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1 of the MCH gene in mice cause hypophagia and leanness.
2  loss of MCH neurons can lead to an acquired leanness.
3 ctile dysfunction were physical activity and leanness.
4 ng a single orexigenic peptide can result in leanness.
5 man brown fat activity levels correlate with leanness.
6 riate physiological conditions is related to leanness, aerobic fitness, and insulin sensitivity.
7  functional analyses of this novel candidate leanness and antidiabetic gene.
8 riction that was started in midlife promoted leanness and glucose tolerance in aged males but not fem
9 ased risk of diabetes in humans, and promote leanness and glycaemic control in both rodents and human
10               Here, we show that PR promotes leanness and glycemic control in 3xTg mice, specifically
11                                         Body leanness and hypertension are additional etiological fac
12 se) responsible for the Fob3b2 QTL effect on leanness and improved metabolic parameters.
13                   Reduced histidine promotes leanness and increased energy expenditure in male mice.
14 nstream gene expression changes that promote leanness and increased energy expenditure.
15 mutation, designated supermodel, that caused leanness and kyphosis associated with myopathy and adipo
16 We show that Tbc1d1, a gene known to mediate leanness and linked to obesity, is down-regulated in the
17 ue (BAT) has been suggested as a promoter of leanness and metabolic health.
18 ndividual cell types in the adipose niche in leanness and obesity.
19 +/- 0.4 y were studied according to parental leanness and overweight or obesity.
20 nditure, and increased satiety, resulting in leanness and reduced body fat.
21 der bidirectional selection for the distinct leanness and reproduction traits between CHD and EUD.
22  in AgRP neurons partially recapitulates the leanness and resistance to diet-induced obesity of RIIbe
23         In contrast, C. albicans resulted in leanness and resistance to diet-induced obesity.
24 of NEAT dissipates excess energy to preserve leanness and that failure to activate NEAT may result in
25           We examined the health benefits of leanness and the hazards of obesity while simultaneously
26 ts genetic loss results in reduced appetite, leanness, and blood glucose burden, leading to protectio
27                       The health benefits of leanness are limited to fit men, and being fit may reduc
28 For each woman, ponderal index (indicator of leanness at birth) was calculated.
29 tary patterns or nutrient intakes that favor leanness [BMI (in kg/m(2)) </=25] in free-living populat
30                                              Leanness can result from deficiency of stored energy or
31 -deficient mice have reduced body weight and leanness due to hypophagia (reduced feeding) and an inap
32 ylcarboxypeptidase (PRCP) is associated with leanness, hypertension, and thrombosis.
33 rons of mice results in reduced food intake, leanness, improved glucose homeostasis, and increased se
34 rons of mice results in reduced food intake, leanness, improved glucose homeostasis, and increased se
35                        Mex3c mutation caused leanness in both heterozygous and homozygous transgenic
36 in-1 (UCP1) and BAT activity correlates with leanness in human adults.
37 ncluding obesity, in mice and correlate with leanness in humans.
38 enomic and non-genomic actions of E2 promote leanness in OVX mice independently of reduced energy int
39                                              Leanness in PTP-1B-deficient mice is accompanied by incr
40 ants, previously shown to be associated with leanness in the HERITAGE Family Study, are also associat
41 l CoA carboxylase-2 (Acc2) reportedly causes leanness in the setting of hyperphagia.
42             We found that a LP diet promotes leanness, increases energy expenditure, and improves gly
43                    Clinical features include leanness, intellectual disability, autistic features and
44 c when maintained on regular chow, and their leanness is a consequence of hyperactivity and altered m
45 otic and dizygotic twin pairs concordant for leanness or obesity, and their mothers, to address how h
46           In the Augalpha-knockout mice, the leanness phenotype is coupled to increased physical acti
47                                              Leanness secondary to caloric restriction is known to be
48        Because the MCH(-/-) model represents leanness secondary to increased energy expenditure rathe
49 y metabolism evolved to favor adiposity over leanness, the availability of palatable, easily attainab
50 me characterized by low appetite and extreme leanness; this is phenocopied by mice carrying similar m
51 Furthermore, cecal transplantation conferred leanness to germ-free recipients.
52                                              Leanness was characterized by hypophagia and increased e
53                                     Instead, leanness was secondary to a marked increase in energy ex
54 adipose-Stra6(-/-) mice fed chow resulted in leanness, which may contribute to their increased insuli
55 erested in determining whether this model of leanness would be associated with beneficial metabolic e