コーパス検索結果 (1語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 fection of one cell line (DEL) with a murine leukemia virus.
2 RNAs packaged by a model retrovirus, murine leukemia virus.
3 ation and packaging domain of Moloney murine leukemia virus.
4 those of oncoretroviral RTs, such as murine leukemia virus.
5 n and also enhanced the production of murine leukemia virus.
6 onto unintegrated DNAs of the Moloney murine leukemia virus.
9 Proviral Integration site for Moloney murine leukemia virus 1) has emerged as a key regulator of hypo
11 proviral integration site for Moloney murine leukemia virus-1 (PIM-1), which in turn regulates NFkapp
12 zes the foreign Ag Tax from the human T cell leukemia virus-1 when presented by the class I MHC HLA-A
13 ink with the autoimmune disease human T cell leukemia virus-1-associated myelopathy/tropical spastic
14 reported that ectopic expression of Moloney leukemia virus 10 (MOV10) protein strongly inhibits retr
19 ipeline, can identify integrations of murine leukemia virus, adeno-associated virus, Tol2 transposons
22 e a high-resolution atlas of m(5)C in murine leukemia virus and reveal a functional role of m(5)C in
23 livery by wild-type ecotropic Moloney murine leukemia virus and vesicular stomatitis virus (VSV) G gl
24 to what extent virions of HERV-Kcon, murine leukemia virus, and HIV-1 have the ability to transduce
25 rived from three retroviruses (HIV-1, murine leukemia virus, and Mason-Pfizer monkey virus), two hepa
29 V and the intensively studied Moloney murine leukemia virus, architectures of the regulatory domains
30 immunodeficiency (SCID-X1), a Moloney murine leukemia virus-based gamma-retrovirus vector expressing
31 virus infections: sheep infected with Bovine Leukemia Virus (BLV) and humans infected with Human T Ly
34 arged residues in the deltaretrovirus bovine leukemia virus (BLV) matrix (MA) and NC domains affects
36 est that the leader region of Moloney murine leukemia virus contains inhibitory/regulatory sequences,
37 by reverse transcription via Moloney murine leukemia virus, degradation of chromosomal DNA with McrB
38 as the proto-oncogene from which the Abelson leukemia virus derived its Gag-v-Abl oncogene, recent re
40 EC3 does not catalyze base changes in murine leukemia virus DNA, it can be recovered from these virus
43 Ecotropic, xenotropic, and polytropic mouse leukemia viruses (E-, X-, and P-MLVs) exist in mice as i
44 his hypothesis, infection of HIV-1 or murine leukemia virus Env (MLV-Env)-pseudotyped HIV-1 particles
45 defect in proteolytic cleavage of the murine leukemia virus Env cytoplasmic tail in pseudotyped virio
49 viral glycoproteins, including Friend murine leukemia virus envelope (F-MLV Env) are actively recruit
53 ing sequence of the ecotropic Moloney murine leukemia virus envelope glycoprotein with the peptide li
54 d how the protomeric units of Moloney murine leukemia virus envelope protein (Env) are activated in r
55 viral entry/infection of pseudotyped murine leukemia viruses expressing pathogenic NWA glycoproteins
56 rovirus glycoproteins, such as Friend murine leukemia virus (F-MLV) Env, but not with the related gib
57 e envelope glycoprotein of the Friend murine leukemia virus (F-MLV) ISD has been reported to abolish
58 ing viral spillover events.IMPORTANCE Feline leukemia virus (FeLV) can infect a variety of felid spec
60 of FeLV infectious disease.IMPORTANCE Feline leukemia virus (FeLV) is a member of the genus Gammaretr
65 exposure to gammaretroviruses such as feline leukemia viruses (FeLVs) occurs worldwide, but the basis
66 uses that infect human cells in vitro Feline leukemia viruses (FeLVs) rank high on this list, but nei
71 LV) Env, but not with the related gibbon ape leukemia virus (GaLV) Env or with a chimeric F-MLV Env w
72 etrovirus-related virus (XMRV) or gibbon ape leukemia virus (GALV) infection, even when their respect
73 a close relative of KoRV and the gibbon ape leukemia virus (GALV), with virion morphology and Mn(2+)
76 e shown to restrict the expression of murine leukemia virus genomes but not retroviral genomes of the
77 parental wild-type ecotropic Moloney murine leukemia virus glycoprotein through the ecotropic recept
78 variants to promote the modified gibbon ape leukemia virus glycoprotein-pseudotyped lentiviral vecto
81 overed the antisense protein of human T-cell leukemia virus (HTLV) type 2 (APH-2), whose messenger RN
82 trate that driver mutations for human T-cell leukemia virus (HTLV)-associated adult T-cell leukemia l
83 and partial characterization of human T-cell leukemia virus (HTLV; now known as HTLV-1) produced by a
84 , human immunodeficiency virus, human T cell leukemia virus, human papilloma virus, hepatitis B and C
85 ic resonance structure of the Moloney murine leukemia virus IN (M-MLV) C-terminal domain (CTD) and a
89 oes not cause acute leukemia on its own, and leukemia virus insertion frequencies predict that RASGRP
91 bidopsis thaliana) B lymphoma Moloney murine leukemia virus insertion region1 homolog (BMI1) POLYCOMB
92 nfirmed that the transcription factor Friend leukemia virus integration 1 (Fli-1) is a target of miR-
101 of proviral insertion site of Moloney murine leukemia virus kinases (Pim-1, -2, and -3) in cancers, p
102 d thrombopoietin/cellular myeloproliferative leukemia virus liganding is dispensable for definitive t
105 In the case of the ecotropic Moloney murine leukemia virus (M-MLV), the Nef-like effect is mediated
108 dispensable for in vivo infectivity, bovine leukemia virus microRNAs represent approximately 40% of
109 release of HIV-1, as well as that of murine leukemia virus (MLV) and Ebola virus (EBOV); knockdown o
111 gene of gammaretroviruses, including murine leukemia virus (MLV) and xenotropic murine leukemia viru
114 nvelope protein (Env) from the CasBrE murine leukemia virus (MLV) can cause acute spongiform neurodeg
116 eins previously identified to inhibit murine leukemia virus (MLV) demonstrated an ability to induce N
118 p between two recent additions to the murine leukemia virus (MLV) ecotropic subgroup: Mus cervicolor
121 generated chimeric constructs between murine leukemia virus (MLV) Gag and HBV Core to determine if th
125 eping Beauty (SB) transposons and the murine leukemia virus (MLV) in mouse embryonic stem cells (ESCs
127 tly affects HIV-1 replication but not murine leukemia virus (MLV) infection and that miR-128 modulati
129 rstanding the molecular mechanisms of murine leukemia virus (MLV) integration into host chromatin is
130 nd that the preintegration complex of murine leukemia virus (MLV) interacts with the dynein complex a
132 ron-alpha-responsive manner, captures murine leukemia virus (MLV) particles and mediates their transf
133 this glycoprotein is compatible with murine leukemia virus (MLV) particles but incompatible with hum
134 iciencies of restriction of HIV-1 and murine leukemia virus (MLV) particles containing various propor
135 ious anemia virus (EIAV), or N-tropic murine leukemia virus (MLV) postentry and supported late HIV-1
136 cosylated Gag (glycoGag) protein of a murine leukemia virus (MLV) similarly enhance the infectiousnes
137 , we engineered a fluorescent Moloney murine leukemia virus (MLV) system consisting of MLV-integrase
138 ated by measuring the cleavage of the murine leukemia virus (MLV) transmembrane Env protein by the vi
140 ement of the photonic inactivation of Murine Leukemia Virus (MLV) via 805 nm femtosecond pulses throu
143 vectors bearing the envelope of 10A1 murine leukemia virus (MLV), a murine retrovirus that can use P
144 For retroviruses such as HIV-1 and murine leukemia virus (MLV), active receptor recruitment and tr
146 the generation of pseudotyped HIV-1, murine leukemia virus (MLV), and vesicular stomatitis virus (VS
148 For simple retroviruses, such as murine leukemia virus (MLV), the identities of the cellular pro
149 lycosylated Gag (glycoGag) protein of murine leukemia virus (MLV), the S2 protein of equine infectiou
150 ations present in a model retrovirus, murine leukemia virus (MLV), using mass spectrometry and sequen
152 n the long terminal repeats (LTRs) of murine leukemia virus (MLV)-based vectors and the vector-specif
153 therapy approaches utilize HIV-1- or murine leukemia virus (MLV)-based vectors, which preferentially
154 odeficiency virus type 1 (HIV-1)- and murine leukemia virus (MLV)-derived viral vectors, respectively
157 related virus (XMRV) as well as other murine leukemia virus (MLV)-related viruses, though not all stu
166 irus-induced leukemogenesis, ecotropic mouse leukemia viruses (MLVs) recombine with nonecotropic endo
167 ers of the gammaretroviruses--such as murine leukemia viruses (MLVs), most notably XMRV [xenotropic m
168 An RNA kissing loop from the Moloney murine leukemia virus (MMLV) exhibits unusual mechanical stabil
170 y inhibits the replication of Moloney murine leukemia virus (Mo-MLV) and is required for the antiretr
172 prototypical gammaretrovirus Moloney murine leukemia virus (MoMLV) favors strong enhancers and activ
173 reverse transcriptase (RT) of Moloney murine leukemia virus (MoMLV) is expressed in the form of a lar
175 t not the RNase H function of Moloney Murine Leukemia Virus (MoMLV) RT and also inhibited Escherichia
176 that tetherin does not affect Moloney murine leukemia virus (MoMLV) spread, and only minimally affect
178 calreticulin (CALR), and myeloproliferative leukemia virus (MPL), abnormally activate the cytokine r
179 gainst clade C HIV-1 gp140, gp120, or murine leukemia virus (MuLV) gp70-scaffolded V1/V2 and toward b
181 arce, femtomole quantities of Moloney murine leukemia virus (MuLV) RNA inside authentic virions and f
184 mouse mammary tumor virus (MMTV) and murine leukemia virus (MuLV) via an adaptive immune mechanism,
185 ammaretroviruses, typified by Moloney murine leukemia virus (MuLV), gag and pol are in the same readi
186 rus 1 (Bxv1), a xenotropic endogenous murine leukemia virus (MuLV), is present in these 2 recently de
189 the mechanism of APOBEC inhibition of murine leukemia viruses (MuLVs) does not appear to be G-->A hyp
190 with mixtures of mouse retroviruses (murine leukemia viruses [MuLVs]) exhibit dramatically altered p
192 , we had previously selected N-tropic murine leukemia virus (N-MLV) mutants escaping from rhesus maca
194 Calreticulin (CALR) and myeloproliferative leukemia virus oncogene (MPL) mutations are specific to
195 calreticulin (CALR), and myeloproliferative leukemia virus oncogene (MPL) mutations; respective freq
196 ng via the thrombopoietin/myeloproliferative leukemia virus oncogene (MPL) pathway and impaired propl
197 Thrombopoietin (Thpo)/myeloproliferative leukemia virus oncogene (Mpl) signaling controls hematop
198 cellular homologue of the myeloproliferative leukemia virus oncogene (Mpl), is the major cytokine reg
199 LNK deficiency increases myeloproliferative leukemia virus oncogene signaling and AKT activation, wh
200 t promotes thrombopoietin/myeloproliferative leukemia virus oncogene signaling and platelet and leuko
202 , including JAK2 exon 12, myeloproliferative leukemia virus oncogene, LNK (also known as SH2B3) mutat
206 oad windows of small RNA sizes in the bovine leukemia virus ovine model of leukemia/lymphoma, we prov
207 provirus integration site for Moloney murine leukemia virus (Pim) 1 kinase is an oncogenic serine/thr
208 proviral integration site for Moloney murine leukemia virus (PIM) 1, 2, and 3 kinases in a NF-kappaB-
209 Proviral integration site for Moloney murine leukemia virus (Pim) kinases are serine/threonine/tyrosi
210 proviral integration site for Moloney murine leukemia virus (PIM) kinases PIM1 and PIM2 have been imp
212 S has been associated with xenotropic murine leukemia virus-related virus (XMRV) as well as other mur
222 irus type-1 (HIV-1) and of xenotropic murine leukemia virus-related virus (XMRV), a gammaretrovirus t
225 a retrovirus called XMRV (xenotropic murine leukemia virus-related virus) was present in the blood o
226 xpectedly high levels of m(5)C in the murine leukemia virus RNA, precisely mapped its location, and s
227 These compounds also inhibit Moloney murine leukemia virus RT but not the Klenow fragment of Escheri
228 cation in vivo, I constructed a novel murine leukemia virus strain (FMLV-IL-1beta) that encodes the m
231 et al. reveal that an isoform of the feline leukemia virus subgroup C receptor (FLVCR1) exports heme
232 mRNA expression of the heme exporter feline leukemia virus subgroup C receptor 1 (beta = -0.30; P =
238 chniques in real time with both human T-cell leukemia virus type 1 (HTLV-1) and human immunodeficienc
241 report that vaccination against human T-cell leukemia virus type 1 (HTLV-1) basic leucine zipper (bZI
243 o HTLV-1 infectivity.IMPORTANCE Human T-cell leukemia virus type 1 (HTLV-1) causes a variety of disea
245 tegrated form of the retrovirus human T-cell leukemia virus type 1 (HTLV-1) contains identical DNA se
260 kawa et al demonstrate that the human T-cell leukemia virus type 1 (HTLV-1) oncoprotein Tax induces a
261 present study, we show that the Human T-cell Leukemia Virus Type 1 (HTLV-1) oncoprotein Tax is a subs
262 d to be largely dispensable for human T-cell leukemia virus type 1 (HTLV-1) particle biogenesis.
266 t of humanized mice infected by human T-cell leukemia virus type 1 (HTLV-1) that recapitulate adult T
270 retroviral oncoprotein Tax from human T-cell leukemia virus type 1 (HTLV-1), an etiological factor th
271 found that HBZ, encoded by the Human T-cell Leukemia Virus type 1 (HTLV-1), binds to multiple domain
272 on by HIV-1, HIV-1Deltavif, and human T-cell leukemia virus type 1 (HTLV-1), while significantly inhi
273 ptosis, we used TRAIL-resistant human T cell leukemia virus type 1 (HTLV-1)-associated adult T cell l
275 to the total viral burden in 22 human T cell leukemia virus type 1 (HTLV-1)-infected individuals by a
281 Tax oncoprotein encoded by the human T-cell leukemia virus type 1 plays a pivotal role in viral pers
282 ions in three viral infections: Human T cell Leukemia Virus type 1, Human Immunodeficiency Virus type
286 s in the transplant population: human T-cell leukemia virus type 1; hepatitis E virus; bocavirus; KI
293 Stavrou et al. (2015) reveal how the murine leukemia virus uses a sugar-protein shield to protect fr
296 e APOBEC3 protein blocks infection by murine leukemia viruses without catalyzing this base change, an
297 nonpermissive to XMRV and xenotropic murine leukemia virus (X-MLV) infection, suggesting that the xe
298 (MLVs), most notably XMRV [xenotropic murine leukemia virus (X-MLV)-related virus--have been reported
301 e leukemia virus (MLV) and xenotropic murine leukemia virus (XMRV), named the CAE (cytoplasmic accumu