ライフサイエンスコーパス: laminectomy

1 rapy than were seen in children managed with laminectomy.

2 chemotherapy compared to those managed with laminectomy.

3 the lumbosacral spinal cord was exposed by a laminectomy.

4 (62%) had surgery, most often decompressive laminectomy (320 of 394 [81%]).

5 nal stenosis to undergo either decompressive laminectomy alone (decompression-alone group) or laminec

6 physical health-related quality of life than laminectomy alone.

7 l surface of C2 spinal cord was exposed by a laminectomy and an electrode placed for recording evoked

8 exposing the spinal cord at T8-T10 level by laminectomy and applying 35 g clip for 1 min.

9 Also in these neurons at 1 week after laminectomy and SCI, Kv3.4 channel inactivation is impai

10 IS) into the thoracic gray matter (T8-9), 2) laminectomy and spinal exposure and penetration without

11 vaginal, and abdominal hysterectomy, colon, laminectomy, and spinal fusion surgeries.

12 Three days after a standard contusive SCI or laminectomy, animals received intraperitoneal BrdU injec

13 ce received complete crush injury or control laminectomy at either thoracic level 3, which disrupts d

14 of 24 assessable patients who had undergone laminectomy developed scoliosis, whereas spinal deformit

15 Bony realignment and laminectomy did not effectively lower intraspinal pressu

16 l laminectomy while lesioned rats received a laminectomy followed by complete spinal transection.

17 The patient subsequently underwent T6 laminectomy followed by treatment with a glucocorticoid

18 The patient subsequently underwent T6 laminectomy followed by treatment with glucocorticoids a

19 reduce soft-tissue trauma compared with open laminectomy for lumbar spinal stenosis (LSS).

20 ive care or surgery (primarily decompressive laminectomy for stenosis and decompressive laminectomy w

21 At 2-6 weeks after laminectomy, however, Kv3.4 channel inactivation returns

22 rative lumbar spinal stenosis, decompressive laminectomy improved symptoms more than nonoperative the

23 r spinal fusion in addition to decompressive laminectomy in patients with symptomatic lumbar grade I

24 nee replacement; lumbar, cervical, or sacral laminectomy; lower extremity arterial bypass surgery; op

25 rol groups consisted of animals subjected to laminectomy only (shams) and to lesion either untreated

26 ly activated in both models when compared to laminectomy-only uninjured rats.

27 ne of 49 assessable patients managed without laminectomy (P =.001).

28 ck or NFS session, rats were anesthetized, a laminectomy performed and extracellular single-unit reco

29 Following laminectomy, the spine is affixed to a stereotactic fram

30 sis, the addition of lumbar spinal fusion to laminectomy was associated with slightly greater but cli

31 A laminectomy was performed to expose the spinal cord and

32 Laminectomy was potentially detrimental by exposing the

33 undergoing MI tubular decompression or open laminectomy were included.

34 Control rats received a T6 vertebral laminectomy while lesioned rats received a laminectomy f

35 e laminectomy for stenosis and decompressive laminectomy with fusion for stenosis associated with deg

36 Dorsal surgery involved laminectomy with instrumented fusion or open-door lamino

37 nectomy alone (decompression-alone group) or laminectomy with posterolateral instrumented fusion (fus

38 Treatment was standard decompressive laminectomy (with or without fusion) or usual nonsurgica