ライフサイエンスコーパス: lipoma

1 nal carcinoma, two chromophobe type, and one lipoma).

2 mour cell types, including fat-cell tumours (lipomas).

3 r (p = 0.044) of a histologically non-benign lipoma.

4 confidence rating of 4 or 5 as positive for lipoma.

5 nign tumors of mesenchymal origin, including lipomas.

6 HMGI-C truncation in the generation of human lipomas.

7 ng multiple angiofibromas, collagenomas, and lipomas.

8 sible role in development and progression of lipomas.

9 well differentiated liposarcomas or atypical lipomas.

10 l segment rearranged in a subset of ordinary lipomas.

11 mosomal segment often rearranged in ordinary lipomas.

12 gn ocular tumors, and central nervous system lipomas.

13 e and efficacious treatment of large colonic lipomas.

14 ce imaging (MRI) findings of rare pancreatic lipomas.

15 , macroscopic fat (<-30 HU) was present in 9 lipomas.

16 bile duct, stomach, oral cavity tumors, and lipomas.

17 low accuracy in the diagnosis of soft-tissue lipomas.

18 ental ages, a cerebellar tumor, and multiple lipomas.

19 on, and have an abnormally high incidence of lipomas.

20 ntial role for HMG I-C in the development of lipomas.

21 nt to perturb adipogenesis and predispose to lipomas.

22 pathy (1 patient in each group), and gastric lipoma (0 and 1 patients, respectively).

23 tients had myelodysplastic lesions (19 filar lipoma, 14 syringomyelia, 10 intradural lipoma, eight de

24 astomas, 18 (6%) with fibromas, 12 (4%) with lipomas, 28 (9%) with other benign primary cardiac tumor

25 expression was detected in 7 of 11 ordinary lipomas (63.6%) with alterations at 12q14-15 and in one

26 Two patients presented with symptomatic cord lipomas after laparoscopic hernia repair.

27 ding frontonasal dysplasia, interhemispheric lipoma, agenesis of the corpus callosum, tibial hemimeli

28 tical tumors, angiofibroma, collagenoma, and lipoma, also occur in some patients.

29 Both lipoma and liposarcoma demonstrated thin septa and regio

30 The incidence of cord lipoma and relationship to inguinal hernia were evaluate

31 Histologic examination yielded 25 lipomas and 14 nonlipomas.

32 39 well differentiated adipose neoplasms (19 lipomas and 20 ALTs) of known karyotype using polyclonal

33 ith histologically verified fatty tumors (35 lipomas and 25 well-differentiated liposarcomas) were re

34 motor dysfunction, subcutaneous and visceral lipomas and hemangiomas, and intestinal juvenile polypos

35 pertrophy of the interatrial septum, cardiac lipomas and liposarcomas) conditions, with or without ex

36 ibroma, 14 myxoma, 6 vascular, 4 teratoma, 3 lipoma, and 15 other.

37 to distinguish between liposarcoma subtypes, lipoma, and normal fat samples.

38 nal juvenile polyposis, developmental delay, lipomas, and pigmentation spots of the male genitalia.

39 Lipomas are benign fatty tumors with a high prevalence r

40 er syndromes characterized by overgrowth and lipomas are part of the PTEN syndrome spectrum, we ascer

41 Lipomas are rare, small, homogenous and well-circumscrib

42 Pancreatic lipomas are reported to be rare but their prevalence is

43 There were 32 (51%) cord lipomas associated with indirect hernias, 11 (17%) with

44 that demonstrated imaging characteristics of lipoma, benign peripheral nerve sheath tumor, and vascul

45 that demonstrated imaging characteristics of lipoma, benign peripheral nerve sheath tumor, and vascul

46 , progressive skeletal deformities, invasive lipomas, benign and malignant tumors, and deep venous th

47 ertained six individuals with overgrowth and lipomas but who did not meet the diagnostic criteria for

48 the level of confidence in the diagnosis of lipoma by using a five-point scale.

49 Epidural lipomas causing cord compression is documented in patien

50 mobility group proteins, is disrupted in 3/3 lipomas characterized by 12q14-q15 rearrangements.

51 Colonic lipomas (CL) are rare benign adipose tumours usually fou

52 Nonendocrine mesenchymal tumors, such as lipomas, collagenomas, and angiofibromas have also been

53 s tumors, epithelioid sarcomas, spindle cell lipomas, dermatofibrosarcoma protuberans, and myofibrobl

54 methemoglobin), 3.lipid-containing lesions (lipoma, dermoid cyst, implanted fatty materials, laminar

55 rtoma tumor syndrome (PHTS), associated with lipoma development in children.

56 ilar lipoma, 14 syringomyelia, 10 intradural lipoma, eight dermal sinus, five diastematomyelia, five

57 lity and focal soft tissue tumors (fibromas, lipomas, etc).

58 Translocations of 12q13-15 in lipomas (fat cell tumors) disrupt HMGI-C and fuse its DN

59 n particular, increased abundance of fat and lipomas, features strikingly similar to those observed i

60 differentiated mesenchymal tumors, including lipomas, fibroadenomas of the breast, salivary gland ade

61 nal regulatory domains was not necessary for lipoma formation.

62 Chromosomal translocations in human lipomas frequently create fusion transcripts encoding hi

63 sion of other mesenchymal tumors (leiomyoma, lipoma, gastrointestinal stromal tumor, leiomyosarcoma,

64 feration of mesenchymal cells in leiomyomas, lipomas, hamartomas,and other diseases has been linked t

65 sferase 3) with stroke, and the LHFPL2 gene (lipoma high mobility group protein I-C fusion partner-li

66 Lhfpl2 (lipoma HMGIC fusion partner-like 2) encodes a tetra-tran

67 localisation from P9 onwards coincides with lipoma HMGIC fusion partner-like 5 (LHFPL5), a protein t

68 MET components Protocadherin 15a (Pcdh15a), Lipoma HMGIC fusion partner-like 5 (Lhfpl5), and Transme

69 everal accessory proteins, including LHFPL5 (lipoma HMGIC fusion partner-like 5).

70 f hair cell stereocilia (TMHS, also known as lipoma HMGIC fusion partner-like 5, LHFPL5), both though

71 ex, including protocadherin 15, cadherin 23, lipoma HMGIC fusion partner-like 5, transmembrane inner

72 nding genes, nucleolar protein 4 (NOL4), and lipoma HMGIC fusion partner-like protein 4 (LHFPL4), whi

73 In conclusion, whole exome sequencing in lipomas identified mutations in genes with a possible ro

74 We report a growth of lipoma in an unusual site in a 28-year-old female renal

75 "loop and let go" technique for large colon lipomas in a large case series.

76 study examined the prevalence of pancreatic lipomas in a sample of patients undergoing abdominal com

77 dministered, are implicated in the growth of lipomas in different anatomic locations including the ep

78 elopment of angiofibromas, collagenomas, and lipomas in patients with MEN1.

79 ontaining preferred translocation partner in lipoma), induced by constitutive activation of STAT5, bu

80 Suspicion of malignant change within a lipoma is a common and increasing workload within the UK

81 ic the mesenchymal submucosal tumors such as lipoma, leiomyoma, and gastrointestinal stromal tumors.

82 Paratesticular tumours can be benign (lipoma, leiomyoma, haemangioma) or malignant (rhabdomyos

83 69%), including those with either classic or lipoma-like histology, had rearrangements of the 8q12 PL

84 n that lipoblastomas can evolve into mature, lipoma-like, lesions.

85 s, range 8-24), there was one small residual lipoma (<1 cm).

86 The lipomas measured between 4 and 12 mm.

87 Lipomas measured from 6 mm to 32 mm (average 12.8 mm) an

88 Until now, reason for lipoma occurrence not been identified.

89 A lipoma of the cord is herniated fat that appears to orig

90 This is the first case of intramedullary lipoma of the spinal cord that may be related to steroid

91 during continuous culture, whereas APCs from lipomas of patients with PHTS retain their adipogenic po

92 Lipomas of the cord and round ligament occur with a sign

93 Sixty-three lipomas of the cord were identified for an incidence of

94 tivity was demonstrated in only two ordinary lipomas: one with 6p21 rearrangement and one with normal

95 In well-differentiated liposarcomas, lipomas, or normal adipose tissue, perilipin 2 was virtu

96 al loci that may be perturbed during cardiac lipoma pathogenesis.

97 , depth, growth noticed by patient, previous lipoma, patient felt pain), Ultrasonographic (size, dept

98 ing to define the mutational spectrum in ten lipoma patients along with their matching control sample

99 a family of proteins that also includes the lipoma preferred partner (LPP) and thyroid receptor-inte

100 Lipoma preferred partner (LPP) has been identified as a

101 Lipoma preferred partner (LPP) is a proline rich LIM dom

102 the adhesion plaque protein, zyxin, and the lipoma preferred partner (LPP).

103 Here, we found that CAFs upregulated the lipoma-preferred partner (LPP) gene in microvascular end

104 SHC adaptor protein (SHCA) and lipoma-preferred partner (LPP) mediate transforming grow

105 d receptor-interacting protein 6 (TRIP6) and lipoma-preferred partner (LPP), but not to zyxin itself.

106 a being the most frequent and others such as lipoma) present as endoluminal growth, with defined bord

107 The echogenicity of lipomas ranged from hypoechoic to hyperechoic relative t

108 rare, karyotypically distinct group of human lipomas, rearrangements of 6p21-23 produce internal dele

109 ed with development of human tumors, such as lipomas, relatively few examples exist of germline mutat

110 igations of primary cardiac tumors (myxomas, lipomas, rhabdomyomas, and fibromas) have provided insig

111 Pancreatic lipomas seem to be quite a common finding and can be fou

112 x angiofibromas, three collagenomas, and one lipoma showed allelic deletion of the MEN1 gene.

113 pillary fibroelastomas (SMR 3.17, P=0.0003), lipomas (SMR 5.0, P=0.0003), other benign tumors (SMR 4.

114 Currently, there is no lipoma-specific data, with regard to which clinical or r

115 Our patient had an intradural lipoma that had merged with the medulla of the spinal co

116 k features of BRR: macrocephaly and multiple lipomas, the latter of which occur in a minority of indi

117 nted genomic insight into the development of lipomas, the most common benign tumor of soft tissue.

118 ons consisted of six adenomatoid tumors, two lipomas, two epidermoid inclusion cysts, two cases of sa

119 differentiated mesenchymal tumors including lipomas, uterine leiomyomas, and pulmonary chondroid ham

120 that is rearranged in several solid tumors, lipomas, uterine leiomyomata, and liposarcomas.

121 ons were studied, the majority of which were lipomas, vascular anomalies, and epidermoid cysts.

122 he tendon sheath, epidermal inclusion cysts, lipomas, vascular lesions, peripheral-nerve tumours, ski

123 eases within the CPA, including meningiomas, lipomas, vascular malformations, hemangiomas, epidermoid

124 Additionally, the presence of lipomas was correlated with the presence of PTEN mutatio

125 peritoneal hernia repair, the anatomy of the lipomas was studied both at the time of surgery and agai

126 Forty-five lipomas were associated with hernias and were characteri

127 Overall, 18 cord lipomas were found in groins without hernias, and these

128 Nine lipomas were found in women, seven presenting with groin

129 In 6 patients small pancreatic lipomas were found.

130 All lipomas were incidental findings unrelated to patients'

131 Following diagnosis, lipomas were looped and ligated by endoloop.

132 y required for tumorigenesis, is bypassed in lipomas which express chimeric HMGI proteins.

133 88, average: 65.6 years), with 13 pancreatic lipomas, whose cases constituted the basis for 10 contra

134 A significant number of lipomas will have prominent non-adipose areas and will d

135 ibed, consisting of looping and ligating the lipoma with a nylon snare.

136 ( > 25 kb) and is the site of disruption in lipomas with 12q14-q15 translocations.