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1 is Rv3775 (LipE) was annotated as a putative lipase.
2 r cholesterol esters are cleaved off by acid lipase.
3 ion of the oral phase and the use of gastric lipase.
4 tion of endocannabinoids by monoacylglycerol lipase.
5 cterial genera and the activity of exoenzyme lipase.
6 Candida antartica lipase B, and Mucor miehei lipase.
7 ene PLIP1, which encodes a plastid-localized lipase.
8 g than quinoa, especially against pancreatic lipase.
9 ch as Streptomyces chromofuscus PLD or serum lipase.
10 hanase with a conserved domain of family I.3 lipase.
11 se in structural dynamics of the immobilized lipase.
12 enerated the same kind of inhibitors against lipase.
13 s and glycerol through the action of neutral lipases.
14 ere studied using exogenous or overexpressed lipases.
15  modifications by kinases, phosphatases, and lipases.
16 n enol ester link and tested against several lipases.
17  for NAPE-PLD versus other tissue-associated lipases.
18                 Worms deficient in lysosomal lipase 2 (lipl-2), a lysosomal enzyme that is transcript
19 ive Hnf4 signaling leads to up-regulation of lipase 3 and enzymes for mitochondrial beta-oxidation.
20 a (4%), thrombocytopenia (3%), and increased lipase (3%).
21 ivolumab and ipilimumab group were increased lipase (57 [10%] of 547), increased amylase (31 [6%]), a
22 e more effective at inhibiting of pancreatic lipase (74.5%) and of alpha-glucosidase (98.2%).
23 ition tests of alpha-glucosidase, pancreatic lipase, acetylcholinesterase and 15-lipoxygenase were pe
24  and ABHD5 ligands, demonstrating that ABHD5 lipase activation could be dissociated from its other fu
25 ctivity for Nape-pld compared with nontarget lipase activities such as Streptomyces chromofuscus PLD
26 anced protease and alpha-amylase and reduced lipase activities were observed in sprouted oat powder,
27 sorder that is caused by loss of lipoprotein lipase activity and characterized by chylomicronemia and
28   The fungal infection significantly reduced lipase activity and lipid mobilization, thus impairing t
29 shows that water molecules essential for the lipase activity can be replaced by the hydroxyl groups a
30  free fatty acids and attenuated lipoprotein lipase activity consistent with hallmarks of dyslipidemi
31                                 However, its lipase activity has never been characterized, and its pr
32 pression of the identified lipases and their lipase activity in a time-dependent manner.
33 ated with increased postprandial lipoprotein lipase activity in adipose tissue.
34 97, Gly342, and His363 are essential for the lipase activity of rLipE.
35                                    Bacterial lipase activity on mammalian lipids and phospholipids ca
36     It is concluded that polyurethanase with lipase activity represents a potential enzyme for the de
37 ongenital ichthyoses; and (v) lipoxygenases, lipase activity, and LIPN co-localize within putative la
38 h suppressed Gbeta expression exhibit higher lipase activity, and show phenotypes similar to plants o
39 ike protein 3 (ANGPTL3) inhibits lipoprotein lipase activity, increasing triglycerides and other lipi
40 olesterol micellar solubility and pancreatic lipase activity, was recorded.
41  showed the highest cellulase and the lowest lipase activity, while C. noveboracense had the highest
42 ctive than conventional roasting in reducing lipase activity.
43  antioxidant activity and PLE for inhibitory lipase activity.
44 a/alpha topology of members of the GDSL-like lipase/acylhydrolase family.
45                 No activity was observed for lipase AK Amano 20, Candida antartica lipase B, and Muco
46  of the hydrolytic enzymes alpha-amylase and lipase along with stored food reserves (lipids, carbohyd
47 -specific phospholipase C and diacylglycerol lipase alpha is known, alternative pathways remain unset
48  the 2-AG-synthesizing enzyme diacylglycerol lipase alpha.
49 r pharmacologic inhibition of diacylglycerol lipase-alpha (DAGL-alpha) impairs hippocampal CA1 LTP, d
50                               Diacylglycerol lipase-alpha (DAGL-alpha), the principal biosynthetic en
51 l the extracts showed inhibitory activity of lipase, although those from T. molitor and extracted by
52      Their inhibitory activity on pancreatic lipase and alpha-amylase was assessed by traditional in
53 inistered a control diet with restored serum lipase and amylase levels (p < 0.05).
54                                        Serum lipase and amylase levels were analyzed at the end of th
55 g multibioactive products against pancreatic lipase and cholesterol absorption simultaneously.
56 fied that the mutant strain lost most of its lipase and esterase activities and displayed reduced vir
57 thers without DM (n = 18), were analysed for lipase and fatty acid transport proteins and fatty acid
58 ibition of both TG hydrolysis by lipoprotein lipase and hepatic uptake of remnant lipoproteins.
59 ere assessed on the inhibition of pancreatic lipase and interference on the bioaccessibility of chole
60    Our data suggest that LipE functions as a lipase and is important for M. tuberculosis intracellula
61 ignaling mediated by a LIPL-4 lysosomal acid lipase and its lipid chaperone LBP-8 increases mitochond
62                 The faba bean exhibited high lipase and lipoxygenase (LOX) activities, with pH optima
63         However, the remaining activities of lipase and lipoxygenase after 30 min steam autoclaving w
64  gas to nitrogen, the residual activities of lipase and lipoxygenase after the same time of atmospher
65 rgon as plasma gas, the residual activity of lipase and lipoxygenase decreased to 42.50% and 87.72%,
66 on in activity of fat destabilizing enzymes (lipase and lipoxygenase), contaminants heavy metals (As,
67 ited along with the lipid degrading enzymes, lipase and lipoxygenase, resulting in a relatively short
68 FCS); a deficiency in the enzyme lipoprotein lipase and some associated proteins, termed familial chy
69 iocatalytic approaches based on the use of a lipase and transaminase, respectively, the combination o
70 sters were positive for the exocrine enzymes lipase and trypsinogen.
71                     The effects of different lipases and galloyl donors/acceptors on the transesterif
72 ydrolyze host lipid substrates with secreted lipases and phospholipases for nutrient acquisition, col
73 elated with the expression of the identified lipases and their lipase activity in a time-dependent ma
74 pha-amylase, -alpha-glucosidase, -pancreatic lipase) and antioxidant potential (FRAP, ORAC), phenolic
75 was one (3%) grade 4 adverse event (elevated lipase) and no treatment-related deaths occurred.
76 sociated perilipins 2/3/5, hormone-sensitive lipase, and 1-acylglycerol-3-phosphate O-acyltransferase
77 atty acids were hydrolyzed by Candida rugosa lipase, and more selectivity is observed with Porcine Pa
78 three different classes of enzymes (amylase, lipase, and sulfatase), relying on two distinct mechanis
79 gically assessed amylase, insulin, glucagon, lipase, and/or trypsinogen in 78 organ donor pancreata f
80 ous downstream molecules, including kinases, lipases, and linkers, is crucial for B cell selection, s
81 rt and metabolism-related genes (lipoprotein lipase, apolipoprotein A1, apolipoprotein A4, apolipopro
82 zation of gas cell interfaces in dough since lipase application did not change GSB LVs.
83 y responsible for loaf volume increases upon lipase application.
84                                              Lipases are associated with food spoilage and are also u
85                                              Lipases are enzymes necessary for the proper distributio
86                                  Immobilized lipases are excellent biocatalysts for the enzymatic syn
87 in and hederacoside C slightly inhibited the lipase (around 10%) and protodioscin reduced the bioacce
88 g fatty acid uptake and identify lipoprotein lipase as a potential therapeutic target in melanoma.
89 sing four commercially available immobilized lipases as catalysts and two acyl donors: ferulic acid (
90 via activation of the conserved triglyceride lipase ATGL-1, triggers a feedback transcriptional loop
91  through repressing the adipose triglyceride lipase (ATGL) activity in neutrophils in prostaglandin E
92               Silencing adipose triglyceride lipase (ATGL) in human pseudoislets (shATGL) increased t
93               Silencing adipose triglyceride lipase (ATGL) in human pseudoislets with shRNA targeting
94 red lipolysis in global adipose triglyceride lipase (ATGL) knockout mice reduced free PAHSA levels an
95 of the lipolysis enzyme adipose triglyceride lipase (ATGL) resulted in large cytoplasmic LDs, whereas
96 ehydrogenase (POX), and adipose triglyceride lipase (ATGL), as well as markedly reduced lipid droplet
97 ride (TG) hydrolysis by adipose triglyceride lipase (ATGL), the major lipase in the liver.
98 a specific inhibitor of adipose triglyceride lipase (ATGL), the rate-limiting enzyme for intracellula
99  conserved regulator of adipose triglyceride lipase (ATGL)-mediated lipolysis that plays important ro
100 sis in the absence of adipocyte triglyceride lipase (ATGL).
101 y metabolic regulators, adipose triglyceride lipase (ATGL/bmm) and transcriptional cofactor PGC-1.
102 pharmacologic inhibition of monoacylglycerol lipase attenuates GBM proliferation.
103              By employing Candida antarctica lipase B (CALB) as the model enzyme with the Ser-His-Asp
104 d using tert-butanol as a solvent, 20 g/L of lipase B from Candida Antarctica, and vinyl cinnamate as
105 ed for lipase AK Amano 20, Candida antartica lipase B, and Mucor miehei lipase.
106 ally available food-grade Candida antarctica lipase B, Lipozyme(R) 435, was used as the biocatalyst.
107                           Candida antarctica lipase B-catalysed synthesis of lipophilic esters of pol
108 ty acid conjugate using a Candida antarctica lipase B-rich extract, without further purification and
109  with octanoic acid using Candida antarctica lipase B.
110 oth sexes identified a role for triglyceride lipase brummer (bmm) in the regulation of sex difference
111 vation of insulin receptor (InR) and adipose lipase brummer (bmm).
112          They all regulate key intracellular lipases but do so to significantly different extents.
113                        We found that the Geh lipase, but not other S. aureus lipases, prevents activa
114 stinal conditions worsened the inhibition of lipase, but slightly catalyzed the alpha-amylase.
115 y extension Ras, allosterically activate the lipase by promoting and stabilizing interactions between
116 increased HDL levels (P = 7.7 x 10(-7)), but lipase C risk variants (rs2043085, rs2070895) were assoc
117       The formation of NVOFAs was related to lipase catalysed lipid hydrolysis and the formation of v
118  into egg-yolk phosphatidylcholine (PC) in a lipase-catalyzed acidolysis and interesterification proc
119                                        Using lipase-catalyzed hydrolysis of tributyrin as a model rea
120 e 4-carboxyindanone followed by a subsequent lipase-catalyzed resolution turned out to be the most ef
121 ted protein 2 (PNLIPRP2), and carboxyl ester lipase (CEL), which may leak into the visceral fat or sy
122                         Encapsulation of the lipase-coated lipid/PDMS droplets into a model protocell
123    Herein, we prepare aqueous suspensions of lipase-coated oil globules comprising a mixture of a tri
124 ibition of alpha-glucosidase, alpha-amylase, lipase, cyclooxygenases-1 and -2 (COX-1/COX-2), and lipo
125  triazole urea derivatives as diacylglycerol lipase (DAGL)-alpha inhibitors.
126 vity of the enzyme, the specific activity of lipase decreased.
127                             Monoacylglycerol lipase deficiency affects diet-induced obesity, fat abso
128                          Besides cytoplasmic lipase-dependent adipocyte fat mobilization, the metabol
129 d extracted chia seed oil and with different lipase derivatives to compare the omega-3 FAEE yield and
130                Furthermore, reutilization of lipase derivatives was studied to evaluate the stability
131                      This occurs through its lipase domain.
132 e functions that extends from the N-terminal lipase domains to the C-terminal EDS1-PAD4 domains and m
133  brans suggesting their inhibitory effect on lipase during bran storage.
134  were used to monitor the clearance of these lipases during biologics process development.
135 it is a lipase, which we name TleV1 (type VI lipase effector Vibrio).
136                                  Endothelial lipase (EL) hydrolyzes phospholipids in high-density lip
137 ter adverse events related to treatment were lipase elevation (n = 7 [10.8%]), fatigue (n = 4 [6.2%])
138 s occurred in 12 (29%) patients and included lipase elevation, atrial fibrillation, hypophosphataemia
139 e D (PLD), a ubiquitously expressed membrane lipase enzyme activity in modulating phagocyte functions
140 pE) protein and demonstrated that LipE has a lipase/esterase activity.
141 nce shares similarities with other bacterial lipase/esterases and we demonstrated that it has esteras
142 e active sites of 43 active SHs encompassing lipases/esterases, GDSL lipases, proteases, Ser carboxyp
143                                         This lipase exhibited high efficiency for omega-3 (n-3), and
144                         Adipose triglyceride lipase expression was decreased and insulin-mediated per
145 identified a gene encoding a novel class III lipase family member, Sl-LIP8, that is associated with a
146 2 interactions has important implications on lipases for screening of stilbenoid.
147 d population in BM was studied by applying a lipase from Fusarium oxysporum in the process.
148 ught to purify, identify, and characterize a lipase from S. liquefaciens isolated from cold raw cow's
149                                              Lipase from Thermomyces lanuginosus (TLL) was immobilize
150  0.4-25 muM) and glycerol ester hydrolase or lipase (gehB, IC(50) 1.5-25 muM).
151  through specific repression of ATGL/Brummer lipase gene expression in adipose (fat body).
152  is encoded by a member of the GDSL esterase/lipase gene family.
153 RNA level of hexokinase-2, hormone sensitive lipase, glutathione peroxidase-1, and myosin heavy chain
154                                     Purified lipase had Michaelis-Menten constant (Km) and catalytic
155 sp catalytic triad, a highly active cysteine-lipase having a Cys-His-Asp catalytic triad and addition
156         Inhibition of human Monoacylglycerol Lipase (hMGL) offers a novel approach for treating neuro
157 ymatic studies showed that hormone sensitive lipase (HSL) hydrolyzes luteal cholesterol esters.
158 ance of lipid droplets and hormone-sensitive lipase (HSL) in regulating the aggressive nature of panc
159 atty acid synthase (FASN), hormone-sensitive lipase (HSL), and acyl dehydrogenases was analyzed in vi
160 f LD through regulation of hormone-sensitive lipase (HSL), which was downregulated in human PDAC.
161            All extracts inhibited pancreatic lipase (IC(50) between 1.15 and 0.59 mg/mL), although th
162                                              Lipase immobilized in the GO aerogel exhibits a 5 to 10-
163                 This allowed concluding that lipase impact on LV is exclusively related to stabilizat
164                                         That lipase impact on LV strongly depended on the flour used
165                           Further studies on lipase in food models are needed.
166 of LDs indicating that ATGL is the principal lipase in human beta cells.
167 f LDs, indicating that ATGL is the principal lipase in human beta-cells.
168 of ATGL, the rate-limiting triglyceride (TG) lipase in many cell types.
169 hepatic flare in two patients, and increased lipase in one patient.
170 reatment-related adverse event was increased lipase in the durvalumab group (seven [2%] of 345 patien
171 dipose triglyceride lipase (ATGL), the major lipase in the liver.
172 ng alveoli, albumin in liver parenchyma, and lipase in the stomach lining.
173  on PIN-lipid interactions and the impact of lipases in bread making.
174 focus on specific functions of intracellular lipases in lipid partitioning, covering basic and transl
175 orophosphonate-binding serine hydrolases and lipases in S. aureus and synthesized target-selective ac
176  of 126 patients) and asymptomatic increased lipase (in 10 [8%]).
177 ein profiling approach to unravel the active lipases, including other Serine hydrolases (SHs), expres
178 ents in the ripretinib group (n=85) included lipase increase (four [5%]), hypertension (three [4%]),
179            Three DLTs were reported: grade 3 lipase increase (n = 2; 100 mg and 200 mg twice a day) a
180 or worse treatment-related adverse event was lipase increase, which occurred in 14 (4%) of 353 patien
181 otransferase increased (33.9% vs 22.8%), and lipase increased (32.2% vs 27.4%); 13% of patients in th
182         Notably, as the thermal stability of lipase increased, as indicated by the temperature optimu
183        We studied the genotypic diversity of lipase induced hydrolytic rancidity (HR) level in the br
184 esulted in protein extracts with the highest lipase inhibition (~ 70%) in a dose-dependent way.
185                                              Lipase inhibition by all smoothies was over 50%.
186                Adding rabbit gastric lipase, lipase inhibitor (orlistat) and tocopherols to cod liver
187 eir functional roles and substrates, the pan-lipase inhibitor isopropyl dodecylfluorophosphonate was
188 overed the repeated loss of the triglyceride lipase inhibitor PNLIPRP1, suggesting enhanced triglycer
189 tat measurements in presence of a pancreatic lipase inhibitor proved to be an efficient way to widen
190 administration of JZL184, a monoacylglycerol lipase inhibitor, blocked SI deficits associated with in
191 ts potently with a covalent monoacylglycerol lipase inhibitor.
192 erine hydrolases by using a panel of neutral lipase inhibitors to identify an enzyme that reacts pote
193 xidant activity whereas a negative impact on lipase inhibitory activity was observed (p<.0001).
194 , antioxidant activity (DPPH) and pancreatic lipase inhibitory capacity were assayed.
195                                   Pancreatic lipase injected into mouse visceral adipose tissue hydro
196       We have examined the trans-resveratrol/lipase interaction by quantitative and qualitative analy
197  investigated the possibility that S. aureus lipases interface with the host immune system to blunt i
198 the activity of hepatic adipose triglyceride lipase, intrahepatic lipolysis, hepatic acetyl-CoA conte
199                            We show that this lipase is located in the intermembrane space of the mito
200 st by immobilization of Candida antarctica B lipase is reported, coating single-core magnetic nanopar
201 ound that VPA0226, a constitutively secreted lipase, is required for escape of V. parahaemolyticus fr
202 been reported to allosterically activate the lipase, it is not known whether Rap1A has the same abili
203 ular dichroism measurement confirms that the lipase keeps its native conformation in the aerogel, and
204                      Because dual pancreatic lipase knockouts are lethal, exocrine parotid acini lack
205 y that simultaneously monitors two high risk lipases known to impact biologics product quality, Phosp
206 e and day 3 compliance with triglyceride and lipase laboratory monitoring per protocol and time to di
207                               Lysosomal acid lipase (LAL) hydrolyzes cholesteryl ester (CE) and retin
208                               Lysosomal acid lipase (LAL) is a critical lipid hydrolase that generate
209 zation, the metabolic role of lysosomal acid lipase (LAL), highly expressed in adipocytes, is unclear
210 tream transcriptional target, lysosomal acid lipase (LAL).
211 ce protocol compliance with triglyceride and lipase level monitoring and mitigate propofol-related ha
212 ormal range, 30-130 U/L [0.5-2.2 ukat/L]), a lipase level of 172 U/L (2.9 ukat/L) (normal range, 0-60
213 opofol in the setting of triglyceride and/or lipase levels exceeding protocol cutoffs.
214         Outcome measures included pancreatic lipase levels, the systemic inflammatory response (conce
215                                              Lipase lid domains cover the active site to control both
216       Heterodimeric complexes containing the lipase-like protein ENHANCED DISEASE SUSCEPTIBILITY1 (ED
217       The EDS1 family of structurally unique lipase-like proteins EDS1, SAG101, and PAD4 evolved in s
218 CED DISEASE SUSCEPTIBILITY1 (EDS1) family of lipase-like proteins for all resistance outputs.
219                        Adding rabbit gastric lipase, lipase inhibitor (orlistat) and tocopherols to c
220  (allele frequency=0.36), mapping to hepatic lipase (LIPC), to be associated with a smaller increase
221                              The endothelial lipase LIPG possesses serine phospholipase activity and
222 is of propyl gallate (PG) using a food-grade lipase (Lipozyme(R) 435).
223 des, markers of lipogenesis, and lipoprotein lipase (LPL) activity in adults participating in a doubl
224 anged in ppHF dams, but systemic lipoprotein lipase (LPL) activity was increased, suggesting that inc
225 or, capturing the soluble enzyme lipoprotein lipase (LPL) during export from the TGN.
226                                  Lipoprotein lipase (LPL) hydrolyzes fatty acids (FAs) from triglycer
227                                  Lipoprotein lipase (LPL) is active in capillaries, where it plays a
228                                  Lipoprotein lipase (LPL) is an enzyme responsible for clearing trigl
229                                  Lipoprotein lipase (LPL) is central to triglyceride metabolism.
230                       The enzyme lipoprotein lipase (LPL) is responsible for breaking down triglyceri
231                                  Lipoprotein lipase (LPL) is responsible for the intravascular proces
232                     We show that lipoprotein lipase (LPL) may more efficiently hydrolyze medium lengt
233                                  Lipoprotein lipase (LPL) plays a central role in triglyceride (TG) m
234                   The binding of lipoprotein lipase (LPL) to GPIHBP1 focuses the intravascular hydrol
235 Using RNA in situ hybridization, lipoprotein lipase (LPL) was found to be expressed in endothelial ce
236 pillary endothelial cells, binds lipoprotein lipase (LPL) within the subendothelial spaces and shuttl
237 rexpression of the gene encoding lipoprotein lipase (LPL), which was upregulated in zebrafish melanoc
238 bition (DSI) was limited by monoacylglycerol lipase (MAGL) but not by fatty acid amide hydrolase.
239 ith regional differences in monoacylglycerol lipase (MAGL) expression in postmortem brain tissue, suc
240                             Monoacylglycerol lipase (MAGL) is the enzyme degrading the endocannabinoi
241 ve focused on inhibition of monoacylglycerol lipase (MAGL) to enhance signaling of the most abundant
242 ies of tetrazine probes for monoacylglycerol lipase (MAGL) were synthesized and the most reactive one
243                             Monoacylglycerol lipase (MAGL), a serine hydrolase extensively expressed
244 nt actions of inhibitors of monoacylglycerol lipase (MAGL), the major degradative enzyme of the endoc
245 ional target MGLL, encoding monoacylglycerol lipase (MAGL), to regulate the self-renewal and tumorige
246 y acid synthase (FASN), and monoacylglycerol lipase (MAGL).
247 n 1 (GPIHBP1) and with its chaperone protein lipase maturation factor 1 (LMF1), we obtained a stable
248                            Rhizomucor miehei lipase mediated-hydrolysis of sardine oil was conducted
249                             Monoacylglycerol lipase (MGL) is the last enzymatic step in triglyceride
250 t (HFD)-induced obesity for monoacylglycerol lipase (MGL), an enzyme that is also known to hydrolyze
251               Inhibitors of monoacylglycerol lipase (MGL), the enzyme that deactivates the endocannab
252 at are potent inhibitors of monoacylglycerol lipase (MGL), the primary degrading enzyme for the endoc
253                                         This lipase must be delivered into the host cytoplasm where i
254 al-specific knockout of adipose triglyceride lipase not only reduced fetal beta-cell area and blood i
255  determinants such as exoenzymes (proteases, lipases, nucleases) and downregulate the expression of s
256 nity were not due to direct functions of the lipase on mammalian cells, but rather a result of inacti
257 aminotranferases (one [2%] of 46), increased lipase or amylase (two [4%]), and pancreatitis (one [2%]
258  in different organic solvents without using lipase or other enzymes.
259  High glucose levels did not alter placental lipase or transporter expression or the profile and abun
260                      Specifically, catalase, lipase, or alkaline phosphatase-filled colloidosomes are
261 , CEE extracts being stronger (fenugreek for lipase -p = 0.009-, and quinoa for alpha-amylase -p < 0.
262                                          The lipase partially purified by ultrafiltration and gel fil
263 ity lipoprotein cholesterol, the lipoprotein lipase pathway or circulating lipoprotein(a) may be effi
264 itors for other serine hydrolases, including lipases, peptidases, and proteases.
265                      Pancreatic triglyceride lipase (PNLIP) increased in adipose tissue during pancre
266 ancreas expresses pancreatic triacylglycerol lipase (PNLIP), pancreatic lipase-related protein 2 (PNL
267 se in apparent activity than the lyophilized lipase powder in transesterification of geraniol and vin
268 oteins, lactoferrin and bile salt-stimulated lipase, presented different kinetics of release during d
269 that the Geh lipase, but not other S. aureus lipases, prevents activation of innate cells in culture.
270 ive SHs encompassing lipases/esterases, GDSL lipases, proteases, Ser carboxypeptidases, ABHD protein,
271 mpathectomy and loss of adipose triglyceride lipase protect mice from GDF15-induced weight loss.
272  enzymatic resolution with immobilized Amano Lipase PS-30.
273 olvent-free glycerolysis of anchovy oil with lipase PS-DI from Burkholderia cepacia.
274 rol/triacylglycerol ratio of 3/1, 9.0% (w/w) Lipase PS-DI, a stirring rate of 200 rpm, and a reaction
275 nstrate the impact of HCP-mAb interaction on lipase quantification.
276 c triacylglycerol lipase (PNLIP), pancreatic lipase-related protein 2 (PNLIPRP2), and carboxyl ester
277                       The corresponding acid lipase(s) catalyzing these reactions remains to be ident
278  We, thus, aimed to determine the pancreatic lipase(s) regulating lipotoxicity during AP.
279  Staphylococcus aureus secretes at least two lipases, Sal1 and glycerol ester hydrolase (Geh), with s
280 e promoter regions of genes encoding the TAG lipases SDP1 and DALL5 and acyl-thioesterase KAT5.
281                   Addition of rabbit gastric lipase significantly increased the release of HNE during
282 yelin and a proteoglycan mediate lipoprotein lipase sorting in the TGN.
283 atidylethanolamines were the most accessible lipase substrates.
284 -18 followed by a physicochemical coating of lipase surface with a dense layer of PEG.
285 , with sequence similarity to plant esterase/lipase/thioesterase (ELT) proteins, is essential for tri
286 atment-related adverse events were increased lipase (three [7%]) and febrile neutropenia (three [7%])
287 ves and compared with commercial immobilized lipases to produce omega-3 rich FAEE.
288               A method involving extraction, lipase treatment, clean-up, and detection and quantifica
289 electivity is observed with Porcine Pancreas lipase type II.
290    Here, we showed that the stabilization of lipase upon MPCI to polymer brush surfaces resulted from
291 il catalysed by polyethylene glycol-modified lipases using a biocatalyst with higher stability than c
292 r shrimp fed diets 3 and 7, while intestinal lipase was significantly higher in shrimp fed diets 7 an
293 nzymes (alpha-amylase, alpha-glucosidase and lipase) was evaluated.
294 and the phosphorylation of hormone-sensitive lipase were attenuated in iWAT of Gcgr(-/-) mice.
295 upon enzymatic activity and are selective to lipases were developed under the name of shifting-nitrox
296 c wheat lines differing in PIN haplotype and lipases were used in bread making.
297 s are lethal, exocrine parotid acini lacking lipases were used to verify the results.
298 n of the putative effector indicates it is a lipase, which we name TleV1 (type VI lipase effector Vib
299 of pancreatic disease and normal amylase and lipase who underwent the examination with the same proto
300 of this method is demonstrated for two other lipases with different structures, promising unprecedent

 
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