchus) and sometimes sympatric killer whales (Orcinus orca) were also found.

292 s (Balaena mysticetus; n = 7), and predator, killer whales (Orcinus orca; n = 3), in a large (63,000

293 and humans, and that GR-LBD is identical in killer whales and minke whales and that the LBD of THRB

294 poral availability for acoustic detection by killer whales to <25%.

295 acked bowheads during the entire 3-wk period killer whales were present, constituting a nonconsumptiv

296 ales and that the LBD of THRB is the same in killer whales, white whales, and humans, it is likely th

297 binding domain (LBD) of PPARG is the same in killer whales, white whales, polar bears, and humans, an

298 der of magnitude the risk of interception by killer whales.

299 taceans to occupy a niche similar to that of killer whales.

300 lly defenceless against their main predator, killer whales.