ライフサイエンスコーパス: leukocytosis

1 swelling at the site, purulent drainage, and leukocytosis).

2 as well as the chemokine CXCL12, leading to leukocytosis.

3 smell, peri-wound erythema, hypotension, and leukocytosis.

4 tion (e.g., serum elastase) accompanies this leukocytosis.

5 r in mice, characterized by splenomegaly and leukocytosis.

6 despite the expected induction of peripheral leukocytosis.

7 with fever, arthralgias, conjunctivitis, and leukocytosis.

8 with lung or colorectal cancer often exhibit leukocytosis.

9 valuable in the evaluation of posttransplant leukocytosis.

10 30% (14/47) of the patients never developed leukocytosis.

11 hritis, granulomatous hepatitis, anemia, and leukocytosis.

12 associated with nausea, vomiting, fever, and leukocytosis.

13 tested, while causing no adverse symptoms or leukocytosis.

14 due to abdominal pain and 66% presented with leukocytosis.

15 patients with essential thrombocythaemia and leukocytosis.

16 ge, history of diverticulosis, vomiting, and leukocytosis.

17 baboons developed illness (with 1 death) and leukocytosis.

18 Laboratory tests were remarkable for leukocytosis.

19 s (45%) required either GO or idarubicin for leukocytosis.

20 E, except for the low levels of BALP and the leukocytosis.

21 larization or obesity-induced adipose tissue leukocytosis.

22 blood myeloid cells in patients with febrile leukocytosis.

23 tive protein level (127 mg/L [1209 nmol/L]), leukocytosis (13 800/mm(3)), and microcytic anemia (hemo

24 isib (all causality) were neutropenia (18%), leukocytosis (14%), thrombocytopenia (12%), pneumonia (1

25 r than that for patients who did not develop leukocytosis (2,100 cells/microL; range, 500 to 5,400 ce

26 riate Cox proportional hazards models showed leukocytosis (2.85 [hazard ratio]; P = .02), hypoalbumin

27 ) and significantly better in the absence of leukocytosis (37% v 8%; P = .01); JAK2V617F or cytogenet

28 ] vs 35/256 [13.7%]; p = 0.04) and lower CSF leukocytosis (4 cells/mm3 [3-25 cells/mm3] vs 52 cells/m

29 criterion, although thrombocytosis (53%) and leukocytosis (49%) are common.

30 Leukocytosis (53%) and fever (34%) were the most common

31 ardiogram QT prolongation (26%), and grade 3 leukocytosis (9%).

32 iogram QT prolongation (26%), and grade >= 3 leukocytosis (9%).

33 the underlying mechanisms of heparin-induced leukocytosis, a side effect occurring in 0.44% of patien

34 osis in hypercholesterolemic mice, displayed leukocytosis, a transplantable myeloproliferative disord

35 Leukocytosis affects CHD through multiple pathologic mec

36 mouse had significantly increased peritoneal leukocytosis after intraperitoneal challenge with thiogl

37 Because hydroxyurea also reduces leukocytosis, an understanding of the impact of this tre

38 27% of the patients and was associated with leukocytosis and a high percentage of bone marrow blast

39 ection of Ptx in mice elicits a long-lasting leukocytosis and a progressive increase in circulating c

40 G-CSF produced a dose-dependent neutrophil leukocytosis and a proportional increase in oxidase acti

41 connect expansion of these populations with leukocytosis and accelerated atherosclerosis.

42 Leukocytosis and activated monocytes are also observed i

43 a dominant-negative p53 construct developed leukocytosis and blastic infiltration of lymph nodes, sp

44 s and CD34(+) HSPCs that was associated with leukocytosis and central nervous system infiltration, bo

45 The aCML defining features of leukocytosis and circulating myeloid precursors, but not

46 sponse to systemic immune cell depletion and leukocytosis and could have prognostic relevance.

47 pound mutation was associated with increased leukocytosis and disease progression of the patient.

48 Blood tests showed mild anemia, neutrophilic leukocytosis and elevated inflammation markers.

49 Initial laboratory findings included leukocytosis and elevated liver enzymes.

50 proved erythropoiesis and anemia, alleviated leukocytosis and endothelial damage, diminished cell adh

51 rosol exposure, as determined by reduced BAL leukocytosis and eosinophilia, decreased tissue inflamma

52 Three of the four patients presented with leukocytosis and eosinophilia; their leukemia cells carr

53 Lsc-deficient mice have a peripheral leukocytosis and extramedullary hematopoiesis, demonstra

54 of floxed alleles, results in multi-lineage leukocytosis and features of autoimmunity.

55 Leukocytosis and hemoconcentration were noted in both ca

56 t a chronic inflammatory state manifested by leukocytosis and increased circulating levels of proinfl

57 18, which lack all four CD11 integrins, have leukocytosis and increased susceptibility to bacterial i

58 Novel disease-related determinants such as leukocytosis and JAK2V617F mutational status and/or muta

59 Lab work was significant for leukocytosis and lactic acidosis.

60 with significantly higher treatment-induced leukocytosis and more pronounced decrease of nodal disea

61 IL-10 modestly attenuated neutrophilic leukocytosis and neutrophil degranulation (plasma concen

62 Increased NETosis correlated with leukocytosis and neutrophilia, and neutrophils and NETs

63 kade with anakinra accelerates resolution of leukocytosis and neutrophilia.

64 Laboratory evaluation was notable for leukocytosis and neutrophilia.

65 , whereas hematopoietic loss of Ptch2 drives leukocytosis and promotes LKS maintenance and replating

66 that MLL-ENL fusion is sufficient for lethal leukocytosis and proof of genome integrity comes from ge

67 LPS-induced ALI, as indicated by alveolar leukocytosis and protein leak, inhibition of surfactant

68 acute wounds that fail to heal are excessive leukocytosis and reduced matrix deposition.

69 In conclusion, both leukocytosis and renal failure are useful predictors, al

70 regarding the association between persistent leukocytosis and risk of thrombosis and disease evolutio

71 mmatory response and modulate the subsequent leukocytosis and secondary tissue damage.

72 with palbociclib alone significantly reduced leukocytosis and splenomegaly and inhibited bone marrow

73 p40 expression (IL-12p40(-/-)) resulted in a leukocytosis and splenomegaly that was significantly les

74 anti-IL-1R1 antibodies significantly reduces leukocytosis and splenomegaly, and ameliorates bone marr

75 tion in nasopharyngeal washes and monitoring leukocytosis and symptoms.

76 s RA and arsenic trioxide is associated with leukocytosis and the RA syndrome.

77 n result in stimulation of both neutrophilic leukocytosis and the release of immature granulocytic po

78 Leukocytosis and thrombocytosis were also detected in in

79 ly characterized by erythrocytosis and often leukocytosis and thrombocytosis(1).

80 the lung, attributable in part to background leukocytosis and to intravascular sequestration.

81 b72F+AS01B induced better protection against leukocytosis and weight loss, suggesting that the polypr

82 Elevated leukocyte cell numbers (leukocytosis), and monocytes in particular, promote athe

83 mic inflammatory response syndrome of fever, leukocytosis, and a hyperdynamic state is common in trau

84 ormal bone remodeling, elevated CRP level or leukocytosis, and a neutrophilic infiltrate on skin biop

85 inflammatory disease characterized by fever, leukocytosis, and a rash with a neutrophilic infiltrate.

86 Anemia, leukocytosis, and abnormal liver enzyme tests were labor

87 otension, edema, hemoconcentration, profound leukocytosis, and absence of fever.

88 ions (arthritis, splenomegaly, hepatomegaly, leukocytosis, and acute-phase reaction) (P<0.01) in grou

89 ith all-trans RA is commonly associated with leukocytosis, and approximately 50% of patients develop

90 arked hepatosplenomegaly, myeloid dysplasia, leukocytosis, and biphenotypic surface markers.

91 was a marked reduction in TNF-alpha levels, leukocytosis, and brain pathology.

92 iated with the APL differentiation syndrome, leukocytosis, and electrocardiographic abnormalities.

93 e syndrome criteria (tachycardia, tachypnea, leukocytosis, and fever) in surgical ICU patients for IC

94 until 2 days after the resolution of fever, leukocytosis, and ileus, with a maximum of 10 days of th

95 d stage, central nervous system involvement, leukocytosis, and LDH >3 times the upper limit of normal

96 acilli from the cerebrospinal fluid, reduced leukocytosis, and less pathology of the brain and lungs

97 Our findings indicate that erythrocytosis, leukocytosis, and moderate splenomegaly with mild periva

98 vels of tumor necrosis factor-alpha , higher leukocytosis, and more-severe clinical manifestations.

99 tive disease, characterized by splenomegaly, leukocytosis, and myeloid hypercellularity, which progre

100 Nonspecific symptoms included fever, leukocytosis, and neurologic abnormalities.

101 al pathology findings included coagulopathy, leukocytosis, and profound liver destruction as indicate

102 which is characterized by thrombocytopenia, leukocytosis, and rapid onset of respiratory distress ca

103 igen test result, a vaginal pH >4.5, vaginal leukocytosis, and recurrent (vs initial) T. vaginalis in

104 e CDI suggested by recent guidelines (fever, leukocytosis, and renal failure), we used the database o

105 tion of BCR-ABL resulted in neutrophilia and leukocytosis, and the mice became moribund within 29 to

106 Higher bilirubin, coagulopathy, leukocytosis, and thrombocytopenia were independently as

107 risk factors for survival include older age, leukocytosis, and thrombosis, whereas JAK2 mutation in E

108 e headache; nonheadache, nonmeningeal signs; leukocytosis; and bacteremia.

109 s characterized in part by recurrent fevers, leukocytosis, anemia, and elevated acute phase proteins

110 ead, patients show progressive organomegaly, leukocytosis, anemia, and thrombocytosis.

111 evere hypoxemia (aOR 4.25, 95%CI 2.36-7.64), leukocytosis (aOR 2.35, 95%CI 1.35-4.11), thrombocytopen

112 [95% confidence interval {CI}, 2.36-7.64]), leukocytosis (aOR, 2.35 [95% CI, 1.35-4.11]), thrombocyt

113 (aOR, 1.53; 95% CI, 1.08-2.17), log of serum leukocytosis (aOR, 3.38; 95% CI, 2.48-4.61), and pyuria

114 In many cases, the mechanisms underlying leukocytosis are not known.

115 , including immunodeficiency, and persistent leukocytosis are risk factors for radiological recurrenc

116 Although causes of acute leukocytosis are well-described, chronic environmental d

117 15.5 vs 11.0 days; P < .01), consistent with leukocytosis as a prognostic marker in CDI.

118 10(9) white blood cells per L), progressive leukocytosis (at least 100% increase if baseline count i

119 in over 800 patients, suggest that not just leukocytosis but also tendency to "anemia of inflammatio

120 LFA-1-deficient mice exhibit leukocytosis but do not develop spontaneous infections,

121 and humans with atherosclerosis from chronic leukocytosis but does not compromise emergency hematopoi

122 Stable patients with leukocytosis, but without fever or documented infections

123 ng of onset and awareness of the patterns of leukocytosis can be valuable in the evaluation of posttr

124 ears of life (dactylitis, severe anemia, and leukocytosis) can help to predict the possibility of sev

125 r expression of vIL-10 significantly reduced leukocytosis, cartilage matrix degradation, and levels o

126 Double deficiency yields an extreme leukocytosis characterized by decreased neutrophil turno

127 eloping anemia, thrombocytopenia, and marked leukocytosis compared with other subtypes.

128 lacking non-neuronal serotonin, showed mild leukocytosis compared with wild-type (WT), primarily dri

129 MI-associated blood leukocytosis correlates inversely with patient survival,

130 flow of leukocytes to inflamed tissues, and leukocytosis correlates with cardiovascular mortality.

131 est that leukocytes may play a role, because leukocytosis correlates with clinical severity and early

132 f physiological responses including a marked leukocytosis, disruption of glucose regulation, adjuvant

133 area and reduced lesion size without causing leukocytosis, dry eye, hair loss, or a reduced life span

134 Sustained leukocytosis, due to an increase in neutrophils counts,

135 as well as low-risk patients who experienced leukocytosis during induction.

136 e, the doubly deficient mice present extreme leukocytosis, elevated cytokine levels, and alterations

137 Within minutes of trauma, a comprehensive leukocytosis, elevated serum pro- and anti-inflammatory

138 The fourth patient had leukocytosis, eosinophilia, and a t(5;12) translocation

139 features of MPNs, including thrombocytosis, leukocytosis, Epo-independent colony formation, characte

140 circulation (also known as "exercise-induced leukocytosis"), especially cytotoxic T cells and natural

141 L disease was characterized by splenomegaly, leukocytosis, extramedullary hematopoiesis (EMH) in sple

142 rinogen levels; tachycardia; thrombocytosis; leukocytosis; fever; leg edema; lower Barthel Index (BI)

143 ower incidence of marked anemia (31% v 39%), leukocytosis greater than 25 x 10(9)/L (9% v 13%), and b

144 ere associated with worse prognosis, whereas leukocytosis greater than grade 1 was associated with be

145 Twenty patients (50%) had leukocytosis (> 10,000 WBC/microL) during induction ther

146 300-7995] vs 5900 [4910-7200]; P < .01), but leukocytosis (>11 000/uL) was uncommon (4.3% vs 2.1%; P

147 tomatic progressive splenomegaly, persistent leukocytosis (>15 x 10(9) white blood cells per L), prog

148 In summary, leukocytosis has been consistently shown to be an indepe

149 lasm characterized by sustained neutrophilic leukocytosis, hepatosplenomegaly and bone marrow granulo

150 temic inflammation (anemia, thrombocytopenia leukocytosis, high erythrocyte sedimentation rate, eleva

151 urine TNF-alpha, as demonstrated by high CSF leukocytosis, high protein accumulation, severe meningea

152 lude abdominal pain, watery diarrhea, fever, leukocytosis, hypoalbuminemia, and hypovolemia.

153 ligand deficiency is distinguished by blood leukocytosis, impaired leukocyte extravasation in inflam

154 n beta2 subunit of CD11/CD18 integrins, have leukocytosis, impaired transendothelial neutrophil emigr

155 ptomatic aEPEC infection was associated with leukocytosis in 24% of patients.

156 Fifty consecutive episodes of leukocytosis in 47 liver transplant recipients were pros

157 Cyclophilin-inhibitors reduced leukocytosis in infant mouse model of pertussis, indicat

158 hemokines may be important in the control of leukocytosis in inflammatory disorders of the central ne

159 While investigating the source of leukocytosis in mice, we discovered that stress activate

160 kin-1 [IL-1] receptor antagonist) suppresses leukocytosis in myocardial infarction.

161 -) mice and show that, apart from anemia and leukocytosis in older mice, the production of mature blo

162 cent confidence interval, 1.14 to 5.33), and leukocytosis in the absence of infection (relative risk,

163 recruited myeloid cells to the CP, increased leukocytosis in the CSF and blood, but infiltration into

164 reported that dactylitis, severe anemia, and leukocytosis in very young children with sickle cell dis

165 ditional vcam-1-deficient mice revealed mild leukocytosis, including elevated immature B cell numbers

166 edentary lifestyle, chronic inflammation and leukocytosis increase atherosclerosis; however, it remai

167 tivariate analysis, severe thrombocytopenia, leukocytosis, increasing marrow blast percentage, unfavo

168 djustment for presence of abscess, fever, or leukocytosis; infection size; diabetes; patient age; and

169 L-12, and transfusion need; IL-2R, IL-8, and leukocytosis; IP-10 and thrombocytopenia; HGF, MIG, IL-1

170 While leukocytosis is a common feature of severe periodontitis

171 Leukocytosis is a common finding in patients with ST ele

172 Leukocytosis is associated with increased cardiovascular

173 Leukocytosis is associated with the proliferation of hem

174 ioxide, with minimal chemotherapy to control leukocytosis, is very effective therapy for newly diagno

175 Correlations are emerging between leukocytosis, JAK2(V617F) mutation, BM fibrosis, and dif

176 Bloods showed multi-organ derangement with leukocytosis, lactic acidosis, haemolytic anaemia and hy

177 as found for tenosynovitis (lambda(s) 29.5), leukocytosis (lambda(s) 25), rheumatoid factor (lambda(s

178 at the loss of Ccr7 results in a significant leukocytosis, leading to hypercellularity within the CNS

179 article-enabled in vivo RNAi mitigated blood leukocytosis, limited inflammation in the ischemic heart

180 racterized included impaired erythropoiesis, leukocytosis, loss of early progenitor cells in the bone

181 R, 1.4 [95% CI, 1.1-2.0]), nor does isolated leukocytosis (LR, <1.7).

182 ent monocytosis and is often associated with leukocytosis, lymphoproliferation, and autoimmune phenom

183 accounted for 28% (14/50) of the episodes of leukocytosis; median time to onset was 25 days after tra

184 ng elevation in hemoglobin level/hematocrit, leukocytosis, megakaryocyte hyperplasia, extramedullary

185 istent with an atypical MPN characterized by leukocytosis, monocytosis, splenomegaly, and progressive

186 counting methods do not distinguish between leukocytosis, neutrophil sequestration, and activation.

187 with significantly greater reticulocytosis, leukocytosis, neutrophilia and thrombocytosis, marked ex

188 re/critically ill cases were associated with leukocytosis, neutrophilia, lymphopenia, elevated creati

189 activation and develop a MPN-phenotype with leukocytosis (neutrophils and monocytes), strong progeni

190 2% (11/50) of the episodes, a characteristic leukocytosis occurred 7-14 days after transplantation (i

191 Leukocytosis of more than 30 x 10(9)/L was found in 26%,

192 dies are needed to assess the true impact of leukocytosis on CHD, compare it with other inflammatory

193 She had no evidence of leukocytosis or anemia and no symptoms of early satiety,

194 fter LT and was infrequently associated with leukocytosis or fever.

195 Infection was defined as leukocytosis or leukopenia, with a positive culture requ

196 The majority of patients with leukocytosis or thrombocytosis at baseline (n = 28 and n

197 discharge, pyrexia, uterine tenderness, and leukocytosis) or by the presence of one of these feature

198 r-related sepsis (CRS) was defined as fever, leukocytosis, or hypotension which resolved with cathete

199 splenomegaly, concurrent thrombocytosis and leukocytosis, or intolerance of phlebotomy can constitut

200 graphic infiltrate with at least 2 of fever, leukocytosis, or purulent sputum increases the likelihoo

201 se of postoperative (P < 0.01) or infectious leukocytosis (P < 0.05).

202 ms and signs, with more vomiting (P = .003), leukocytosis (P = .003), and blood in the stool (P = .00

203 ldren were less likely than controls to have leukocytosis (P<.001) and more likely to have isolates w

204 Prevalent at diagnosis were leukocytosis, peripheral blast exceeding marrow blast pe

205 crease as viral latency increased and as the leukocytosis phase of the disease progressed.

206 al or laboratory parameter, including fever, leukocytosis, pleocytosis, or CSF protein and glucose, c

207 sia, and marked thrombocythemia, but without leukocytosis, polycythemia, or marrow fibrosis, displayi

208 erium bovis Ravenel intracisternally induced leukocytosis (predominantly mononuclear cells), high pro

209 and laboratory data studied, prevalences of leukocytosis, prior antibiotic use, and hospitalizations

210 The presence of leukocytosis, prior antibiotic use, and previous hospita

211 NF-alpha in the CSF in association with high leukocytosis, protein accumulation, and severe meningeal

212 use severe disease, manifested as pronounced leukocytosis, pulmonary hypertension, and even death.

213 Deaths relate to leukocytosis, pulmonary hypertension, and pneumonia.

214 ents with a clinical diagnosis of PN (fever, leukocytosis, purulent sputum, and new or changing infil

215 All 8 patients developed leukocytosis (range, 14.5-27.6 x 10(9)/L) with eosinophi

216 sis accompanied by features of MF, including leukocytosis, reduced hematocrit, splenomegaly, and incr

217 Leukocytosis refers to an increase in leukocyte count ab

218 We conclude that heparin-induced leukocytosis requires glucosamine 6-O-sulfation and is c

219 cytotoxic therapy was administered, and the leukocytosis resolved in all cases.

220 ceptibility to the lethal effects of PTX and leukocytosis response were observed.

221 ce susceptibility to the lethal effects, the leukocytosis response, disruption of glucose regulation,

222 This leukocytosis resulted in a significant enhancement of le

223 veloping 24 to 40 h postexposure followed by leukocytosis resulting from a high percentage of neutrop

224 d hemoglobin, hematocrit, platelet count and leukocytosis, resulting in increased blood viscosity.

225 ultiple selectins display varying degrees of leukocytosis, resulting in part from alterations in leuk

226 Quantification of adipose tissue leukocytosis revealed that elimination of Chop during ob

227 (HDL) suppressed the LSK population, reduced leukocytosis, reversed the myeloproliferative disorder,

228 Leukocytosis (risk ratio [RR], 2.29; 95% confidence inte

229 The modest leukocytosis seen during paclitaxel cycles was attributa

230 patients with persistent fevers, increasing leukocytosis, sepsis or septic shock, advanced age, preg

231 However, after controlling for age, leukocytosis, sex, laparoscopic approach, and perforatio

232 ML-RAR alpha leukemias were characterized by leukocytosis, similar to human APL with FLT3 mutations.

233 mainly characterized by high spiking fevers, leukocytosis, skin rash, arthralgia and myalgia.

234 Clinically, JMML manifests as monocytic leukocytosis, splenomegaly with consequential thrombocyt

235 e disorder characterized by myeloid-dominant leukocytosis, splenomegaly, and an increase of hematopoi

236 ythematosus (SLE)-like disease that included leukocytosis, splenomegaly, hypergammaglobulinemia, anti

237 F/P(+) HSC/Ps (IL-5Tg-F/P) developed intense leukocytosis, strikingly high eosinophilia, and eosinoph

238 Leukocytosis strongly correlated with the adrenaline res

239 anifestations included intense inflammation, leukocytosis, synovial hypertrophy and hyperplasia, and

240 PT promoted colonization, leukocytosis, T cell phenotypic changes, systemic pathol

241 e thermal imaging and the ALT-70 (asymmetry, leukocytosis, tachycardia, and age >=70 years) predictio

242 sed reduced emergency hematopoiesis and less leukocytosis than a first MI.

243 SFBL had more severe cecal inflammation and leukocytosis than TG SHAM controls.

244 unknown link between insulin sensitivity and leukocytosis that can affect the predisposition to ather

245 rmosets suggestive of thrombosis, as well as leukocytosis that consisted mostly of granulocytes.

246 epducins selective for CXCR4 cause a massive leukocytosis that does not affect survival.

247 he loss of Ccr7 resulted in an immediate pan-leukocytosis that remained elevated throughout the infec

248 for mortality, like renal insufficiency and leukocytosis, the network identified laboratory markers

249 Among the patients with leukocytosis, there was no observed relation between the

250 stent fever or chills, those with increasing leukocytosis, those older than 80 years, those who are p

251 Animals that died had profound leukocytosis, thrombocytopenia, and elevated serum creat

252 sive reduction in inflammation (reduction in leukocytosis, thrombocytosis, and circulating interleuki

253 resent initially as isolated erythrocytosis, leukocytosis, thrombocytosis, or any combination of thes

254 n and hematocrit, increased red blood cells, leukocytosis, thrombocytosis, splenomegaly, reduced seru

255 pidemiologic and clinical studies have shown leukocytosis to be an independent predictor of future ca

256 tachypnea (OR 1.9, P = 0.001), leukopenia or leukocytosis (total white blood cell count of <4500 or >

257 ate the impact of pretreatment tumor-related leukocytosis (TRL) on the diagnostic performance of 18F-

258 n baseline leukocyte count for patients with leukocytosis was 3,900 cells/microL (range, 1,200 to 72,

259 Peripheral blood leukocytosis was also enhanced in the type-1 but not the

260 Peritoneal leukocytosis was decreased in infected A1-a-deficient mi

261 Leukocytosis was detected with circulating "atypical" ly

262 Leukocytosis was documented in 70% (33/47) of the patien

263 nt mobilization of stem/progenitor cells and leukocytosis was elicited in selectin-deficient mice (L(

264 ore common (P< .01 for both comparisons) and leukocytosis was less common (P< .01) in patients with P

265 Leukocytosis was observed in 15 patients (58%).

266 a trend of shorter survival in patients with leukocytosis was observed.

267 Simultaneously, a prominent leukocytosis was seen; 1 day before being moribund, maca

268 were significantly better protected against leukocytosis, weight loss, and proliferation of B. pertu

269 n of priming with markers of illness such as leukocytosis, weight loss, bacterial proliferation, and

270 Previous TIA and leukocytosis were also independently associated with cli

271 , lower mean arterial pressure, and baseline leukocytosis were associated with higher 90-day mortalit

272 agnosis (specifically immunodeficiency), and leukocytosis were independently associated with reinfarc

273 Patients who developed leukocytosis were significantly more likely to develop t

274 duced O(2) saturation, along with low Hb and leukocytosis, were important predictors for the developm

275 ents with relatively low Alvarado scores and leukocytosis, when physical examination is confusing.

276 evated cytokine expression, and neutrophilic leukocytosis, which augments antilisterial defense.

277 T significantly reduced B. pertussis-induced leukocytosis, which is a hallmark of infant infection an

278 ase for RANKL and TRAP; reduction of OPG and leukocytosis, which were significantly prevented by MRE,

279 Laboratory evaluation revealed leukocytosis (white blood cell count, 15.4 x 10(9)/L; no

280 atients with essential thrombocythaemia with leukocytosis who are intolerant or resistant to hydroxyu

281 ein (16 mg/dL; normal range, 0-5 mg/dL), and leukocytosis with an eosinophilia level of 8710/muL (nor

282 TEL/PDGFbetaR transplanted mice developed leukocytosis with Gr-1(+) granulocytes, splenomegaly, ev

283 There is leukocytosis with lymphocytosis and apneic episodes.

284 Leukocytosis with lymphocytosis and pneumonia are common

285 sed on culture/polymerase chain reaction and leukocytosis with lymphocytosis.

286 features of human chronic myeloid leukemia: leukocytosis with maturing neutrophils, splenomegaly, he

287 All patients showed prompt leukocytosis with maximum blood neutrophils and lymphocy

288 y findings were elevated C-reactive protein, leukocytosis with neutrophilia, and anemia.

289 iferative disorder manifested by significant leukocytosis with neutrophilia, myeloid hyperproliferati

290 myeloproliferative syndrome characterized by leukocytosis with normal maturation of myeloid lineage c

291 in ligand formation, there was no finding of leukocytosis with these single ST3Gal deficiencies.

292 vera-like phenotype (elevated hematocrit and leukocytosis) with a 2.7% average donor cell chimerism i

293 At admission, laboratory studies revealed leukocytosis, with a white blood cell count of 15.1 x 10

294 At admission, laboratory studies revealed leukocytosis, with a white blood cell count of 15.1 x 10

295 athy, graft dysfunction, and either fever or leukocytosis within three weeks after transplantation.

296 Leukocytosis without a recognizable etiology often poses

297 ed for adult patients with isolated fever or leukocytosis without considering the pretest probability

298 le phenotypes, including hair loss, dry eye, leukocytosis, xanthomatosis, and a reduced life span.