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1 natural immunity conferred by carriage of N. lactamica.
2 the challenge group became colonized with N. lactamica.
3 of 17 strains examined but was present in N. lactamica.
4 rizontal gene transfer with the commensal N. lactamica.
5  and Y, Neisseria gonorrhoeae, and Neisseria lactamica.
6 restriction endonuclease NlaIII in Neisseria lactamica.
7 methyltransferases, including M.NlaIII in N. lactamica.
8  of whom seven were already colonised with N lactamica.
9 ly in participants who were colonised with N lactamica.
10            Two weeks after inoculation of N. lactamica, 33.6% (48/143) of the challenge group became
11 lted in the acquisition of 26 consecutive N. lactamica alleles.
12                  In the second study, anti-N lactamica and anti-N meningitidis serum IgG titres incre
13  between colonization by commensal Neisseria lactamica and meningococcal disease, we investigated whe
14 in four individuals cocolonized by Neisseria lactamica and Neisseria meningitidis One HGT event resul
15 h all participants were reinoculated with N. lactamica and resampled 2 weeks later.
16 f 20 participants who were inoculated with N lactamica and six (55%) of 11 participants who were inoc
17 gitidis, Neisseria gonorrheae, and Neisseria lactamica are often regarded as highly recombining bacte
18 male participants naturally colonised with N lactamica at baseline were followed up, but not inoculat
19             Here, we show that engineered N. lactamica, chromosomally transformed to express a hetero
20 udy, we measured the effect of duration of N lactamica colonisation on seroconversion by terminating
21            The primary endpoint was infant N lactamica colonisation.
22 s were inoculated, of whom 15 (71%) became N lactamica-colonised, and no sustained N lactamica Y92-10
23 e other commensals, whole bacteria of two N. lactamica commensal strains had low inflammatory potenti
24                      Genetically modified N. lactamica could therefore be used to generate beneficial
25 nce of adaptive cell-mediated immunity to N. lactamica during the peak age of carriage.
26                                  When the N. lactamica expressing the meningococcal antigen Neisseria
27 ly in participants who were colonised with N lactamica for 14 days.
28 strain, colonization with NadA-expressing N. lactamica generated NadA-specific immunoglobulin G (IgG)
29 hree additional adverse events (two in the N lactamica group and one in the PBS group) and no serious
30 8 and AcrIC9 that strongly inhibit Neisseria lactamica I-C function.
31  with either 10(5) colony-forming units of N lactamica in 1 mL phosphate-buffered saline (PBS) or 1 m
32 us of N. meningitidis, N. gonorrheae, and N. lactamica in the light of the PCE model.
33 imed to evaluate whether colonisation with N lactamica induces N lactamica-specific B-cell responses
34 isation by the commensal bacterium Neisseria lactamica inhibits colonisation by Neisseria meningitidi
35 ific plasma cells and memory B cells after N lactamica inoculation (day 7-28) compared with baseline
36                       We hypothesised that N lactamica inoculation in pregnancy induces mother-to-inf
37                                 Intranasal N lactamica inoculation is a safe and well characterised c
38                                    Neisseria lactamica is a commensal bacteria that colonizes the hum
39                                    Neisseria lactamica is a harmless pharyngeal commensal that correl
40 e inhibition of meningococcal carriage by N. lactamica is even more potent than after glycoconjugate
41 urified lipooligosaccharide nor PorB from N. lactamica is likely to be responsible for this activity.
42                                    Neisseria lactamica is not associated with disease, but N. meningi
43 loid, while the commensal organism Neisseria lactamica maintains chromosomes in single copy.
44 ead, outer membrane vesicles derived from N. lactamica mediate a B cell-dependent proliferative respo
45  to N meningitidis after colonisation with N lactamica might be due to cross-reactive adaptive respon
46 dered to be nonpathogenic, such as Neisseria lactamica, N. subflava, and N. sicca.
47 h 3, 2019, 31 were randomly assigned (n=20 N lactamica, n=11 PBS).
48  six commensal Neisseria isolates, Neisseria lactamica, Neisseria subflava, Neisseria flava, Neisseri
49 tion of a minimal CRISPR-Cas3 from Neisseria lactamica (Nla) type I-C system to create targeted large
50                                    Neisseria lactamica or its components could be a novel bacterial m
51 milar or identical to alleles from Neisseria lactamica or Neisseria gonorrhoeae.
52 ulated with 10(4) colony-forming units of N. lactamica or were sham-inoculated, and carriage was moni
53                 However, commensal Neisseria lactamica organisms and purified N. lactamica PorB (publ
54     These effects are reproduced by whole N. lactamica organisms.
55 s abrogated the proliferative response to N. lactamica outer membrane vesicles, suggesting that this
56 sseria meningitidis (P < 0.05) and Neisseria lactamica (P < 0.002) (2-sided Fisher's exact test) was
57 eisseria lactamica organisms and purified N. lactamica PorB (published elsewhere as Nlac PorB) induce
58  loop residues have been switched between N. lactamica PorB and N. meningitidis PorB, we identified r
59 himeric N. meningitidis strain expressing N. lactamica PorB induces lower levels of interleukin 8 (IL
60                              Furthermore, N. lactamica PorB is a poor inducer of proinflammatory medi
61                     Our studies show that N. lactamica PorB is a TLR2 ligand but its binding specific
62 rolled infection of human volunteers with N. lactamica prevents colonization by Neisseria meningitidi
63 of human adults with the commensal Neisseria lactamica produced safe, sustained pharyngeal colonizati
64 I restriction enzyme isolated from Neisseria lactamica recognizes the sequence 5'-CATG-3', cleaving a
65    Crossover inoculation of controls with N. lactamica replicated the result.
66                                            N lactamica selective culture, genome sequencing, and sero
67                                       Anti-N lactamica serum IgG titres increased in seven (88%) of e
68 ther colonisation with N lactamica induces N lactamica-specific B-cell responses that are cross-react
69                                     Median N lactamica-specific IgG memory-B-cell frequencies (percen
70  endpoint was the frequency of circulating N lactamica-specific plasma cells and memory B cells after
71 lines compared with peak post-colonisation N lactamica-specific plasma-cell frequencies (per 10(5) pe
72                    Analysis of the LPS of N. lactamica strain NL4 revealed two distinct and interconv
73                   Conversely, non-Y92-1009 N lactamica strain sharing was observed in four (57%) of s
74 ghly conserved property and is present in N. lactamica strains derived from diverse clonal complexes.
75 cB were also found in N. meningitidis and N. lactamica strains, and an insertion in the meningococcal
76 ve an influx of oral taxa, such as Neisseria lactamica, Streptococcus, Prevotella nanceiensis, Fusoba
77 he 17 participants who were colonised with N lactamica, the median baselines compared with peak post-
78                                 In Neisseria lactamica, this promoter involves another dcw-associated
79 of N. polysaccharea (Np), four strains of N. lactamica, three strains of N. subflava and nine strains
80 tion in pregnancy induces mother-to-infant N lactamica transmission postnatally.
81 infant M catarrhalis and sibling-to-mother N lactamica transmission, challenges conventional percepti
82         Lack of sustained mother-to-infant N lactamica transmission, despite evidence supporting moth
83 carriage was only observed in carriers of N. lactamica, was due both to displacement of existing meni
84  of N. meningitidis, the commensal Neisseria lactamica, which chiefly colonizes infants not adults.
85 gitidis, Neisseria gonorrhoeae and Neisseria lactamica, which exhibit very different phenotypes with
86 nd signaling, commensal organisms such as N. lactamica would benefit from expressing a product that i
87 station with 10(5) colony-forming units of N lactamica Y92-1009 at University Hospital Southampton Cl
88 s model in pregnancy is feasible, and that N lactamica Y92-1009 can safely and efficiently colonise p
89 me N lactamica-colonised, and no sustained N lactamica Y92-1009 transmission to their infants was obs
90 G titres increased in seven (88%) of eight N lactamica Y92-1009-colonised female participants, but no