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1 stitutional symptoms, and elevated values of lactic dehydrogenase.
2 ll viability was accompanied by an increased lactic dehydrogenase activity in the culture medium.
3  flow, reduced release of the cardiac enzyme lactic dehydrogenase, and reduced myocardial concentrati
4 (1%) or CORM-3 (10-100 iM) and cytotoxicity (lactic dehydrogenase assay) measured at 24 hrs.
5         Bilirubin, alkaline phosphatase, and lactic dehydrogenase concentrations could not be measure
6  age >/= 60 years, stages III to IV disease, lactic dehydrogenase (LDH) > normal, extranodal sites (E
7 5%] of eight, P: <.005) and higher levels of lactic dehydrogenase (LDH) (339 U/L +/- 65 [SD] vs 258 U
8 inase (AST), alanine transaminase (ALT), and lactic dehydrogenase (LDH) (but not alkaline phosphatase
9 oorer failure-free survival (FFS) included a lactic dehydrogenase (LDH) level greater than normal (P
10 horter survival included age >/=60, elevated lactic dehydrogenase (LDH) or beta-2-microglobulin (beta
11                            The admission ALT/lactic dehydrogenase (LDH) ratio, when levels of both en
12 dependent increase in cell death measured by lactic dehydrogenase (LDH) release and vital dye exclusi
13         Cell injury was assessed in terms of lactic dehydrogenase (LDH) release, 45Ca2+ uptake, and M
14 ers such as creatine kinase (CK), myoglobin, lactic dehydrogenase (LDH), and N-terminal pro brain nat
15                   A dose-related increase in lactic dehydrogenase (LDH)-5 isoenzyme concentrations wa
16   Combining elevated beta2 microglobulin and lactic dehydrogenase (neither elevated v one or both ele
17 ded sex, stage, Karnofsky performance score, lactic dehydrogenase, number of extranodal sites, B symp
18 (glyceraldehyde-3-phosphate dehydrogenase or lactic dehydrogenase) or even 3 mM magnesium ions can al
19                                              Lactic dehydrogenase (P < 0.01) and D-dimer (P < 0.01) l
20 id not injure T84 monolayers, as assessed by lactic dehydrogenase release and protein synthesis assay
21 ts of cell integrity (ATP levels and percent lactic dehydrogenase release).
22 ubular segment injury (up to 92, 71, and 30% lactic dehydrogenase release, respectively) and massive
23 etastases and the presence of elevated serum lactic dehydrogenase to be used in the M category; (4) a
24 arameters of the labile enzyme rabbit muscle lactic dehydrogenase were determined as a function of th