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1 ch as multiple sclerosis and periventricular leukomalacia.
2 crotizing enterocolitis, and periventricular leukomalacia.
3  brain injury in the form of periventricular leukomalacia.
4 (P=0.028) than those without periventricular leukomalacia.
5 e intracranial hemorrhage or periventricular leukomalacia.
6 traventricular hemorrhage or periventricular leukomalacia.
7 raventricular hemorrhage and periventricular leukomalacia.
8 de of 3 or greater or cystic periventricular leukomalacia.
9 e period of highest risk for periventricular leukomalacia.
10 lockade in a rodent model of periventricular leukomalacia.
11 e simultaneous occurrence of periventricular leukomalacia.
12 ial hemorrhage and/or cystic periventricular leukomalacia.
13 ntracerebral hemorrhage, and periventricular leukomalacia.
14 raventricular hemorrhage and periventricular leukomalacia (12.4 percent vs. 23.5 percent; relative ri
15 raventricular hemorrhage and periventricular leukomalacia (33.3 percent and 38.2 percent, respectivel
16 er injury, previously called periventricular leukomalacia, a major form of brain injury affecting pre
17 er injury, previously called periventricular leukomalacia, a major form of brain injury affecting pre
18 23.9 percent, P=0.03) and of periventricular leukomalacia alone (5.2 percent vs. 9.0 percent, P=0.048
19 injury is thought to mediate periventricular leukomalacia, an age-dependent white matter lesion seen
20 recovery in insults, such as periventricular leukomalacia and multiple sclerosis.
21 te matter disorders, such as periventricular leukomalacia and multiple sclerosis.
22 te matter disorders, such as periventricular leukomalacia and multiple sclerosis.
23 ytes and myelin as occurs in periventricular leukomalacia and multiple sclerosis.
24 s a therapeutic strategy for periventricular leukomalacia and that the mechanism of protective effica
25 e sclerosis, cerebral palsy (periventricular leukomalacia), and spinal cord injury.
26 ntraventricular haemorrhage, periventricular leukomalacia, and necrotising enterocolitis.
27 e, thin corpus callosum, and periventricular leukomalacia, and quantitative volumetry demonstrated si
28 ere intracranial hemorrhage, periventricular leukomalacia, and ventriculomegaly.
29 racranial hemorrhage, cystic periventricular leukomalacia, and/or severe retinopathy of prematurity.
30 raventricular hemorrhage, or periventricular leukomalacia; and death or necrotizing enterocolitis.
31 (n = 10), DFNB9 (n = 10) and periventricular leukomalacia associated with congenital CMV infection (n
32 n animal models, features of periventricular leukomalacia can be induced by hypoxia and activation of
33 ay, intracranial hemorrhage, periventricular leukomalacia, chronic lung disease, patent ductus arteri
34                       Cystic periventricular leukomalacia (cPVL) is believed to be a precursor of cer
35  stillbirth, neonatal death, periventricular leukomalacia, early respiratory morbidity, intraventricu
36 ural brain abnormalities and periventricular leukomalacia, fetal and postnatal cerebral blood flow is
37 (bronchopulmonary dysplasia, periventricular leukomalacia > grade 1, intraventricular hemorrhage > gr
38 ricular hemorrhage >grade 2, periventricular leukomalacia >grade 1, or necrotizing enterocolitis).
39  BAS; however, neonates with periventricular leukomalacia had lower preoperative oxygenation (P=0.026
40 congenital infection, cystic periventricular leukomalacia, hemorrhagic parenchymal infarction, and po
41 respect to the mechanisms of periventricular leukomalacia in infants and of persistent demyelination
42 traventricular hemorrhage or periventricular leukomalacia in premature infants are associated with ab
43 ial hemorrhage and/or cystic periventricular leukomalacia in the neonatal period.
44 ential for disorders such as periventricular leukomalacia in the preterm and neonatal seizures in the
45                              Periventricular leukomalacia is a form of hypoxic-ischemic cerebral whit
46                              Periventricular leukomalacia is characterized by a reduction in brain ma
47                              Periventricular leukomalacia is the predominant injury in the preterm in
48 on or remyelination, such as periventricular leukomalacia leading to cerebral palsy, stroke, and seco
49 hemorrhage grade 3-4, cystic periventricular leukomalacia, necrotizing enterocolitis, retinopathy of
50 o noted in children with the periventricular leukomalacia of cerebral palsy, another major source of
51 racranial hemorrhage, cystic periventricular leukomalacia, or other complications.
52  of intracranial hemorrhage, periventricular leukomalacia, or ventriculomegaly (17.5 percent vs. 23.9
53 traventricular hemorrhage or periventricular leukomalacia (PIVH/PVL), retinopathy of prematurity (ROP
54 al or physical impairment in periventricular leukomalacia (pre- or perinatal white matter injury lead
55 intraventricular hemorrhage, periventricular leukomalacia, proven sepsis, necrotizing enterocolitis,
56 including the development of periventricular leukomalacia (PVL) and cerebral palsy (CP).
57 atal brain injury leading to periventricular leukomalacia (PVL) and cerebral palsy.
58 tive MRI examinations showed periventricular leukomalacia (PVL) in 4 patients (16%) and infarct in 2
59                              Periventricular leukomalacia (PVL) is a structural loss of white matter
60 ties in preterm infants with periventricular leukomalacia (PVL) is uncertain.
61 ite matter, which results in periventricular leukomalacia (PVL), a condition associated with abnormal
62 alsy in premature infants is periventricular leukomalacia (PVL), a disorder of the immature cerebral
63 ught to consist primarily of periventricular leukomalacia (PVL), a distinctive form of cerebral white
64 alsy in premature infants is periventricular leukomalacia (PVL), a lesion of the immature cerebral wh
65 f Huntington's disease (HD), periventricular leukomalacia (PVL), and kidney dysfunction; Fer-1 inhibi
66 hite matter injury, that is, periventricular leukomalacia (PVL), the dominant form of brain injury in
67 o white matter injury termed periventricular leukomalacia (PVL), the leading cause of subsequent neur
68 ding with the peak period of periventricular leukomalacia (PVL), the major disorder underlying cerebr
69 rtant in the pathogenesis of periventricular leukomalacia (PVL), the major pathological substrate of
70 age of immaturity, including periventricular leukomalacia (PVL), was the most common finding (42.5%),
71 e brain pathologies, notably periventricular leukomalacia (PVL), which is distinguished by bilateral
72 white matter injury, such as periventricular leukomalacia (PVL).
73  and loss, a disorder termed periventricular leukomalacia (PVL).
74 ound in newborn infants with periventricular leukomalacia (PVL).
75 o the cerebral white matter [periventricular leukomalacia (PVL)] is a common and leading cause of bra
76 cular cerebral white matter [periventricular leukomalacia (PVL)] results in cerebral palsy and is the
77 ntricular hemorrhage, cystic periventricular leukomalacia, severe bronchopulmonary dysplasia, retinop
78                              Periventricular leukomalacia should be added to the differential diagnos
79 rade of 3 or greater, cystic periventricular leukomalacia, surgical necrotizing enterocolitis, or sta
80 ., microcephaly, atrophy, or periventricular leukomalacia) that were consistently identified across s
81 In preterm infants (n = 44), periventricular leukomalacia was associated with larger vCD (1084 vs. 82
82                              Periventricular leukomalacia was not associated with BAS; however, neona
83  premature birth and typical periventricular leukomalacia, who had a dramatic improvement in motor fu