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1 dividuals will spontaneously recover partial language function.
2 brupt and mild and affected motor-sensory or language function.
3 vival probability and improved cognitive and language function.
4 als evaluated the baseline and postoperative language function.
5 pecific influence of structure and myelin on language function.
6 le paradigms that probe different aspects of language function.
7 ranscription factor implicated in speech and language function.
8 lities potentially underlying differences in language function.
9 ave debated the role of key brain regions in language function.
10 d resection will spare areas associated with language function.
11 nization insufficiently account for observed language function.
12 eurodegenerative disease primarily affecting language function.
13 as of Foxp2 expression are critical to human language function.
14 ed by an isolated and gradual dissolution of language function.
15 ntal gyrus, which shared both inhibitory and language functions.
16 es in human-specialized social-emotional and language functions.
17  regions that contribute to oral and written language functions.
18    LNM scores did not improve prediction for language functions.
19 cal tissue can take on visual perception and language functions.
20 losal inhibition that does not contribute to language functions.
21 ort specific cognitive capabilities, such as language functions.
22  of the role of the hemispheres in different language functions.
23 es is not known but may relate to speech and language functions.
24 dentified the lateralization and location of language functions.
25 tical representation that underlies multiple language functions.
26 ntal areas in a task emphasizing "receptive" language functions.
27  of language is not uniformly detrimental to language functioning.
28 les, and this decline differentially affects language functioning.
29 c tissue without compromising post-operative language functioning.
30 e causal relations between brain regions and language functions absent in functional neuroimaging, bu
31 ds are required to improve the evaluation of language function across a spectrum of psychiatric and n
32  the right hemisphere actively contribute to language function after a focal left hemisphere lesion.
33 e suggested that recovery or compensation of language function after a lesion in the left hemisphere
34                                     To study language function after brain-tumor resection with langu
35 in the white matter, and how these relate to language function after surgery remain unknown.
36  prosody and also typically left-lateralized language functions after an early LH stroke?
37                                  Maintaining language functions after left hemisphere lesions has bee
38 tion at a site interrupted motor, sensory or language function, afterdischarges were more likely to o
39    While LF depict positive association with language function and cognitive flexibility, LR shows a
40 ere less likely to lose their gross motor or language function and demonstrated significantly lower M
41                        Cerebral dominance of language function and hand preference are suggested to b
42 esidual and new neural mechanisms to improve language function and that neuroimaging modalities may h
43                                              Language functioning and NfL were the earliest abnormal
44  reproducible networks critical for specific language functions and often damaged in primary progress
45 ith speech and language therapy in enhancing language functions and quality of life in individuals wi
46 ned into two cognitive composites -- one for language functions and the other for executive functions
47 pecifically involved in executive functions, language functions and verbal fluency to ascertain wheth
48 aled that his literacy skills, certain basic language functions and vocabulary development were arres
49 Function Classification in MLD), loss of any language function, and magnetic resonance imaging (MRI)
50 reatment optimize outcome, and that gains in language function are most likely to be seen years, rath
51     In central alexic syndromes, where other language functions are also involved, the emphasis has b
52                    Normal sex differences in language functions are disrupted in schizophrenia.
53 eft hemisphere sustained in early childhood, language functions are likely to reorganize and develop
54 udies of healthy people have shown that some language functions are mediated by the right hemisphere
55         Christiansen & Chater (C&C) envision language function as a hierarchical chain of transformat
56 schizophrenia is associated with deficits in language function, as well as structural and functional
57       We found that individual cognitive and language functions assessed at the age of 2 years were r
58 F), are responsible for the deterioration in language functioning associated with age.
59 ng and dissociations from other frontal lobe language functions, (b) the organization of categories o
60 ve provided 3-D and 4D mapping of speech and language function based upon the results of direct corti
61 rome characterized by progressive decline in language function but relative sparing of other cognitiv
62 ocal dementia syndrome with deterioration of language functions but relative preservation of other co
63 by impairments in social, behavioral, and/or language function, but postmortem studies indicate that
64  right hemisphere may actively contribute to language functions by supporting disrupted processing in
65 brain plasticity process with acquisition of language functions by the non-dominant hemisphere speech
66 icated in autism spectrum disorder, in which language function can be severely affected.
67 tation, which is the common basis of the two language functions, can be generalized to fundamental ce
68 s in various white matter tracts relevant to language function despite no significant difference in V
69 entifying the language cortex and preserving language function during epilepsy and neuro-oncological
70  the stability of intellectual, academic and language functioning during development in children with
71  characterized by the gradual dissolution of language functions, especially in the early stages of di
72 also time and patient age, sites specific to language function for presurgical evaluation of focal ep
73 ighlights the importance of right hemisphere language functions for successful social communication a
74 , both historical and contemporary models of language function have assigned primacy to superior temp
75  current dual-steam neurocognitive models of language function have coalesced around the view that di
76 es while other aspects of cognition, such as language functioning, have received less attention.
77 the left hemisphere, which typically support language functions, have a more protracted course of mat
78 rain lesions in regions subserving motor and language functions help explain stroke severity in both
79 ort here a direct demonstration of preserved language function in a stroke patient (LF1) apparently d
80 fies protein sequence, structure and natural language function in a trimodal language model through c
81 y meaningful slowing of decline of motor and language function in children with CLN2 disease.
82 rences for future identification of aberrant language function in children with various disorders.
83  intracranial tumours significantly improves language function in dysphasic patients, and is unlikely
84 d in some centres to lateralize and localize language function in epilepsy surgery candidates.
85 al systems, which play a significant role in language function in modern humans, originally evolved t
86 cephalography offers the prospect of mapping language function in real time.
87 motor functioning than with either memory or language functioning in men.
88 he immediate effects of resective surgery on language functions in a heterogeneous group of patients
89 her asymmetric brain regions associated with language functions in humans including the planum tempor
90 of domain-general capacities underlying core language functions in humans.
91 ific communication in non-human primates and language functions in humans.
92 ysphasic patients, and is unlikely to impair language functions in non-dysphasic patients.
93 ization of eloquent cortex enables rescue of language functions in patients who sustain brain injury.
94 ues, highlighting the presence of supportive language functions in the right hemisphere.
95 ciation of 3'-SL and cognition, particularly language functions, in typically developing children who
96 igate neurofunctional correlates of improved language functions induced by atDCS over a core language
97                Research into the recovery of language function is almost exclusively focused on monol
98 eceptive and, to a lesser extent, expressive language functions is attributed to plasticity of the ri
99 jority of participants in the SLI groups had language function lateralized to the right hemisphere (5
100 n characterized as a selective impairment in language function, left hemisphere lesions may cause imp
101                             The higher order language functions mediated by the right hemisphere are
102 f abnormal lateralization of left hemisphere language functions need to take account of the consequen
103 erial, which will indicate the postoperative language function of future patients and facilitate exte
104 emporal processing tasks predicted receptive language function (p<0.05).
105 t these are predictive of certain aspects of language function, recovery and rehabilitation.
106                              Many studies of language function rely on priming paradigms that use pai
107             One week after surgery, baseline language function remained in 194 patients (77.6%), it w
108                                        Other language functions remained severely impaired until at l
109     In patients with behavioural evidence of language function, responses to language and music were
110 aphasic patients, who have largely recovered language function, show increased frontoparietal integra
111 e findings are consistent with the different language functions subserved by subcomponents of the lan
112 tes of recovery and relationships to overall language function, suggesting that specific domains diff
113 quired to localize accurately those specific language functions that are most at risk following surge
114 ndividuals show an atypical brain control of language functions that differs from the typical lateral
115 mary progressive aphasia (PPA), a decline in language functions that remains isolated for at least 2
116 oral connectivity displayed better receptive language function; these patients also showed greater th
117 nsequences of a failure of lateralization of language functions to the right as well as the left hemi
118     Study 1 examined IQ, academic skills and language functions using the same test version over the
119                                              Language function was most affected, with 35% of 146 chi
120                                 In contrast, language function was relatively preserved in the female
121 t predictor of postoperative improvements in language function was that of length of time since surge
122                                              Language functioning was measured using the Peabody Pict
123 mporale volumes with performance on tests of language function were examined.
124 athway of the cANN was essential for the two language functions, whereas cANN variants with further b
125 entia characterized by gradual impairment of language function with initial sparing of the memory dom
126 rhagic stroke and evaluated their speech and language function within 5 days using the Quick Aphasia
127  extent) and speech/language domain (overall language function, word comprehension, sentence comprehe

 
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