ライフサイエンスコーパス: kiss

1 ng teleregulation and interchromosomal "gene kissing".

2 that mediate positive regulation of GnRH by Kiss.

3 sites located on chromosome 1 by chromosome kissing.

4 ght in adult humans, as tested in the act of kissing.

5 sm of regulation of Fob1-mediated chromosome kissing.

6 rous tissue results in rescued expression of KiSS-1 and reduced metastatic phenotype.

7 human chromosome 6q16.3-q23 is essential for KiSS-1 expression in normal tissues.

8 Furthermore, we demonstrate that KiSS-1 expression is regulated by Sp1 elements within th

9 RIP-130-modulated transcriptional control of KiSS-1 expression.

10 Loss of the metastasis suppressor gene, KiSS-1 has been strongly correlated to the progression o

11 The mechanism through which KiSS-1 is lost during metastasis, however, is still not

12 an chromosome 6q16.3-q23, results in reduced KiSS-1 promoter activation in highly malignant melanoma

13 ements within the first 100-bp region of the KiSS-1 promoter and that targeted deletion of a single G

14 suggest that DRIP-130 is a key regulator in KiSS-1 transactivation in normal tissue, and that the lo

15 Genes affected by WNT5A include KISS-1, a metastasis suppressor, and CD44, involved in t

16 Metastin, also known as KiSS-1, the cognate ligand for the metastin receptor GPR

17 ty (income inequality) predicts frequency of kissing across romantic relationships.

18 e, we define genetic mechanisms that mediate Kiss action on target gene expression.

19 o, 1.54; 95% CI, 1.28 to 1.86), and intimate kissing (adjusted odds ratio, 1.65; 95% CI, 1.33 to 2.05

20 In an Editorial, Ligia Kiss and Cathy Zimmerman discuss the need for research o

21 Activity-evoked kiss and run exocytosis opens synaptic DCV fusion pores

22 ese data suggest the presence of a selective kiss and run mechanism of insulin release.

23 Transient "kiss and run" interactions between endosomes containing

24 frequent cycles of fusion and fission in a 'kiss and run' pattern.

25 hormones are released through a transient ('kiss and run') or an irreversibly dilating pore (full fu

26 , mutually engaging with facial expressions, kisses and greetings.

27 nnealing can initiate through both loop-loop kissing and a distinct "zipper" pathway involving nuclea

28 a significant male bias in the initiation of kissing and a significant bias in head-turning to the ri

29 d its phosphorylation antagonized chromosome kissing and recombination and enhanced the RLS.

30 Data from mathematical models suggest that kissing and saliva exchange during sexual activity might

31 d structural changes, such as dimerization, "kiss", and cyclization.

32 or the first time that Otx-2 is regulated by Kiss, and plays a role in mediating the transcriptional

33 Visually guided eating, biting and kissing, and avoiding objects moving toward the face and

34 kiss initiators and kiss recipients for lip kissing, and took into consideration differences due to

35 Kiss-and-coat exocytosis entails prolonged maintenance o

36 ence for a third form of exocytosis, dubbed "kiss-and-coat," which is characteristic of a broad varie

37 The rapid kiss-and-hop interaction explains why tau, although bind

38 Our data imply a novel kiss-and-hop mechanism by which tau promotes neuronal mi

39 t a new membrane feature resulting from the "kiss-and-merge" granule fusion.

40 Kiss-and-run (KR) is an unconventional fusion between se

41 Kiss-and-run and reuse could enable hippocampal nerve te

42 k single vesicles through multiple rounds of kiss-and-run and reuse, without perturbing vesicle cycli

43 The increased incidence of kiss-and-run at lower frequencies may ensure that vesicl

44 meter, Qdots will not escape vesicles during kiss-and-run but only with full collapse fusion.

45 pocampal synapses and that the prevalence of kiss-and-run can be modulated by stimulus frequency.

46 Kiss-and-run dominated at the beginning of stimulus trai

47 The importance of kiss-and-run during efficient neurotransmission has rema

48 forms of compensatory endocytosis, including kiss-and-run endocytosis and a mechanism for efficient r

49 sed including clathrin-mediated endocytosis, kiss-and-run endocytosis, cavicapture, and bulk endocyto

50 his form of recycling is not compatible with kiss-and-run endocytosis; moreover, it is 200-fold faste

51 y synaptic vesicle endocytosis including any kiss-and-run events.

52 radox is explained by a fourfold increase in kiss-and-run exocytosis (as determined by single-granule

53 he upregulation of STXBP6 and an increase in kiss-and-run exocytosis at the expense of full fusion.

54 synaptotagmin isoform activated, and because kiss-and-run exocytosis can filter small molecules throu

55 ely slow rate of release of glutamate during kiss-and-run exocytosis shifts the population of AMPA re

56 ion pores and survive intact for future use (kiss-and-run exocytosis).

57 f a fusing vesicle are fates associated with kiss-and-run exocytosis, and we find that these are the

58 An intact cortex favors kiss-and-run exocytosis, whereas disrupting the cortex f

59 dent partial emptying of DCVs, suggestive of kiss-and-run exocytosis.

60 ) in Dictyostelium is carried out by a giant kiss-and-run focal exocytic event during which the two m

61 uminescence change allowed us to distinguish kiss-and-run from full-collapse fusion and to track sing

62 selectively release catecholamines through a kiss-and-run fusion event.

63 l cortex plays a key role in stabilizing the kiss-and-run fusion event.

64 Kiss-and-run fusion events were concentrated near the ce

65 Further work is needed to determine whether kiss-and-run is a major mode of fusion and has a major r

66 Kiss-and-run is a mode of membrane fusion and retrieval

67 echnique to provide compelling evidence that kiss-and-run is the dominant mode of vesicle fusion at h

68 thin the readily releasable pool (RRP) via a kiss-and-run mechanism that involves rapid opening and c

69 eatedly deliver material to the vacuole by a kiss-and-run mechanism.

70 Basal sympathetic firing elicits a transient kiss-and-run mode of exocytosis and modest catecholamine

71 pore dilation and maintains the granule in a kiss-and-run mode of exocytosis.

72 cles, with long-dwelling vesicles preferring kiss-and-run rather than full-collapse fusion.

73 However, the existence of kiss-and-run remains highly controversial, as revealed b

74 parison with FM dye destaining revealed that kiss-and-run strongly prevailed over full-collapse fusio

75 otein receptor (SNARE) complex that promotes kiss-and-run vesicle fusion.

76 Quickening of kiss-and-run vesicle reuse was also observed at higher f

77 pore, the activation of isoforms that favor kiss-and-run will select smaller molecules over larger m

78 vents associated with "full fusion" events, "kiss-and-run" and "kiss-and-stay" exocytosis, confirming

79 By extension, clathrin-independent "kiss-and-run" endocytosis does not sustain synaptic tran

80 "Kiss-and-run" events and tubule connections mediate tran

81 me and revealed to precisely mark organelle "kiss-and-run" events.

82 In contrast, a nonclassical mode known as "kiss-and-run" features fusion by a transient fusion pore

83 onclusions dispute previous assertions that "kiss-and-run" is a major mechanism of vesicle recycling

84 they released their contents, indicating a "kiss-and-run" pathway.

85 Transient fusion ("kiss-and-run") is accepted as a mode of transmitter rele

86 id opening and closing of a fusion pore (or "kiss-and-run") with a median opening time of 2.6 s, whic

87 ely 1 s by the reversal of fusion pores via 'kiss-and-run' endocytosis.

88 ore fleeting mode of vesicle fusion, termed 'kiss-and-run' exocytosis or 'flicker-fusion', indicates

89 tsynaptic consequences, such that so-called 'kiss-and-run' exocytosis results in negligible activatio

90 cle fusion, we found both full collapse and 'kiss-and-run' fusion at calyx-type synapses.

91 These results show that 'kiss-and-run' fusion occurs at synapses and that it can

92 ' remains controversial, and the ability of 'kiss-and-run' fusion to generate rapid synaptic currents

93 'Kiss-and-run' fusion was seen as a brief capacitance fli

94 es completely, or close rapidly to generate 'kiss-and-run' fusion.

95 t evidence argues against the occurrence of 'kiss-and-run' in hippocampal synapses.

96 es, the size of the fusion pore is unclear, 'kiss-and-run' remains controversial, and the ability of

97 Such 'kiss-and-run' vesicle fusion can in principle result in

98 cargo discharge and reducing pore closure ('kiss-and-run').

99 cycled by a second, faster mechanism called 'kiss-and-run', which operates in 1 s or less to retrieve

100 tion of flickering and closing fusion pores (kiss-and-run) is very well explained by the observed beh

101 are recovered by either fusion pore closure (kiss-and-run) or clathrin-mediated endocytosis directly

102 ucture (Omega-profile), followed by closure (kiss-and-run) or merging of the Omega-profile into the p

103 Alternatively, the pore may close (kiss-and-run), but the triggering mechanisms and its end

104 ivities, their preference for full fusion or kiss-and-run, and their sensitivity to inhibition by syn

105 action of fusion events has been shown to be kiss-and-run, as determined using cell-attached capacita

106 ne invagination and vesicle reformation; (b) kiss-and-run, in which the fusion pore opens and closes;

107 as that proposed to support the presence of kiss-and-run, is likely explained by the stochastic natu

108 During kiss-and-run, syt IV increased the conductance and durat

109 retory vesicle collapses into the PM; or by "kiss-and-run," where the fusion pore does not dilate and

110 n pathway via alternative mechanisms such as kiss-and-run.

111 via two distinct mechanisms: full-fusion and kiss-and-run.

112 sis was shifted from full-collapse fusion to kiss-and-run.

113 th "full fusion" events, "kiss-and-run" and "kiss-and-stay" exocytosis, confirming that the device ha

114 regulation of postsynaptic signaling and a 'kiss-and-wait' mode of regulated membrane protein insert

115 Oral sex and open-mouthed kissing are associated with the development of oral HPV

116 ndant in thalamo-recipient cortical layers ("kissing" astrocytes), overlap markedly less.

117 (culotte or crush techniques) with mandatory kissing balloon dilatation.

118 ain vessel was stented, followed by optional kissing balloon dilatation/T-stent.

119 l success (provisional 97%, culotte 94%) and kissing balloon inflation (provisional 95%, culotte 98%)

120 cases were completed successfully with final kissing balloon inflations.

121 simple group (n=250), 66 patients (26%) had kissing balloons in addition to main-vessel stenting, an

122 ller than that of Lai, suggesting that fewer kissing base pairs are broken at the transition state of

123 Protein-mediated "chromosome kissing" between two DNA sites in trans (or in cis) is k

124 scapularis), body louse (Pediculus humanus), kissing bug (Rhodnius prolixus) and tsetse fly (Glossina

125 l and South America, Rhodnius prolixus, the "kissing bug".

126 ginal JH that Wigglesworth described for the kissing-bug R. prolixus remained unidentified.

127 A heat exchange mechanism in the head of kissing bugs helps to prevent stress and regulate their

128 by a protozoan transmitted by haematophagous kissing bugs.

129 We used thermography to examine how kissing-bugs Rhodnius prolixus actively protect themselv

130 ree steps with two obligatory intermediates (kissing complex and bent intermediate) and driven by Mg(

131 proach (referred to as enzyme-linked aptamer kissing complex assay (ELAKCA)) relied on the kissing co

132 g 2+ greatly slows down the unfolding of the kissing complex but has moderate effects on the formatio

133 We also show that loop-loop kissing complex formation becomes more efficient at phys

134 An RNA kissing complex formed by the dimerization initiation si

135 trong mechanical stability of even a minimal kissing complex indicates the importance of such loop-lo

136 nsistent with observations that the Lai-type kissing complex is more stable and requires significantl

137 , FMRP binds intramolecular G-quadruplex and kissing complex RNA (kcRNA) ligands via the RGG box and

138 Furthermore, formation of the kissing complex was dominated by base pairing, whereas i

139 igated the mechanical unfolding of a minimal kissing complex with only two G.C base pairs.

140 nt role in genome dimerization by forming a 'kissing complex' between two complementary hairpins.

141 issing complex assay (ELAKCA)) relied on the kissing complex-based recognition of the target-bound ha

142 The kinetics and thermodynamics of kissing-complex formation and their subsequent strand-di

143 obe the factors that govern the stability of kissing complexes and their subsequent structural rearra

144 rogenic ligand (MG) exclusively when RNA-RNA kissing complexes are formed, whereas MG does not bind t

145 g of RNA duplex formation can be extended to kissing complexes but that kissing complexes display an

146 In some cases, kissing complexes can be a prelude to strand displacemen

147 Formation and stability of these RNA kissing complexes depend crucially on cationic condition

148 an be extended to kissing complexes but that kissing complexes display an unusual level of stability

149 In minimal RNA kissing complexes formed between hairpins with cognate G

150 The two kissing complexes have distinct unfolding transition sta

151 primary difference between stable and labile kissing complexes is based almost completely on their of

152 ither Mal-type (GUGCAC) or Lai-type (GCGCGC) kissing complexes under various ionic conditions.

153 tion and dissociation dynamics of individual kissing complexes, as well as the formation of the matur

154 sites that are complementary partners of a "kissing" contact across the dimer interface.

155 timulates refolding of SL1 from a metastable kissing dimer (KD) into thermodynamically stable linear

156 on of reverse transcription, can promote the kissing dimer but not the extended dimer.

157 nce in its apical loop, forming a metastable kissing dimer form.

158 First, a metastable kissing dimer is formed via a loop-loop interaction and

159 First, a metastable kissing dimer is formed via standard Watson-Crick base p

160 iciency virus type I that forms a metastable kissing dimer that is converted during viral maturation

161 ich facilitates the in vitro conversion from kissing dimer to extended dimer.

162 Our studies indicate that a kissing dimer-mediated structure, if formed, exists only

163 ization mass spectrometry was used to detect kissing dimers in a multiequilibria mixture, whereas opt

164 ly conserved internal loop that promotes the kissing-duplex transition by a mechanism that remains po

165 ing, Mg2+ may increase the dependence of the kissing-duplex transition on NC binding thus preventing

166 loop in an SL1 monomer that may promote the kissing-duplex transition.

167 putative expression of estrogen receptors in kiss-expressing cells and, finally, we investigated whet

168 d relationship between income inequality and kissing frequency (r = 0.67, BCa 95% CI[0.32,0.89]) was

169 Most teleosts have two kiss genes, kiss1 and kiss2, but their sites of expressi

170 signal (DIS) that has been proposed to form kissing hairpin and/or extended duplex intermolecular co

171 Kissing hairpin interactions form when the loop residues

172 follows: the loop-loop interactions found in kissing hairpins or the stem-stem interactions of a cruc

173 Kisspeptins (Kiss) have been shown to be key components in the regula

174 We investigated head-turning bias in both kiss initiators and kiss recipients for lip kissing, and

175 nt bias in head-turning to the right in both kiss initiators and kiss recipients, with a tendency amo

176 dimerization is initiated through an RNA-RNA kissing interaction formed via the dimerization initiati

177 e distribution, the kinetics of breaking the kissing interaction is calculated as a function of force

178 The force required to disrupt the kissing interaction of the two structures, the rip force

179 This loop has been implicated in a kissing interaction with a complementary stem-loop struc

180 interacts with the CRE in the absence of the kissing interaction.

181 ucleosides and examined their effects on the kissing interaction.

182 The inhibitor induces a "kissing" interaction between two capsid dimers.

183 hairpins participate in intermolecular cross-kissing interactions (SL-C to SL-D' and SLC' to SL-D) an

184 In a unimolecular context, kissing interactions are important for tertiary folding

185 the cellular environment and that loop-loop kissing interactions involving Stem 3 modulate -1 PRF an

186 s and that 5BSL3.2 engages simultaneously in kissing interactions using its apical and internal loops

187 another (similar to characterized stem-loop kissing interactions) forming a hitherto undescribed, hi

188 t allows us to treat a variety of miRNA-mRNA kissing interactions, which have been ignored in the cur

189 t, in isolated RNAs, are capable of forming "kissing" interactions stabilized by two intermolecular G

190 Oligomerization of Fob1 caused synaptic (kissing) interactions between pairs of terminator (Ter)

191 way that it can engage loop-loop (so-called kissing) interactions with RNA hairpins displaying partl

192 Romantic mouth-to-mouth kissing is culturally widespread, although not a human u

193 allow, and blow normally whereas pouting and kissing is still difficult.

194 Our data reveal that kissing is valued more in established relationships than

195 iss1 gene and a single Kiss1r gene, multiple Kiss ligand and receptor genes are found in nonmammalian

196 t allows RNA dimerization via intermolecular kissing loop (KL) base pairing.

197 ribozyme core via three tertiary contacts: a kissing loop (P14), a metal core-receptor interaction, a

198 structure assembled from common elements, a kissing loop and two three-way junctions.

199 ormation of an extended duplex rather than a kissing loop complex because the short stems are not sta

200 The degree of cation accumulation around the kissing loop complex was also inversely proportional to

201 two other RNAs, an adenine riboswitch and a kissing loop complex, become more stable by 2-3 kcal/mol

202 m-loop interface to allow the formation of a kissing loop complex.

203 op interface is critical in the formation of kissing loop complexes and that in the absence of Mg(2+)

204 Kissing loop complexes are loop-loop complexes where two

205 Our results show that although kissing loop complexes form more readily in the presence

206 rived from the ColE1 plasmid to associate as kissing loop complexes in the presence and absence of di

207 is exposed, and the genome is competent for kissing loop dimerization and packaging into assembling

208 interactions such as intermolecular RNA-RNA kissing loop dimerization, RNA-protein binding, and intr

209 Readily reversible kissing loop formation combined with slow cleavage of th

210 However, we showed that kissing loop formation improves ligand binding efficienc

211 An RNA kissing loop from the Moloney murine leukemia virus (MML

212 A long-range, 5-nucleotide, base-pairing kissing loop interaction between the 3'BTE and a 5'UTR s

213 Recognition of the substrate involves a kissing loop interaction between the substrate and the c

214 To analyze the role of the kissing loop interaction in the riboswitch regulatory me

215 We found that the kissing loop interaction is not essential for ligand bin

216 Escherichia coli btuB riboswitch contains a kissing loop interaction that is in close proximity to t

217 A kissing loop is a highly stable complex formed by loop-l

218 ubstrate stem loop I (SLI)-stem loop V (SLV) kissing loop junction of the Varkud Satellite ribozyme h

219 to analyze the binding and dynamics of this kissing loop junction.

220 ranscriptional expression possibly through a kissing loop model bridging TRX 3'- and 5'-UTRs through

221 he basis of the palindromic nature of SL1, a kissing loop model has been proposed.

222 indromic nature of the apical loop of SL1, a kissing loop model has been proposed.

223 ection pathway resembles that of an isolated kissing loop similar to P14, and the rate along the indu

224 alter the observed counterion specificity in kissing loop stability.

225 These oligomers self-associate to form a kissing loop that thermally rearranges into a more stabl

226 ed metastable configuration consisting of a "kissing loop" stabilized by flanking helical domains; th

227 In contrast, a 'kissing loop' interaction between the terminal loop of S

228 The presence of the 'kissing loop' interaction inhibited the formation of SL9

229 By monitoring the folding of the aptamer, kissing loop, and riboswitch expression platform, we est

230 n that the Tar-Tar(*) complex, an archetypal kissing loop, can form without Mg(2+), so long as high c

231 ion of the MMLV dimerization initiation site kissing loop.

232 involves the fast formation of an unstable "kissing" loop intermediate, followed by a slower convers

233 the fast formation of an unstable extended "kissing" loop intermediate, followed by a slower strand

234 d that SARS-CoV RNA dimers assemble through 'kissing' loop-loop interactions.

235 nucleocapsid (NC) to the hinge region of the kissing-loop (KL) dimer formed by stemloop 1 (SL1) can h

236 complementary sequences to form a metastable kissing-loop (KL) dimer.

237 sociated with the disruption of a long-range kissing-loop (KL) interaction is substantially decreased

238 it requires as much force to break the MMLV kissing-loop complex as is required to unfold an 11-bp R

239 in the unusual stability of other retroviral kissing-loop complexes such as the HIV dimerization site

240 we use to construct a Markov state model for kissing-loop dissociation.

241 ly as an unstructured spacer to position the kissing-loop elements.

242 Using this system, we determine that the kissing-loop interaction between 5BSL3.2 and 3' SL2 is r

243 bilizes an unusual long-range intramolecular kissing-loop interaction that controls mRNA expression.

244 o engages in a stable, long-distance RNA-RNA kissing-loop interaction with a 12-bp 5'-coding-region h

245 contains an apical loop capable of forming a kissing-loop interaction with a 5' proximal hairpin and

246 Most 3'CITEs participate in a long-distance kissing-loop interaction with a 5' proximal hairpin to d

247 the PEMV PTE that engages in a long-distance kissing-loop interaction with a coding sequence hairpin

248 or eIF4F complex and to engage in an RNA-RNA kissing-loop interaction with a hairpin loop located at

249 The resulting kissing-loop interaction, common in tick-borne flaviviru

250 iform is not an absolute requirement for the kissing-loop interaction, suggesting a model in which tr

251 ed through an artificially designed branched kissing-loop motif, involving Watson-Crick base pairing

252 Finally, we show that both the poly(U) and kissing-loop RNA elements can function outside of their

253 slated region (3'UTR), the ribosome-binding, kissing-loop T-shaped structure (kl-TSS) and eukaryotic

254 EMV) 3' translational enhancer, known as the kissing-loop T-shaped structure (kl-TSS), binds to 40S s

255 tifunctional element is designated a kl-TSS (kissing-loop T-shaped structure) to distinguish it from

256 B coding region, 5BSL3.2, forms a functional kissing-loop tertiary structure with part of the 3' NTR,

257 two classes of tertiary interactions, namely kissing loops and a pseudoknot.

258 The cationic uptake by kissing loops depends on the number of basepairs between

259 nal self-assembly of tiles based on branched kissing loops, and show that tiles inserted into a trans

260 that were fluorescently labelled in and near kissing loops.

261 Use and appreciation of kissing may therefore vary according to whether the envi

262 that a family of F-box proteins, called the kiss me deadly (KMD) family, targets type-B ARR proteins

263 wide kinase-interacting substrate screening (KISS) method, we identified that YopO phosphorylates a w

264 ty of both hairpin functional nucleic acids, kissing motifs, and enzyme-based signaling systems, ELAK

265 CaMP6 fiber photometry, we find that the ARN(KISS) neuron population exhibits brief ( approximately 1

266 s aimed at resetting the activity of the ARN(KISS) neuron population with halorhodopsin were found to

267 These observations indicate the ARN(KISS) neurons as the long-elusive hypothalamic pulse gen

268 The selective optogenetic activation of ARN(KISS) neurons for 1 min generated pulses of LH in freely

269 role of the arcuate nucleus kisspeptin (ARN(KISS)) neurons in LH pulse generation.

270 pi6), we show that Spi6 protects DC from the kiss of death by inhibiting granzyme B (GrB) delivered b

271 ugh contact-mediated cytotoxicity (so-called kiss of death) has proved to be controversial.

272 , G4-based secondary structure we term a 'G4 Kiss or G4K'.

273 ignificant differences in the frequencies of kissing or sexual and relationship satisfactions.

274 mes to late endosomes followed by transient (kissing) or complete fusions between late endosomes and

275 er risk of infection than those reporting no kissing (P < .01).

276 matched odds ratio [mOR], 15.0; P = .03), >1 kissing partner (mOR, 13.66; P = .03), and attending bar

277 rtners (P = .046, for trend) or open-mouthed kissing partners (P = .023, for trend) but not vaginal s

278 These ARN(KISS) population events were found to be near-perfectly

279 his issue of Genes & Development, Vitali and Kiss (pp.

280 competition among large groups and predicts kissing prevalence in remote societies, this gesture may

281 oupled receptor, GPR54 (kisspeptin receptor, Kiss-R), are critical for the control of reproduction in

282 d to saporin (SAP) to selectively inactivate Kiss-R1-expressing neurons.

283 ead-turning bias in both kiss initiators and kiss recipients for lip kissing, and took into considera

284 s and kiss recipients, with a tendency among kiss recipients to match their partners' head-turning di

285 ing to the right in both kiss initiators and kiss recipients, with a tendency among kiss recipients t

286 nding hypothesis of the function of romantic kissing, relative poverty (income inequality) predicts f

287 Two dimerization initiation site kissing sequences, Mal and Lai, have been found in most

288 ic RNA and subgenomic RNA1 via long-distance kissing stem-loop interaction to facilitate translation.

289 The BTE forms a kissing stem-loop interaction with the 5' UTR to mediate

290 orientations, for pseudoknots and loop-loop kissing structures.

291 rated an increase in GnRH gene expression by Kiss suggesting regulation of GnRH at both the secretory

292 d by Santiago Ramon y Cajal as "protoplasmic kisses that appear to constitute the final ecstasy of an

293 Saccharomyces cerevisiae promoted chromosome kissing that initiated rDNA recombination and controlled

294 ity to the mRNA 5' end of the stem-loop that kisses the 3' BTE.

295 in which the CROPS domain bends toward and "kisses" the delivery domain.

296 mRNA, protein, and binding to the KsRE after Kiss treatment were demonstrated.

297 To define the mechanism of regulation, Kiss was first shown to induce nucleosome-depleted DNA w

298 Kissing was a significant risk for primary EBV infection

299 Fob1 oligomerization and Ter-Ter kissing were regulated by intramolecular inhibitory inte

300 Subjects reporting deep kissing with or without coitus had the same higher risk