ライフサイエンスコーパス: leukocyte migration

1 d the potential role of ALF in regulation of leukocyte migration.

2 investigate the role of Plg in inflammatory leukocyte migration.

3 several innate immune functions that include leukocyte migration.

4 ed in the regulation of barrier function and leukocyte migration.

5 e for DARC expressed on endothelial cells in leukocyte migration.

6 d proteins with roles in axonal guidance and leukocyte migration.

7 nhibits alpha4 integrin signals that support leukocyte migration.

8 chemokines in the key physiologic process of leukocyte migration.

9 nchanged, suggesting that bilirubin inhibits leukocyte migration.

10 hich in turn provide directional signals for leukocyte migration.

11 quired for immune cell-cell interactions and leukocyte migration.

12 hown to regulate neuronal and CXCR4-mediated leukocyte migration.

13 NF in the mouse air pouch reduced SP-induced leukocyte migration.

14 n pathologic conditions may adversely affect leukocyte migration.

15 several aminoacyl-tRNA synthetases to induce leukocyte migration.

16 large family of cytokines that direct normal leukocyte migration.

17 rins, alpha9beta1 plays an important role in leukocyte migration.

18 ar acidification rate, CD11b expression, and leukocyte migration.

19 omologues are known to be potent signals for leukocyte migration.

20 ts arachidonic acid metabolism, and inhibits leukocyte migration.

21 ligand very late antigen-4 (VLA-4), in such leukocyte migration.

22 ignaling role for these molecules in the CNS leukocyte migration.

23 rin expressed on leukocytes, is important in leukocyte migration.

24 te in re-establishing vessel integrity after leukocyte migration.

25 pha had no significant affect on the overall leukocyte migration.

26 rgistic regulation of these receptors during leukocyte migration.

27 sion molecules involved in the regulation of leukocyte migration.

28 ody labeling allowed for precise tracking of leukocyte migration.

29 In vitro, PI16 promotes transendothelial leukocyte migration.

30 forming a physical barrier to intravascular leukocyte migration.

31 mma and promotes Gbetagamma signaling during leukocyte migration.

32 e capable of inducing directed intravascular leukocyte migration.

33 es, and assessment of thioglyccolate-induced leukocyte migration.

34 es in organ development and orchestration of leukocyte migration.

35 xhibit differences in thioglyccolate-induced leukocyte migration.

36 ways critically involved in transendothelial leukocyte migration.

37 hion without interfering with intraembryonic leukocyte migration.

38 es reported that CB2R signaling also reduces leukocyte migration.

39 crosis factor-alpha in an air-pouch model of leukocyte migration.

40 o control many cellular processes, including leukocyte migration.

41 ulator, in binding Gbetagamma and inhibiting leukocyte migration.

42 for the efficacy of Gbetagamma signaling and leukocyte migration.

43 Leukocyte migration across endothelial cell borders (par

44 ix and cell death, in addition to regulating leukocyte migration across extracellular matrix barriers

45 Chemokines may play a role in leukocyte migration across the blood-brain barrier (BBB)

46 he pathogenesis of neuroinflammation, namely leukocyte migration across the blood-brain barrier.

47 Leukocyte migration across the endothelial lining is a c

48 The basic route and mechanisms for leukocyte migration across the endothelium remain poorly

49 alpha4 integrins are important mediators of leukocyte migration across vascular endothelium.

50 the nasal airway impacting barrier function, leukocyte migration activation, and mucus secretion that

51 tibility complex (MHC) protein function; (2) leukocyte migration, activation and cytokine responses;

52 nervous system (CNS)-specific mechanisms of leukocyte migration, activation, and MV clearance.

53 ulation of pathways of mucus hypersecretion, leukocyte migration/activation, and endoplasmic reticulu

54 es are small, soluble proteins that regulate leukocyte migration, adhesion, and proliferation.

55 ent zebrafish larvae for in vivo analysis of leukocyte migration after morpholino knockdown of FAN.

56 sation, focal adhesion, platelet activation, leukocyte migration, amino acid biosynthesis, carbon met

57 Leukocyte migration analysis in vivo showed that Acat1(-

58 ase type III alpha pathway in the control of leukocyte migration and actin dynamics.

59 incipal cell adhesion receptors that mediate leukocyte migration and activation in the immune system.

60 Leukocyte migration and activation is orchestrated by ch

61 In addition to their role as regulators of leukocyte migration and activation, chemokines and their

62 s to the lung, and appropriate regulation of leukocyte migration and adhesion is integral to this pro

63 gulation of CXCL14-a chemokine that controls leukocyte migration and angiogenesis, and whose expressi

64 adhesion molecule 1 (PECAM-1) is involved in leukocyte migration and angiogenesis, which are key comp

65 Stromal and parenchymal cells accentuated leukocyte migration and antiviral defenses.

66 Chemokines are the principal regulators of leukocyte migration and are essential for initiation and

67 Chemokines and other chemoattractants direct leukocyte migration and are essential for the developmen

68 Chemokines are the principal regulators of leukocyte migration and are essential in the initiation

69 or near genes involved in cellular adhesion, leukocyte migration and atherosclerosis (PECAM1, rs18676

70 e important adhesion molecules necessary for leukocyte migration and cellular interactions.

71 apeutic approach to treat diseases involving leukocyte migration and chemotactic factors.

72 The role of leukocyte migration and chimerism in organ allograft acc

73 P or MMP-14) is a collagenase that is key in leukocyte migration and collagen destruction.

74 e of neuro-immune interactions that regulate leukocyte migration and consequences for protective immu

75 ed from eicosapentaenoic acid that regulates leukocyte migration and enhances macrophage phagocytosis

76 of soluble mediators important in regulating leukocyte migration and extravasation, including the CXC

77 How leukocyte migration and function are coordinated is unkn

78 ifestyle factors modulate haematopoiesis and leukocyte migration and function in the context of cardi

79 are the cell adhesion receptors that mediate leukocyte migration and functions.

80 Chemokines mediate leukocyte migration and homeostasis and are key targets

81 degrading Galpha(i2), Nef directly subverts leukocyte migration and homing.

82 D18 is a key adhesion receptor that mediates leukocyte migration and immune functions.

83 ell (plt) mutation leads to abnormalities in leukocyte migration and immune response.

84 es a basis for their severe abnormalities in leukocyte migration and immune response.

85 NO-np modified leukocyte migration and increased tumor growth factor-be

86 ity of a neuronal guidance cue in regulating leukocyte migration and indicate that there may be a gen

87 n RCD motif have shown promise in modulating leukocyte migration and inflammation presumably by block

88 d immune functions in blood cells, including leukocyte migration and inflammatory responses, and decr

89 restricted signaling molecule that regulates leukocyte migration and integrin-mediated adhesion.

90 ing Csf-1 and Lgals3bp, directly involved in leukocyte migration and invasion, were significantly upr

91 s and circuitry, including lipid metabolism, leukocyte migration and olfaction.

92 well as angiogenesis, vascular permeability, leukocyte migration and platelet formation in vivo.

93 t of morphine, a known immunosuppressant, on leukocyte migration and recruitment to conditioned media

94 nt inhibition may be effective in preventing leukocyte migration and subsequent local and remote orga

95 nsplantation is directly related to enhanced leukocyte migration and that early islet graft survival

96 first time morphine's inhibitory effects on leukocyte migration and their ability to transmigrate ac

97 rs an avenue for precise characterization of leukocyte migration and therapeutic modulators.

98 alpha(4) integrins play a pivotal role in leukocyte migration and tissue-specific homing.

99 ory pathway based on its ability to modulate leukocyte migration and to inhibit the expression of inf

100 Leukocyte migration and trafficking is dynamically regul

101 f E-selectin receptor/ligand interactions in leukocyte migration and vascular pathology.

102 phenotypic and functional changes to support leukocyte migration and, in some cases, aggregation into

103 a beta-arrestin-dependent one that promotes leukocyte migration, and a G-protein/Ca(2+) one that is

104 these QTL, including the glycan degradation, leukocyte migration, and antigen-presenting pathways.

105 ial for neutrophil migration and chemotaxis, leukocyte migration, and cell chemotaxis.

106 iched in immune pathways such as chemotaxis, leukocyte migration, and cytokine signaling.

107 otein that costimulates T cells, facilitates leukocyte migration, and inhibits macrophage scavenger f

108 1, a chemokine that regulates cerebellar and leukocyte migration, and its receptor CXCR4 are expresse

109 lar injury, operating after transendothelial leukocyte migration, and presumably binding to alternate

110 meability, endothelial inflammatory markers, leukocyte migration, and susceptibility to LPS-induced d

111 ses, including embryogenesis, hematopoiesis, leukocyte migration, and tumor metastasis.

112 t perceptions concerning the role of PI3K in leukocyte migration are based predominantly around evide

113 with lymphocytes and its in-depth effects on leukocyte migration are poorly understood.

114 Chemokines play a pivotal role in regulating leukocyte migration as well as other biological function

115 glycollate model of peritoneal inflammation, leukocyte migration at 72 hours increased significantly

116 lly related, low m.w. proteins that regulate leukocyte migration both in vitro and in vivo.

117 teins do not only serve as a stop signal for leukocyte migration but also can propagate the extravasa

118 Previously, LEC was shown to induce leukocyte migration but the responsible signaling recept

119 issue hydration, release of collagenase, and leukocyte migration, but their roles in cervical ripenin

120 Ca2+ signals coordinate leukocytes migration, but whether Ca2+ fluxes mediated b

121 The glycocalyx is thought to affect leukocyte migration by masking adhesion molecules and re

122 helial migration, and that polymorphonuclear leukocyte migration can occur without permeability alter

123 This ligand bias correlates with changes in leukocyte migration, consistent with different mechanism

124 In addition to its role in leukocyte migration, CXCL10 inhibits the angiogenic func

125 iated with NK cytotoxicity, Ag presentation, leukocyte migration, cytokine activity, protein kinases,

126 pressed genes (DEGs) including enrichment in leukocyte migration, cytokine-cytokine receptor interact

127 a key role in immune homeostasis regulating leukocyte migration, differentiation, and function.

128 es, shear stress is a contributing factor to leukocyte migration directionality.

129 y contribute to neuropil ECM degradation and leukocyte migration during HAD.

130 oduction by endothelial cells and suppresses leukocyte migration during inflammation.

131 on, in embryonic tissue morphogenesis and in leukocyte migration during inflammation.

132 r of the gelatinase family of MMPs, mediates leukocyte migration during inflammation.

133 spases that regulate cytokine production and leukocyte migration during pathogen infection.

134 tokines studied to date in its regulation of leukocyte migration during primary listeriosis.

135 MIF-2 promotes leukocyte migration, endothelial arrest, and foam-cell f

136 The effect of strawberry extract and P3G, on leukocyte migration, exudation levels and many inflammat

137 Leukocyte migration from a hemopoietic pool across marro

138 Leukocyte migration from blood into lymphoid and non-lym

139 Leukocyte migration from bloodstream to tissue requires

140 Leukocyte migration from bloodstream to tissue requires

141 ivo lung perfusion (EVLP) to study passenger leukocyte migration from donor lungs into the recipient

142 Leukocyte migration from the bloodstream to a site of in

143 ization of MCP-1 significantly reduced total leukocyte migration (>50% reduction), whereas neutraliza

144 of small, homologous proteins that regulate leukocyte migration, hemopoiesis, and HIV-1 absorption.

145 ing adhesion dynamics, with implications for leukocyte migration, immune responses and potentially pa

146 ial cells and/or to induce polymorphonuclear leukocyte migration in a tissue culture model of mammali

147 own, and impaired both monocyte adhesion and leukocyte migration in a transwell system (p < 0.0001).

148 N, HCoV-OC43 N inhibits CXCL12beta-mediated leukocyte migration in chemotaxis assays, as do all high

149 a causative role for negative regulators of leukocyte migration in chronic inflammation.

150 To better understand the mechanism of leukocyte migration in complex environments, model extra

151 nes CCL2 and CX3CL1 probably are involved in leukocyte migration in diabetic nephropathy.

152 ls and macrophages, collagen deposition, and leukocyte migration in fibroblasts, chemotaxis in endoth

153 oteinase 9 (MMP-9) is a critical mediator of leukocyte migration in hepatic ischemia/reperfusion (I/R

154 Whether this motility pattern applies for leukocyte migration in inflamed tissue is still unknown.

155 ns plays a central role in the regulation of leukocyte migration in inflammatory responses.

156 te/endothelial adhesion and transendothelial leukocyte migration in inflammatory states.

157 g, pro-inflammatory cytokine production, and leukocyte migration in intestinal immune (myeloid) and e

158 undly inhibited MMP-9 activity and depressed leukocyte migration in livers after I/R injury.

159 Evaluation of fluorescently labeled leukocyte migration in mice revealed that DCs travel via

160 lex, hyper-IL-6, and to effectively modulate leukocyte migration in murine acute peritonitis.

161 to excessive neutrophil TEM and uncontrolled leukocyte migration in murine inflammatory models, while

162 s, and upregulated gene pathways involved in leukocyte migration in presymptomatic experimental autoi

163 in human and its mouse homolog mFPR2 mediate leukocyte migration in response to agonists associated w

164 formylpeptide receptor (FPR), which mediates leukocyte migration in response to bacterial and host-de

165 To study leukocyte migration in response to surface-bound chemoki

166 erum concentration of molecules that control leukocyte migration in serial samples from 34 patients f

167 ht the utility of this approach for defining leukocyte migration in the context of homeostasis and ca

168 mors, thereby advancing our understanding of leukocyte migration in the context of tumor development.

169 is approach unveiled the substantial role of leukocyte migration in tumor progression using a preclin

170 a metalloproteinase (ADAM) 10 and ADAM17 for leukocyte migration in vitro and in a murine model of ac

171 cell-cell interactions necessary to sustain leukocyte migration in vitro and tissue infiltration in

172 grin CD18 has been shown to be important for leukocyte migration in vitro.

173 d cholinergic agonists significantly blocked leukocyte migration in vivo.

174 fects of PECAM-1 and alphavbeta3 blockade on leukocyte migration in vivo.

175 vivo imaging technique for visualization of leukocyte migration into and out of corneal stroma, we s

176 The chemokine receptor CCR7 drives leukocyte migration into and within lymph nodes (LNs).

177 ta (IL-1beta), to predict chemotactic driven leukocyte migration into and within the artery wall.

178 e studies suggest that autoantigens initiate leukocyte migration into damaged and inflamed tissue tha

179 Chemokine receptors mediate leukocyte migration into inflamed rheumatoid arthritis (

180 ttractant receptors synergistically regulate leukocyte migration into lymphoid tissues and sites of i

181 otein receptor mediates the initial steps of leukocyte migration into secondary lymphoid organs and s

182 n and ICAM-1 expression dramatically reduced leukocyte migration into sites of inflammation beyond wh

183 Leukocyte migration into sites of inflammation involves

184 ammatory response involved polymorphonuclear leukocyte migration into the alveolar space and the accu

185 toxicity in ALS, and stimulating peripheral leukocyte migration into the brain in inflammatory condi

186 An analysis of leukocyte migration into the brain revealed that adminis

187 activity with brain capillaries and to block leukocyte migration into the brain was used to identify

188 llowing infection and for the restriction of leukocyte migration into the brain.

189 model of MS, lovastatin treatment inhibited leukocyte migration into the CNS and significantly atten

190 CCL7, in directing monocyte mobilization and leukocyte migration into the CNS is unclear.

191 This leukocyte migration into the cornea when erosions develo

192 Selective blockade of leukocyte migration into the gut is a promising strategy

193 f HIFs that myeloid HIFs are dispensable for leukocyte migration into the inflamed eye.

194 0) = 0.75 nM), suggesting that inhibition of leukocyte migration into the knee joint is a likely mech

195 with rheumatoid arthritis, and they promote leukocyte migration into the synovial tissue.

196 ctive absence of Mac-1 impairs transplatelet leukocyte migration into the vessel wall, reducing leuko

197 Leukocyte migration into tissues is characteristic of in

198 t collagen-binding integrins are involved in leukocyte migration into tissues.

199 cytes to vascular endothelium is crucial for leukocyte migration into tissues.

200 Anticancer immunity is predicated on leukocyte migration into tumors.

201 ed that poly(I:C) induced T/B cell-dependent leukocyte migration into white pulp regions.

202 conditions, as well as in various diseases, leukocyte migration is a crucial issue for the immune sy

203 Leukocyte migration is critical for the infiltration of

204 Chemokine-directed leukocyte migration is crucial for effective immune and

205 Efficient leukocyte migration is important for an effective host r

206 The control of leukocyte migration is of key interest during conditions

207 Leukocyte migration is the hallmark of inflammation, and

208 The multistep sequence leading to leukocyte migration is thought to be locally regulated a

209 Thus, with regard to leukocyte migration, leukocyte-expressed ADAM10 but not

210 tor agonist can inhibit the normal vectorial leukocyte migration mediated by chemokines.

211 y ligands such as VCAM-1 markedly stimulates leukocyte migration mediated by LFA-1 (integrin alpha(L)

212 involve vessel infiltration by inflammatory leukocytes, migration of medial vascular smooth muscle c

213 onsiveness was not associated with increased leukocyte migration or mucous production in the lung but

214 tion, and iii) transcriptional enrichment of leukocyte migration pathways.

215 production, intracellular antioxidation, and leukocyte migration plus genes for proinflammatory cytok

216 (< 40-microns diameter), whereas most of the leukocyte migration (predominantly neutrophils) occurred

217 This blockade in the leukocyte migration process is consistent with the obser

218 e conventional multistep paradigm holds that leukocyte migration represents a cascade of events, init

219 ted with CFTR(172) corrected the exaggerated leukocyte migration seen in these animals.

220 s also displayed defective polymorphonuclear leukocyte migration, suggesting mast cells as one source

221 These studies define a distinct process of leukocyte migration that is initiated by homotypic adhes

222 e, we identified a fasting-induced switch in leukocyte migration that prolongs monocyte lifespan and

223 both remodeling of extracellular matrix and leukocyte migration, their influence on the outcome of i

224 Leukocyte migration through activated venular walls is a

225 cultured cells revealed that sVAP-1 promotes leukocyte migration through catalytic generation of ROS,

226 nd suggested a role for the DDR1a isoform in leukocyte migration through extracellular matrix.

227 f the ImmunoCloak also significantly reduced leukocyte migration through the endothelial cell layer b

228 Leukocyte migration through the endothelial cell wall in

229 The Slit protein guides neuronal and leukocyte migration through the transmembrane receptor R

230 s most likely induce cytokine production and leukocyte migration through TLR7 signaling.

231 The mechanism of leukocyte migration through venular walls in vivo is lar

232 Neutralization of CCL5 and CXCL10 decreases leukocyte migration to areas of infection by 70%.

233 h some, but not all, chemokines and controls leukocyte migration to inflammatory sites.

234 Thus, MCK-1/MCK-2 appears to promote host leukocyte migration to initial sites of infection and ma

235 t serum levels of IL-6 and TNF alpha and the leukocyte migration to lungs and peritoneal cavity in LP

236 kocyte activation and may directly influence leukocyte migration to peripheral lymphoid tissues or to

237 G-protein-coupled receptor that facilitates leukocyte migration to regional lymph nodes.

238 uronan substrates and has been implicated in leukocyte migration to sites of inflammation.

239 cross lung grafts, responded to infection by leukocyte migration to small airways and alveoli of the

240 r investigating the mechanisms that regulate leukocyte migration to the joint in systemic models of R

241 dity, survival, clearance of bacteremia, and leukocyte migration to the peritoneal cavity and organs

242 Intravenous 4F-GalNAc infusion reduced leukocyte migration to the peritoneum in a murine model

243 compound was highly effective at inhibiting leukocyte migration toward CypA in vitro as well as in t

244 Leukocyte migration towards injury sites is directed by

245 Chemokines, 8 kDa proteins implicated in leukocyte migration via oligomerization, bind to glycosa

246 in the severe/profound hearing loss group of leukocyte migration, viral infection, and migration of c

247 onse to synthetic dsRNA was acute, extensive leukocyte migration was observed.

248 the bell-shaped concentration dependence of leukocyte migration was shown to arise from the agonist

249 study the in vivo effects of IP-10 on human leukocyte migration, we then examined the ability of rec

250 a new experimental concept for understanding leukocyte migration within the wounded cornea.

251 recruitment of this membrane to the zones of leukocyte migration, without affecting the constitutive