ライフサイエンスコーパス: killer T-cell

1 clude the gamma-delta T cell and the Natural Killer T cell.

2 n of alphabeta T cells and invariant natural killer T cells.

3 t of alphabeta T cells and invariant natural killer T cells.

4 f conventional T cells and invariant natural killer T cells.

5 CD1d presents lipid antigens to natural killer T cells.

6 t T-cell subsets to favor regulatory natural killer T cells.

7 e conventional CD4+CD3+ T cells, not natural killer T cells.

8 of tumoricidal immune cells such as natural killer T cells.

9 restricted CD4+ T cells but, rather, natural killer T cells.

10 subgroup of T cells, CD1d-restricted natural killer T cells.

11 high affinity to CD1d and stimulates natural killer T cells.

12 t to increased cytolytic activity of natural killer T cells.

13 t of B, alphabetaT, gammadeltaT, and natural killer T cells.

14 out (beta2M-/-) mice lack CD8+ T and natural killer T cells.

15 al killer cells but lack Valpha14(+) natural killer T cells.

16 or dying cells for eliciting tumor-specific killer T cells.

17 ding a block in EBNA1 presentation to CD8(+) killer T cells.

18 oenvironment block IL-2-induced expansion of killer T cells.

19 umor-infiltrating natural killer and natural killer T cells.

20 ecialized subset of T cells known as natural killer T cells.

21 dependent of CTLs, natural killer or natural killer T cells.

22 kemic cells by human CD5(+) cytokine-induced killer T cells.

23 of normal frequencies of circulating natural killer T cells.

24 e innate immune system and invariant natural killer T cells.

25 are the major endogenous ligands of natural killer T cells.

26 osa-associated invariant T cells and natural killer T cells.

27 ransmission of HIV and stimulator of natural killer T-cells.

28 esents IL-15 in trans to neighboring natural killer/T cells.

29 poptosis and elevated natural killer/natural killer T cell and macrophage cell markers.

30 Naive CD8 T cells, natural killer T cells and a subset of memory CD4 T cells bind C

31 s whose maintenance depends on help (such as killer T cells and B cells).

32 regulatory T cells, dendritic cells, natural killer T cells and B cells.

33 c functions of CD4(+) T helper cells, CD8(+) killer T cells and CD4(+) regulatory T cells are also es

34 T cells, including a large subset of natural killer T cells and CD69(+) tissue-resident memory T cell

35 CD4 T cells, together with invariant natural killer T cells and gammadelta T cells, receive strong TC

36 T cell apoptosis, depleting hepatic natural killer T cells and inducing proinflammatory cytokine pol

37 llergy include the role of invariant natural killer T cells and influences of dietary components, suc

38 specialized T-cell lineages such as natural killer T cells and innate mucosal-associated invariant T

39 As natural killer T cells and liver injury are central in liver reg

40 ers of microparticles from invariant natural killer T cells and macrophages/monocytes (CD14(+)), whic

41 In addition to natural killer T cells and mucosal-associated invariant T (MAIT

42 ly confined to TCRs from innate-like natural killer T cells and mucosal-associated invariant T cells,

43 luding gammadelta T cells, invariant natural killer T cells and mucosal-associated invariant T cells-

44 CD4 and natural killer T cells and neutrophils were markedly reduced in

45 is (P < 0.01), together with reduced natural killer T cells and raised interleukin (IL)-12 and IL-18.

46 d for development of CD4(+) T cells, natural killer T cells and regulatory T cells.

47 ponse of natural killer cells and of natural killer T cells and the Th1 polarization of antigen-speci

48 er T-cell response, characterized by natural killer T-cell and neutrophilic inflammation.

49 to effectively stimulate CD8 T cell, natural killer T cell, and natural killer cell immunity.

50 CD4+CD25+ Foxp3+ regulatory T cells, natural killer T cells, and approximately 25% of NK cells.

51 les for conventional CD4(+) T cells, natural killer T cells, and CD4(+)CD25(+)FoxP3(+) Tregs in AKI p

52 t on the presence of regulatory host natural killer T cells, and expression of CD1d on donor marrow b

53 atural killer cells, tissue-resident natural killer T cells, and helper-like innate lymphoid cells.

54 lations, including B cells, T cells, natural killer T cells, and innate lymphoid cells.

55 al killer cells, gammadelta T cells, natural killer T cells, and innate-like CD8+ T cells) are spatia

56 repertoire, near-to-absent invariant natural killer T cells, and severely diminished mucosal-associat

57 irculating regulatory T cells, lower natural killer T cells, and stabilization of inflammatory monocy

58 esidual host T cell subsets favoring natural killer T cells, and the low incidence of GVHD was associ

59 arge cell; and 1 each had extranodal natural killer/T cell, angioimmunoblastic, and precursor T-lymph

60 ent norepinephrine increases hepatic natural killer T cell apoptosis, depleting hepatic natural kille

61 Natural Killer T cells are a distinct lymphocyte lineage that re

62 ta T lymphocytes and CD1d-restricted natural killer T cells are classified as innate T lymphocytes, w

63 Invariant natural killer T cells are found in low numbers in the airways o

64 Natural killer T cells are immunoregulatory cells, which have im

65 Natural killer T cells are mediators of important regulator and

66 els of allergic asthma indicate that natural killer T cells are required for the development of aller

67 autoreactive recognition of CD1d by natural killer T cells, are indispensable for the binding of an

68 luding both natural killer cells and natural killer T cells, are unusually abundant in the liver.

69 anced interferon-gamma production by natural killer T cells as well as number of viable CD4 T cells.

70 d-tetramers, antibodies specific for natural killer T cells, as well as reverse-transcriptase-polymer

71 n Th2 inflammation such as invariant natural killer T cells, basophils, and interleukin-9 are importa

72 (GFP), revealed CD4+ memory T cells, natural killer T cells, basophils, mast cells, and eosinophils a

73 ressed in activated human CD8(+) and natural killer T cells, both of which have been implicated in as

74 essor NKAP is required for invariant natural killer T cell but not conventional T cell development.

75 c lineage T cells and into invariant natural killer T cells but did not signal the differentiation of

76 We enumerated invariant natural killer T cells by flow cytometry with the use of CD1d te

77 cells before and after activation of natural killer T cells by ligand alpha-galactosylceramide.

78 ion of CD1d-restricted semiinvariant natural killer T cells by using the CD1d ligand alpha-galactosyl

79 gammadelta and natural killer T cells can also play a role in protective immuni

80 We show that antitumor killer T cells can be detected in patients with both pro

81 mmune cells, such as macrophages and natural killer T cells, can have both deleterious and protective

82 The ability of killer T cells carrying the CD8 antigen to detect tumour

83 urally occurring regulatory T cells, natural killer T cells, CD4(+) and CD8(+) T lymphocytes, margina

84 c Th2 cells did not require B cells, natural killer T cells, CD8(+) T cells, or IFNgamma.

85 o deficient in natural killer cells, natural killer T cells, CD8+ T lymphocytes, and TCRgammadelta in

86 )-binding kinase/T-LAK (lymphokine-activated killer T cell) cell originating protein kinase (PBK/TOPK

87 ytokines expression of dendritic and natural killer T cells co-culture.

88 particles from CD14(+) and invariant natural killer T cells correlated with levels of alanine aminotr

89 The frequency of natural killer T cells correlated with the level of anti-HBs (P

90 Killer T cells (cytotoxic T lymphocytes, CTLs) maintain

91 ted into transgenic mice deficient in NK and killer T-cell cytotoxicity generated by expressing dipht

92 lls, goblet cells, and proliferating natural killer/T cells decrease with age, whereas alveolar fibro

93 4 days before partial hepatectomy to natural killer T cells- deficient mice or to anti CD1d1-treated

94 Natural killer T cells- deficient or mice injected with anti CD1

95 Alcohol-fed natural killer T cell-deficient Jalpha281(-/-) mice express a de

96 Hepatic natural killer T cell depletion leads to Th-1 polarization of he

97 conditional knockout mice, invariant natural killer T cell development is blocked at the double-posit

98 a role of the SAP-Fyn interaction in natural killer T cell development through the ability of SAP-Fyn

99 ses a similar block in the invariant natural killer T cell development, indicating that NKAP and hist

100 e defects in natural killer cell and natural killer T cell development, suggesting a role for MEF in

101 onally interact to control invariant natural killer T cell development.

102 ature thymocytes for trafficking and natural killer T cell development.

103 sphate receptor S1pr1 as well as for natural killer T cell development.

104 Stimulation of natural killer T cells during alcohol consumption induces seriou

105 Natural killer T cells express a conserved, semi-invariant alpha

106 The natural killer T cells expressed an invariant T-cell receptor an

107 Natural killer T cells expressing 'invariant' T cell receptor al

108 Natural killer T cells expressing an invariant T-cell receptor (

109 ding regulatory T and CD1d-dependent natural killer T cells, fail to develop.

110 PE DHC enhances EAE in mice lacking natural killer T cells, fails to enhance EAE in Toll-like recept

111 This report analyzes the role of natural killer T cells, Fas, and TNF-alpha in a model of chronic

112 syl ceramide (alpha-GalCer) to mouse natural killer T cells, formally demonstrating both the in vitro

113 We show here that natural killer T cells from UV-irradiated donor mice function as

114 lopment of colitis involves not only natural killer T-cell functions, but also requires IL-13 product

115 ulates the generation or function of natural killer T cells, gammadelta T cells, innate lymphoid cell

116 Invariant natural killer T cells have a distinct developmental pathway fro

117 ualitative defects in CD1-restricted natural killer T cells have been reported in several autoimmune-

118 10 years since the first workshop on natural killer T cells helped to launch a growth phase for this

119 Natural killer T cell hybridomas with identical T cell antigen r

120 r, little is known about the role of natural killer T cells in alcohol-induced liver injury.

121 the role of vitamin D and CD8(+) and natural killer T cells in asthma exacerbation in a genome-wide g

122 generated and the pathogenic role of natural killer T cells in asthma.

123 rent study is to explore the role of natural killer T cells in impaired liver regeneration.

124 tion between the number of invariant natural killer T cells in the airway and disease severity.

125 e studied the frequency of invariant natural killer T cells in the airways of subjects with mild or m

126 ial cells, macrophages, T cells, and natural killer T cells in the CNV.

127 in mice indicating a requirement for natural killer T cells in the development of allergen-induced ai

128 l consumption induces an increase of natural killer T cells in the liver and a high sensitivity of he

129 in 4- and interferon gamma-producing natural killer T cells in the liver and spleen and enhanced gran

130 ss the frequency and distribution of natural killer T cells in the lungs and in the circulating blood

131 ly activated hepatic macrophages and natural killer T cells, in the absence of obesity or insulin res

132 In alcohol-consuming animals, liver natural killer T cells increase, and further activation by alpha

133 Here, we report that cytotoxicity of killer T cells induces activation of FAO and upregulatio

134 Using natural killer T cells (iNKT cell) as a model of a T cell subset

135 Invariant natural killer T cells (iNKT cells) are a small subset of immuno

136 de (alphaGC) that activate invariant natural killer T cells (iNKT cells) are being developed as poten

137 Invariant natural killer T cells (iNKT cells) are critical for host defens

138 Invariant natural killer T cells (iNKT cells) are innate-like lymphocytes

139 Invariant natural killer T cells (iNKT cells) are innate-like T cells impo

140 Invariant natural killer T cells (iNKT cells) are innate-like T cells that

141 Invariant natural killer T cells (iNKT cells) are innate-like T lymphocyte

142 Invariant natural killer T cells (iNKT cells) are involved in the host def

143 Invariant natural killer T cells (iNKT cells) are lipid-sensing innate T c

144 Glycolipid ligands for invariant natural killer T cells (iNKT cells) are loaded onto CD1d molecul

145 lock in the development of invariant natural killer T cells (iNKT cells) at their earliest progenitor

146 echanisms of activation of invariant natural killer T cells (iNKT cells) by microbes: direct activati

147 Invariant natural killer T cells (iNKT cells) can produce copious amounts

148 Invariant natural killer T cells (iNKT cells) differentiate into thymic an

149 Invariant natural killer T cells (iNKT cells) express a restricted T cell

150 Invariant natural killer T cells (iNKT cells) have a prominent role during

151 Invariant natural killer T cells (iNKT cells) have an innate immunity-like

152 Mouse invariant natural killer T cells (iNKT cells) provide cognate and noncogna

153 Invariant natural killer T cells (iNKT cells) rapidly produce effector cyt

154 Invariant natural killer T cells (iNKT cells) respond to CD1d-presented gl

155 nteracted with innate-type invariant natural killer T cells (iNKT cells) to regulate B cell responses

156 romoted the interaction of invariant natural killer T cells (iNKT cells) with those neutrophils, a pr

157 cells, which encompass 70% invariant natural killer T cells (iNKT cells), have been found primarily p

158 pment and proliferation of invariant natural killer T cells (iNKT cells).

159 ing glycolipid antigens to invariant natural killer T cells (iNKT cells).

160 hat the cross-talk between invariant natural killer T cells (iNKT) and CD8(+) T cells in the spleen,

161 (iT) cell subsets, such as invariant natural killer T cells (iNKT) and mucosal-associated invariant T

162 V alpha14 invariant natural killer T cells (iNKT) are localized in peripheral tissue

163 kedly decreased numbers of invariant natural killer T cells (iNKT) as defined by staining with an alp

164 Invariant natural killer T cells (iNKT) cells are T lymphocytes displaying

165 ments for invariant Valpha14-bearing natural killer T cells (iNKT) in the thymus are poorly understoo

166 re regulated via recipient invariant natural killer T-cell (iNKT) interleukin-4-driven expansion of d

167 Invariant Natural Killer T-cells (iNKT-cells) are an attractive target for

168 CD1d-restricted Valpha24-invariant natural killer T cells (iNKTs) are important in immunoregulation

169 have previously found that invariant natural killer T cells (iNKTs) are involved in CM allergy sensit

170 stricted Valpha24-Jalpha18-invariant natural killer T cells (iNKTs) are potentially important in tumo

171 Invariant natural killer T cells (iNKTs), a small population characterized

172 cells, regulatory T cells, invariant natural killer T cells [iNKTs], NK cells, and dendritic cell sub

173 nnate lymphocyte lineages, including natural killer T cell, innate lymphoid cell, mucosal-associated

174 identified CD1d-restricted invariant natural killer T cells is a matter of controversy.

175 as been shown that the proportion of natural killer T cells is markedly elevated during liver regener

176 ber of the innate immune system, the natural killer T cell, is activated during bacterial infections.

177 , expressed in T cells, B cells, and natural killer T cells, is essential for cell activation and imm

178 subset of CD4+ T helper 1 cells and natural killer T cells, is involved in lymphocyte homing into th

179 uding the Hut 78 T-cell leukemia, JY natural killer T-cell leukemia, Daudi B-cell lymphoma, HeLa, and

180 enhanced by coadministration of the natural killer T-cell ligand 7DW8-5, which heightened the produc

181 stocompatibility complex (MHC) nonrestricted killer T-cell line (TALL-104) as a new marrow purging ag

182 NKAP in selection into the invariant natural killer T cell lineage.

183 (thymocytes) to either the CD4 helper or CD8 killer T-cell lineages correlates precisely with their T

184 of 55%, 95% CI 46-65) and extranodal natural killer T-cell lymphoma (108 deaths and 3-year overall su

185 The clinical outcome of extranodal natural killer T-cell lymphoma (ENKTL) has improved substantiall

186 Extranodal natural killer T-cell lymphoma (NKTCL), nasal type, is a rare an

187 Extranodal natural killer T-cell lymphoma (NKTCL; nasal type) is an aggress

188 leukaemia or lymphoma and extranodal natural killer T-cell lymphoma.

189 Extranodal natural killer-T-cell lymphoma (NKTCL) of nasal type is a malign

190 Natural killer/T cell lymphoma (NKTCL) is a rare and aggressive

191 mic and transcriptional landscape of natural killer/T cell lymphoma (NKTCL), a rare form of non-Hodgk

192 Extranodal natural-killer/T-cell lymphoma (ENKTL) is a rare and aggressive

193 Extranodal natural killer/T-cell lymphoma (ENKTL), nasal type, generally af

194 ell lymphoma (n = 2), and extranodal natural killer/T-cell lymphoma (n = 2).

195 eripheral T-cell lymphoma (PTCL) and natural killer/T-cell lymphoma (NKTCL) are rare and heterogeneou

196 Natural killer/T-cell lymphoma (NKTCL) is a rare, aggressive for

197 Extranodal natural killer/T-cell lymphoma (NKTCL) is an aggressive malignan

198 In the current study, using natural killer/T-cell lymphoma (NKTL) as a disease model, we fou

199 tly overexpressed in the majority of natural killer/T-cell lymphoma (NKTL), an aggressive lymphoid ma

200 fferent cancers including extranodal natural killer/T-cell lymphoma (NKTL).

201 ; 95% CI, 14.5-18.1), and extranodal natural killer/T-cell lymphoma (SIR, 44.3; 95% CI, 5.37-160).

202 dence of AITL, extranodal nasal-type natural killer/T-cell lymphoma and NK-cell leukemia (ENKCL), and

203 Extranodal natural killer/T-cell lymphoma rarely presents as intraocular ma

204 luding acute lymphoblastic leukemia, natural killer/T-cell lymphoma, and acute myeloid leukemia, as w

205 of mycosis fungoides and extranodal natural killer/T-cell lymphoma.

206 ll as non-Hodgkin, Hodgkin and nasal natural killer/T-cell lymphomas, although all three TET family g

207 ly caused by Wegener granulomatosis, natural killer/T-cell lymphomas, cocaine abuse, or infections.

208 rovided evidence that T lymphocytes, natural killer T cells, mast cells, and B cells also enter adipo

209 IL-9 is produced by T cells, natural killer T cells, mast cells, eosinophils, and innate lymp

210 the activation of T lymphocytes and natural killer T cells, maturation of DCs, secretion of proinfla

211 remains slow, but IL-13 produced by natural killer T cells may be involved.

212 d T helper type 2-like properties of natural killer T cells may originate largely from differences in

213 Highly active killer T cells mediate a stable standoff during controll

214 Natural killer T cell-mediated apoptosis proceeds by the Fas pat

215 nity and compromised the function of natural killer T-cell-mediated innate immunity.

216 Hepatic natural killer T cells modulate liver injury by balancing local

217 ymphoid cells, natural killer cells, natural killer T cells, mucosal-associated invariant T cells, an

218 ences in the percentages of T cells, natural killer T cells, natural killer (NK) cells, macrophages a

219 Depletion of natural killer-T cells, natural killer cells, plasma cells, and

220 tration, while natural killer cells, natural killer T cells, neutrophils and dendritic cells hepatic

221 is essential for the development of natural killer T cell (NKT cell) effector functions.

222 a type of white blood cell known as natural killer T cell (NKT cell).

223 eceptors can affect innate immunity [natural killer T cell (NKT) and gamma-delta T-cell receptor], an

224 uced expression of genes critical to natural killer T cell (NKT) and gammadelta T-cell differentiatio

225 (hi)Ly6G(-) cells also enhance liver natural killer T cell (NKT) death in an Fas-dependent manner.

226 colipid activates innate Valpha14(+) natural killer T cell (NKT) lymphocytes, which drive DC maturati

227 mpaired thymic selection of Valpha14 natural killer T cells (NKT cells) and a subsequent reduction of

228 antigen receptor (TCR) expressed by natural killer T cells (NKT cells) and the antigen-presenting mo

229 ass I homolog CD1d are recognized by natural killer T cells (NKT cells) characterized by either a sem

230 lls (T regulatory cells [Tregs]) and natural killer T cells (NKT cells) each protect against graft-ve

231 Natural killer T cells (NKT cells) expressing a semi-invariant C

232 ingolipids (GSLs) to activate type I natural killer T cells (NKT cells) has been known for 2 decades.

233 Natural killer T cells (NKT cells) have stimulatory or inhibitor

234 Loss of natural killer T cells (NKT cells) in CD1 knockout mice resulted

235 this study is to identify invariant natural killer T cells (NKT cells) in cellular infiltrate of hum

236 CD1d-restricted natural killer T cells (NKT cells) possess a wide range of effec

237 Natural killer T cells (NKT cells) recognize glycolipid antigens

238 Natural killer T cells (NKT cells) restricted by the antigen-pre

239 of a niche shared by MAIT cells and natural killer T cells (NKT cells).

240 nd thereby the effector functions of natural killer T cells (NKT cells).

241 The antigen receptor for natural killer T cells (NKT TCR) binds CD1d-restricted microbial

242 n with an IL-15-mediated increase of natural killer T-cells (NKT) and the up-regulation in liver prod

243 n-presenting cell CD1d with distinct natural killer T-cell ("NKT") populations can induce rapid gamma

244 However, after exclusion of natural killer T cells (NKTs) and gammadelta T cells by FACS, li

245 Human natural killer T cells (NKTs) are innate-like T lymphocytes incr

246 Valpha24-invariant natural killer T cells (NKTs) localize to tumors and have inhere

247 lls, macrophages, natural killer and natural killer T cells, nonclassical T cells, and memory B cells

248 By chronic exposure to killer T cells or ICI therapy, we derived acquired-resis

249 red mitochondrial priming and sensitivity to killer T cells or ICIs.

250 duction from CD4(+) T cells, but not natural killer T cells or innate lymphoid cells, suggesting a TH

251 by heat shock proteins, glycolipids, natural killer T cells, or dendritic cells in disease pathogenes

252 r results strongly suggest that CD4+ natural killer T cells play a prominent pathogenic role in human

253 Natural killer T cells play a role in the pathogenesis of NAFLD

254 yperreactivity, we hypothesized that natural killer T cells play an important role in human asthma.

255 Natural killer T cells play an important role in liver regenerat

256 ectly in the expansion of protective natural killer T-cell populations.

257 ndritic cells, and type 1 T cells or natural killer T cells potentially drives pathogenic inflammatio

258 mount, which results in expansion of natural killer T cells producing IL-13 (and perhaps IL-5).

259 ed expression of interferon gamma by natural killer T cells, promoting hepatocyte proliferation.

260 antibodies specific to the invariant natural killer T-cell receptor in samples of bronchoalveolar-lav

261 of gene expression of the invariant natural killer T-cell receptor.

262 n of messenger RNA for the invariant natural killer T-cell-receptor domains Valpha24 and Vbeta11 was

263 Mouse type I natural killer T cell receptors (iNKT TCRs) use a single V alpha

264 following the ligation of invariant natural killer T cell receptors to sphingolipids (SLs).

265 How viruses evade natural killer T cell recognition remains unclear.

266 Tolerance required host natural killer T-cell recognition of CD1d on donor marrow cells.

267 Natural killer and natural killer T cells remove virus-infected hepatocytes by deat

268 expressing, but not TIM-1-deficient, natural killer T cells responded to apoptotic airway epithelial

269 have evolved mechanisms to avoid the CD8(+) killer T cell responses by interfering with MHC class I

270 ion of protective CD4(+) T cells and natural killer T-cell responses against ehrlichiae.

271 al type I/III interferon and blunted natural killer-/T-cell responses, reflecting a potential novel i

272 ates a role for TCR beta in defining natural killer T cell specificity, despite the more restricted d

273 Identification of a natural killer T-cell subtype associated with cell-mediated immu

274 ctor properties resembling invariant natural killer T cells, such as copious production of cytokines

275 senting cell-mediated stimulation of natural killer T cells, supporting the idea that this mechanism

276 g the activity of natural killer and natural killer T cells that normally contribute to tumor surveil

277 t T cell subsets to favor regulatory natural killer T cells that suppress GVHD and prevent organ allo

278 t T-cell subsets to favor regulatory natural killer T cells that suppress GvHD by polarizing donor T

279 diators in proliferating T cells and natural killer T cells, that also expressed the antimicrobial cy

280 -recognition receptor, conferring on natural killer T cells the ability to sense and respond in an in

281 The hepatic natural killer T cells themselves are regulated by Kupffer-cell-

282 promote the progression of invariant natural killer T cells through the thymic maturation process and

283 the innate-like effector program of natural killer T-cell thymocytes, is prominently associated with

284 of the invariant T-cell receptor of natural killer T cells to assess the frequency and distribution

285 that provides a continuous supply of potent killer T cells to curb Toxoplasma gondii growth during l

286 sponsible for processing antigens that allow killer T cells to distinguish between healthy and compro

287 ild-type CD4(+) Th cells depleted of natural killer T cells to Lck(cre)IL-4Ralpha(-/lox) mice restore

288 sess the contribution of the vaccine-induced killer T cells to the control of HIV-1.

289 hat recruits T cell help from type I natural killer T cells under mRNA-vaccination conditions resulte

290 V(alpha)14 invariant natural killer T cells (V(alpha)14iNKT cells) are thymus-derived

291 er gamma-delta T cells and invariant natural killer T cells were found to be involved in IL-17A hyper

292 e conventional T cells and invariant natural killer T cells were reduced in the spleen.

293 FN-alpha, whereas natural killer and natural killer T cells were the main source of IFN-gamma product

294 Natural killer T cells, which are stimulated by lipids presented

295 , Kupffer cells), natural killer and natural killer T cells, which constitute the innate immune syste

296 Glycolipid reactive natural killer T cells with an invariant TCR alpha-chain (iNKT c

297 Two populations of natural killer T cells with invariant TCR alpha-chains (iNKT cel

298 d innate-like lymphocytes, including natural killer T cells, with an emphasis on the known requiremen

299 ated by IFN-gamma and CD4(+) Th1 and natural killer T cells, with lower IL-10 secretion by T cells.

300 and sputum induction were invariant natural killer T cells, with no significant differences among th