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1 romyotonia (NMT), Morvan syndrome (MoS), and limbic encephalitis.
2  with GABAB encephalitis have presented with limbic encephalitis.
3 s, and a mixed movement disorder rather than limbic encephalitis.
4 he cognitive impairment associated with this limbic encephalitis.
5 age-gated potassium channel complex antibody limbic encephalitis.
6 dentify the real autoantigen associated with limbic encephalitis.
7  be the target of antibodies associated with limbic encephalitis.
8 patients (P < 0.0001), who predominantly had limbic encephalitis.
9                       Only 26% had classical limbic encephalitis.
10 y been reported in three cases of reversible limbic encephalitis.
11               MRI showed signs indicative of limbic encephalitis.
12 ented as classic paraneoplastic syndromes (5 limbic encephalitis, 1 paraneoplastic encephalomyelitis,
13 e neurological presentations (5 brainstem or limbic encephalitis, 3 cerebellar ataxia, 2 Lambert-Eato
14 ts with neuronal surface antibodies (49%) or limbic encephalitis (46%) as opposed to non-limbic seron
15 cohort of patients (n = 38) after autoimmune limbic encephalitis, a syndrome associated with focal st
16       A group of individuals with autoimmune limbic encephalitis-a condition known to focally affect
17  reactivation is well established as causing limbic encephalitis after haematopoietic stem cell trans
18                                   Autoimmune limbic encephalitis (ALE) presents with new-onset mesial
19                                   Autoimmune limbic encephalitis (ALE) represents a heterogeneous dis
20  seen with symptoms suggestive of autoimmune limbic encephalitis, although they can be paucisymptomat
21  whereas LGI1 autoantibodies are involved in limbic encephalitis, an acquired epileptic disorder asso
22 ients with neuromyotonia, Morvan's syndrome, limbic encephalitis and a few cases of adult-onset epile
23 GI1 autoantibodies are known to present with limbic encephalitis and additionally faciobrachial dysto
24                             One patient with limbic encephalitis and antibodies against LGI1 also had
25 We analysed sera and CSF of 57 patients with limbic encephalitis and antibodies attributed to voltage
26 cerebrospinal fluid of patients with ataxia, limbic encephalitis and certain forms of epilepsy.
27 with various clinical presentations, such as limbic encephalitis and complex and diffuse encephalopat
28 od B-cell populations from two patients with limbic encephalitis and faciobrachial dystonic seizures
29 ctivated 1 (LGI1) are found in patients with limbic encephalitis and focal seizures.
30 ies are detected in subjects with autoimmune limbic encephalitis and peripheral nerve hyperexcitabili
31 vant, but not all patients had a 'classical' limbic encephalitis and some did not receive immunothera
32 re found in patients with different forms of limbic encephalitis, and in a few patients with epilepsy
33 h VGKC antibodies described in patients with limbic encephalitis, and the subsequent seminal paper de
34 re considered definitely autoimmune, 10 with limbic encephalitis (antibody specificity: 5 LGI1, 1 con
35                                     The term limbic encephalitis associated with antibodies against v
36                                              Limbic encephalitis associated with antibodies to compon
37 ated potassium channels should be changed to limbic encephalitis associated with LGI1 antibodies, and
38 ibodies, 79 had the presumptive diagnosis of limbic encephalitis, dementia, cognitive dysfunction, or
39  uncertainty and effort, patients with acute limbic encephalitis demonstrated blunted sensitivity to
40 otein-2 (CASPR2), are found in patients with limbic encephalitis, faciobrachial dystonic seizures, Mo
41 es with other more common non-paraneoplastic limbic encephalitis (five with antibodies against leucin
42 otassium channel complex antibody-associated limbic encephalitis has recently been recognized as a fo
43 ness in 13 patients with VGKC antibodies and limbic encephalitis in 2004.
44 a single institution, who developed subacute limbic encephalitis initially considered of uncertain ae
45                                              Limbic encephalitis is typically a paraneoplastic syndro
46                                              Limbic encephalitis (LE) associated with antibodies to t
47                                              Limbic encephalitis (LE) is often associated with lung,
48 actor-related receptor) in PCD and mGluR5 in limbic encephalitis (LE).
49 ons without peripheral involvement is called limbic encephalitis (LE).
50 -responsive clinical presentations including limbic encephalitis, Morvan's syndrome and acquired neur
51 sociated disorders is more diverse including limbic encephalitis, Morvan's syndrome, peripheral nerve
52 onopathy (n = 7), K(+)-channel antibody with limbic encephalitis (n = 1) or neuromyotonia (n = 1), an
53 erebellar ataxia (N=55), epilepsy (N=35) and limbic encephalitis (N=7) could occur either in isolatio
54                                              Limbic encephalitis, one of the most common manifestatio
55 ge: 64.0 +/- 2.55 years, median 4 years post-limbic encephalitis onset; n = 18 controls].
56 and two as stiff person syndrome; five had a limbic encephalitis or epileptic encephalopathy, two had
57                            Syndromes such as limbic encephalitis or opsoclonus-myoclonus should alway
58                               Paraneoplastic limbic encephalitis (PLE) is a rare disorder characteriz
59      LGI1 is the autoantigen associated with limbic encephalitis previously attributed to voltage-gat
60 es showed that antibodies from patients with limbic encephalitis previously attributed to voltage-gat
61 gnificantly upregulated proteins in anti-AK5 limbic encephalitis, respectively mapping to apoptosis p
62 that the clinical manifestations of anti-AK5 limbic encephalitis result from a distinct T cell-mediat
63 mplex encephalitis, Korsakoff's syndrome and limbic encephalitis should be considered.
64                                              Limbic encephalitis that is associated with anti-voltage
65 ated 1, are associated with a common form of limbic encephalitis that presents with cognitive impairm
66  description of focal cortical dysplasia and limbic encephalitis, the pathology of status epilepticus
67                    Other phenotypes included limbic encephalitis (three), encephalopathy with/without
68 same period, only one paraneoplastic case of limbic encephalitis was identified between the two main
69  in the BRAF(V600) mutation-positive cohort (limbic encephalitis) was considered to be related to ate
70 ed to have paraneoplastic or immune-mediated limbic encephalitis were clinically assessed.
71 -treated LGI1 VGKC-complex antibody-mediated limbic encephalitis were investigated using in vivo ultr
72                       Patients with anti-AK5 limbic encephalitis were mostly male (20/26, 76.9%) with
73                                  Symptoms of limbic encephalitis were observed in all patients, inclu
74                                              Limbic encephalitis with antibodies against adenylate ki
75 ium channel (VGKC) complex antibody-mediated limbic encephalitis with generalized hippocampal atrophy
76                                              Limbic encephalitis with leucine-rich glioma inactivated
77                                              Limbic encephalitis with leucine-rich, glioma-inactivate
78  with high potassium channel antibodies have limbic encephalitis without tumours.