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1 the markers of thrombosis, inflammation, and leukocyte adhesion.
2 onal change in beta2 integrins, facilitating leukocyte adhesion.
3 m by which LA1-activated CD11b/CD18 mediates leukocyte adhesion.
4  and is critical for firm integrin-dependent leukocyte adhesion.
5 t captures CTCs while minimizing nonspecific leukocyte adhesion.
6                          LFA-1 also mediates leukocyte adhesion.
7 ptors to assess their relevance for rhythmic leukocyte adhesion.
8  of mobility for the onset of LFA-1 mediated leukocyte adhesion.
9 the inhibition of exocytosis, and suppresses leukocyte adhesion.
10 red to endothelium and subsequently mediated leukocyte adhesion.
11 othelium (P = 0.03) matching the increase in leukocyte adhesion.
12 lammatory signals with those associated with leukocyte adhesion.
13 ed ICAM-1 in endothelial cells (EC) promotes leukocyte adhesion.
14 ation into plaques through platelet-mediated leukocyte adhesion.
15 s an important regulator of CX3CL1-dependent leukocyte adhesion.
16  a preceding activation step to trigger firm leukocyte adhesion.
17 finity triggering mediates chemokine-induced leukocyte adhesion.
18  by integrin-dependent sickle-red blood cell-leukocyte adhesion.
19  microglial activation, and reduced cerebral leukocyte adhesion.
20 tion of HC-HA cable structures and abolishes leukocyte adhesion.
21 ry factor, which inhibits integrin-dependent leukocyte adhesion.
22 ent with metformin decreased the SFA-induced leukocyte adhesion.
23         APN-KO mice also exhibited increased leukocyte adhesion (2.3+/-0.4-fold) and tumor necrosis f
24 e (CX3CL1) and its receptor, CX3CR1, mediate leukocyte adhesion, activation, and trafficking.
25 ional biology: a mechanics-based modeling of leukocyte adhesion (adhesive dynamics) and signal transd
26 and partly prevented increases in VCAM-1 and leukocyte adhesion after treatment with tumour necrosis
27 creased permeability, ICAM-1 expression, and leukocyte adhesion, all of which are features of an infl
28 ated a crucial role for CatG during arterial leukocyte adhesion, an effect not found during venular a
29 uorescence microscopy, we observed excessive leukocyte adhesion and accelerated atherosclerotic plaqu
30 nflammation, vascular lesions with increased leukocyte adhesion and capillary degeneration.
31               In contrast, polymorphonuclear leukocyte adhesion and chemotaxis were normal.
32 gulates many biological processes, including leukocyte adhesion and chemotaxis.
33 clerosis in ApoE(-/-) mice, there is reduced leukocyte adhesion and decreased CD18 expression on Gr1(
34 9)/L platelets, showing reduced signaling in leukocyte adhesion and diapedesis and increased compleme
35  DPA and RvD5n-3 DPA decreased the extent of leukocyte adhesion and emigration following ischemia-rep
36 ntified role for Panx1 channels in promoting leukocyte adhesion and emigration through the venous wal
37 ade body secretion, and in the regulation of leukocyte adhesion and extravasation during inflammation
38 ion in the genetic absence of Coro1A impairs leukocyte adhesion and extravasation in inflamed cremast
39 muscle venules revealed severely compromised leukocyte adhesion and extravasation in MK(-/-) mice com
40  control endothelial cell (EC) permeability, leukocyte adhesion and inflammation are pivotal for athe
41 10-treated mice showed reduced expression of leukocyte adhesion and inflammatory markers, respectivel
42                                Functionally, leukocyte adhesion and integrin activation are abrogated
43 show that N-glycosylation of JAM-A regulates leukocyte adhesion and LFA-1 binding.
44  ligands and their ability to intervene with leukocyte adhesion and migration across brain endothelia
45   CX3CR1 is a chemokine receptor involved in leukocyte adhesion and migration and hence a mediator of
46 egment of the microvasculature that supports leukocyte adhesion and migration expands through remodel
47                                 Constitutive leukocyte adhesion and migration in murine bone marrow (
48 Cs) are well recognized as key regulators in leukocyte adhesion and migration in response to bacteria
49    The finding that Cdk inhibitors can block leukocyte adhesion and migration may expand the clinical
50 t human fetal HSCs employ mechanisms used in leukocyte adhesion and migration to mediate HSC self-ren
51 anscripts encoding for molecules involved in leukocyte adhesion and migration, cytotoxic functions, a
52 nd its ligand CX3CL1 have been implicated in leukocyte adhesion and neuronal protection.
53 dothelial activation manifested as increased leukocyte adhesion and plasma extravasation in response
54 ta2-integrin activation is indispensable for leukocyte adhesion and recruitment to sites of infection
55     Intravital microscopy showed decrease in leukocyte adhesion and rolling after ethanol consumption
56                         L-selectin regulates leukocyte adhesion and rolling along the endothelium.
57  new role of fibrinogen in integrin-mediated leukocyte adhesion and suggest that this mechanism may p
58 tify alpha 4 integrin/CD49d as a mediator of leukocyte adhesion and the resultant early signature abn
59 protein I (beta2GPI) induce endothelial cell-leukocyte adhesion and thrombus formation via unknown me
60 ould be used as a therapeutic tool to reduce leukocyte adhesion and thus reduce neuroinflammation.
61 or investigation of lymphatic involvement in leukocyte adhesion and trafficking during the immune res
62 the effects of hemodynamics and cytokines on leukocyte adhesion and trans-endothelial migration (TEM)
63 t human JAM-C to determine its role in human leukocyte adhesion and transendothelial cell migration u
64 ion of endothelial ABPs to the regulation of leukocyte adhesion and transendothelial migration remain
65 e their endothelial cell ligands and support leukocyte adhesion and transendothelial migration.
66                                Consequently, leukocyte adhesion and transmigration are significantly
67 synthase and fibrinolytic activity, decrease leukocyte adhesion and transmigration, and enhance resis
68 ound LYVE-1 molecules and thereby facilitate leukocyte adhesion and transmigration.
69  functions including macrophage recognition, leukocyte adhesion and transmigration.
70 beta while an anti-ICAM-1 mAb inhibited both leukocyte adhesion and transmigration.
71 F-induced proliferation and thrombin-induced leukocyte adhesion and triggers a STAT3-dependent increa
72 dase is positively correlated with increased leukocyte adhesion and vascular leakage in diabetes and
73 s and reduced significantly diabetes-induced leukocyte adhesion and vascular leakage.
74  and smooth muscle cell apoptosis as well as leukocytes adhesion and proliferation.
75          Cell polarization is a key step for leukocytes adhesion and transmigration during leukocytes
76 is, cell growth and apoptosis, angiogenesis, leukocyte adhesion, and cytokine activation and inhibiti
77  in atherosclerotic plaques, blocks arterial leukocyte adhesion, and inhibits atherosclerosis and inf
78 hamber protein concentration, retinal vessel leukocyte adhesion, and protein leakage were measured 24
79 at plays a role in tight junction formation, leukocyte adhesion, and transendothelial migration.
80 cells is essential for platelet aggregation, leukocyte adhesion, and transmigration through endotheli
81 a-AR stimulation can promote both SS RBC and leukocyte adhesion as well as vaso-occlusion, suggesting
82              AnxA1Ac2-26 treatment decreased leukocyte adhesion at 40 min and all L-E interactions at
83 ies in the murine inflamed microcirculation (leukocyte adhesion being the readout) and in skin traffi
84 , the retinas were extracted and assayed for leukocyte adhesion; blood-retinal barrier breakdown; VEG
85 hoG by small interfering RNA does not affect leukocyte adhesion but decreases cup formation and inhib
86 tyrosine phosphatase 1 (Shp1) show increased leukocyte adhesion, but the interpretation of these data
87  of the immunoglobulin superfamily, supports leukocyte adhesion by binding alpha4beta1 integrins and
88 3 cells, suggesting that these miRs regulate leukocyte adhesion by modulating the expression of adhes
89                         Understanding of the leukocyte adhesion cascade and interactions of leukocyte
90           Despite expanded definition of the leukocyte adhesion cascade and mechanisms underlying ind
91    These data provide unique insights in the leukocyte adhesion cascade and the potential for time-ba
92               Current in vitro models of the leukocyte adhesion cascade cannot be used for real-time
93                                          The leukocyte adhesion cascade is important in chronic lymph
94 tion in vivo and add additional steps to the leukocyte adhesion cascade that supports leukocyte traff
95  be used for real-time studies of the entire leukocyte adhesion cascade, including rolling, adhesion,
96 N-glycans contributing to all aspects of the leukocyte adhesion cascade, O-glycans only being importa
97 sses have been identified in the endothelial-leukocyte adhesion cascade.
98  the endothelium commonly referred to as the leukocyte adhesion cascade.
99 llular MRP8/14 as an autocrine player in the leukocyte adhesion cascade.
100    In view of the capacity of FKN to mediate leukocyte adhesion, chemoattraction, and transmigration,
101                              This diminished leukocyte adhesion correlated with the magnitude of gran
102 le and a prolongation of chemokine-dependent leukocyte adhesion could be observed.
103                                              Leukocyte adhesion, crawling, and transmigration are reg
104 s in this disappearance, a dog having canine leukocyte adhesion deficiency (CLAD) or CLAD dogs who ha
105                                       Canine leukocyte adhesion deficiency (CLAD) represents the cani
106 e use of foamy virus vectors to treat canine leukocyte adhesion deficiency (CLAD).
107 ect in different human cell types, including leukocyte adhesion deficiency (LAD) patient-derived immo
108 umans, mutations in beta(2) integrin lead to leukocyte adhesion deficiency (LAD) syndrome and mutatio
109 activation on neutrophils is the hallmark of leukocyte adhesion deficiency (LAD) syndrome in humans,
110                       PURPOSE OF REVIEW: The leukocyte adhesion deficiency (LAD) syndromes are rare g
111 the canine counter-part of the human disease leukocyte adhesion deficiency (LAD).
112 ed IL-17 has also been shown in SLE, RA, and leukocyte adhesion deficiency and may contribute to oral
113 in inherited and acquired disorders, such as leukocyte adhesion deficiency and myelodysplasia.
114            Chronic granulomatous disease and leukocyte adhesion deficiency are the major primary immu
115 ontrasts with the phenotype seen in type III leukocyte adhesion deficiency caused by the absence of k
116 nd identify CF as a new, cell type-selective leukocyte adhesion deficiency disease, providing new ins
117      The protein kindlin 3 is mutated in the leukocyte adhesion deficiency III (LAD-III) disorder, le
118                              In the disorder leukocyte adhesion deficiency III (LAD-III), integrins o
119 e severe than those reported for people with leukocyte adhesion deficiency III (LAD-III).
120 de an effective, but less toxic approach for leukocyte adhesion deficiency in children.
121 ivation on leukocytes is the hallmark of the leukocyte adhesion deficiency syndrome in humans, charac
122 f young dogs with the genetic disease canine leukocyte adhesion deficiency that were treated with a n
123              Immune cells from patients with leukocyte adhesion deficiency type 1 (LAD-1) had reduced
124     Previous studies using phagocytes from a leukocyte adhesion deficiency type 1 (LAD-I) patient ide
125                               A patient with leukocyte adhesion deficiency type 1 (LAD1) had severe p
126 e the contribution of CD18 on neutrophils to leukocyte adhesion deficiency type I (LAD-I), a complex
127 se), severe congenital neutropenia (SCN) and leukocyte adhesion deficiency type I confer a predisposi
128 lts imply that the immunodeficiency found in leukocyte adhesion deficiency type III patients, in addi
129  accumulation of HSPCs in the circulation of leukocyte adhesion deficiency type III patients, who lac
130 hat is mutated in the rare genetic disorder, leukocyte adhesion deficiency type III, a disorder chara
131 n-3 is mutated in the rare genetic disorder, leukocyte adhesion deficiency type III, which is charact
132                                              Leukocyte adhesion deficiency type-1 (LAD-1) is an autos
133 enital origin (e.g., congenital neutropenia, leukocyte adhesion deficiency, and Chediak-Higashi syndr
134 cell activity is impaired, but not absent in leukocyte adhesion deficiency, may lead to the developme
135 which die perinatally of severe bleeding and leukocyte adhesion deficiency, mice expressing as little
136                                Subjects with leukocyte adhesion deficiency-1 (LAD-I) do not express b
137                            A canine model of leukocyte adhesion deficiency-I facilitated development
138         Preimplantation genetic diagnosis of leukocyte adhesion deficiency-I led to the birth of a no
139 adhesion deficiency-I; prenatal diagnosis of leukocyte adhesion deficiency-I; and association of auto
140 e adhesion deficiency-I; transplantation for leukocyte adhesion deficiency-I; prenatal diagnosis of l
141 herapy for chronic granulomatous disease and leukocyte adhesion deficiency-I; transplantation for leu
142 ng kindlin-3, the protein that is mutated in leukocyte adhesion deficiency-III patients.
143    Patients with Glanzmann thrombasthenia or Leukocyte Adhesion Deficiency-III syndrome (LAD-III or L
144 tudying a patient with kindlin-3 deficiency (leukocyte adhesion deficiency-III).
145 nderlie the human immune deficiency known as leukocyte adhesion deficiency-III.
146 matous disease and beta2 integrin defects in leukocyte adhesion deficiency.
147 ll transplant and gene therapy approaches to leukocyte adhesion deficiency.
148 E) abolished the inhibitory effect of gAd on leukocyte adhesion, demonstrating the obligatory role of
149                                              Leukocyte adhesion during inflammation is initiated by t
150 electin, ICAM-1, and EphB4 and were sites of leukocyte adhesion during lipopolysaccharide-induced acu
151           Superfusion with IS induced strong leukocyte adhesion, enhanced extravasation, and interrup
152 lence at least in part by impeding Fg-driven leukocyte adhesion events.
153                                   Similarly, leukocyte adhesion, extravasation, and bacterial clearan
154 of temporal patterns of rhodamine-6G-labeled leukocyte adhesion/extravasation, was used.
155 molecules, including interleukin (IL)-6, the leukocyte adhesion factor ICAM1, and chemokines that att
156 ter B lymphocytes from mice with a defect in leukocyte adhesion fail to divide asymmetrically.
157 ctionally important endogenous inhibitors of leukocyte adhesion have not been identified.
158           Intravitreal IL-6 directly induced leukocyte adhesion in both wild-type and IL-6-deficient
159 muscle confirmed the defect of CXCL1-induced leukocyte adhesion in HPK1-deficient mice.
160  rolling on unstimulated veins and increased leukocyte adhesion in inflamed veins.
161 ceded clinical signs of anterior uveitis and leukocyte adhesion in iris vasculature.
162 eceptors in endothelial cells also increased leukocyte adhesion in mesenteric venules and increased t
163 ition of KDM7A and UTX significantly reduces leukocyte adhesion in mice, establishing the biological
164 rteriogenesis, thrombosis, inflammation, and leukocyte adhesion in patients with intermittent claudic
165 rupts capillary fine structure and increases leukocyte adhesion in postcapillary venules.
166      Insulin decreased VCAM-1 expression and leukocyte adhesion in quiescent tumour endothelial cells
167 ]-3-methyl-cyclohexanol), greatly attenuated leukocyte adhesion in surface pial vessels and in deep a
168 n leukocyte rolling and a 5-fold increase in leukocyte adhesion in the microcirculation.
169                                              Leukocyte adhesion in the microvasculature influences bl
170 d protein content in the aqueous fluid, firm leukocyte adhesion in the retinal vessels, and the numbe
171 rm during inflammatory processes and mediate leukocyte adhesion in the synovial fluids of arthritis p
172 d the mechanistic roles of hyperglycemia and leukocyte adhesion in this process.
173 l Del-1 deficiency increased LFA-1-dependent leukocyte adhesion in vitro and in vivo.
174 ion molecules (ICAMs) in cis, which inhibits leukocyte adhesion in vitro and in vivo.
175                                      Using a leukocyte adhesion in vitro assay under shear forces mim
176 pproximately 33 mumol) mitigated LPS-induced leukocyte adhesion in WT and AnxA1-null animals without
177 h soluble fibrinogen exerts its influence on leukocyte adhesion indicated that it did not block integ
178 studied endothelial permeability, intravital leukocyte adhesion, involvement of the Akt/WNT/beta-cate
179                            Integrin-mediated leukocyte adhesion is central to this process.
180                                              Leukocyte adhesion is determined by the balance between
181                                              Leukocyte adhesion is mediated mainly by selectins, cell
182 organization of endothelial ICAM-1 regulates leukocyte adhesion is not well understood.
183                  A key parameter influencing leukocyte adhesion is the shear stress acting on the leu
184                                              Leukocyte adhesion is triggered by the interaction of va
185 platelet dysfunction in addition to impaired leukocyte adhesion, is now known to be due to absence of
186 m from obese mice expressed higher levels of leukocyte adhesion markers and lower levels of cell-cell
187 ignificantly increased the expression of the leukocyte adhesion markers LFA-1 and VLA-4, consistent w
188                                     Platelet-leukocyte adhesion may contribute to thrombosis and infl
189            Evidence suggests that disrupting leukocyte adhesion may prevent the impairment of renal m
190 macrophage-1 antigen complex, which mediates leukocyte adhesion, migration and phagocytosis as part o
191  and functions in diverse capacities such as leukocyte adhesion, migration, and cell survival on liga
192 dose of natalizumab, an antibody against the leukocyte adhesion molecule alpha4 integrin, in patients
193 ) and increased expression of membrane-bound leukocyte adhesion molecule CD11b, leading to enhanced i
194  this report, we have determined that the EC-leukocyte adhesion molecule E-selectin is a key target f
195                                E-selectin, a leukocyte adhesion molecule expressed on endothelium, is
196 irectly suppressed endothelial activation of leukocyte adhesion molecule expression and inflammation.
197 or the synthesis of functional selectin-type leukocyte adhesion molecule ligands.
198 ical surface, where JAM-A played a role as a leukocyte adhesion molecule participating in transendoth
199                                    Increased leukocyte adhesion molecule VCAM-1 expression and leukoc
200  observed elevated expression of endothelial leukocyte adhesion molecule-1, a human glaucoma marker,
201  but under inflammatory conditions acts as a leukocyte adhesion molecule.
202 ulatory role in cytokine induction of the EC-leukocyte adhesion molecules (ELAM) E-selectin and vascu
203                 Therefore, the expression of leukocyte adhesion molecules and secretion of proinflamm
204                      The characterization of leukocyte adhesion molecules and their control by proinf
205 eta2 integrins (CD11/CD18) are heterodimeric leukocyte adhesion molecules expressed on hematopoietic
206                            TNF-alpha induces leukocyte adhesion molecules on endothelial cells (ECs),
207 t, Rho GTPases affect the expression of some leukocyte adhesion molecules on endothelial cells, such
208                   In contrast, the genes for leukocyte adhesion molecules showed a significant upregu
209 esion, and extravasation by up-regulation of leukocyte adhesion molecules such as E-selectin and P-se
210 e-inducible, NF-kappaB-dependent endothelial-leukocyte adhesion molecules that participate in the leu
211 tigated the role of p75 in TNF-alpha-induced leukocyte adhesion molecules using cultured ECs derived
212 ir ability to regulate the expression of key leukocyte adhesion molecules, on both leukocytes and end
213 capillaries enlarged into venules expressing leukocyte adhesion molecules, sprouting angiogenesis and
214 that the phenolic content of RW may modulate leukocyte adhesion molecules, whereas both ethanol and p
215  associated with endothelial upregulation of leukocyte adhesion molecules, which persist even after i
216                               Transient mild leukocyte adhesion occurred in mCMV-ND venules at 7 and
217 or IL-6, and their retinas were analyzed for leukocyte adhesion or for the expression and localizatio
218 a(2+)]i in endothelial cells does not affect leukocyte adhesion or locomotion but selectively blocks
219 strains lacking Pra1p were unable to support leukocyte adhesion or migration.
220                               No significant leukocyte adhesion or rolling, nor changes in capillary
221 scular permeability, tissue edema, augmented leukocyte adhesion, platelet aggregation, and dysregulat
222 scular complications, the role that rhythmic leukocyte adhesion plays in different vascular beds has
223 w glycocalyx damage or removal will increase leukocyte adhesion potential during inflammation.
224 ndogenous inhibitors, as well as the role of leukocyte adhesion-promoting molecules) has provided new
225                       Mechanisms controlling leukocyte adhesion, propulsion and directional migration
226 matory markers, neutrophil chemoattractants, leukocyte adhesion proteins, and matrix metalloproteases
227 coli strains, targets a broad range of human leukocyte adhesion proteins.
228 form to the TNFalpha-induced upregulation of leukocyte adhesion proteins.
229     We report that in addition to supporting leukocyte adhesion, provision of specific substrate to V
230 shear stress responses, and TNFalpha-induced leukocyte adhesion rates characteristic of arterial endo
231 y increases in endothelial expression of the leukocyte adhesion receptor E-selectin and in microvascu
232 xide production, and by direct modulation of leukocyte adhesion receptor expression.
233                                    CD44, the leukocyte adhesion receptor for hyaluronan, has been con
234                                          The leukocyte adhesion receptor L-selectin forms bonds with
235 Pases are activated by engagement of several leukocyte adhesion receptors and contribute to both earl
236                       The selectin family of leukocyte adhesion receptors is principally recognized f
237 G, interact with ICAM-1 and are recruited to leukocyte adhesion sites.
238 tivity C-reactive protein, osteopontin), and leukocyte adhesion (soluble vascular cell adhesion molec
239  addressed this problem, two key features of leukocyte adhesion, such as cell deformation and rolling
240 n endothelial cells and, in turn, to augment leukocyte adhesion, thus exacerbating S1P-mediated proad
241 chronic inflammatory disease associated with leukocyte adhesion to and extravasation through vascular
242 tation of ICAM and VCAM expression, elevated leukocyte adhesion to and migration across BMVEC monolay
243                                              Leukocyte adhesion to brain endothelial cells, the blood
244 a inhibition in vivo substantially decreased leukocyte adhesion to brain endothelium under inflammato
245                 PolyI:C-induced, HA-mediated leukocyte adhesion to colon SMCs from STAT1-null mice wa
246 AM-1 is best known for its role in mediating leukocyte adhesion to endothelial cells and guiding leuk
247 treated SS RBCs induced activation of murine leukocyte adhesion to endothelium as well.
248 ich SS RBCs act via LW and CD44 to stimulate leukocyte adhesion to endothelium, and suggest that RBC
249 so purified LFA-1, an integrin that mediates leukocyte adhesion to endothelium.
250 expression in renal vascular endothelium and leukocyte adhesion to endothelium.
251 is study investigated the regulation of firm leukocyte adhesion to human brain endothelium by two dif
252 igation rapidly activated integrin-dependent leukocyte adhesion to immobilized ICAM-1 and fibronectin
253  in multiple leukocyte populations, impaired leukocyte adhesion to inflamed vessels, and accumulation
254                                              Leukocyte adhesion to P-selectin on activated platelets
255 ation of AnxA1(2-50) markedly reduced (>60%) leukocyte adhesion to postcapillary venules in wild type
256 e blockade of NAD(P)H oxidase in turn blocks leukocyte adhesion to retinal vessels during diabetes an
257 tent with these findings, we found increased leukocyte adhesion to sdc-1 knock-out endothelial cells
258 rst description of a mechanism that prevents leukocyte adhesion to the endothelium by a parasite and
259                 Furthermore, IL-35 inhibited leukocyte adhesion to the endothelium in the vessels of
260                                     However, leukocyte adhesion to the fibrin clot can be detrimental
261                                  It promotes leukocyte adhesion to the liver in vivo and drives lymph
262 dose-dependently inhibited TNF-alpha-induced leukocyte adhesion to the murine cremasteric arterioles
263 ed increases in IL-6 mRNA and protein and in leukocyte adhesion to the retinal vessels.
264     ET-1-treated PbN-infected mice displayed leukocyte adhesion to the vascular endothelia and petech
265 ology of diabetic retinopathy is mediated by leukocyte adhesion to the vascular endothelium of the di
266                                Inhibition of leukocyte adhesion to the vascular endothelium represent
267                                              Leukocyte adhesion to the vascular wall is a critical ea
268 iferation of plaque resident macrophages and leukocyte adhesion to the vascular wall were significant
269                                Intriguingly, leukocyte adhesion to the vasculature is significantly i
270 t limits endothelial activation and prevents leukocyte adhesion to the vessel wall.
271 dition of heavy chains to HA cables enhanced leukocyte adhesion to these cables, but it also had seve
272                       Finally, HDL inhibited leukocyte adhesion to TNFalpha-stimulated ECs isolated f
273 to nude mice promotes both SS RBC and murine leukocyte adhesion to vascular endothelium in vivo.
274 s (such as VCAM-1 the ligand for VLA-4), and leukocyte adhesion to vascular endothelium.
275                      Human polymorphonuclear leukocytes adhesion to endothelial cells during the earl
276 nd hemorrhagic shock did not further amplify leukocytes adhesion to intestinal venules compared with
277 oxemia and hemorrhagic shock did not amplify leukocytes adhesion to the endothelium further than eith
278     Furthermore, hemorrhagic shock triggered leukocytes adhesion to the venular endothelium to the sa
279                     Hypoxemia also triggered leukocytes adhesion to the venular endothelium.
280 y for rapid activation of integrin-dependent leukocyte adhesion, triggered by toll-like receptor (TLR
281 the essential role for TXNIP in mediating EC-leukocyte adhesion under d-flow, as well as define a nov
282 c IgG and assembled immune complexes amplify leukocyte adhesion under dynamic conditions.
283  core-1/Galbeta1,3GalNAc glycan may regulate leukocyte adhesion under fluid shear.
284 llular adhesion molecule-1 (ICAM-1) mediates leukocyte adhesion under force.
285 ppressed the LPS-induced increase in retinal leukocyte adhesion; vascular leakage; NF-kappaB, HIF-1al
286 r contributed to activation of NF-kappaB and leukocyte adhesion via the EC induction of intercellular
287 glycosylation, sialyl Lewis-X formation, and leukocyte adhesion via the selectins.
288                                              Leukocyte adhesion was assayed by concanavalin A labelin
289                                              Leukocyte adhesion was assessed by labeling adherent leu
290                                 Platelet and leukocyte adhesion was assessed in cortical venules with
291                                              Leukocyte adhesion was evaluated by chemotaxis and in vi
292           Unexpectedly, although in arteries leukocyte adhesion was highest in the morning, it peaked
293 ferase FUT9 played an important role because leukocyte adhesion was reduced by 50-60% in FUT9-HL-60,
294 tically upregulated at the onset of hypoxia, leukocyte adhesion was unaffected.
295   Differences in cell adhesion molecules and leukocyte adhesion were ablated when disrupting sympathe
296 expression on retinal neovascularization and leukocyte adhesion were abolished in mice lacking TNF-al
297                                   Similarly, leukocyte adhesions were significantly lower, with eleva
298 hyaluronan production or hyaluronan-mediated leukocyte adhesion when treated with the viral mimic pol
299 ith SFA or inhibition of autophagy increased leukocyte adhesion, whereas treatment with metformin dec
300 rolling phase resulting in the transition to leukocyte adhesion, which appear to contribute to chemok
301 tion of CatG specifically abrogated arterial leukocyte adhesion without affecting myeloid cell adhesi

 
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