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1      We also show that the LTB(4) precursors leukotriene A(4) (LTA(4)) and 5-hydroperoxyeicosatetreno
2 meras generated from 5-lipoxygenase(-/-) and leukotriene A(4) (LTA(4)) hydrolase(-/-) mice, we demons
3 nes is the conversion of arachidonic acid to leukotriene A(4) (LTA(4)) in two successive reactions ca
4 ntitate the major product in the reaction of leukotriene A(4) (LTA(4)) with deoxyguanosine (dGuo).
5 iene biosynthesis, the addition of exogenous leukotriene A(4) (LTA(4); the precursor of LTB(4)) to re
6  such as the prostaglandin endoperoxides and leukotriene A(4) epoxide of mammalian biology and the al
7                            Activation of the leukotriene A(4) hydrolase (LTA(4)H) aminopeptidase (AP)
8                                   We studied leukotriene A(4) hydrolase (LTA(4)H) as a relevant targe
9      The aminopeptidase activity (AP) of the leukotriene A(4) hydrolase (LTA(4)H) enzyme has emerged
10                                              Leukotriene A(4) hydrolase (LTA(4)H) is a proinflammator
11                                          The leukotriene A(4) hydrolase (LTA(4)H) protein is regarded
12  patients with a C/T promoter variant in the leukotriene A(4) hydrolase (LTA4H) gene encoding an enzy
13                  The cytosolic metalloenzyme leukotriene A(4) hydrolase (LTA4H) is the final and rate
14                            Modulation of the leukotriene A(4) hydrolase (LTA4H) locus, which controls
15  peripheral blood neutrophils contained both leukotriene A(4) hydrolase and 5-lipoxygenase exclusivel
16  results demonstrate for the first time that leukotriene A(4) hydrolase can be accumulated in the nuc
17                   In this study, we asked if leukotriene A(4) hydrolase co-localizes with 5-lipoxygen
18                      The finding of abundant leukotriene A(4) hydrolase in the soluble nuclear fracti
19 ipoxygenase, the subcellular distribution of leukotriene A(4) hydrolase is cell-specific and dynamic,
20 uited into inflamed appendix tissue, whereas leukotriene A(4) hydrolase remained cytosolic.
21 s rapidly imported into the nucleus, whereas leukotriene A(4) hydrolase remained cytosolic.
22                                              Leukotriene A(4) hydrolase was also found to accumulate
23 e (5-LO), 5-LO activating protein (FLAP) and leukotriene A(4) hydrolase, are packaged and released in
24 emistry and immunofluorescence revealed that leukotriene A(4) hydrolase, like 5-lipoxygenase, was mos
25 ible mutant maps to the lta4h locus encoding leukotriene A(4) hydrolase, which catalyzes the final st
26  generated by the enzymes 5-lipoxygenase and leukotriene A(4) hydrolase.
27 al action of two enzymes: 5-lipoxygenase and leukotriene A(4) hydrolase.
28          Human M2 macrophages also converted leukotriene A(4) to lipoxins.
29 step transformation of arachidonic acid into leukotriene A(4), leading to the synthesis of various le
30 shows similarity to the N-terminal domain of leukotriene A-4 hydrolase.
31                                              Leukotrienes, a class of inflammatory bioactive lipids,
32 ls and augmented transcellular conversion of leukotrienes, a disturbance in platelet-leukocyte intera
33 ominent among such signals are the cysteinyl leukotrienes, a family of potent proinflammatory lipid m
34 on of an arachidonic acid hydroperoxide to a leukotriene A (LTA) type epoxide by specific lipoxygenas
35 ,13trans,15cis-C18.omega3, an epoxide of the leukotriene A type.