ライフサイエンスコーパス: killing of

1 either LCK to enhance the kinetics of tumor killing of 4-1BB CAR-T cells or SHP1 to tune down cytoki

2 AR23 greatly augments myeloid cell-dependent killing of a collection of hematopoietic and nonhematopo

3 Here, I report facultative matricide (worker killing of a colony's queen) in the social wasp Dolichov

4 innate immunity, mediating CXCR3-independent killing of a diverse array of pathogenic microorganisms.

5 y contrasting mechanisms and efficiencies of killing of a range of tumor cell lines.

6 c fibrosis neutrophils, wortmannin inhibited killing of a rough clinical isolate and not a smooth iso

7 auses ROS production and, in turn, apoptotic killing of a wide variety of melanoma cells, irrespectiv

8 e effective than AMX at enhancing phagocytic killing of A. actinomycetemcomitans by neutrophils.

9 ve transport system for AZM that may enhance killing of A. actinomycetemcomitans.

10 e induction of optimal antifungal neutrophil killing of A. fumigatus spores.

11 persistent microfilaridermia and in enhanced killing of adult worms after repeated standard IVM treat

12 as no better than IVM alone for sterilizing, killing of adult worms or achieving sustained MF clearan

13 ith neutrophils, enhancing the intracellular killing of ALF-exposed M. tuberculosis and up-regulating

14 CD3xCD123 DART also induces a dose-dependent killing of AML cell lines and primary AML blasts in vitr

15 itor (CHK1i) GDC-0575 enhances AraC-mediated killing of AML cells both in vitro and in vivo, thus abr

16 ML blasts and enables T cell recognition and killing of AML.

17 y been shown to enhance complement-dependent killing of, and facilitate bacterial clearance in, anima

18 roscovitine increases CF macrophage-mediated killing of antibiotic-resistant bacteria.

19 tabolism-sensitive staining to report on the killing of antibiotic-tolerant bacteria.

20 n initiating specific CD8(+) T-cell-mediated killing of antigen-loaded target cells in vivo.

21 Inhibition of PfCLK3 mediated rapid killing of asexual liver- and blood-stage P. falciparum

22 ar trap (NET) formation, are involved in the killing of Aspergillus fumigatus conidia and hyphae, usi

23 TCR transfer to T cells of patients enabled killing of autologous and nonautologous melanoma.

24 antigens and found these cells could mediate killing of autologous lymphoma cells.

25 s and were equally capable of MHC-restricted killing of autologous target cells.

26 to CD22 and selectins promote both targeted killing of B lymphoma cells and improved trafficking to

27 iciently internalized via hCD22 resulting in killing of B-cell lymphoma cells.

28 )-roscovitine and M3 significantly increased killing of B. cenocepacia and P. aeruginosa in CF MDMs i

29 (R)-roscovitine-mediated killing of B. cenocepacia was enhanced by combination wi

30 observed in healthy individuals and improved killing of B. pseudomallei in vitro.

31 g proteins (GBPs); this led to intracellular killing of bacteria and DNA release.

32 less efficient at inducing opsonophagocytic killing of bacteria by neutrophils in vitro, and were wo

33 echanism of reactive oxygen species-mediated killing of bacteria by neutrophils.

34 X) mediated signaling enhanced intracellular killing of bacteria by neutrophils.

35 hemical processes including, in our opinion, killing of bacteria by protozoa.

36 clearance, and iv) increased phagocytic cell killing of bacteria compared with tail trauma.

37 f bacteria throughout the colony exceeds the killing of bacteria on the surface and pinpoints how the

38 ly due to induction of pyroptosis and direct killing of bacteria rather than production of cytokines.

39 odrugs we describe here synergize the direct killing of bacteria with a simultaneous rapid immune res

40 le for the opsonization and subsequent lytic killing of bacteria, work over the last century firmly e

41 hils, and correspondingly, there was limited killing of bacteria.

42 how these steps in turn influence host cell killing of bacteria.

43 parable ingestion and initial phagolysosomal killing of bacteria.

44 ism of membrane-tension-induced (mechanical) killing of bacterial cells by non-translocating NPs.

45 ng a well-controlled platform for systematic killing of bacterial cells.

46 g minimal cell toxicity, displaying superior killing of bacterial reservoirs harboured within bladder

47 Moreover, neutrophil-mediated killing of biofilm bacteria correlated with the evident

48 Impaired killing of biofilm parallels the decrease in NET product

49 oprotection was proliferation-independent as killing of both 5-ethynyl-2'-deoxyuridine-positive (or K

50 vents that culminate in Perforin-2-dependent killing of both intracellular and extracellular, cell-ad

51 s to radiation, and mediate synthetic lethal killing of BRCA2-deficient cancer cells, effects that we

52 4-targeted conjugate demonstrated acute cell killing of breast cancer cells with high CD44 expression

53 In contrast, killing of bronchial epithelial and renal cortical cells

54 (2003-2008) provided fecal samples to assess killing of C. difficile and changes to components of the

55 oring phage03 showed, on average, 17% higher killing of C. elegans (P = 4.4e(-6)).

56 ial role of C5a-C5aR signaling in neutrophil killing of C. neoformans in real time.

57 nd CCL5/RANTES and more efficient uptake and killing of C. neoformans These findings suggest that DAP

58 ophage lysosome damage and enabled increased killing of C. neoformans.

59 ired neutrophil migration and its subsequent killing of C. neoformans.

60 on in IECs, and abrogated STING-mediated IEC killing of C. rodentium.

61 intervention that could increase immunotoxin killing of cancer cells and enhance our understanding of

62 nical cancer therapy, due to their selective killing of cancer cells and high safety characteristics.

63 herapy can enhance T cell-dependent targeted killing of cancer cells both by stimulating the hematopo

64 e metabolism results in dramatic synergistic killing of cancer cells both in vitro and in vivo.

65 Effective immunotherapy promotes the killing of cancer cells by cytotoxic T cells.

66 is and immunogenic cell death contributed to killing of cancer cells in DBC-UiO-induced PDT.

67 CD8(+) T cell-dependent killing of cancer cells requires efficient presentation

68 bility that could be leveraged for selective killing of cancer cells through targeting of an unlinked

69 ossibly transcription), leading to selective killing of cancer cells with BRCA1/2 mutations.

70 circuits triggered selective T cell-mediated killing of cancer cells, but not of normal cells, in vit

71 toimmunity and activate lymphocyte-dependent killing of cancer cells, but the mechanisms remain poorl

72 ntratumoral vessels as a result of increased killing of cancer cells, setting up a positive feedback

73 D-28 had a high specificity in the selective killing of cancer cells, without any apparent effects on

74 vation-induced apoptosis and T cell-mediated killing of cancer cells.

75 ibition in sensitizing anticancer agents for killing of cancer cells.

76 umor fibrosis, and unleashed T cell-mediated killing of cancer cells.

77 engineering enhances avidity and direct cell killing of cancer-targeting anti-glycan antibodies to cr

78 f AB307.30 but failed to increase phagocytic killing of capsule-positive strains.

79 apsidCas13a(s), capable of sequence-specific killing of carbapenem-resistant Escherichia coli and met

80 HIV killing of CD4 T cells that replicate HIV can involve HI

81 ls, which inhibited IFN-gamma production and killing of CEF-pulsed monocytes.

82 moiety and axial ligands) contribute to the killing of cells and not just one dominant component.

83 cific receptors, such as Ly49A, that inhibit killing of cells expressing self-MHC-I.

84 NHE mediates killing of cells from multiple lineages and hosts, highl

85 for selective and efficient light-controlled killing of cells which are positive for epidermal growth

86 ranzyme B production in vitro and by in vivo killing of CFSE-labeled targets.

87 They also effectively mediated killing of chronically and acutely HIV-1 infected T cell

88 ved D-lactate promotes Kupffer-cell-mediated killing of circulating pathogens.

89 imetics were highly synergistic in restoring killing of CLL cells.

90 igate the role of p53 dynamics in fractional killing of colon cancer cells in response to chemotherap

91 rovide an example of pathogen T6SS-dependent killing of commensal bacteria as a mechanism to successf

92 This activation results in T6SS-mediated killing of competing bacteria but renders A. baumannii s

93 ode to avoid cannibalism while promoting the killing of competing nematodes.

94 dent bacterial weapon that allows for direct killing of competitors through the translocation of prot

95 plicated an NKG2D-dependent mechanism for NK killing of CSCs.

96 teins is a prerequisite for efficient serial killing of CTLs and identify key events in this process.

97 Using this assay, the levels of killing of different T. parva-specific CD8(+) T cell clo

98 Here we report a natural cancer killing of different types of cancer cells by sera from

99 Rather, rapid killing of donor lymphocytes by host alloreactive NK cel

100 was reduced, consistent with their impaired killing of EBV-infected cells.

101 ed activation by, rather than effects during killing of, EBV-transformed B cells.

102 ongoing autoimmune inflammation by selective killing of effector autoreactive T cells.

103 nding resulted in an almost 10-fold superior killing of EGFR-overexpressing tumor cells compared with

104 phants comes from the Monitoring the Illegal Killing of Elephants (MIKE) program, which reports numbe

105 Genetic killing of embryonic eCN throughout the cerebellum also

106 fic opsonic antibodies in mice, resulting in killing of encapsulated bacteria by phagocytic activity.

107 Enhanced killing of eosinophils in the presence of IL-5 might lea

108 These results demonstrate the killing of Escherichia coli and Salmonella typhimurium,

109 s factor and interleukin 6 and intracellular killing of Escherichia coli by monocytes and peritoneal

110 ng BTN3A1 therefore orchestrates cooperative killing of established tumors by alphabeta and gammadelt

111 tudies have shown that NOX2 is essential for killing of G. bethesdensis by neutrophils and monocytes

112 unized fish were preincubated with CC41 mAb, killing of G14D-CCV targets was reduced by approximately

113 genes, which are essential for the enhanced killing of ganetespib treated melanoma cells by T cells.

114 onstrated decreased resistance to phagocytic killing of GAS lacking EndoS in vitro and decreased viru

115 ogenic survival assays demonstrated enhanced killing of GBM cells in response to a combination of PDI

116 ests have been happening for weeks since the killing of George Floyd in Minneapolis.

117 ease 2019 (COVID-19) pandemic and the police killings of George Floyd, Breonna Taylor, and multiple o

118 ctin as a key functional arm of NET-mediated killing of gonococci.

119 CD3+ T cells to GPRC5D+ MM cells and induces killing of GPRC5D+ cells.

120 ent structural features are relevant for the killing of Gram-negative and Gram-positive bacteria.

121 es to delineate antibody-mediated uptake and killing of Gram-positive pathogens remain extremely limi

122 , this serum facilitated complement-mediated killing of H. pylori and E. cloacae, indicating its pote

123 The security challenge has led to the killing of health workers, destruction of health facilit

124 c drug doxorubicin (Dox) proved effective in killing of HeLa cancer cells and multicellular tumor sph

125 g drug storage, drug release (via heat), and killing of HeLa cells in culture is investigated.

126 cific antibody-drug conjugates for selective killing of HER2-positive breast cancer cells.

127 ndogenous Ag processing and presentation and killing of HIV-infected CD4 T cells by CD8 T cells in a

128 ll maturation and improved effector-mediated killing of HIV-infected CD4 T cells by the HIV envelope-

129 ary CD4 T cells, venetoclax causes selective killing of HIV-infected cells, resulting in decreased nu

130 ells underwent clonal expansion and mediated killing of HIV-infected cells.

131 ombinations of 3 mAbs mediated more than 30% killing of HIV-infected primary CD4+ T cells in the pres

132 the BsAbs were able to efficiently redirect killing of HLA-DR(+) leukemic cells by human CD5(+) cyto

133 Direct killing of host cells by the amoebae is likely to be the

134 ulation is associated with cytokine-mediated killing of human beta-cells, a process partially prevent

135 T cells are more potent in antigen-specific killing of human GBM cells.

136 activation by IL-18 results in uncontrolled killing of human hepatocytes in vitro.

137 The killing of human leukemia lymphoblasts by NK cells depen

138 AdRGD-PG-hIFNbeta vector provides extensive killing of human melanoma cells in vitro and a potent an

139 ical for Staphylococcus aureus targeting and killing of human neutrophils ex vivo and is produced in

140 EBV miRNAs strongly inhibit recognition and killing of infected B cells by EBV-specific CD8(+) T cel

141 xic EBV-specific CD4(+) effector T cells and killing of infected B cells.

142 ified T cells will hopefully provide durable killing of infected cells and sustained virus suppressio

143 It promotes killing of infected cells by Fc-mediated antibody-depend

144 gger the clearance of viruses and subsequent killing of infected cells(1-4).

145 2 to 24 hours resulted in increased specific killing of infected cells, even with neutralization-resi

146 cytotoxic T lymphocyte (CTL) recognition and killing of infected cells.

147 ntrol intracellular infection leading to the killing of infected cells.

148 s, in deflecting antigen-specific CD8 T cell-killing of infected cells.

149 ) T cell degranulation may impede the proper killing of infected cells.

150 responses to influenza infection include the killing of infected epithelial cells and generation of a

151 nd contribute to protecting the host through killing of infected, foreign, stressed or transformed ce

152 Type 1 diabetes mellitus is caused by the killing of insulin-producing beta cells by CD8+T cells.

153 ole for neutrophil calprotectin in uptake or killing of intracellular A. fumigatus conidia either in

154 share structural semblances and both induce killing of intracellular Leishmania, we surmised that pl

155 ly taken up by infected macrophages, enhance killing of intracellular Mtb, and are efficiently delive

156 ctivation, and, most importantly, macrophage killing of intracellular Mtb.

157 al was required for phagosome maturation and killing of intracellular Mycobacterium tuberculosis (Mtb

158 antituberculosis treatment would enhance the killing of intracerebral Mycobacterium tuberculosis orga

159 mental conditions, TM7x can exhibit virulent killing of its host bacterium.

160 crophages (BMMs) exhibit defective cytosolic killing of L. monocytogenes We further showed that P2X5

161 c Leukaemia (RBL-2H3) MCs led to significant killing of L. tropica and to a lesser extent of L. donov

162 these new sources of energy resulted in the killing of latent HIV infected macrophages.

163 ediated cell depletion, therapeutic antibody-killing of LCMV infected cells and human CD20-expressing

164 show that this system can achieve effective killing of leukemic cells.

165 We thus hypothesized that the killing of liver-stage malarial parasites by IFN-gamma i

166 We also observed effective killing of lymphoid tumour cell lines in vitro following

167 Moreover, COMP inhibits phagocytic killing of M. catarrhalis by human neutrophils.

168 The enhanced killing of M. tuberculosis in macrophages in vivo by CD4

169 ibition of MDR1 improves antibiotic-mediated killing of M. tuberculosis.

170 ruit and activate myeloid cells for enhanced killing of mAb-opsonized tumors.

171 both males and females despite the one-sided killing of male reproductives by larval female soldiers

172 hrough checkpoint inhibition to boost immune killing of malignant cells.

173 recruitment of NK cells and TRAIL-dependent killing of melanoma cells by macrophages.

174 ored degranulation, IFNgamma production, and killing of MHC class I negative hematopoietic grafts.

175 revent killing of self-cells, while enabling killing of MHC I-deficient cells.

176 red leukocyte recruitment, ingestion, and/or killing of microbes.

177 rimary MM, at low E:T ratios (56.2% +/- 3.9% killing of MM.1s at 48 h, E:T ratio 1:32; P < .01) and o

178 ny forming unit analysis, motif Pep-H led to killing of more than 90% M. tb in vitro at 10 mug/ml, wh

179 binding mode; consequently, the direct cell killing of mouse IgG3 mAb is lost upon chimerization or

180 cubation of linezolid significantly improved killing of MRSA by dysfunctional neutrophils, which was

181 EC = 0.2-12.5 muM), as well as the effective killing of MRSA persister cells in non-biofilm cultures.

182 urn lead to increased susceptibility to host killing of MRSA.

183 Killing of Mtb by the compound is potentiated by another

184 teine to isoniazid treatment potentiated the killing of Mtb Furthermore, we demonstrate that the addi

185 Curiously, BDQ-mediated killing of Mtb lags significantly behind its inhibition

186 ns of anisomycin induced selective apoptotic killing of Mtb-infected human macrophages, which was com

187 Finally, selective killing of Mtb-infected macrophages and subsequent bacte

188 in-induced autophagy increased intracellular killing of Mtb.

189 argeting the ETC can be exploited to enhance killing of Mtb.

190 covitine's impact on macrophage function and killing of multi-drug resistant bacteria.

191 encapsulation of clofazimine (CFZ), enhance killing of mycobacteria in vitro and in infected zebrafi

192 gized with rifampin to promote intracellular killing of mycobacteria.

193 ls (DCs) that impairs T-cell recognition and killing of myeloma cells.

194 azepine dimers demonstrated potent bystander killing of neighboring CD30(-) cells.

195 , such as fratricide, the kin-discriminatory killing of neighboring cells, and competence-mediated gr

196 anti-Rmp Abs) can block complement-dependent killing of Neisseria gonorrhoeae by otherwise bactericid

197 e proven to be involved in contact-dependent killing of NF2 in vitro, as well as in its elimination a

198 ulated process to allow for effective serial killing of NK cells.

199 nd pyridoxine did not only cause synergistic killing of NSCLC cells but also elicited signs of immuno

200 Phagocytosis and killing of NTHi by macrophages were evaluated by an in v

201 These aggregates can drive contact-dependent killing of other organisms, or Caulobacter cells not pro

202 1 function, we further demonstrate selective killing of p53-deficient cells with camptothecin while s

203 s associated with greater-than-additive cell killing of pancreatic tumor cells.

204 ) showed enhanced phagocytosis and bacterial killing of PAO1.

205 teristic of candidate antimalarials is rapid killing of parasites.

206 and granzyme B confirmed that CD8(+) T cell killing of parasitized cells is dependent on granule exo

207 ected defense strategies that complement the killing of parasitized cells.

208 protective microenvironment, amplifying the killing of pathogenic bacteria within.

209 chanism of host defense, responsible for the killing of pathogens and the production of potent anaphy

210 o be strictly controlled to ensure efficient killing of pathogens but avoid the induction of negative

211 o the peritoneum, or improve phagocytic cell killing of pathogens.

212 ar traps (NETs) facilitate the extracellular killing of pathogens.

213 e proteins are involved in the extracellular killing of pathogens.

214 P90 with ganetespib enhances T-cell-mediated killing of patient-derived human melanoma cells by their

215 the intestinal epithelium and the effective killing of penetrant microorganisms, while suppressing t

216 pproximately 40% compared to that of HC, and killing of periodontal pathogens, including Porphyromona

217 le nitric oxide synthase (iNOS) and enhanced killing of persistent pathogens, suggesting that inhibit

218 ompared to wild-type cells, showed increased killing of phagocytosed E. coli and M. smegmatis Polypho

219 ation plays a vital role in opsonophagocytic killing of pneumococci in blood.

220 e plus AC significantly reduced in vitro AMo killing of pneumococci, relative to other conditions, in

221 elates with the efficacy of opsonophagocytic killing of pneumococci.

222 dal antibodies resulting in opsonophagocytic killing of prevalent and invasive GAS serotypes of diffe

223 Moreover, direct recognition and killing of primary autologous lymphoma cells by activate

224 This damage occurs primarily by killing of proximal tubule kidney cells and mechanosenso

225 Hypercapnia also reduced macrophage killing of Pseudomonas aeruginosa.

226 In contrast, killing of purified eosinophils without IL-5 was only se

227 DNA cleavage in vitro impair P2 OLD-mediated killing of recBC-Escherichia coli hosts, indicating that

228 RT elicits effects that go beyond the direct killing of residual tumor cells.

229 Increased phagocytosis promotes killing of S aureus and interleukin-8 release by neutrop

230 oration with treatment significantly enabled killing of S aureus both ex vivo and in vivo.

231 efense against S. aureus both through direct killing of S. aureus and enhancing the antimicrobial fun

232 ce macrophage phagocytosis and intracellular killing of S. aureus In this study we report evidence in

233 associated with enhanced neutrophil-mediated killing of S. aureus, increased C5a release, and modulat

234 We concluded that killing of S. mutans by ME promotes effective reminerali

235 mediated and cell-dependent opsonophagocytic killing of S. Typhimurium in vitro.

236 logous O-antigen, which mediated significant killing of Salmonella Choleraesuis and provided full pro

237 lf-MHC class I (MHC I) molecules and prevent killing of self-cells, while enabling killing of MHC I-d

238 ed serotype 6A and 6B antibodies mediate the killing of serotype 6E pneumococci.

239 d serotype 6B antibodies were able to elicit killing of serotype 6E pneumococci.

240 Furthermore, PRELP enhances phagocytic killing of serum-opsonized M. catarrhalis by human neutr

241 plicing fidelity and results in preferential killing of SF-mutant leukemias over wild-type counterpar

242 itment is indispensable for opsonophagocytic killing of staphylococci and for protection against bloo

243 pecific antibodies, which trigger phagocytic killing of staphylococci and protect mice against lethal

244 immunity are not yet known, opsonophagocytic killing of staphylococci by phagocytic cells offers oppo

245 uld enhance phagocyte oxidant production and killing of Staphylococcus aureus, a significant pathogen

246 white blood cells showed defective in vitro killing of Staphylococcus aureus, consistent with a spec

247 n-induced signals in stellate cells and thus killing of stellate cells by bile acids might have impor

248 tains epitopes that promote opsonophagocytic killing of streptococci.

249 ages enhances lysosome-based proteolysis and killing of subsequently phagocytosed E. coli compared to

250 pansion that causes, but also relies on, the killing of surrounding cells.

251 troy target cells while avoiding nonspecific killing of surrounding healthy cells.

252 at oxidative processes are involved in TLF-1 killing of T. brucei brucei.

253 ULK1 and Beclin 1 to stimulate autophagy and killing of T. gondii.

254 me B and determine its role in mediating the killing of T. parva-parasitized cells.

255 type 1 envelope (Env)-specific ADCC-mediated killing of target cells (P<.001) was observed after 96 w

256 -subunit resulted in a significantly reduced killing of target cells and a >50% reduction in CG fusio

257 ice was associated with reduced IAV-specific killing of target cells and a reduction in the number of

258 However, the killing of target cells sometimes requires multiple enga

259 r function (e.g., production of IFNgamma and killing of target cells) in NK cells, whereas C-HIV and

260 MART1 TCR-engineered T cells potentiated the killing of target cells.

261 for inflammatory cytokine production and the killing of target cells; however, much less is known abo

262 cytotoxic machinery of Ly49s(+) NK cells in killing of target T cells.

263 which may account for the enhanced host cell killing of that mutant.

264 synthesized drug that mediated bactericidal killing of the bacterium Mycobacterium tuberculosis, a m

265 ells, selectively recognizes and induces the killing of the cells.

266 ression of FHbp ID 896 resulted in augmented killing of the clade by LL-37.

267 hance bacterial phagocytosis but did enhance killing of the few bacteria ingested by neutrophils.

268 Despite lack of killing of the Gal-Gal-Glc-HepI mutants, mAb 2C7 deposit

269 use of venetoclax, which causes preferential killing of the HIV-expressing cells.

270 he increase in viral production will lead to killing of the infected cell either by the virus itself

271 . Typhimurium to effect in vitro and in vivo killing of the invasive African S. Typhimurium strain D2

272 almost completely abolished phagocytosis and killing of the organisms by neutrophils.

273 x compartments with lysosomes and subsequent killing of the pathogen.

274 to 3 to initiate a delayed contact-dependent killing of the proteinosomes and cessation of glucose ox

275 nditions enhanced the efficacy of tobramycin killing of the resident wound biofilms by up to 5-logs.

276 r of these showed significant dose-dependent killing of the schistosome larvae and markedly impaired

277 antiserum mediated in vitro opsonophagocytic killing of the strain harboring the pIP501 plasmid and a

278 In this regard, potencies (EC(50) killing) of the best immunoligands were substantially im

279 layed by dendritic cells (DCs) in the direct killing of this bacterium.

280 ations achieved similar complement-dependent killing of three gonococcal mutants with glycan extensio

281 rtant stimulator of autophagy and autophagic killing of Toxoplasma gondii in host cells.

282 CD40-mediated autophagic killing of Toxoplasma gondii is known to require TNF-alp

283 nts enable the detection and photomechanical killing of triple negative breast cancer cells that are

284 adiotherapy may result from increased direct killing of tumor cells and effects on nontumor cell popu

285 essing the ability to elicit T cell-mediated killing of tumor cells and prevent disease recurrence.

286 The killing of tumor cells by CD8(+) T cells is suppressed b

287 ly improved NK cell-mediated, EGFR-dependent killing of tumor cells compared with the NK cell engager

288 -1) disruption augmented CAR T cell mediated killing of tumor cells in vitro and enhanced clearance o

289 ndothelial cell death, resulting in indirect killing of tumor cells through nutrient depletion.

290 T-cell-directed killing of tumor cells using bispecific antibodies is a

291 scription was previously shown to potentiate killing of tumor cells.

292 oach that potentiates human NK cell-mediated killing of tumor cells.

293 ly studied with regard to NK recognition and killing of tumors.

294 also mediate antitumour responses by direct killing of tumour cells and by participating in cellular

295 BCL-2 antagonist venetoclax induced minimal killing of uninfected CD4 T cells but markedly increased

296 we have shown that natural killer (NK) cell killing of various tumors is inhibited in the presence o

297 ells have been defined by nonspecific innate killing of virus-infected and tumor cells.

298 d virus neutralization or cytotoxic CD8 cell killing of virus-infected cells and may be mediated in p

299 Using an assay designed to measure the killing of virus-infected cells by antibody-dependent ce

300 onocytes, generation of TipDC, and phagocyte killing of yeast.