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1 ger tricuspid valve area z-score, and larger left ventricular volume.
2 in left atrial pressure and little change in left ventricular volume.
3 h assessment of the corresponding changes in left ventricular volume.
4 ity, respectively, independently of baseline left ventricular volume.
5 d after the surgery to assess the changes in left ventricular volume.
6 ium and concomitant substantial decreases in left ventricular volume.
7      Postmortem MI size was 13.5 +/- 2.6% of left ventricular volume.
8  therapy, resulting in a marked reduction of left ventricular volumes.
9 occurred early and did not result in smaller left ventricular volumes.
10 ular function, whereas L-NAME did not affect left ventricular volumes.
11 s]/[functional right ventricular+left atrial+left ventricular volumes]).
12 ntricular ejection fraction and reduction of left ventricular volumes after cardiac resynchronization
13 is study was to determine absolute right and left ventricular volume and ejection fraction measuremen
14                                              Left ventricular volume and ejection fraction were measu
15  were made of end-systolic and end-diastolic left ventricular volume and ejection fraction.
16 la: see text] ([Formula: see text]), and the left ventricular volume and endocardial area were found
17                    Blood 5-HT, infarct size, left ventricular volume and function, MR fraction and mi
18 rbidity and mortality also lead to decreased left ventricular volume and mass and a more normal ellip
19 nalysis was used retrospectively to evaluate left ventricular volume and mass and systolic pump funct
20 nts, we performed quantitative assessment of left ventricular volume and mass, wall thickness, segmen
21       Yet zoniporide prevented reductions in left ventricular volume and wall thickening while favori
22 e changes were accompanied by an increase in left ventricular volumes and a reduction in LVH and cham
23  Heart Association clinical class and higher left ventricular volumes and arterial pressure both at r
24  orifice area is likely related to decreased left ventricular volumes and decreased annular distentio
25                                              Left ventricular volumes and EF improved significantly a
26  provide rapid, reproducible measurements of left ventricular volumes and EF, as well as average bipl
27                                     Although left ventricular volumes and ejection fraction are stron
28 e echocardiographic measurements showed that left ventricular volumes and ejection fraction were sign
29            Gated tomographic data, including left ventricular volumes and ejection fraction, were pro
30 SPECT (QGS) has been used for computation of left ventricular volumes and ejection fraction.
31  with echocardiography for the assessment of left ventricular volumes and ejection fraction.
32                DMD patients exhibited normal left ventricular volumes and ejection fractions but mani
33 QGS, compared with cine MRI, for determining left ventricular volumes and ejection fractions in dogs
34 d blood-pool scintigraphy absolute right and left ventricular volumes and ejection fractions show goo
35 d by Doppler echocardiography and tonometry: left ventricular volumes and end-systolic elastance (Ees
36    The purpose of this study was to evaluate left ventricular volumes and function by gated SPECT usi
37 R) is the reference standard for quantifying left ventricular volumes and function from which SVi by
38 on, myocardial delayed enhancement (DE), and left ventricular volumes and function is unclear.
39 iographic (RT3DE) quantitative evaluation of left ventricular volumes and function.
40 e and clinically feasible tool for assessing left ventricular volumes and function.
41 for these baseline differences, reduction of left ventricular volumes and hypertrophy was greater and
42 omaterial, we observed substantially reduced left ventricular volumes and improved wall-motion scores
43 s associated with a significant reduction in left ventricular volumes and improvement in the ejection
44 gnificantly (P < 0.001) greater reduction in left ventricular volumes and increase in ejection fracti
45  cardiovascular events as well as changes in left ventricular volumes and infarct size.
46 e and post-cycle 17 for the determination of left ventricular volumes and left ventricular ejection f
47                      Both groups had similar left ventricular volumes and mass and normal global syst
48                                              Left ventricular volumes and mass were compared against
49                      Correlation between QGS left ventricular volumes and MRI was good for group I (e
50 chronic aortic regurgitation, a reduction in left ventricular volumes and regurgitant fraction, with
51 f CD4+ T cells was associated with increased left-ventricular volumes and deterioration of systolic f
52 na contracta area, mitral annular dimension, left ventricular volume, and inter-papillary muscle dist
53 iltration rate, and larger left atrial size, left ventricular volume, and mass.
54 terial tree, noninvasive aortic flow curves, left ventricular volumes, and E/e' as inputs were used t
55 th, tenting area, mitral annular dimensions, left ventricular volumes, and MR severity were quantifie
56  heart failure and correlated with survival, left ventricular volumes, and other markers of fibrosis.
57 ntricular reconstruction to CABG reduced the left ventricular volume, as compared with CABG alone.
58 Super-resolved low-resolution images yielded left ventricular volumes comparable to those from full-r
59 rease in RV volume and a 12+/-9% decrease in left ventricular volume compared to baseline.
60               For example, they have smaller left ventricular volumes compared to lowlanders despite
61                             Model inputs are left ventricular volume curves from cardiovascular magne
62 lic pressure, and non-invasive estimation of left ventricular volume data.
63                        Ejection fraction and left ventricular volumes determined by echocardiography
64                                              Left ventricular volumes, diastolic pressures, and diast
65                                 In contrast, left ventricular volume did not change significantly unt
66 d from automatically derived stress and rest left ventricular volumes during stress technetium-99m (T
67 condary outcome measures included changes in left ventricular volumes, echocardiographic peak global
68 LVEF varies with the change in end-diastolic left ventricular volume (EDV) and in particular to verif
69    In computer simulations the end-diastolic left ventricular volume (EDV) and the targeted LVEF (tLV
70                             We characterized left ventricular volume, effective arterial elastance, l
71 a hematologic malignancy (n=14), we measured left ventricular volumes, ejection fraction, and contras
72 rs in 100 patients, were analyzed to measure left ventricular volumes, ejection fraction, and global
73                                              Left ventricular volumes, ejection fraction, risk area (
74                         Assessments included left ventricular volumes, ejection fraction, strain, reg
75         There was a significant reduction of left ventricular volumes (end-systolic volume: -4.3 [11.
76 arning model that automatically extracts the left ventricular volume from the continuous image record
77 cular volume (HR, 1.25 [95% CI, 1.20-1.31]), left ventricular volume (HR, 1.27 [95% CI, 1.23-1.35]),
78  disease process (e.g., ejection fraction or left ventricular volume in HF).
79 n is a specific procedure designed to reduce left ventricular volume in patients with heart failure c
80 ng the first 5 minutes of untreated VF, mean left ventricular volume increased by 34%.
81 ropic effect on left ventricular mass index, left ventricular volume index and maximal left atrial an
82 ificant impact on the cardiac traits such as left ventricular volume index, parasternal long axis int
83  left ventricular end-diastolic pressure and left ventricular volume indexes.
84                      Secondary measures were left ventricular volumes, infarct size (assessed in a su
85 l and left ventricular pressure (Millar) and left ventricular volume (Leycom) were measured over 8 in
86 , high PVC burden was associated with larger left ventricular volumes, lower ejection fraction, and h
87                                              Left ventricular volume, LVEF and LV function categories
88 SPECT is a reproducible method for assessing left ventricular volume (LVV) and left ventricular eject
89                                              Left ventricular volume, mass and ejection fraction were
90 s undeniably the gold standard for assessing left ventricular volume, mass, and function, the assessm
91                                              Left ventricular volume, mass, ejection fraction and mit
92 mputing left ventricular function, including left ventricular volumes, mass and ejection fraction, ha
93 cluding age; duration of cardiomyopathy; and left ventricular volumes, mass, and ejection fraction (h
94 ng of these 2 groups were similar, including left ventricular volumes, mass, maximal wall thickness,
95 e for accurate and repeatable measurement of left ventricular volumes, mass, regional left ventricula
96 ncluding valve anatomy, flow quantification, left ventricular volumes, mass, remodeling, and function
97 magnetic resonance (CMR) imaging measures of left ventricular volumes/mass.
98 FISP produces small but significantly higher left ventricular volume measurements, as compared with F
99 is scanty regarding the accuracy of absolute left ventricular volumes measurements by this technique.
100 ction decreased strikingly and end-diastolic left ventricular volume more than doubled within 30 mins
101  and PCWP, particularly in those with larger left ventricular volumes, more impaired cardiac indexes,
102 % (p < 0.05) and an absolute reduction of IS/left ventricular volume of 6.2% (p = 0.15).
103   NIMR creation did not significantly change left ventricular volume or inter-papillary muscle distan
104 iency is a valvular disease characterized by left ventricular volume overload.
105  more severe MR (p = 0.0005) despite smaller left ventricular volumes (p = 0.005) and higher right ve
106 ion depth and tenting area (P<0.001), larger left ventricular volumes (P<0.001), and worse left ventr
107                      Furthermore, changes in left ventricular volume, pressure, and contractility aff
108  (r = 0.91, slope = 0.90, s.e.e. = 15.7) and left ventricular volumes (r = 0.96, slope = 0.88, s.e.e.
109                                          The left ventricular volumes ranged from 97.8 to 166.2 cm(3)
110            A servomotor was used to initiate left ventricular volume reduction (VR) at end systole, w
111 alve repair or replacement, cardiomyoplasty, left ventricular volume reduction surgery, and bridging
112 g thickening of the interventricular septum, left ventricular volume reduction, left ventricular hype
113 progressive remodeling process, with reduced left ventricular volumes, relatively maintained contract
114                     beta-AR blockers reduced left ventricular volume (reverse remodeling) and restore
115                                              Left ventricular volume studies were performed in 10 pat
116 lamipretide resulted in favorable changes in left ventricular volumes that correlated with peak plasm
117                                   At similar left ventricular volumes, their diastolic pressures are
118          The XVE was associated with greater left ventricular volume unloading.
119  assessed the effects of beta-AR blockade on left ventricular volume using isolated perfused hearts a
120 emodeling, assessed by infarct thickness and left ventricular volume, was mitigated by hydrogel treat
121                                              Left ventricular volumes were determined echocardiograph
122                                 In addition, left ventricular volumes were echocardiographically esti
123 lative MR studies, and ejection fraction and left ventricular volumes were further investigated using
124   In contrast, neither ejection fraction nor left ventricular volumes were independently predictive.
125      For evaluation of clinical performance, left ventricular volumes were measured, and statistical
126        Progressive and striking increases in left ventricular volumes were observed after successful
127 43 microm, P<0.02), whereas infarct size and left ventricular volumes were similar.
128                   Fully automated and manual left ventricular volumes were strongly correlated for en
129 es, namely aortic and mitral valve sizes and left ventricular volume, were significantly larger in th
130 provides highly reproducible measurements of left ventricular volumes, which are significantly larger
131                             VEGF-B increased left ventricular volume without compromising cardiac fun

 
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