ライフサイエンスコーパス: learned helplessness

1 osure to inescapable electric shock in rats (learned helplessness).

2 e Th17 cell function, reduced Th17-dependent learned helplessness.

3 sed feeding (NSF), social defeat stress, and learned helplessness.

4 vioral paradigms for anhedonia, despair, and learned helplessness.

5 d induction of escape deficits in a model of learned helplessness.

6 sion, chronic unpredictable stress (CUS) and learned helplessness.

7 tidepressant effects of sleep deprivation on learned helplessness.

8 ct on the landscape of the hippocampus after learned helplessness.

9 roach to be critical to induce resilience to learned helplessness.

10 Whereas synaptic potentiation was linked to learned helplessness, a depression-like behavior, synapt

11 selectively bred line of rats susceptible to learned helplessness, a model of depression, presents an

12 ng biases, anhedonia, despair-like behavior (learned helplessness) - affords unique opportunities for

13 nce of the transcription factor DeltaFosB on learned helplessness, an animal model of affective disor

14 ed increased motivation for food, heightened learned helplessness and anhedonia, and altered stress s

15 fear processing, arousal, social avoidance, learned helplessness and anhedonia.

16 ibility to social aversion, "anhedonia," and learned helplessness and causes impaired glucocorticoid-

17 Mouse brain Th17 cells were elevated by learned helplessness and chronic restraint stress, two c

18 neuroregulation as a proximate mechanism in learned helplessness and conservation-withdrawal.

19 our lives, which might be related to social learned helplessness and depression.

20 lso has important implications for models of learned helplessness and depression.

21 nregulated in the hippocampus after both the learned helplessness and forced swim test (FST) paradigm

22 ing to identify gut bacteria associated with learned helplessness and to quantify the level of the qu

23 digm where no vehicle-treated mice developed learned helplessness, and impaired novelty suppressed fe

24 Animals underwent learned helplessness, and subsequently immobility time w

25 To evaluate learned helplessness behavior, we used an active avoidan

26 threshold unpredictable stress and increased learned helplessness behavior.

27 n brain areas that control the expression of learned helplessness behaviors.

28 in brain slices and can significantly reduce learned helplessness behaviour in rats.

29 Three groups were studied: (1) rats bred for learned helplessness (cLH); (2) rats resistant to learne

30 ed helplessness (cLH); (2) rats resistant to learned helplessness (cNLH); and (3) control Sprague Daw

31 rlier, in agreement with previously reported learned-helplessness effects.

32 heels showed antidepressant-like behavior in learned helplessness, forced-swim (FST) and tail suspens

33 Th17 cell production exhibited resistance to learned helplessness, identifying modulation of RORgamma

34 Th17 cell administration promoted learned helplessness in 89% of mice in a paradigm where

35 unsolvable anagrams have been used to induce learned helplessness in humans, this finding may provide

36 Inescapable stress can induce learned helplessness in many species of animals.

37 r CIS, specifically: struggling, aggression, learned helplessness, inhibitory avoidance, and escape b

38 Learned helplessness is a phenomenon which has some beha

39 fect in behavioral models of depression, the learned helplessness (LH) and forced swim test (FST) par

40 al consequences of uncontrollable stress, or learned helplessness (LH) behaviors, are thought to invo

41 he nucleus (DRN) are implicated in mediating learned helplessness (LH) behaviors, such as poor escape

42 e tested the response of brain FoxO3a in the learned helplessness (LH) paradigm and tested signaling

43 lase promoter, showed protection against the learned helplessness (LH) paradigm, an animal model to t

44 ST), the tail suspension test (TST), and the learned helplessness (LH) paradigm-as well as in the fem

45 underlying the acquisition and expression of learned helplessness (LH), a robust stress model.

46 g a chronic social defeat (SD) stress model, learned helplessness (LH), and a chronic corticosterone

47 r either contextual fear conditioning (CFC), learned helplessness (LH), stress enhanced fear learning

48 sfer), whereas yoked rats failed to learn (a learned helplessness-like effect).

49 In learned helplessness, mice were exposed to a shuttle box

50 ens CREB-dynorphin influence behavior in the learned helplessness model and suggest that this signali

51 n onto LHb neurons contributes to the rodent learned helplessness model of depression.

52 Here we show that in two learned helplessness models of depression, excitatory sy

53 Both in learned helplessness paradigm and after peripheral infla

54 tidepressant-like behavioural effects in the learned helplessness paradigm and regulates molecular ev

55 th their susceptibility or resilience to the learned helplessness paradigm in a sex- and microbiota-d

56 d signaling following aversive learning in a learned helplessness paradigm in mice.

57 ificance of this transcription factor in the learned helplessness paradigm, a behavioral model of dep

58 proximate mediator of escape deficits in the learned helplessness paradigm, suggesting that neuronal

59 In the learned helplessness paradigm, the SCN-Bmal1-KD mice wer

60 rformance following inescapable shock in the learned helplessness paradigm.

61 c-Fos-like immunoreactivity (FLI) using the learned helplessness paradigm.

62 elease, compensating blunted plasticity in a learned helplessness paradigm.

63 Here we used the learned helplessness procedure in mice to examine the ro

64 ehavioral responses to stress induced by the learned helplessness procedure, in which animals are sub

65 hological (illness behavior, social support, learned helplessness, smoking, drinking), clinical, sero

66 d the role of ACh signaling in the mPFC in a learned helplessness task in which mice were exposed to

67 e, progressive ratio responding to food, and learned helplessness task were normal, such avolition-li

68 the Porsolt, novelty induced hypophagia, and learned helplessness tests in rats without exhibiting su

69 cientific contributions-from conceptualizing learned helplessness to uncovering the neural and immune

70 ing study of the effects of fluoxetine after learned helplessness training.

71 In foot-shock groups, learned helplessness was more robust in heterozygotes th