ライフサイエンスコーパス: leukocyte rolling

1 Weibel-Palade bodies and P-selectin-mediated leukocyte rolling.

2 dministration of a compound that antagonizes leukocyte rolling.

3 etics, thereby failing to accurately predict leukocyte rolling.

4 nd tether extraction of leukocytes stabilize leukocyte rolling.

5 surable effect on P- or L-selectin-dependent leukocyte rolling.

6 pits, which enhances its ability to support leukocyte rolling.

7 ital microscopy induced P-selectin-dependent leukocyte rolling.

8 (EC) leukocyte receptor P-selectin initiates leukocyte rolling.

9 be limited by inhibiting selectin-dependent leukocyte rolling.

10 the firm adhesion of sticking cells than for leukocyte rolling.

11 oninflamed skin venules support constitutive leukocyte rolling.

12 on by rapid induction of P-selectin-mediated leukocyte rolling.

13 ile not affecting the selectin-mediated slow leukocyte rolling.

14 deficient mouse almost completely eliminated leukocyte rolling.

15 electin-deficient mice significantly reduces leukocyte rolling.

16 ditional physiologic regulatory parameter of leukocyte rolling.

17 ement, particularly during selectin-mediated leukocyte rolling.

18 locations and potentiate selectin-dependent leukocyte rolling.

19 sis factor-alpha (TNF-alpha) and causes slow leukocyte rolling.

20 ed torque is critical for the maintenance of leukocyte rolling.

21 flow, whereas pCS caused a rapid increase in leukocyte rolling.

22 contribute significantly to the velocity of leukocyte rolling.

23 ally vasodilation, platelet aggregation, and leukocyte rolling.

24 e required for downstream signaling and slow leukocyte rolling.

25 /-) mice exhibited reduced histamine-induced leukocyte rolling.

26 eparation range to be 0.01-0.1 microm during leukocyte rolling.

27 ich partially inhibited P-selectin-dependent leukocyte rolling.

28 In contrast, leukocyte rolling 2 h after tumor necrosis factor alpha

29 ulted in a several-fold systemic increase in leukocyte rolling 2 to 4 hours after infusion.

30 ed a significant, time-dependent increase in leukocyte rolling (56 +/- 8 cells/min; P < 0.01 vs. cont

31 208075 dose-dependently reduced postischemic leukocytes rolling (7.3+/-2.3 vs. 3.3+/-1.4 vs. 0.7+/-0.

32 Leukocyte recruitment requires leukocyte rolling, activation, firm adhesion, and transm

33 tenuated thrombin-induced and L-NAME-induced leukocyte rolling, adherence, and transmigration in rat

34 bited leukocyte-endothelium interaction (ie, leukocyte rolling, adherence, and transmigration) induce

35 activation is crucial for the regulation of leukocyte rolling, adhesion and trans-vessel migration d

36 local vascular endothelial cells to mediate leukocyte rolling, adhesion, and extravasation by up-reg

37 mmation is a multistep process that involves leukocyte rolling, adhesion, and extravasation mediated

38 oaminooxidase that is involved in modulating leukocyte rolling, adhesion, and migration.

39 s pCRP administration significantly enhanced leukocyte rolling, adhesion, and transmigration via loca

40 ated and TNF-alpha-treated Galnt1(-/-) mice, leukocyte rolling, adhesion, and transmigration were sig

41 capacity of TNF-stimulated HUVECs to support leukocyte rolling, adhesion, and transmigration.

42 ocyte-endothelial cell interactions, such as leukocyte rolling, adhesion, and ultimately transendothe

43 ation, NF-kappaB activation, vaso-occlusion, leukocyte rolling/adhesion, and heme lethality.

44 Leukocyte rolling after surgical trauma was assessed in

45 (only E-selectin present) show an absence of leukocyte rolling after trauma and severely reduced roll

46 Selectins and their ligands mediate leukocyte rolling, allowing interactions with chemokines

47 s of inflammation and tissue injury involves leukocyte rolling along the endothelial wall, followed b

48 Leukocyte rolling along the endothelium in inflammation

49 Senp2(+/-) mice exhibited increased leukocyte rolling along the endothelium, and accelerated

50 fragments, followed by E-selectin-dependent leukocyte rolling along the endothelium.

51 electin) have been shown to be essential for leukocyte rolling along the vascular endothelium, the fi

52 Leukocyte rolling along the walls of inflamed venules pr

53 cell surface adhesion receptor that mediates leukocyte rolling along vascular endothelium at sites of

54 ncy was associated with a 2-fold increase in leukocyte rolling and a 5-fold increase in leukocyte adh

55 D-Glucose significantly increased leukocyte rolling and adherence in mesenteric postcapill

56 logs at 10 nmol/liter reduces L-NAME-induced leukocyte rolling and adherence in the mesenteric rat mi

57 However, the increased leukocyte rolling and adherence that occurred in cholest

58 n nitrate-fed mice there is reduced systemic leukocyte rolling and adherence, circulating neutrophil

59 (50 microM) significantly increased venular leukocyte rolling and adherence, which were also signifi

60 PS resulted in significant increases in both leukocyte rolling and adherence, which were maintained f

61 and wall shear rates, coupled with increased leukocyte rolling and adherence.

62 ice with pentostatin significantly decreased leukocyte rolling and adhesion (6.02 +/- 0.09 versus 1.7

63 However, leukocyte rolling and adhesion almost trebled by 7 days

64 of both agents led to marked improvements in leukocyte rolling and adhesion and decreased heterotypic

65 of the lung allograft with TNF-alpha induced leukocyte rolling and adhesion in both arterioles and ve

66 .001), and NOS inhibition further increased leukocyte rolling and adhesion in both septic and contro

67 ulature demonstrated significant (P < 0.001) leukocyte rolling and adhesion in brain venules of P. be

68 tion in brain myeloperoxidase, and increased leukocyte rolling and adhesion in cerebral venules of wi

69 with LPS significantly inhibited LPS-induced leukocyte rolling and adhesion in mesenteric postcapilla

70 tion, intravital microscopy revealed reduced leukocyte rolling and adhesion in P2X7-deficient mice.

71 ere, we use intravital microscopy to examine leukocyte rolling and adhesion in Peyer's patch high end

72 escence microscopy revealed highly increased leukocyte rolling and adhesion in postcapillary skin ven

73 sive imaging revealed a dramatic increase in leukocyte rolling and adhesion in veins near the optic n

74 shed the inhibitory effect of adiponectin on leukocyte rolling and adhesion in vivo.

75 idized, but not native, EPA markedly reduced leukocyte rolling and adhesion to venular endothelium of

76 reased mesenteric vessel adhesion molecules, leukocyte rolling and adhesion to vessels, leukocyte and

77 Leukocyte rolling and adhesion was increased in septic r

78 The effects of L-arginine on leukocyte rolling and adhesion were also measured, both

79 The effects of NOS inhibition on leukocyte rolling and adhesion were also measured.

80 Leukocyte rolling and adhesion were measured in cremaste

81 Leukocyte rolling and adhesion were significantly increa

82 esenteric microcirculation demonstrated that leukocyte rolling and adhesion were unaffected by the ab

83 l but not vitamin E, significantly decreased leukocyte rolling and adhesion, as well as capillary end

84 is, but NO is also an important inhibitor of leukocyte rolling and adhesion.

85 lecules including E-selectin, which mediates leukocyte rolling and adhesion.

86 n function, completely inhibited LPS-induced leukocyte rolling and adhesion.

87 rum serotonin by > 80% and similarly reduced leukocyte rolling and adhesion.

88 Leukocyte rolling and arrest on the vascular endothelium

89 al shear force in diverse situations such as leukocyte rolling and arrest on the vasculature, capture

90 d adhesion molecules, resulting in increased leukocyte rolling and atherosclerotic plaque size.

91 l microscopy of gingival vessels showed that leukocyte rolling and attachment to the vascular endothe

92 low rates, reduced microvascular endothelial leukocyte rolling and attachment, and minimized endothel

93 a cell-surface adhesion molecule involved in leukocyte rolling and attachment, has been hypothesized

94 ndothelial venules of lymph nodes to mediate leukocyte rolling and binds to a ligand on neutrophils t

95 d 10E9.6) on neutrophil recruitment in vivo, leukocyte rolling and circulating leukocyte concentratio

96 synovial microcirculation revealed enhanced leukocyte rolling and diminished adherence in mice lacki

97 Leukocyte rolling and emigration in response to inflamma

98 the role of the 3 selectins (E, L, and P) in leukocyte rolling and emigration, a null mutation for L-

99 function synergistically to mediate optimal leukocyte rolling and entry into tissues, which is essen

100 or P- plus E-selectins, lead to deficits in leukocyte rolling and extravasation.

101 WT cremaster in p75-/- mice showed a robust leukocyte rolling and firm adhesion upon TNF-alpha activ

102 amics, a computational method for simulating leukocyte rolling and firm adhesion, we have developed a

103 Interestingly, in ERK5-EKO mice, increased leukocyte rolling and impaired vessel reactivity were re

104 K5 knockout (ERK5-EKO) mice showed increased leukocyte rolling and impaired vessel reactivity.

105 lectins (PL(-/-)) show drastic reductions in leukocyte rolling and in extravasation of neutrophils in

106 We examined the quality of leukocyte rolling and L-selectin-mediated signaling.

107 Virtual lack of leukocyte rolling and low extravasation at sites of infl

108 Selectins mediate leukocyte rolling and may represent good anti-inflammato

109 gical role in cell adhesion as a mediator of leukocyte rolling and migration during inflammation, we

110 nd factor (vWF) and P-selectin, which induce leukocyte rolling and platelet adhesion and aggregation.

111 that the loss of ppGalNAcT-1 led to reduced leukocyte rolling and recruitment and increased rolling

112 mice exhibit a significant reduction in both leukocyte rolling and recruitment and we show a failure

113 raction of P-selectin with PSGL-1 results in leukocyte rolling and recruitment of leukocytes to sites

114 tosis, resulting in part from alterations in leukocyte rolling and recruitment.

115 h discrete steps involving selectin-mediated leukocyte rolling and subsequent firm adhesion mediated

116 ursodeoxycholic acid attenuated HNE-induced leukocyte rolling and their firm adhesion to the endothe

117 surface protrusion is an important aspect of leukocyte rolling, and it should not be ignored when leu

118 apid P-selectin up-regulation and associated leukocyte rolling, and suggest that endothelial SK-1 is

119 (IVM) demonstrated that RvE1 rapidly reduced leukocyte rolling (approximately 40%) in venules of mice

120 tor CGP77175A inhibited E-selectin-dependent leukocyte rolling at a dose of 3 mg/kg.

121 forces, and successfully explains the stable leukocyte rolling at a wide range of shear rates over th

122 The observed differences in leukocyte rolling behavior demonstrated that ICAM-1 expr

123 and microvillus elasticity actively modulate leukocyte rolling behavior.

124 y gene targeting to lack CCR2 exhibit normal leukocyte rolling but have a pronounced defect in MCP-1-

125 ing mAb only partially inhibited LPS-induced leukocyte rolling, but completely inhibited LPS-induced

126 matically inhibited LPS-induced increases in leukocyte rolling, but unlike the P-selectin mAb did not

127 Inhibition of leukocyte rolling by 2- and 4-GSP-6 lasted 2-4 min.

128 Fucoidan also interferes with leukocyte rolling by binding to L-selectins (expressed o

129 , chemokine interactions with receptors, and leukocyte rolling cell adhesion.

130 In parallel plate flow assays used to model leukocyte rolling, cells expressing CD44/TSG-6 failed to

131 ing significantly reduced the "jerkiness" of leukocyte rolling, defined as the variability of velocit

132 ntegrins play a critical role in stabilizing leukocyte rolling during a protracted cellular activatio

133 obilize it to the plasma membrane to mediate leukocyte rolling during inflammation.

134 f the L-, E-, and P-selectins, which promote leukocyte rolling during inflammation.

135 ocyte migration to peripheral LNs (PLNs) and leukocyte rolling during inflammation.

136 PSGL-1) binding to P-selectin controls early leukocyte rolling during inflammation.

137 simulations are continued for at least 1s of leukocyte rolling during which the instantaneous quantit

138 yond its well-established roles in mediating leukocyte rolling, E-selectin is emerging as a multifunc

139 n-ligand bonds are primarily responsible for leukocyte rolling, experimental evidence suggests that c

140 TNF-alpha-stimulated leukocyte rolling, firm adhesion to ECs, and transmigrat

141 steps of the inflammatory response; namely, leukocyte rolling, firm adhesion, and transmigration.

142 Leukocyte rolling flux and rolling velocity were assesse

143 esis of functional selectin ligands in vivo, leukocyte rolling flux and velocity were studied in venu

144 y (0-15 min) after surgical trauma, the mean leukocyte rolling flux fraction was lower (10 +/- 3 vs 3

145 cremaster muscle venules indicated that the leukocyte rolling flux fraction was reduced at blood cen

146 Although the leukocyte rolling flux fraction was reduced by 70%, Peye

147 icrovascular flow velocities and reduced the leukocyte rolling flux.

148 L-selectin-mediated leukocyte rolling has been proposed to require a high ra

149 tagogue of Weibel-Palade bodies, slowed down leukocyte rolling in Adamts13(-/-) but not in WT mice.

150 y functional to support L-selectin-dependent leukocyte rolling in Core2GlcNAcT-I-deficient mice.

151 pergammaglobulinemia, severe deficiencies of leukocyte rolling in cremaster venules with or without a

152 expression of selectin ligands required for leukocyte rolling in dermal microvessels.

153 ium-dependent lectin on leukocytes mediating leukocyte rolling in high endothelial venules and inflam

154 hat beta(2)-integrins are necessary for slow leukocyte rolling in inflamed venules.

155 -1 and LFA-1 play a cooperative role in slow leukocyte rolling in inflamed vessels, and that, althoug

156 Whether disrupted leukocyte rolling in L-selectin and ICAM-1 double-defici

157 Leukocyte rolling in liver tumor was twofold lower than

158 Leukocyte rolling in P-selectin/ICAM-1-deficient mice tr

159 TNF-alpha induced leukocyte rolling in P-selectin/ICAM-1-deficient mice, b

160 The defect in leukocyte rolling in PAR2-deficient mice did not persist

161 By contrast, leukocyte rolling in Peyer's patch HEV was not significa

162 studies have proposed that the abundance of leukocyte rolling in postcapillary venules is due to int

163 Leukocyte rolling in postcapillary venules of inflamed t

164 In contrast, 10E9.6 had no effect on leukocyte rolling in RB40.34-treated Balb/c or C57BL/6 m

165 ed on the wall of a flow chamber can support leukocyte rolling in shear flow.

166 ) significantly attenuated histamine-induced leukocyte rolling in the cremaster muscle.

167 However, a secretagogue failed to upregulate leukocyte rolling in the DeltaCT mice, indicating an abs

168 Selected compounds showed decrease in leukocyte rolling in the IVM mouse model.

169 The atherogenic diet stimulated leukocyte rolling in the mesenteric venules in both geno

170 The atherogenic diet stimulated leukocyte rolling in the mesenteric venules of LDLR-defi

171 e of mediating cell adhesion as it supported leukocyte rolling in the mutant mice.

172 We also investigated leukocyte rolling in the presence of PSGL-1 antibody or

173 Leukocyte rolling in the vasculature is mediated by the

174 ions with fucosylated glycan ligands mediate leukocyte rolling in the vasculature under shear forces.

175 Neither 9A9 nor 10E9.6 alone blocked leukocyte rolling in tumor necrosis factor-alpha-treated

176 We investigated trauma-induced leukocyte rolling in venules (diameter, 23 to 58 microns

177 face expression of P-selectin and subsequent leukocyte rolling in venules can be induced by mast cell

178 ns during inflammation, we have investigated leukocyte rolling in venules of tumor necrosis factor-al

179 electin ligand biosynthesis, we investigated leukocyte rolling in venules of untreated and TNF-alpha-

180 We report a strong reduction of leukocyte rolling in venules of vWf-deficient mice.

181 profiles, inhibition of P-selectin-dependent leukocyte rolling in vivo by such a compound has yet to

182 e of both SK-1 and SK-2 in histamine-induced leukocyte rolling in vivo was assessed using pharmacolog

183 duce but do not abolish P-selectin-dependent leukocyte rolling in vivo whereas PSGL-1-deficient mice

184 Leukocyte rolling in vivo, studied by intravital microsc

185 function synergistically to mediate optimal leukocyte rolling in vivo, which is essential for the ge

186 ctin ligand can inhibit P-selectin-dependent leukocyte rolling in vivo.

187 whether GSP can inhibit P-selectin-dependent leukocyte rolling in vivo.

188 WT and AnxA1-null animals without affecting leukocyte rolling, in comparison to saline control.

189 vated P-selectin-/- platelets did not induce leukocyte rolling, indicating that platelet P-selectin w

190 h angiogenesis suppression and inhibition of leukocyte rolling induced by gallbladder tumors.

191 Leukocyte rolling is also facilitated by members of the

192 Selectin-mediated leukocyte rolling is crucial for the proper function of

193 e made in L-selectin-deficient mice in which leukocyte rolling is entirely P-selectin dependent.

194 e observations show that P-selectin-mediated leukocyte rolling is not required for the development of

195 This severe defect in leukocyte rolling may explain the absence of leukocyte r

196 AM-1 is the main ligand responsible for slow leukocyte rolling mediated by Mac-1, but not LFA-1.

197 ized in contributing to the dynamic range of leukocyte rolling observed in vivo during inflammatory p

198 Selectin adhesion molecules mediate leukocyte rolling on activated endothelium, a prerequisi

199 toplasmic viscosity plays a critical role in leukocyte rolling on an adhesive substrate.

200 dorsal skinfold chamber revealed unaffected leukocyte rolling on anti-VWF treatment.

201 rce to receptor-ligand molecules involved in leukocyte rolling on blood vessel walls.

202 CD44 enables slow leukocyte rolling on E-selectin expressed on inflamed en

203 The selectins and their ligands mediate leukocyte rolling on endothelial cells, the initial step

204 L-Selectin mediates leukocyte rolling on endothelium and immobilized leukocy

205 ctivated endothelial cells, is essential for leukocyte rolling on endothelium which leads to extravas

206 nction as an adhesion receptor that mediates leukocyte rolling on hyaluronan (HA).

207 ix glycosaminoglycan hyaluronan, can mediate leukocyte rolling on hyaluronan substrates and has been

208 synergistic increase in E-selectin-dependent leukocyte rolling on microvascular endothelium in vivo.

209 These functional studies show that leukocyte rolling on P- and L-selectin is ablated in cel

210 PSGL-1 cytoplasmic domain is dispensable for leukocyte rolling on P-selectin but is essential to acti

211 lectin requires a threshold shear to support leukocyte rolling on P-selectin glycoprotein ligand-1 (P

212 ocyte cortical cytoskeleton is essential for leukocyte rolling on P-selectin.

213 tween catch and slip bonds might explain why leukocyte rolling on selectins first increases and then

214 Leukocyte rolling on the endothelium via selectin molecu

215 During leukocyte rolling on the endothelium, surface protrusion

216 presents a generic mechanism for stabilizing leukocyte rolling on the endothelium.

217 Leukocyte rolling on the vascular endothelium requires i

218 hat ADAMTS13 deficiency results in increased leukocyte rolling on unstimulated veins and increased le

219 cyte migration to peripheral lymph nodes and leukocyte rolling on vascular endothelium during inflamm

220 L-selectin mediates leukocyte rolling on vascular endothelium during inflamm

221 , has dual function as a selectin ligand for leukocyte rolling on vascular selectins expressed in inf

222 nteractions of PSGL-1 with selectins mediate leukocyte rolling on vascular surfaces.

223 Selectin-ligand interactions (bonds) mediate leukocyte rolling on vascular surfaces.

224 to be far less than velocities observed for leukocytes rolling on L-selectin in vivo.

225 Nevertheless, the number of leukocytes rolling on postcapillary venules in an E-sele

226 Leukocytes rolling on selectins extrude thin membrane te

227 ti-LFA-1 significantly reduced the number of leukocytes rolling on venule endothelial surfaces, but t

228 ngly, unlike VEGF-A, VEGF-C did not increase leukocyte rolling or adhesion in tumor vessels.

229 suppressed leukocyte extravasation, but not leukocyte rolling or firm adhesion, elicited by IL-1beta

230 a strategy that did not significantly reduce leukocyte rolling or sticking in iris vessels but blocke

231 Leukocytes rolling or adhering to endothelium were count

232 vascular leakage but did not further enhance leukocyte rolling over pCS alone.

233 sentation of adhesion molecules relevant for leukocyte rolling (P-selectin) and platelet capture (von

234 tal microscopy, we showed that the number of leukocytes rolling per minute in unstimulated veins was

235 om inflammatory regions might form part of a leukocyte rolling response, increasing the plaque volume

236 We conclude that the residual trauma-induced leukocyte rolling seen in P-selectin-deficient mice is c

237 e rolling, and it should not be ignored when leukocyte rolling stability is studied systematically.

238 of the presumed glycocalyx rolled more like leukocytes: rolling steps were more uniform and shear re

239 n of platelets in Adamts13(-/-) mice reduced leukocyte rolling, suggesting that platelet interaction

240 E-Selectin-null mice showed more rapid leukocyte rolling than wild-type or ICAM-1-deficient mic

241 We conclude that slow leukocyte rolling through E-selectin results in long tra

242 -1) is expressed as a homodimer and mediates leukocyte rolling through interactions with endothelial

243 ng drastically increases the transit time of leukocytes rolling through an inflamed tissue and thus a

244 The addition of cyanoborohydride to leukocytes rolling through L-selectin on mildly oxidized

245 microscopy of mesenteric venules showed that leukocyte rolling time was decreased, whereas rolling ve

246 Leukocyte rolling time, defined as the time it takes for

247 ently and markedly attenuated L-NAME-induced leukocyte rolling to 10 +/- 4 (P < 0.01), 4 +/- 1 (P < 0

248 increasing levels of shear stress stabilize leukocyte rolling under flow.

249 dly enhances E-selectin's ability to mediate leukocyte rolling under flow.

250 ytokine-activated endothelial cells mediates leukocyte rolling under flow.

251 lectin mediates, in part, the early event of leukocyte rolling under hydrodynamic flow, the contribut

252 via long-lived catch-bonds that support slow leukocyte rolling under shear stress.

253 intercellular adhesion molecule (ICAM) 1 in leukocyte rolling using gene-targeted mice deficient in

254 udied the effect of cytoplasmic viscosity on leukocyte rolling using our three-dimensional numerical

255 Specifically, leukocyte rolling velocities during inflammation are sig

256 Leukocyte rolling velocities in cremaster muscle venules

257 Consistent with previous studies, leukocyte rolling velocities on P-selectin were observed

258 Increased leukocyte rolling velocities presumably translated into

259 Leukocyte rolling velocities were significantly reduced

260 Leukocyte rolling velocity (Vwbc) and the number of adhe

261 ouse cremaster muscle venules show increased leukocyte rolling velocity and reduced leukocyte recruit

262 This extracellular activation loop reduces leukocyte rolling velocity and stimulates adhesion.

263 The IC-mediated slow leukocyte rolling velocity and subsequent adhesion and e

264 Rolling flux and average leukocyte rolling velocity in ICAM-1-deficient mice was

265 a role for L-selectin shedding in regulating leukocyte rolling velocity in vivo.

266 In contrast, leukocyte rolling velocity is significantly decreased an

267 alysis of the mesenteric venules showed that leukocyte rolling velocity was markedly decreased and nu

268 The median leukocyte rolling velocity was reduced in L-/- mice and

269 injection--we recorded significantly reduced leukocyte rolling velocity, which suggests PSGL-1 up-reg

270 he earliest steps in leukocyte adhesion (ie, leukocyte rolling) via administration of a recombinant s

271 ainst P- and E-selectin, L-selectin-mediated leukocyte rolling was almost completely abolished in cre

272 he majority of observed L-selectin-dependent leukocyte rolling was between free flowing leukocytes an

273 L-selectin-dependent leukocyte rolling was completely abolished in Galnt1(-/-

274 In untreated core 2(-/-) mice, leukocyte rolling was dramatically reduced with markedly

275 Leukocyte rolling was mediated solely by the interaction

276 w chamber assay, P- and E-selectin-dependent leukocyte rolling was mildly reduced in St3gal6-null mic

277 A significant decrease in leukocyte rolling was observed at 30-min and 5-hr reperf

278 Leukocyte rolling was significantly slower for leukocyte

279 With a biomechanical model of leukocyte rolling, we calculate the force history on the

280 With a biomechanical model of leukocyte rolling, we predict the force history of the a

281 /c mice, but 9A9 almost completely inhibited leukocyte rolling when combined with the function-blocki

282 substantially reversed P-selectin-dependent leukocyte rolling, whereas control GSP, which are not fu

283 ctin-dependent, but not E-selectin-dependent leukocyte rolling, whereas in double-deficient mice, E-s

284 so led to a significant 1.4-fold increase in leukocyte rolling, whereas inhibition of heme oxygenase

285 ds to intermediate affinity and induces slow leukocyte rolling, whereas P-Rex1 is not involved in the

286 Leukocyte rolling, which is increased in CD44-deficient

287 Leukocyte rolling, which is mediated by P-selectin in th

288 lectin-mediated integrin activation and slow leukocyte rolling, which promotes ischemia-reperfusion-i