ライフサイエンスコーパス: lipomannan

1 eplaced instead by a novel truncated form of lipomannan.

2 nown intermediate and of the known precursor lipomannan.

3 ol mannosides and linear and mature branched lipomannan and lipoarabinomannan are prominent phospholi

4 cothiol, but phosphatidylinositol mannoside, lipomannan and lipoarabinomannan levels were not altered

5 sence of the phosphatidylinositol-containing lipomannan and lipoarabinomannan, replaced instead by a

6 levels of IL-17 in response to Mycobacterium lipomannan, and depletion of gammadelta T cells improved

7 lycolipids: phosphatidylinositol mannosides, lipomannan, and lipoarabinomannan (LAM).

8 hway of phosphatidyl-myo-inositol mannoside, lipomannan, and lipoarabinomannan, which are key glycoli

9 way of phosphatidyl-myo-inositol mannosides, lipomannan, and lipoarabinomannan, which are key glycoli

10 glycolipids phosphatidylinositol mannoside, lipomannan, and lipoarabinomannan.

11 n is a biosynthetic precursor of PIM4, PIM6, lipomannan, and lipoarabinomannan.

12 phosphatidyl-myo-inositol mannosides (PIMs), lipomannan, and mannose-capped lipoarabinomannan (ManLAM

13 smegmatis produced increased levels of LAM, lipomannan, and phosphatidylinositol mannosides (PIMs) c

14 e and thus indirectly for lipoarabinomannan, lipomannan, and the higher-order phosphatidyl-myo-inosit

15 rans, attached to either a galactofuran or a lipomannan, are the primary constituents of mycobacteria

16 e lipoglycans, such as lipoarabinomannan and lipomannan, but a mechanism for lipoglycans to traffic f

17 etion of NCgl1505 resulted in the absence of lipomannan (Cg-LM-A), lipoarabinomannan (Cg-LAM) and a m

18 sulted in the formation of a novel truncated lipomannan (Cg-t-LM) and a complete ablation of LM/LAM b

19 We found that both LPS and lipomannan enhanced CAIA more potently in the presence o

20 The best ligand was Micrococcus luteus lipomannan, followed by Enterococcus spp. LTA containing

21 only 5 to 20 Manp residues as compared with lipomannan from the wild type strain consisting of 21-34

22 unrecognized feature of the biosynthesis of lipomannan/lipoarabinomannan allows a significant revisi

23 he phosphatidylinositol-anchored lipoglycans lipomannan (LM) and lipoarabinomannan (LAM) are cell env

24 Lipomannan (LM) and lipoarabinomannan (LAM) are key Cory

25 e essential, mycobacterial genes involved in lipomannan (LM) and lipoarabinomannan (LAM) biosynthesis

26 The mycobacterial lipoglycans, lipomannan (LM) and lipoarabinomannan (LAM), are potent

27 the phosphatidylinositol-based lipoglycans, lipomannan (LM) and lipoarabinomannan (LAM).

28 n altered growth and inability to synthesize lipomannan (LM) but accumulation of a previously unchara

29 (PI)-containing lipoarabinomannan (LAM) and lipomannan (LM) of Mycobacterium spp. follows a conserve

30 phosphatidylinositol mannoside-6 (PIM6) and lipomannan (LM) were identified as factors responsible f

31 actions and spheroplasts showed that LAM and lipomannan (LM) were primarily found in a cell wall-enri

32 Lipomannan (LM), a glycophospholipid found on the cell s

33 Phosphatidylinositol mannosides (PIM), lipomannan (LM), and lipoarabinomannan (LAM) are essenti

34 .tb-exposed cell envelope molecules, such as lipomannan (LM), contain subtle structural variations th

35 Lipomannan (LM), found on the surface of Mycobacterium t

36 A related polymer, lipomannan (LM), is composed of only the phosphatidylino

37 strategy was developed for the synthesis of lipomannan (LM), useful for vaccine development.

38 e lipoarabinomannan (LAM) and its precursor, lipomannan (LM), which are trafficked from the bacterium

39 ates that the biosynthetic precursor of LAM, lipomannan (LM), which is also present in the cell wall,

40 lipoglycan on the M. tuberculosis surface is lipomannan (LM).

41 es (PIMs)], and PIMs associated lipoglycans [lipomannan (LM); mannose-capped lipoarabinomannan (ManLA

42 ted Corynebacterineae synthesize a family of lipomannans (LM) and lipoarabinomannans (LAM) that are a

43 using LAMs and their biosynthetic precursor lipomannans (LMs) isolated from different mycobacterial

44 was mannosyl-lipoarabinomannan, followed by lipomannan, phosphatidylinositol mannoside, arabinosyl-l

45 ght/mass spectrometry of the mutated form of lipomannan shows a family of phosphatidylinositol-anchor

46 erase, required for the proper elongation of lipomannan to its normal state and subsequent synthesis

47 In contrast with the MR, the PIM(2)f and lipomannan were recognized by DC-SIGN comparable to high

48 ws a family of phosphatidylinositol-anchored lipomannans with from only 5 to 20 Manp residues as comp