ライフサイエンスコーパス: left ventricular wall

1 al infarction (>75% transmural extent of the left-ventricular wall).

2 roperties of Ca2+ channels across the canine left ventricular wall.

3 nd 41+/-1% of the inner circumference of the left ventricular wall.

4 osis was mid-myocardial in the basal-lateral left ventricular wall.

5 ght and heart/body weight ratio, and thicker left ventricular wall.

6 the epicardium and myocardium typical of the left ventricular wall.

7 n delivery and stimulate angiogenesis in the left ventricular wall.

8 in the anterior and lateral portions of the left ventricular wall.

9 nted images to annotate the location of each left ventricular wall.

10 during rapid growth and morphogenesis of the left ventricular wall.

11 ation of cardiomyocytes within the embryonic left ventricular wall.

12 ort-term effect of injecting material to the left ventricular wall.

13 demonstrated re-entry involving the inferior left ventricular wall.

14 reversing the direction of activation of the left ventricular wall.

15 reversing the direction of activation of the left ventricular wall.

16 t two of the three standard segments in each left ventricular wall.

17 interventricular septum than in the right or left ventricular walls.

18 s exhibited a nonprogressive thinning of the left ventricular wall and a concomitant decrease in card

19 argeted large-scale support of the infarcted left ventricular wall and improvement of heart function.

20 varying degrees was observed in the anterior left ventricular wall and septum.

21 function, interstitial fibrosis, a thickened left ventricular wall and tachycardia with reduced heart

22 severe dilated cardiomyopathy with thickened left ventricular walls and profound impairment of systol

23 Dispersion of APD90 across the left ventricular wall averaged 51+/-19 and 64+/-25 ms at

24 I3:p.R79C carriers had significantly thicker left ventricular walls compared with noncarriers while i

25 smural stress and strain distribution in the left ventricular wall considering it to be made of homog

26 Tissue samples isolated from the left ventricular wall demonstrate that sarcoplasmic reti

27 (HCM) is characterized by thickening of the left ventricular wall, diastolic dysfunction, and fibros

28 assess systolic and diastolic function, and left ventricular wall dimensions.

29 Surgical restoration of the left ventricular wall (Dor procedure) has been advocated

30 ent of a signaling pathway in the control of left ventricular wall edema during sepsis.

31 Interestingly, exaggerated left ventricular wall edema was not coupled with aggrava

32 ith a contrast of 0.25 were simulated in the left ventricular wall for 6 locations.

33 tion of noncontractile material to a damaged left ventricular wall has important effects on cardiac m

34 ity of repolarization that exists across the left ventricular wall, how this dispersion of repolariza

35 depth of ablated lesions reached 90% of the left ventricular wall in both normal and infarcted myoca

36 que were implanted into the anterior-lateral left ventricular wall in C57BL/6J (allogeneic model, n =

37 left ventricular mass, and thickening of the left ventricular wall in IAV-infected HF mice compared t

38 gene construct subepicardially in the canine left ventricular wall in situ.

39 strain rate revealed that the anterior free left ventricular wall is particularly susceptible to T2D

40 eight closed-chest dogs with acute posterior left ventricular wall ischemia either with (MR) or witho

41 ere changes in the following parameters: (a) left ventricular wall mass (LVM), measured in grams; (b)

42 y lead to subendocardial ischemia, increased left ventricular wall mass, and diastolic dysfunction, a

43 ith borderline increases in thickness of the left ventricular wall, mild morphologic expression of hy

44 l pattern that involves the proximal lateral left ventricular wall most severely, with relative spari

45 Left ventricular wall motion (WM) abnormalities have rec

46 Ponatinib produced segmental left ventricular wall motion abnormalities in 33% of wil

47 es, and a typical pattern of circumferential left ventricular wall motion abnormalities that usually

48 chocardiographic abnormalities that included left ventricular wall motion abnormalities, global left

49 A new regional left ventricular wall motion abnormality occurred more f

50 Patients (n = 119) with abnormal left ventricular wall motion and a left ventricular ejec

51 ntraoperative TEE for assessment of regional left ventricular wall motion and measurement of hemodyna

52 logy for the echocardiographic assessment of left ventricular wall motion based on acoustic quantific

53 heart disease or hypertension, Killip class, left ventricular wall motion index, and sex.

54 ted to estimated glomerular filtration rate, left ventricular wall motion index, sex, blood pressure,

55 lide MPI and two supplementary codes (add-on left ventricular wall motion or left ventricular ejectio

56 Left ventricular wall motion score (LVWMS) increased sig

57 y artery disease (P < .0001), global resting left ventricular wall motion score index (P < .0001), in

58 ess echocardiography underwent DCMR in which left ventricular wall motion score index (WMSI), defined

59 Large MIs (based on echocardiographic left ventricular wall motion score index) were created b

60 Left ventricular wall motion scores were similar at base

61 In 78 women (85%), left ventricular wall motion was normal at baseline and

62 re used to reconstruct the three-dimensional left ventricular wall motion.

63 using small-angle x-ray diffraction of mouse left ventricular wall muscle.

64 ed infarcts greater than at least 35% of the left ventricular wall (n = 8).

65 iated cardiac-enriched hESC progeny into the left ventricular wall of athymic rats.

66 d by planar wavefronts on the surface of the left ventricular wall of Langendorff-perfused isolated r

67 rified miRNAs were injected in vivo into the left ventricular wall of mice, and, 48 hours later, the

68 ns (each 0.15 mL, 5 mg.mL-1 saline) into the left ventricular wall of rat hearts before a 60-minute o

69 t of proximal flow constraint induced by the left ventricular wall on the accuracy of calculated flow

70 art uptake allowing clear delineation of the left ventricular wall over 60 min after tracer administr

71 siological properties of myocytes across the left ventricular wall play an important role in both the

72 es and cells are maintained in intact canine left ventricular wall preparations in which the myocardi

73 Nonfailing left ventricular wall samples procured from explanted he

74 ely determine whether mechanical behavior of left ventricular wall segments that contain different de

75 We characterized left ventricular wall stress (LVWS) profiles in pressure

76 Compared with baseline, mean left ventricular wall stress and stroke work were not ch

77 End-systolic left ventricular wall stress was calculated by finite el

78 End-systolic left ventricular wall stress was significantly higher wh

79 ial injury and NT-proBNP levels as marker of left ventricular wall stress were determined.

80 We tested the hypothesis that reducing left ventricular wall stress with a percutaneous left at

81 ter reperfusion in the MI+unload group (mean left ventricular wall stress, 44 658 versus 22 963 dynes

82 nges in left ventricular loading conditions, left ventricular wall stress, desensitization of proinfl

83 ndpoint was change in NT-proBNP, a marker of left ventricular wall stress, from baseline to 12 weeks;

84 ors potentially related to chronic increased left ventricular wall stress, including age, hypertensio

85 al probrain natriuretic peptide, a marker of left ventricular wall stress.

86 sis of the papillary muscles and inferobasal left ventricular wall, suggesting a myocardial stretch b

87 onomicrometric crystals in the region of the left ventricular wall supplied by the occluded left ante

88 emodynamic function, pulmonary gas exchange, left ventricular wall thickening and myocardial blood fl

89 hemodynamic function, myocardial blood flow, left ventricular wall thickening and pulmonary gas excha

90 With LAD constriction, left ventricular wall thickening fell 45+/-8% (P<0.01).

91 The mean percentages of left ventricular wall thickening in infarcted, stunned,

92 phic identification of otherwise unexplained left ventricular wall thickening in the presence of a no

93 e was substantiated by localized patterns of left ventricular wall thickening occurring more commonly

94 hemodynamic function, myocardial blood flow, left ventricular wall thickening or pulmonary gas exchan

95 ere-measured regional myocardial blood flow, left ventricular wall thickening or pulmonary gas exchan

96 However, left ventricular wall thickening was not reversed.

97 ntation, five patients showed progression of left ventricular wall thickening with increased left ven

98 Young female transgenic mice exhibited left ventricular wall thickening without dilatation, whe

99 characterize microstructural dynamics during left ventricular wall thickening, and apply the techniqu

100 The disease is characterized by maladaptive left ventricular wall thickening.

101 Risk factors for SCD were left ventricular wall thickness >/=30 mm (20%), family h

102 nger HCM patients and compared with ATH with left ventricular wall thickness >13 mm.

103 opulation comprised patients with a baseline left ventricular wall thickness >=13 mm and no history o

104 Adults with HCM (left ventricular wall thickness >=15 mm or pathogenic va

105 and prehypertensive participants had higher left ventricular wall thickness (0.83 and 0.78 versus 0.

106 versus 0.85+/-0.13 cm, P:<0.005), posterior left ventricular wall thickness (1.00+/-0.24 versus 0.88

107 vs. 90 +/- 16 g/m(2) , P = 0.03) and maximal left ventricular wall thickness (16 +/- 1 vs. 8 +/- 1 mm

108 Patients in the DE group (n=35) had greater left ventricular wall thickness (2.09+/-0.44 versus 1.78

109 l HCM and 32 had subclinical HCM with normal left ventricular wall thickness (21 with early phenotypi

110 atients with cardiomyopathies with increased left ventricular wall thickness (amyloidosis, septal HCM

111 xrazoxane, relative to doxorubicin alone, on left ventricular wall thickness (difference between grou

112 tly related to NYHA class as well as age and left ventricular wall thickness (each with a value of P=

113 At 4 weeks after MI, left ventricular wall thickness (echocardiography; 0.89+

114 of total population), patisiran reduced mean left ventricular wall thickness (least-squares mean diff

115 pathies, but their contribution to increased left ventricular wall thickness (LVWT) in the community

116 ), family history of sudden death (FHSD) and left ventricular wall thickness (LVWT).

117 Left ventricular wall thickness (mean 16+/-6 mm) and dis

118 -up: (1) an increase in >=15% of the maximal left ventricular wall thickness (MLVWT), both in mm and

119 red in grams per meters squared; (c) maximum left ventricular wall thickness (MLVWT), measured in mil

120 Higher left ventricular wall thickness (odds ratio, 2.61; CI, 1

121 farcted or had infarction comprising <25% of left ventricular wall thickness (P<0.005 for ejection fr

122 01), end-diastolic diameter (r2=.32, P<.05), left ventricular wall thickness (r2=.38, P<.01), left at

123 al HCM; n=36), mutation carriers with normal left ventricular wall thickness (subclinical HCM; n=28),

124 sis (ATTR-CM) from other causes of increased left ventricular wall thickness among patients referred

125 severity as well as confounding variables of left ventricular wall thickness and age.

126 Black race was also associated with greater left ventricular wall thickness and concentricity, diffe

127 ardiography showed a significantly increased left ventricular wall thickness and decreased fractional

128 netic analyses of 24 subjects with increased left ventricular wall thickness and electrocardiograms s

129 decreased in subclinical HCM despite normal left ventricular wall thickness and excellent HRQOL.

130 f left ventricular hypertrophy, with reduced left ventricular wall thickness and heart weight/body we

131 monstrate that LHFS of the MI region altered left ventricular wall thickness and material properties,

132 ined ventricular tachycardia (nsVT), maximum left ventricular wall thickness and obstruction were sig

133 Left ventricular wall thickness and postinfarct scar thi

134 iovascular magnetic resonance to investigate left ventricular wall thickness and the presence of asym

135 cludes T2-weighted imaging and assessment of left ventricular wall thickness in detecting patients wi

136 Left ventricular wall thickness increased from 10 to 14

137 tractile abnormalities are present even when left ventricular wall thickness is normal.

138 a myocardial disease defined by an increased left ventricular wall thickness not solely explained by

139 ntiating features from normal pregnancy were left ventricular wall thickness of >/=1.0 cm, exaggerate

140 Echocardiography demonstrated maximal left ventricular wall thickness of 19.9+/-3.8 mm, systol

141 .7 years, p = 0.0002), had more hypertrophy (left ventricular wall thickness of 24.2 vs. 21.1 mm, p =

142 obstruction of at least 30 mm Hg, and marked left ventricular wall thickness of more than 25 mm-were

143 If left ventricular wall thickness seemed nonuniform, the s

144 Cardiomyopathies with increased left ventricular wall thickness such as cardiac amyloido

145 Median (IQR) left ventricular wall thickness was 13 (12-14) mm, and m

146 In the primary cohort, maximal left ventricular wall thickness was 17+/-4 mm for adults

147 Maximum left ventricular wall thickness was 18 +/- 8 mm, and lef

148 The mean maximal left ventricular wall thickness was 21 mm.

149 The maximum left ventricular wall thickness was 22.9 +/- 8.7 mm and

150 Left ventricular wall thickness was also measured.

151 In addition, heterogeneity of left ventricular wall thickness was greater in the BZ th

152 Using cardiovascular magnetic resonance, the left ventricular wall thickness was measured in all 17 s

153 was good (90%), although CMR measurements of left ventricular wall thickness were approximately 19% l

154 ventricular relative wall thickness and mean left ventricular wall thickness were independent predict

155 Age at diagnosis and maximum left ventricular wall thickness were similar to SP- and

156 reased left ventricular dimension and normal left ventricular wall thickness) and dilated cardiomyopa

157 otocol (with the addition of T2-weighted and left ventricular wall thickness) increased the specifici

158 logic phenotype (by virtue of showing normal left ventricular wall thickness).

159 hortening [FS]), end-diastolic diameter, and left ventricular wall thickness).

160 ses of 20 subjects with massive hypertrophy (left ventricular wall thickness, > or =30 mm) but withou

161 gnostic criteria at baseline (median maximal left ventricular wall thickness, 13 mm; interquartile ra

162 t weight, enlarged cardiomyocytes, increased left ventricular wall thickness, and decreased fractiona

163 nance imaging reveals a dramatic increase in left ventricular wall thickness, as compared with Cav-1-

164 and quantification of left atrial dimension, left ventricular wall thickness, chamber diameter, and e

165 Patisiran decreased mean left ventricular wall thickness, global longitudinal str

166 exploratory cardiac end points included mean left ventricular wall thickness, global longitudinal str

167 twin pairs were followed for 5 to 14 y, and left ventricular wall thickness, left atrial diameter, a

168 imilar increases in systolic blood pressure, left ventricular wall thickness, left ventricular mass,

169 the rest of the cohort in age at diagnosis, left ventricular wall thickness, left ventricular outflo

170 rves as the primary imaging tool, evaluating left ventricular wall thickness, outflow tract gradients

171 ent, revealed significant associations among left ventricular wall thickness, postinfarct scar thickn

172 disease phenotype, as evidenced by decreased left ventricular wall thickness, reduced myocardial fibr

173 ish the effect of carvedilol on standardised left ventricular wall thickness-dimension ratio Z score

174 ined by confounding due to changes in BSA or left ventricular wall thickness.

175 interventricular septum thickness, and mean left ventricular wall thickness.

176 ndividuals and is characterized by increased left ventricular wall thickness.

177 erentiated CA from other causes of increased left ventricular wall thickness.

178 ventricular function and a partial rescue of left ventricular wall thickness.

179 in RTRs regardless of BP, mainly by reducing left ventricular wall thickness.

180 tolic contractile function and reductions in left ventricular wall thickness.

181 agnetic resonance imaging, and assessment of left ventricular wall thickness.

182 ecedent cardiac hypertrophy (average maximal left-ventricular-wall thickness, 8.5 mm) nor histopathol

183 Left ventricular wall thicknesses in the overall study g

184 These mice develop left ventricular wall thinning and chamber dilation, wit

185 hin a myocardial infarct (MI) contributes to left ventricular wall thinning and changes in regional s

186 LV left ventricular wall thinning with LV left ventricular

187 At 60 min, the uptake ratios of left ventricular wall to blood, lung, and liver (mean of

188 f the papillary muscle still attached to the left ventricular wall was also noted but was less sensit

189 Reduction of the akinetic left ventricular wall was observed in BMCeP-treated hear

190 Myocardial blood flow of the anterior left ventricular wall was reduced from 1.00 +/- 0.18 to

191 The left ventricular wall was segmented and characterized us

192 al infarction (<50% transmural extent of the left-ventricular wall), whereas SPECT identified only 31

193 EPCM was sutured to the anterolateral left ventricular wall, which included the region of isch

194 al pressure of carbon dioxide, and thickened left ventricular wall with prolonged professional CPR.

195 ents by advancing the fiberoptic through the left ventricular wall with the laser inactivated.