ライフサイエンスコーパス: leukoplakia

1 skin pigmentation, nail dystrophy, and oral leukoplakia.

2 helial EBV in the pathogenesis of oral hairy leukoplakia.

3 ufficient for the pathogenesis of oral hairy leukoplakia.

4 e tongue epithelium in lesions of oral hairy leukoplakia.

5 BV-associated diseases other than oral hairy leukoplakia.

6 rting a series of cases of marginal gingival leukoplakia.

7 premalignant lesions, particularly high-risk leukoplakias.

8 biopsies and a BL biopsy but not from hairy leukoplakia, a permissive EBV infection.

9 ositive subjects with and without oral hairy leukoplakia, a replicative EBV-associated epithelial dis

10 atio (OR) = 0.68, p = 0.0035) and oral hairy leukoplakia (adjusted OR = 0.67, p = 0.033) when compare

11 virus (EBV) replication characterizes hairy leukoplakia, an oral epithelial lesion typically occurri

12 bited feeder vessels and tended to have more leukoplakia and a gelatinous appearance.

13 tients with either oral candidiasis or hairy leukoplakia and a low CD4:CD8 cell ratio should be caref

14 ks of age, and when homozygous exhibits oral leukoplakia and blistering, and growth retardation.

15 characterized by abnormal skin pigmentation, leukoplakia and nail dystrophy.

16 EBV replication was detected in both hairy leukoplakia and normal tongue tissues.

17 Oral leukoplakia and oral submucous fibrosis are 2 main oral

18 Some oral fibrotic conditions such as leukoplakia and oral submucous fibrosis, after subsequen

19 and its synthetic derivatives, can eradicate leukoplakia and suppress the formation of squamous cell

20 included cases exhibiting marginal gingival leukoplakia, and with accompanying clinical images.

21 n from Zp, with all Z(+) cells in oral hairy leukoplakia being BLIMP1(+).

22 udy is to raise awareness of this pattern of leukoplakia by reporting a series of cases of marginal g

23 All cases presented as well-demarcated leukoplakias, either on the buccal or lingual gingival m

24 unspecified conditions) and oral precancer (leukoplakia/erythroplakia).

25 Hairy leukoplakia (HL) is a proliferative lesion of the tongue

26 Epstein-Barr virus (EBV) causes hairy leukoplakia (HL), a benign lesion of oral epithelium tha

27 C), pseudomembranous candidiasis (PC), hairy leukoplakia (HL), and warts was computed over follow-up

28 mbrane protein (LMP)-1 is expressed in hairy leukoplakia (HL), but data on LMP-1 sequence variation o

29 deficiency virus (HIV)-associated oral hairy leukoplakia (HLP) and Epstein-Barr virus (EBV) replicati

30 udy, EBV strains were identified in 25 hairy leukoplakia (HLP) biopsies and six matched peripheral bl

31 otein expression in vivo in lesions of hairy leukoplakia (HLP) in which there is abundant EBV replica

32 The immunodeficiency-associated hairy leukoplakia (HLP) lesion is the only pathologic manifest

33 (EBV) replicates productively in oral hairy leukoplakia (HLP).

34 sociated with the pathogenesis of oral hairy leukoplakia (HLP).

35 nails, abnormal skin pigmentation, and oral leukoplakia; Hoyeraal-Hreidarsson syndrome (HH), a clini

36 pithelial cell lesions resembling oral hairy leukoplakia in AIDS patients.

37 a high coprevalence of candidiasis and hairy leukoplakia in these subjects.

38 deficiency virus (HIV)-candidiasis and hairy leukoplakia-in 152 HIV-infected blood transfusion recipi

39 h as nasopharyngeal carcinoma and oral hairy leukoplakia, indicating that the virus can infect epithe

40 f HHV-8 DNA in both the EBV-associated hairy leukoplakia lesions and in the EBV-associated AIDS-relat

41 lytically infected with EBV (from oral hairy leukoplakia lesions) express much more FAS than uninfect

42 EBV was detected by Southern blot in hairy leukoplakia lesions, in a subset of AIDS-related lymphom

43 HIV-positive persons but not in pseudohairy leukoplakia lesions, oral aphthous ulcers, or oral KS le

44 ms to raise awareness that marginal gingival leukoplakia may represent potentially malignant lesions,

45 multisystem disorder, characterized by oral leukoplakia, nail dystrophy, and abnormal skin pigmentat

46 867417 p = 0.02 and rs2240308 p = 0.02), and leukoplakia of oral mucosa is associated with both AXIN2

47 Diagnosis of leukoplakia of oral mucosa, unspecified lesions of oral

48 sopharyngeal carcinoma (NPC), and oral hairy leukoplakia (OHL) lesions that have lytic infection, fre

49 The management of oral leukoplakia (OL) is challenging because of a high risk f

50 gnant disorders (OPMDs), represented by oral leukoplakia (OLK), usually precede head and neck squamou

51 Oral leukoplakia (OPL) is a heterogeneous oral lesion with an

52 eal involvement (OR, 2.7 [95% CI, 1.8-4.0]), leukoplakia (OR, 2.6 [95% CI, 1.7-3.9]), papilliform sur

53 e due to lytic infection (such as oral hairy leukoplakia) or latent infection (such as nasopharyngeal

54 Diagnosis of oral leukoplakia, oral erythroplakia, oral submucous fibrosis

55 Patients with oral leukoplakia, oral erythroplakia, oral submucous fibrosis

56 y malignant disorders (OPMDs)-including oral leukoplakia, oral erythroplakia, oral submucous fibrosis

57 has shown potential in the treatment of oral leukoplakia, oral lichen planus, and head and neck cance

58 Some cases of marginal gingival leukoplakia, over time, progress to extensively involve

59 eukoplakia, recently coined as proliferative leukoplakia (PL), is associated with a strong tendency t

60 head and neck lesions, such as premalignant leukoplakia progressing to established oral squamous cel

61 Proliferative verrucous leukoplakia (PVL) is an aggressive oral precancerous dis

62 ing the criteria for proliferative verrucous leukoplakia (PVL).

63 Proliferative verrucous leukoplakia, recently coined as proliferative leukoplaki

64 al premalignant lesions, we examined 84 oral leukoplakia samples from 37 patients who had been enroll

65 diverse pathologies ranging from oral hairy leukoplakia to nasopharyngeal carcinoma, from infectious

66 Oral hairy leukoplakia tongue tissue, which contains the lytic form

67 Oral leukoplakia was found in 1124 patients, with 94 (8.4%) u

68 A history of hairy leukoplakia was less predictive of persistence of that l

69 Leukoplakia was the most common OPMD found (9.7%), while

70 A total of 30 cases of marginal gingival leukoplakia were included.

71 HIV-positive patients with oral leukoplakia were more likely to develop oral cancer (odd

72 on in vivo, resulting in resolution of hairy leukoplakia when it was present.