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1 hat measured the skin (r = -0.48, P < .001), lips (r = -0.21, P = .001), and lip rhytids (r = -0.32,
2 in's lymphoma (HR, 3.0; 95% CI, 1.7 to 5.3), lip cancer (HR, 3.4; 95% CI, 2.0 to 6.0), and nonepithel
3 g and modification of some phonemes although lip closure was still possible allowing food intake.
4                                  We analyzed lip products by inductively coupled plasma optical emiss
5 , P < .001), lips (r = -0.21, P = .001), and lip rhytids (r = -0.32, P < .001) correlated with the re
6 recorded M1 neurons in the anterior bank and lip of the central sulcus as monkeys performed more natu
7 icant positive correlation between blood and lip tissue DNA methylation, indicating blood as a suitab
8   Cancers of the breast, uterine cervix, and lip or oral cavity are three of the most common malignan
9 tial coherence between left motor cortex and lip movements and this partial coherence directly predic
10 ntrained to the auditory speech envelope and lip movements (mouth opening) when listening to a spoken
11 g pain in the lower limb to include face and lip regions.
12 zed patients with dry eyes and dry mouth and lip biopsies from the Sjogren's International Collaborat
13 regulated by PeMYBs in the sepals/petals and lip.
14 stive mouth movements (tongue protrusion and lip smacking) for the purposes of social communication:
15 fects checklists for problems after skin and lip treatment.
16 related with the reporting of more skin- and lip-related adverse effects.
17                       Performance levels and lipping intensity (an appetitive behavior) both showed t
18 ion that was most pronounced on the face and lips and was associated with nail pigmentation, blue pig
19 his, we acquired sound, eye trajectories and lips' dynamics during the reading of consonant-consonant
20 orehead, glabella, lateral periorbital area, lips, and marionette lines), with scores ranging from 0
21  involved multiple body parts (residual arm, lips, and feet).
22 tices at frequencies that match articulatory lip movements; the right angular gyrus then extracts slo
23 sociated increases in affiliative behaviors--lip smacking, visual attention to a caregiver, and time
24  consistently oriented toward the blastopore lip in dorsal and lateral cells, but oriented parallel t
25 e apical surfaces of cells at the blastopore lip, and it functions during blastopore formation and cl
26                            At the blastopore lip, Vangl2 is required for the apical accumulation of t
27                     Visual speech carried by lip movements is an integral part of communication.
28 ounty-level mortality rates from 29 cancers: lip and oral cavity; nasopharynx; other pharynx; esophag
29 acial allograft, including mandible, cheeks, lips, and chin, in November 2009.
30 nsistent with the hypothesis that chimpanzee lip-smacks function to coordinate and prolong social gro
31                                        Cleft lip and/or palate (CL/P) are common structural birth def
32                                        Cleft lip is one of the most common human birth defects, yet o
33                                        Cleft lip is one of the most common human birth defects.
34                                        Cleft lip with or without cleft palate (CL/P) is one of the mo
35                                        Cleft lip with or without cleft palate is the most common cong
36                                        Cleft lip with or without palate (CLP) and isolated cleft pala
37                                        Cleft lip, which results from impaired facial process growth a
38                                     15 cleft lip and palate organisations totalled 26% of both revenu
39 ospadias (0.6%), hydrocephalus (0.6%), cleft lip or palate (0%), and obstetric fistula (0%).
40 tion, Msx1-deficient embryos develop a cleft lip when the mother is transiently exposed to reduced ox
41 observed wider de novo deletions among cleft lip and palate (CLP) cases than seen among cleft palate
42  than seen among cleft palate (CP) and cleft lip (CL) cases.
43  and isolated cleft lip only (CLO) and cleft lip and palate (CLP).
44 ry such as ophthalmology (88, 28%) and cleft lip and palate surgery (70, 22%) were also frequently pe
45  of limb deficiency, cleft palate, and cleft lip cases were, respectively, 1.8 (95% CI: 1.1, 3.1), 1.
46 ase DOT by analyzing breast cancer and cleft lip data, in which our method strengthened levels of pre
47 alyses for cleft palate only (CPO) and cleft lip with or without palate (CL/P) with ~17 million marke
48 persistence of the epithelial seam and cleft lip.
49 owth, epithelial seam persistence, and cleft lip.
50 ctrodactyly, ectodermal dysplasia, and cleft lip/palate (EEC) syndrome is caused by single point muta
51  associated with cognitive defects and cleft lip/palate, its role in mammalian development and physio
52 longed treatment trajectories, such as cleft lip and palate.
53 sed risk of anencephaly, spina bifida, cleft lip with or without cleft palate (CLP), or cleft palate
54 -agonists were found for spina bifida, cleft lip, anal atresia, severe congenital heart defects in ge
55 aniofacial defects including bilateral cleft lip and palate and tongue agenesis, following the loss o
56   The proband presented with bilateral cleft lip and palate, malformed auricles, and bilateral ectrod
57 r Esrp1 have fully penetrant bilateral cleft lip and palate.
58 directed at ophthalmology, followed by cleft lip and palate surgery.
59 ed this new Mendelian syndrome CATIFA (cleft lip, cataract, tooth abnormality, intellectual disabilit
60 n saliva, and patients with the common cleft lip and palate have a higher prevalence of dental caries
61 ing regulatory protein (Esrp1) develop cleft lip and palate.
62  the ectodermal dysplasia-ectrodactyly-cleft lip/palate (EEC) syndrome, comprising SHFM.
63  mutations could increase the risk for cleft lip formation by interacting with an impaired morphogene
64 he cellular mechanism demonstrated for cleft lip pathogenesis, we found that either SHH ligand additi
65  cilium, and these patients often have cleft lip and palate.
66 ed testes, hypospadias, hydrocephalus, cleft lip or palate, and club foot.
67 ed testes, hypospadias, hydrocephalus, cleft lip/palate, and clubfoot) was determined by physical exa
68 sion of genes previously implicated in cleft lip and/or palate, including components of multiple sign
69 ental anomalies were more prevalent in cleft lip with cleft palate than other cleft types.
70 disruption of this sequence results in cleft lip.
71 esult is orofacial clefting, including cleft lip and cleft palate (CL/P).
72 use of abnormal development (including cleft lip and/or palate, craniosynostosis and facial dysostose
73        Three common diseases, isolated cleft lip and cleft palate (CLP), hypothyroidism and thyroid c
74        Among individuals with isolated cleft lip and cleft palate, increased risks of intellectual di
75  three affected siblings with isolated cleft lip and palate, we discovered that they share a novel mi
76 , KIAA1598-VAX1, and MAFB and isolated cleft lip only (CLO) and cleft lip and palate (CLP).
77 ung adults who were born with isolated cleft lip only did not differ significantly from unaffected in
78  mortality among persons with isolated cleft lip only.
79 lateral choanal atresia, hearing loss, cleft lip and/or palate, and other craniofacial dysmorphisms.
80 lly treatable conditions (breast mass, cleft lip/palate, club foot, hernia or hydrocele [adult and pa
81         These mutants developed median cleft lip and palate demonstrating a previously undocumented c
82 nout, expansion of the facial midline, cleft lip, extensive exencephaly, and microphthalmia or anopht
83    We identified a novel mouse mutant (cleft lip/palate, edema and exencephaly; Clpex) with a hypo-mo
84                           Nonsyndromic cleft lip and/or palate (NSCL/P) is a prevalent birth defect o
85 known candidate genes for nonsyndromic cleft lip and/or palate through genome-wide linkage analysis.
86 e genetic contribution to nonsyndromic cleft lip and/or palate through the analysis of family pedigre
87 acial clefts (NSOFCs) are nonsyndromic cleft lip only (NSCLO), nonsyndromic cleft lip with palate (NS
88 d GREM1) specifically for nonsyndromic cleft lip with cleft palate.
89 icated in the etiology of nonsyndromic cleft lip with or without cleft palate (NSCL/P) in populations
90                           Nonsyndromic cleft lip with or without cleft palate (NSCL/P) is a common bi
91                           Nonsyndromic cleft lip with or without cleft palate (nsCL/P) is among the m
92 genetic risk variants for nonsyndromic cleft lip with or without cleft palate (NSCL/P).
93 c cleft lip only (NSCLO), nonsyndromic cleft lip with palate (NSCLP), and nonsyndromic cleft palate o
94                           Nonsyndromic cleft lip with/without cleft palate (nsCL/P) and nonsyndromic
95                     Non-syndromic (NS) cleft lip with or without cleft palate (CL/P) is a common diso
96 s focused on the molecular etiology of cleft lip and clefts of the hard palate, but study of the unde
97 erygium syndrome, 2 syndromic forms of cleft lip and palate.
98 WS), the most common syndromic form of cleft lip and palate.
99                         Median CERs of cleft lip or palate repair ($47.74 per DALY), general surgery
100 isorder; however, our understanding of cleft lip pathogenesis remains incomplete.
101 in a limited number of mouse models of cleft lip that can be leveraged to characterize the genes and
102 es to the etiology and pathogenesis of cleft lip through antagonistic interactions with other gene re
103  (TGFA) polymorphisms with the risk of cleft lip with or without cleft palate (CL/P) or cleft palate
104  understanding the genetic etiology of cleft lip with or without cleft palate, relatively little is k
105 in the upper beak or the equivalent of cleft lip.
106 th syndromic and nonsyndromic forms of cleft lip/palate (CLP).
107 senting 14 specialties (ophthalmology, cleft lip/palate, multidisciplinary teams, orthopaedics, cardi
108 t surgical specialties (ophthalmology, cleft lip/palate, multidisciplinary teams, orthopaedics, cardi
109 steroids were confirmed (cleft palate, cleft lip, anal atresia, and hypospadias).
110 al orofacial clefts and with syndromic cleft lip and/or palate (CL/P).
111                          Non-syndromic cleft lip and/or palate (NSCLP) is a common congenital malform
112 ts (OFCs), which include non-syndromic cleft lip with or without cleft palate (CL/P), are among the m
113 fied in several GWAS for non-syndromic cleft lip with or without cleft palate (NS CL/P).
114 s as candidate genes for non-syndromic cleft lip with or without cleft palate (NSCL/P).
115                          Non-syndromic cleft lip with palate (NSCLP) is the most serious sub-phenotyp
116                                    The cleft lip area, bilateral nostril areas, and the nostril heigh
117 tion in utero can partially rescue the cleft lip phenotype.
118  showed that mutations in IRF6 lead to cleft lip and palate and mandibular abnormalities.
119 investigated the mechanisms leading to cleft lip as well as cleft palate in both existing and new Esr
120   20 patients with complete unilateral cleft lip and palate were prospectively recruited.
121 al outcome assessment in patients with cleft lip and for the rehabilitation of patients with facial n
122 ling pathway have been associated with cleft lip and palate in humans and mice, the mechanisms involv
123 mprove nasal symmetry in patients with cleft lip and palate.
124                          Patients with cleft lip and/or palate (CLP), who undergo numerous medical in
125 rge multi-ethnic human population with cleft lip identified clusters of single-nucleotide polymorphis
126 dentified in both mice and humans with cleft lip.
127                          Patients with cleft lip/palate (CLP) have been reported, in some studies, to
128 ith isolated cleft palate (ie, without cleft lip) had increased mortality (hazard ratio, 3.4; 95% CI,
129      The cleft is defined as type I ("closed lips") if there are fused clefts in cerebral mantle.
130 , seen in a mirror, spoke with corresponding lip movements, and for half of the participants this was
131 irtual mirror, that spoke with corresponding lip movements.
132 )) and liposome-water distribution ratios (D(lip/w)) of the chemicals was used to calculate D(FBS/w)
133  Although this defect alone does not disrupt lip formation, Msx1-deficient embryos develop a cleft li
134 hus the dynamic behavior of epithelia during lip/palate development.
135 le for sonic hedgehog (SHH) signaling during lip fusion.
136 s that are regulated by Shh signaling during lip morphogenesis.
137 and P > .16 for individual symptoms of face, lip, tongue, or eyelid swelling).
138           Most patients had swelling of face/lips (78.3%) and tongue (78.3%).
139 ntaining transmembrane protein known as Fish-lips (Fili) as a novel wiring molecule in the assembly o
140  more chemosensory behaviour (tongue flicks, lip smacks and substrate lickings) when presented with t
141 hic frequencies of lesions were as followed: lips and oral cavity (n = 46 [93%]) and pharynx and vest
142 both kiss initiators and kiss recipients for lip kissing, and took into consideration differences due
143 AC and MEL; RCM and dermoscopy were used for lips analysis.
144 nd sometimes difficult to differentiate from lip melanoma (MEL).
145              Liposomes with Gd-complexes (Gd-lip) co-encapsulated with thrombolytic agents can serve
146                                 Moreover, Gd-lip did not show pro-inflammatory effects, as assessed i
147 In this study, we evaluated nanosafety of Gd-lip containing PE-DTPA chelating Gd(+3) prepared by lipi
148            We detected no cytotoxicity of Gd-lip in human liver cells including cancer HepG2, progeni
149 Furthermore, no potential side effects of Gd-lip were found using a complex system including general
150 udy indicates potential in vivo safety of Gd-lip with respect to hepatotoxicity and immunopathology c
151 sks that depend implicitly on achieving good lip-synch.
152 was seen with vesicles and bullae around her lips, trunk, axillae, arms, and thighs.
153                      Conjunctival hyperemia, lip swelling, cold sweats, and nausea presented later.
154                             The elevation in lip cancer incidence is consistent with an effect of imm
155 ripheral nervous system (PNS), especially in lip and foot regions of the anterior integument.
156 Irf6 cKO), bypassing the role of the gene in lip and palate morphogenesis and thus ensuring survival
157 We identified small yet consistent shifts in lip representation contralateral to the missing hand tow
158 ured the McGurk effect (in which incongruent lip-voice pairs evoke illusory phonemes), and also ident
159 ided elongation of the oligodendrocyte inner lip membrane.
160                               Interestingly, lip-reading in silence still activates the auditory cort
161 e interactions (mutual gaze and intermittent lip-smacking) with human caregivers display increased so
162                                     Invasive lip cancer incidence was associated with male sex (adjus
163 and a clinical trial of a minimally invasive lip treatment in the United Kingdom and France (clinical
164 uitment in vitro and potently elicited lower lip retraction in vivo, a component of "serotonergic syn
165    Stimulation of the nose, upper lip, lower lip, and chin caused a somatotopic lateral-to-medial, ip
166 a (BM), HP, and transition zone of the lower lip (TZ) after six-day culture.
167 with Van der Woude express pits on the lower lip that continuously or intermittently drain saliva, an
168 topographically ordered input from the lower lip.
169 phyrin, in the microcirculation of the lower lip.
170 , often but not always associated with lower lip pits.
171 ween the nipple-areola complex and the lower lips, moves as a rigid body with the cycling motion of t
172                                The mammalian lip and primary palate form when coordinated growth and
173        For example, stimulation of the motor lip representation influences specifically discriminatio
174 und that TMS-induced disruption of the motor lip representation modulated specifically the early audi
175                                     Multiple lip revision operations are often required throughout ch
176 reductase (NarGHI) was examined using a narG-lip (lipase) reporter gene assay in vivo.
177 rded from 10 patients with clinically normal lips (mean [SD] age, 34.5 [6.1] years), 16 patients with
178                                    In normal lips, the draped pattern-a previously described MAC RCM
179 confocal microscopy (RCM) features of normal lips of different phototypes and to identify features th
180        Group 1 included patients with normal lips and different phototypes, and Group 2 consisted of
181  were concentrated around the midface, nose, lips, and eyes.
182                     Objective assessments of lip movement can be beneficial in many disciplines inclu
183 macules (MAC) are the most frequent cause of lip pigmentation and sometimes difficult to differentiat
184  can assist in the differential diagnosis of lip MEL and MAC.
185 on influences specifically discrimination of lip-articulated speech sounds.
186 Match Study, we describe the epidemiology of lip cancer among 261 500 transplant recipients in the Un
187  spectrometry and used previous estimates of lip product usage rates to determine daily oral intakes.
188 gular gyrus then extracts slower features of lip movements, mapping them onto the corresponding speec
189 nsplant recipients have an increased risk of lip cancer, but the reasons are uncertain.
190 reaching the TG contralateral to the side of lip infection.
191 only from the eye ipsilateral to the site of lip infection.
192 ls of taste buds including the epithelium of lips and olfactory epithelium, and ii) mechanosensory ce
193 ssociated angioedema (defined as swelling of lips, tongue, pharynx, or face during ACE inhibitor use
194                       Two hundred thirty-one lip cancers were identified, corresponding to elevated r
195                            In type II ("open lips") the clefts are separated and filled with cerebros
196 on the ear (RR, 4.67; 95% CI, 1.28-17.12) or lip (RR, 4.55; 95% CI, 1.41-14.69), invasion beyond subc
197 4.63), ear (RR, 2.33; 95% CI, 1.67-3.23), or lip (RR, 2.28; 95% CI, 1.54-3.37).
198                  Clefts of the palate and/or lip are among the most common human craniofacial malform
199 than repression and formation of the palate, lip, and characteristic folds of the closed Antirrhinum
200 ecurrent herpes labialis receiving permanent lip makeup.
201 g high-frequency vocalizations at the phonic lips, a constriction in the nasal passages just beneath
202    Groomers were also more likely to produce lip-smacks during face-to-face grooming where the visual
203 traight-sided glasses elicited more 'pursed' lip embouchures, which may partly explain reduced consum
204                                       An RCM lip algorithm was developed that provided 100% sensitivi
205 s, erythema of the palms, and strikingly red lips and conjunctiva.
206 nderstanding of the mechanisms that regulate lip morphogenesis is limited.
207 ulation', which modulates Mnp shape, rescues lip formation and involves a localized abrogation of Bmp
208 nt of Pax6 in the development of all rhombic lip (RL) lineages.
209 al expansion of both ventricular and rhombic lip primary progenitor zones to include subventricular z
210 o the medial ganglionic eminence and rhombic lip, resembling the gnathostome brain.
211  defining molecule of the cerebellar rhombic lip and Pax6 is downstream in the Math1 pathway.
212 reduces the length of the cerebellar rhombic lip.
213                 We also examine five rhombic lip cell markers (Wls, Math1, Pax6, Lmx1a, and Tbr2) to
214                 Disruptions in human rhombic lip development are associated with posterior cerebellar
215                            The human rhombic lip persists longer through cerebellar development than
216 Wls-positive cells in the Math1-null rhombic lip indicates that Wls expression is independent of Math
217 signals is required for induction of rhombic lip-derived cerebellar neurons.
218 on of cerebellar cell types from the rhombic lip and its upregulation inhibits their production.
219 e between columnar epithelium of the rhombic lip and the expanding squamous epithelium of the roof pl
220 e of spatial compartmentation in the rhombic lip and the interplay between Wls, Math1, and Pax6 in th
221 cularly distinct compartments in the rhombic lip during cerebellar development.
222                                  The rhombic lip gives rise to neuronal populations that contribute t
223      These findings suggest that the rhombic lip is dynamically patterned by the expression of Wls, M
224  between Wls, Math1, and Pax6 in the rhombic lip provides novel views of early cerebellar development
225 he cells in the interior face of the rhombic lip throughout normal mouse cerebellar development.
226 ng from the ventricular zone and the rhombic lip, two distinct germinal zones of the embryonic cerebe
227 ives rise to isthmic nuclei, and the rhombic lip, which generates deep cerebellar nuclei and granule
228  that WNT medulloblastomas match the rhombic lip-derived mossy fiber neuronal lineage and embryonal t
229 ule precursor cells derived from the rhombic lip.
230 ocalized to the exterior face of the rhombic lip.
231  precerebellar nuclei originate from rhombic lips, the first analysis of neuronal migrations from the
232 ly arise only in the lower and upper rhombic lips, respectively.
233 ve knock-out of P/Q-type channels in rhombic-lip-derived neurons including the PF and MF pathways (qu
234 superinfection by 4 days after initial right lip inoculation elicited failed superinfecting-virus gen
235 e that form a deep socket rimmed by a robust lip of bone.
236 on rat Sigmodon hispidus via a common route (lip abrasion) can cause multifocal CNS demyelination and
237 een oscillatory brain activity and speaker's lip movements and demonstrated significant entrainment i
238  activity in visual cortex and the speaker's lips.
239 ing underlying oscillatory mechanism: seeing lip movements first modulates neuronal activity in early
240 l at frequencies <1 Hz more than to the seen lip movements.
241 ed rate of other so-called 'stress signals', lip licking and yawning.
242  demonstrate that the brain can use a silent lip-read signal to synthesize a coarse-grained auditory
243 elevated risks for both invasive and in situ lip cancers (standardized incidence ratios of 15.3 and 2
244  scales that measure appearance of the skin, lips, and facial rhytids (ie, overall, forehead, glabell
245                    During continuous speech, lip movements provide visual temporal signals that facil
246                           Most of the tested lip products contained high concentrations of titanium a
247                                We found that lip-smacking, a distinct multimodal oral gesture produce
248        More precisely, the results show that lips on the Target speaker matched to a secondary (Mask)
249 ed syllables were least likely to engage the lip sensorimotor area and they were least impaired by TM
250 r of samples, including human cases from the lip and nasal mucosa.
251 the coherent auditory speech signal from the lip-brain coherence.
252 ptical fibre probe is used to illuminate the lip and acquire fluorescence emission spectra in approxi
253 y cilia of the CNC-derived mesenchyme in the lip and palate region in mice and is a strong candidate
254 demonstrate that inoculation of HSV-1 in the lip propagates virus to both TG, but with delay in reach
255       Virus was detected sequentially in the lip, trigeminal ganglia, and brain of infected animals.
256 dical professionals are trained to "lift the lip" to identify anterior caries lesions.
257  medication played in the development of the lip abscess.
258                   Nonsyndromic clefts of the lip and palate are common birth defects resulting from g
259 l populations participating in fusion of the lip and primary palate, as well as formation of the naso
260                       Surgical repair of the lip is the only treatment and is usually performed durin
261 rus 1 (HSV-1) inoculation in one side of the lip propagates virus to infect the ipsilateral TG.
262 om the vermillion and/or mucosal part of the lip were recorded from 10 patients with clinically norma
263 onsive to the red spots in the callus of the lip, and PeMYB12 participated in the full pigmentation i
264                            For tumors of the lip, oral cavity and pharynx, digestive organs, respirat
265                              Clefting of the lip, with or without palatal involvement (CLP), is assoc
266 EL of the vermillion and mucosal part of the lip.
267 full pigmentation in the central lobe of the lip.
268 for whom lymphocytic foci per 4 mm(2) on the lip biopsies was either 0 (F = 0), <1 (F <1) or > 3 (F >
269  lateral cells, but oriented parallel to the lip in ventral regions.
270 t of multiple articulators (for example, the lips, jaw, tongue and larynx) over rapid time scales.
271 ntal disease, tooth loss, and cancers of the lips and oral cavity.
272 experienced an unequivocal angioedema of the lips as a positive reaction in SBPCPT.
273  and usually present with enlargement of the lips, tongue, nose, hands, and feet and are diagnosed by
274 sented with chronic verrucous papules on the lips, tongue, and buccal mucosa refractory to multiple e
275                         Lightly stroking the lips or gently poking some skin regions can evoke mechan
276 t analysis of neuronal migrations from these lips was performed with markers of neuroblasts.
277 late several morphogenetic events leading to lip fusion.
278 cally the early auditory-cortex responses to lip-articulated speech sounds when they were attended.
279 during non-speaking movements of the tongue, lips, and jaw.
280         The motor cortex in the brain tracks lip movements to help with speech perception.
281 light on the oscillatory dynamics underlying lip-reading.
282                            An abnormal upper lip bite test, which is easily assessed by clinicians, r
283 s that include a prominent maxilla and upper lip that readily reveal the upper gingiva, widely spaced
284 s cNCC mesenchyme proliferation during upper lip morphogenesis, and that disruption of this sequence
285 cell (cNCC) mesenchyme is required for upper lip closure.
286 ty of cells contributing to the future upper lip.
287 passive eruption (APE) and hypermobile upper lip (HUL), has not been reported.
288               Stimulation of the nose, upper lip, lower lip, and chin caused a somatotopic lateral-to
289 f IQR, P-value 0.0371), and wrinkle on upper lip (7.7% more wrinkle on upper lip per increase of IQR,
290 kle on upper lip (7.7% more wrinkle on upper lip per increase of IQR, P-value 0.0218).
291  eyes, a narrow nasal bridge, a tented upper lip, a high palate, an open mouth, tightly adherent skin
292 t anatomic malformations affecting the upper lip and hard and soft palate.
293 ion included a grade of class 3 on the upper lip bite test (lower incisors cannot extend to reach the
294 erm of the medial nasal processes, the upper lip remained intact in mutant mice.
295 er incisors cannot extend to reach the upper lip; positive likelihood ratio, 14 [95% CI, 8.9-22]; spe
296 s hypertrichosis, thick eyebrows, thin upper lip vermilion, and upturned nose.
297 poral narrowing, broad nasal tip, thin upper lip, posteriorly rotated or low-set ears, and microretro
298  broad and smooth philtrum, and a thin upper lip.
299 re-biased patch (MCP) located on the ventral lip of the posterior superior temporal sulcus (STS) in a
300 erium simum), a large African herbivore with lips specialized for grazing in open savannahs, relates

 
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