ライフサイエンスコーパス: lifestyle

1 tently decreased with adherence to a healthy lifestyle.

2 s associated with the mite's minute size and lifestyle.

3 aptations are gained in light of a symbiotic lifestyle.

4 12, with a higher score indicating healthier lifestyle.

5 les in these organisms are affected by their lifestyle.

6 orization to accomplish the same two-pathway lifestyle.

7 o more quickly adapt to a surface-associated lifestyle.

8 be further reduced by adherence to a healthy lifestyle.

9 ayers in control and adjustment of microbial lifestyle.

10 vasion machinery critical for the parasites' lifestyle.

11 eating an atlas of changes in metabolism and lifestyle.

12 nd MI is modified by glucose homeostasis and lifestyle.

13 diet, medications, ethnicity, geography, and lifestyle.

14 n associated with adaptation to a pathogenic lifestyle.

15 ts, neuropsychiatric symptoms, and cognitive lifestyle.

16 be attributed to their spectacular migratory lifestyle.

17 n the transitions to an obligately anaerobic lifestyle.

18 e for linking effector targets to pathogenic lifestyles.

19 to best support their unique visuoecological lifestyles.

20 iloring hearing to eusocial and subterranean lifestyles.

21 s, thus providing insights into its range of lifestyles.

22 o increase fitness in planktonic and biofilm lifestyles.

23 eper insights into microbial functioning and lifestyles.

24 nts prompted by their distinct intracellular lifestyles.

25 with a high genetic risk and an unfavourable lifestyle (0.53 [0.29-0.99], p=0.048), with an absolute

26 rable lifestyle than those with a favourable lifestyle (2.03 [1.46-2.83], p<0.0001).

27 from a strictly terrestrial to an amphibious lifestyle across small mammals (Afrosoricida, Eulipotyph

28 a 1:1 ratio to receive active intervention (lifestyle advice delivered by renal dietitians using beh

29 fragmented and is not linked to constructive lifestyle advice.

30 n mHR is independent of gender, fitness, and lifestyle, affecting in equal measure women and men, ath

31 Management includes classical lifestyle alterations with a role for pharmacological th

32 ional diversity contributes to the symbiotic lifestyle and aids in nutrient cycling.

33 s of UI (stress, urge, and mixed), including lifestyle and behavioral therapy, drug therapy, and mini

34 in the human gut microbiome can reflect host lifestyle and behaviors and influence disease biomarker

35 inverse associations between grand-maternal lifestyle and BMI in grandchildren were mainly mediated

36 ther adjustment for potential confounding by lifestyle and cardiovascular disease risk factors; and M

37 Adherence to a healthy lifestyle and colonoscopy in the preceding 10 years were

38 k can be lowered with incremental changes in lifestyle and diet, e.g. fruit-vegetable intake ameliora

39 lids but contrast with Cambrian taxa in both lifestyle and gross morphology(2,6).

40 of T2D for participants with the healthiest lifestyle and high T2D-GR was 2.24 (95% confidence inter

41 and for participants with the least healthy lifestyle and high T2D-GR, it was 8.72 (95% CI: 7.46, 10

42 attitudes, and habits (KAH) toward a healthy lifestyle and ideal cardiovascular health (ICH) scores o

43 nvestigated the associations between healthy lifestyle and life expectancy in people with and without

44 nvironment with profound effects on cellular lifestyle and longevity.

45 86); for participants with the least healthy lifestyle and low T2D-GR, it was 4.05 (95% CI: 3.56, 4.6

46 mpared with participants with the healthiest lifestyle and low T2D-GR, the relative risk of T2D for p

47 m and collected self-reported information on lifestyle and occupational, exposure, and health histori

48 microbiota profiles and also identify human lifestyle and physiological characteristics that, if not

49 with the suite of adaptations for an aquatic lifestyle and piscivorous diet that have previously been

50 interventions aimed at maintaining a healthy lifestyle and prevention of liver-related morbidity.

51 6500 gut metagenomes spanning geography and lifestyle and reconstruct over 1300 E. rectale high-qual

52 e women and explored their associations with lifestyle and reproductive factors.

53 iven multidisciplinary approach to integrate lifestyle and risk factor management as an integral part

54 ciation between the combination of unhealthy lifestyle and risk of AF.

55 In the main models, adjusted for maternal lifestyle and sociodemographic factors, a higher E-DII s

56 ntribute to the unique attributes of biofilm lifestyle and virulence.

57 all ARDs, associated with exposure to urban lifestyles and environment in Uganda.

58 We discuss how different lifestyles and environmental factors might regulate micr

59 istics vary across populations with distinct lifestyles and following specific childhood events, and

60 ng global population combined with sedentary lifestyles and unhealthy diets has contributed to an inc

61 Independent of developmental stage, lifestyle, and adaptations to specific habitats, it seem

62 Dietary habits are important factors in our lifestyle, and confer both susceptibility to and protect

63 Data on genetics, lifestyle, and diet were harmonized.

64 ultivariable model adjusted for updated BMI, lifestyle, and dietary confounders, a 1-egg/d increase w

65 ted models controlling for sociodemographic, lifestyle, and health characteristics, neither WBC marke

66 r adjustment for demographic, socioeconomic, lifestyle, and morbidity factors, hospitalisation rates

67 Poor nutrition, sedentary lifestyle, and obesity are among the primary risk factor

68 0.81, 0.93) after adjusting for demographic, lifestyle, and other dietary factors (model 1); HR 0.90

69 Diet, lifestyle, and psychosocial factors might influence fert

70 le resources and energy, pursue a low-carbon lifestyle, and reduce energy intensity over the next few

71 ociations with an individual's demographics, lifestyle, and/or genetics are robust and generalizable

72 diate chronic disease and/or promote healthy lifestyle are needed to decrease the prevalence of frail

73 daptations in benefiting distinct pathogenic lifestyles are discussed.

74 olutions in art, cosmetics, fashion, and our lifestyle as a whole.

75 potential to model household consumption and lifestyles as mitigation options through their impact on

76 Among these, lifestyle associated with certain sexual and antimicrobi

77 prevention possible through colonoscopy and lifestyle at a predefined genetic risk.

78 ce, who benefit from the currently available lifestyle-based diabetes prevention programmes.

79 test the effectiveness of a high-intensity, lifestyle-based program for obesity treatment delivered

80 A high-intensity, lifestyle-based treatment program for obesity delivered

81 Overall, chalk is useful for a cementing lifestyle because it enables morphological plasticity ne

82 The current trends of unhealthy lifestyle behaviors in underserved communities are distu

83 We also examined whether separate lifestyle behaviors interact with PRS and affect changes

84 In conclusion, combined unhealthy lifestyle behaviors were associated with substantially i

85 ase due to treatment toxicity and changes in lifestyle behaviors(3-5).

86 of incident CKD, adjusting for demographics, lifestyle behaviors, and cardiometabolic conditions.

87 on models adjusted for demographics, height, lifestyle behaviors, prevalent disease, cognitive functi

88 After adjustment for demographics and lifestyle behaviors, severe obstructive sleep apnea asso

89 and supplement data on socioeconomic status, lifestyle, behaviors, and environmental conditions.

90 Disease and mortality burdens of unhealthy lifestyle behaviours are often reported.

91 ility as an advocacy tool to promote healthy lifestyle behaviours.

92 ers of PD as well as factors related to diet/lifestyle, bowel function, and medication were studied i

93 ive measures on GERD-related factors such as lifestyle can be among the health policies of Iran.

94 ident gastric cancer was observed across the lifestyle categories (p(trend)<0.0001), with a higher ri

95 Understanding of the harm to health from lifestyle causes among the general population is low, wi

96 that diabetes prevention programmes based on lifestyle change have not been successful in preventing

97 of certain preconditions makes a significant lifestyle change more likely.

98 Specific recommendations are provided for lifestyle change, despite social distancing restrictions

99 about hypertension, and support for healthy lifestyle change.

100 matically improve NASH outcomes, significant lifestyle changes can be challenging to sustain.

101 bility can only be effective if far-reaching lifestyle changes complement technological advancements.

102 Given the difficulties in implementing major lifestyle changes or widespread primary prevention strat

103 that, in addition to genetic predisposition, lifestyle changes owing to modernization have an importa

104 lts and addresses the policy adjustments and lifestyle changes required to mitigate their deleterious

105 1D) over the past decades has been linked to lifestyle changes, but the underlying mechanisms are lar

106 Lifestyle changes, medication, and surgery are the main

107 of care for secondary prevention, including lifestyle changes, optimal medical therapy, myocardial r

108 NA therapeutics and complementary multimodal lifestyle changes.

109 ildhood, likely because of environmental and lifestyle changes.

110 ures (Fear-Avoidance Beliefs Questionnaire), lifestyle characteristics (BMI, smoking, alcohol abuse,

111 01), after multivariate adjustment including lifestyle characteristics and BMI.

112 Often, relevant mechanisms that begin with lifestyle choices and lead to cardiovascular events span

113 l contributions of environmental factors and lifestyle choices in AD pathogenesis.

114 are influenced by the gut microbiome(4), by lifestyle choices such as smoking(5), or by diet(6).

115 Their occurrence is increasing with lifestyle choices, globalization of food systems, and ec

116 ariates included demographic, socioeconomic, lifestyle, chronic disease, and medication data.

117 tinct patterns of association with genetics, lifestyle, cognition, physical measures and disease.

118 A cluster of three unhealthy lifestyle components was associated with a 22% higher ri

119 is associated with overtreatment and drastic lifestyle consequences.

120 Individual and daily lifestyle considerations and involvement in decision-mak

121 themes were identified: (i) individual/daily lifestyle considerations, (ii) patient involvement in th

122 ress genetic, biological, socioeconomic, and lifestyle contributors to disease.

123 tline the best CHD prevention strategy using lifestyle control only.

124 rchitecture, environment, clinical measures, lifestyle, cost-effectiveness and treatment burden.

125 orts, suggesting that adherence to a healthy lifestyle could lead to greater absolute risk reduction

126 ed with a particle-attached or free-swimming lifestyle could reflect adaptation to various environmen

127 healthy lifestyle, those who adopt a healthy lifestyle could substantially reduce their risk of incid

128 ing congenital heart defects, and health and lifestyle covariates was linked.

129 end toward increasingly active and predatory lifestyles, culminating in jawed vertebrates that domina

130 and height as well as other demographic and lifestyle data were obtained by standardized interviews.

131 beta-cell function, insulin sensitivity, and lifestyle) data comprised the key input variables.

132 indication for use, demographic factors, and lifestyle/dietary factors.

133 ues for novel treatment options in burden of lifestyle diseases, such as CKD.

134 ding to either an endophytic or a pathogenic lifestyle during colonization of maize (Zea mays) and so

135 to explore regional differences in maternal lifestyle during pregnancy related to congenital heart d

136 To examine associations of healthy lifestyle during pregnancy with body mass index (BMI) an

137 als whose grandmothers had the least healthy lifestyle during pregnancy, individuals whose grandmothe

138 The rates of unhealthy maternal lifestyles during pregnancy associated with CHD differed

139 randchildren of women who had the healthiest lifestyles during pregnancy defined by no excess gestati

140 riched in functions related to the symbiotic lifestyle (e.g., CRISPR system, Eukaryotic-like proteins

141 racted for 106 modifiable factors, including lifestyle (e.g., exercise, sleep, media, diet), social (

142 vely, it may reflect residual confounding or lifestyle effects of parenthood that are of special rele

143 h influences of intrinsic aging and those of lifestyle, environment and disease.

144 ive studies indicate that adopting a healthy lifestyle, especially maintaining a healthy weight, can

145 The lifestyle extended model included menopausal status, fam

146 and remained associated after adjustment for lifestyle factors (beta = 0.219, p = 0.001, R(2) = 0.68%

147 ident gastric cancer associated with healthy lifestyle factors (defined as not smoking, never consumi

148 Our findings suggest that lifestyle factors accompanying having offspring, rather

149 In addition, adverse lifestyle factors accumulated in the higher risk groups,

150 a web-based questionnaire on demographic and lifestyle factors and a validated FFQ.

151 Covariates grouped in terms of demographics, lifestyle factors and co-morbidities and cardiovascular

152 We examined interactions between lifestyle factors and genetic risk of type 2 diabetes (T

153 proach to understanding associations between lifestyle factors and infertility treatment outcomes.

154 ntaneous preterm birth after adjustments for lifestyle factors and pregnancy complications, highest c

155 However, lifestyle factors have a strong influence on the outcome

156 Genetic variants and lifestyle factors have been associated with gastric canc

157 lence and sociodemographic, bio-clinical and lifestyle factors in LMICs settings.

158 along with maternal socioeconomic status and lifestyle factors in the second and third generations ac

159 take was not associated with demographic and lifestyle factors including sex, race, income, physical

160 h care by taking genetic, environmental, and lifestyle factors into account is central to modern medi

161 Changing lifestyle factors may alter traditional modes of microbi

162 ing to the robustness of the contribution of lifestyle factors on health expectancy.

163 s such as family history, sex, age and other lifestyle factors or comorbidities as well as on metabol

164 patients to identify treatable or reversible lifestyle factors or medical conditions.

165 Among the lifestyle factors that are associated with the metabolic

166 Lifestyle factors that are known to promote the metaboli

167 and may represent an archive of exposures to lifestyle factors that are relevant to the prediction of

168 prehensive information on dietary intake and lifestyle factors using validated questionnaires prior t

169 After adjustment for socioeconomic and lifestyle factors, a ceramide score (RR Q4 versus Q1 = 2

170 zards regression, adjusted for demographics, lifestyle factors, and body mass index.

171 exposures, sociodemographic characteristics, lifestyle factors, and chronic condition.

172 ck of detailed information on diabetes type, lifestyle factors, and colonoscopy data.

173 CAD risk after adjustment for demographics, lifestyle factors, and other CAD risk factors than was h

174 the interrelationships between host diet and lifestyle factors, clinical blood markers, and the human

175 me composition and host clinical markers and lifestyle factors, including host-microbe associations t

176 ic measurements, cardio-metabolic variables, lifestyle factors, medication, sleep, and nutrition were

177 sease is influenced by numerous clinical and lifestyle factors, motivating the multifaceted approache

178 For individual lifestyle factors, no current smoking was associated wit

179 ne their associations with socioeconomic and lifestyle factors.

180 rences in diet, environmental exposures, and lifestyle factors.

181 horts: the Australian Imaging, Biomarker and Lifestyle Flagship Study of Ageing (AIBL) (n = 382), the

182 ance of the malaria parasite's intravacuolar lifestyle for successful erythrocyte infection and provi

183 ogical strain is associated with modern 24/7 lifestyles (for example, shift work) and several negativ

184 s was significantly greater in the intensive-lifestyle group (change in body weight, -4.99%; 95% conf

185 Patients in the intensive-lifestyle group participated in a high-intensity program

186 enrolled: 452 were assigned to the intensive-lifestyle group, and 351 were assigned to the usual-care

187 and following interviews on oral hygiene and lifestyle habits.

188 data on sociodemographic, health status, and lifestyle habits.

189 uals whose grandmothers had the most healthy lifestyle had 0.17 (95% CI 0.01, 0.33; P for trend = 0.0

190 ts with a high genetic risk and a favourable lifestyle had a lower risk of gastric cancer than those

191 Drastic changes in modern environments and lifestyles have led to an imbalance of this evolutionari

192 ntentious, with both aquatic and terrestrial lifestyles having been proposed [1, 9-12].

193 ging when controlling for chronological age, lifestyle, health-related, and demographic factors in po

194 Whether a healthy lifestyle impacts longevity in the presence of multimorb

195 Studies on the influence of a modern lifestyle in abetting Coronary Heart Diseases (CHD) have

196 assess the benefits of adhering to a healthy lifestyle in individuals with a high genetic risk.

197 largely disregarding the impact of a healthy lifestyle in mitigating CHD risk.

198 nto 8 groups by the combination of unhealthy lifestyle including current smoking, heavy drinking (> 3

199 the observational design with self-reported lifestyle information with risk of residual or unmeasure

200 nsive data collection based on self-reported lifestyle information, medical documentation and health

201 ), a randomised controlled trial (RCT), of a lifestyle intervention (low glycaemic index (GI) diet pl

202 The impact of a lifestyle intervention and changes in cardiorespiratory

203 of incident HF (n=257) between the intensive lifestyle intervention and the diabetes support and educ

204 allocated either to an intensive weight-loss lifestyle intervention based on er-MedDiet, PA promotion

205 litus in the Look AHEAD trial, the intensive lifestyle intervention did not appear to modify the risk

206 is study is to evaluate BC changes made by a lifestyle intervention in a randomized homogeneous sampl

207 nal environment appeared to be modified by a lifestyle intervention in pregnancy.

208 In adults with type 2 diabetes, a lifestyle intervention is associated with significant lo

209 Active lifestyle intervention led by renal dietitians did not i

210 "individual-focused" or "peer-to-peer-based" lifestyle intervention program for 12 months or control.

211 IMED-Plus) based on an intensive weight-loss lifestyle intervention program.

212 Clinically, active versus passive lifestyle intervention resulted in reduced incidence of

213 In addition, diet and lifestyle intervention studies are needed to confirm our

214 r kidney transplantation, but the benefit of lifestyle intervention to improve glucose metabolism pos

215 to provide patients with either an intensive lifestyle intervention, which focused on reduced caloric

216 entive and antihypertensive effects of major lifestyle interventions (regular physical exercise, body

217 rs, if present, would favor more intensified lifestyle interventions and consideration of initiation

218 rved among the participants in the different lifestyle interventions during months 6-14 (aFMT, 30.4%

219 Growing evidence supports the use of lifestyle interventions for the prevention and adjuvant

220 viding helpful information to improve future lifestyle interventions in adults.

221 rial highlights that high adherence rates to lifestyle interventions may improve health outcomes.

222 s underlying the beneficial effects of these lifestyle interventions on hypertension, which include n

223 tudy was to assess the impact of 2 different lifestyle interventions on parents/caregivers of childre

224 When diagnosed at early stages, lifestyle interventions such as exercise and weight loss

225 otransporter type 2 inhibitors as add-ons to lifestyle interventions with or without metformin in tho

226 disease (AD) are now focusing on multidomain lifestyle interventions.

227 diuretics, mineralocorticoid antagonists and lifestyle interventions.

228 newly diagnosed dyslipidemia who were under lifestyle interventions.

229 ust conserves sufficient energy for a modest lifestyle involves chemically challenging reactions cata

230 A sedentary lifestyle is associated with increased risk for cardiova

231 public health implication is that a healthy lifestyle is important for diabetes prevention, especial

232 ng is degenerate or adaptive to their unique lifestyles is controversial.

233 he transition between planktonic and biofilm lifestyles is modulated by the intracellular secondary m

234 cellulolytic fungi cultivated for food, in a lifestyle known as fungiculture.

235 factors such as unhealthy diet and sedentary lifestyle; limited access to risk-reducing behaviors suc

236 apical complex is essential to the parasitic lifestyle, little is known about the regulation of apica

237 k factors (sociodemographic, anthropometric, lifestyle, medical history, and nutritional factors).

238 nnaires on sociodemographic characteristics, lifestyle, medication use, and medical history; females

239 imary prevention of ischemic stroke includes lifestyle modification and diet, treatment of risk facto

240 LD identification, staging, and promotion of lifestyle modification are imperative.

241 Lifestyle modification is the mainstay of treatment, inc

242 after a weight loss intervention (intensive lifestyle modification or bariatric surgery).

243 ncluding directing patients toward intensive lifestyle modification to promote weight loss and referr

244 ls at high risk of developing T2DM undertake lifestyle modification, irrespective of whether the pred

245 Treatments include lifestyle modification, PPI medication, and laparoscopic

246 ctor and surgical intervention together with lifestyle modification.

247 Lifestyle modifications focused on healthy eating and re

248 y screening, risk stratification approaches, lifestyle modifications, surgical and catheter intervent

249 and implement preventive strategies (healthy lifestyles, modifications to cardiovascular risk factors

250 Lifestyle, obesity, and the gut microbiome are important

251 The sessile lifestyle of plants requires accurate physiology adjustm

252 e the type of coselected traits reflects the lifestyle of the specific microbe.

253 features that may support the extremophilic lifestyle of this sea ice alga include massively expande

254 mes are uniquely fitted to the intracellular lifestyle of this significant human pathogen.

255 and hydrazine (N(2)H(4)) is essential to the lifestyles of diverse organisms.

256 The military lifestyle often includes continuous operations whether i

257 analysis establishes gender independence of lifestyle on CHD, refuting long held assumptions and unq

258 genetic risks, and assessed the influence of lifestyle on genetic outcomes.

259 Data on participant demographics, diet, lifestyle, opium use, and different exposures were colle

260 ering blood pressure, smoking cessation, and lifestyle optimisation are common to all stroke subtypes

261 e of simple or more-intensive evidence-based lifestyle options in an empathetic manner, with encourag

262 ic score individuals, adherence to a healthy lifestyle or use of statins may offset increased inherit

263 iation between certain AGF genera and animal lifestyles, or animal host family was observed.

264 a predominantly aerobic organoheterotrophic lifestyle, perhaps based on scavenging amino acids, nucl

265 Demographic, work-related information, lifestyle questionnaire and the Professional Quality of

266 ing the Victoria Longitudinal Study Activity Lifestyle Questionnaire.

267 Epidemiology (BAMSE) (Sweden), Influences of Lifestyle-Related Factors on the Human Immune System and

268 t cancer cases, exceeding the impact of many lifestyle-related factors.

269 Other rising lifestyle-related risk factors such as use of tobacco pr

270 lood pressure, lipids, diabetes mellitus and lifestyle-related risk factors, as well as the effects o

271 also strongly associated with many of these lifestyle-related traits after correction for population

272 orrelation of birth location with a range of lifestyle-related traits, including body-mass index and

273 ased genetic risk can be offset by a healthy lifestyle remains unknown.

274 he past month were 23%-81% for self-care and lifestyle requirements (diet, exercise, blood pressure m

275 H group showed lower risk of nonadherence to lifestyle requirements (diet/exercise) than the usual ca

276 en and 7.6 years for women; however, not all lifestyle risk factors equally correlated with life expe

277 Individual lifestyle risk factors have been associated with an incr

278 a indicates that unfavorable combinations of lifestyle risk factors lead to a high likelihood of hepa

279 We examined independent and joint effects of lifestyle risk factors on fatty liver index (FLI), a bio

280 bed under the influence of environmental and lifestyle risk factors.

281 effect on CAD through central nervous system-lifestyle risk factors.

282 However, higher lifestyle risk score was not significantly associated wi

283 to the polygenic risk score and the healthy lifestyle score (men, 3.5%-13.4%; women, 2.5%-10.6%).

284 ty, and smoking during pregnancy to create a lifestyle score ranged from 0 to 12, with a higher score

285 luenced by many factors, including genetics, lifestyle, sex and socio-economic status.

286 met this need effectively, as its dimorphic lifestyle showcases distinctive features, including cell

287 In conclusion, maintaining a healthy lifestyle since midlife may help reduce cognitive declin

288 These lifestyle-specific traits require integration into the r

289 gastric cancer in those with an unfavourable lifestyle than those with a favourable lifestyle (2.03 [

290 of age) with obesity and a poor response to lifestyle therapy alone.

291 bo subcutaneously once daily, in addition to lifestyle therapy.

292 t a high genetic risk who adopt an unhealthy lifestyle, those who adopt a healthy lifestyle could sub

293 findings support the promotion of a healthy lifestyle to lower the risk of new-onset AF.

294 nctions and in some cases, underpinned major lifestyle transitions.

295 azards regression adjusting for clinical and lifestyle variables was used.

296 factors, including metabolic biomarkers and lifestyle variables, were assessed in clinical examinati

297 e SNP data, and 27 clinical, demographic and lifestyle variables.

298 e of multimorbidity, engaging in a healthier lifestyle was associated with up to 6.3 years longer lif

299 d have morphologies that indicate epibenthic lifestyles, whereas phylogenomics recovers sessile, infa

300 hunter-gatherer to a sedentary agricultural lifestyle, which ultimately resulted in the development